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1  were identified with a primary diagnosis of pancreatic neoplasm.
2 ma of the pancreas is the most common benign pancreatic neoplasm.
3 eatic ductal adenocarcinoma (PDAC), a deadly pancreatic neoplasm.
4 copic techniques for diagnosing and treating pancreatic neoplasms.
5 cience and clinical advances in the field of pancreatic neoplasms.
6 ly in the ladder of oncogenesis, as in human pancreatic neoplasms.
7 s per year, and they account for 1-2% of all pancreatic neoplasms.
8   Two patients with choledochoceles (7%) had pancreatic neoplasms.
9               These data suggest that cystic pancreatic neoplasms 1) occur in 0.7% of patients, 2) in
10  benign pancreatic neoplasm (34%), malignant pancreatic neoplasm (31%), other neoplasm (15%), chronic
11 imary DP, the indications for DP were benign pancreatic neoplasm (34%), malignant pancreatic neoplasm
12 organ will certainly shape the management of pancreatic neoplasm and holds the promise of improved ou
13 s on this organ is changing the treatment of pancreatic neoplasms and holds the promise of improved o
14 efine the risk of premalignant and malignant pancreatic neoplasms and potential benefits and limitati
15              Acinar cell carcinomas are rare pancreatic neoplasms associated with postresection survi
16  Expression of large T antigen is highest in pancreatic neoplasms, but is also detectable in the norm
17    Screening can detect curable, noninvasive pancreatic neoplasms, but the optimal imaging approach i
18     Only few case reports of mucinous cystic pancreatic neoplasm containing an undifferentiated carci
19                             The treatment of pancreatic neoplasm continues to change and improve as m
20  standard resection for benign and low-grade pancreatic neoplasms, has been described in mainly small
21                         Human PDAC cells and pancreatic neoplasms in mice contain morphologically and
22  APC/beta-catenin pathway in other nonductal pancreatic neoplasms including pancreatoblastomas and ac
23 influence the development and progression of pancreatic neoplasms initiated by an oncogenic allele of
24 accepted and agreed on terminology for solid pancreatic neoplasms, is needed.
25 id-pseudopapillary tumors (SPTs) are unusual pancreatic neoplasms of low malignant potential that mos
26 events or severe hypoglycemia, pancreatitis, pancreatic neoplasms, or allergic reactions than was pla
27 hat have affected the study and treatment of pancreatic neoplasms over the past year.
28 s of ACCs overlap with those of another rare pancreatic neoplasm, pancreatoblastoma.
29                             The treatment of pancreatic neoplasms remains a major challenge for physi
30 These compound mutant mice developed a novel pancreatic neoplasm, serous cystadenoma (SCA), presentin
31              Twenty-two patients with cystic pancreatic neoplasms that were confirmed at surgical res
32 red in locally advanced or centrally located pancreatic neoplasms to achieve complete tumor clearance
33                                              Pancreatic neoplasms were induced in rats by implantatio
34  ductal adenocarcinomas, and 6 miscellaneous pancreatic neoplasms) were microdissected from 29 formal
35 Primary tumors, usually lung, colorectal, or pancreatic neoplasms, were identified in 135 patients (2
36 or the separate clonal evolution of multiple pancreatic neoplasms within individual patients.

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