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1 sk of lymph node metastases in patients with pancreatic neuroendocrine tumors.
2 ould be independent prognostic biomarkers of pancreatic neuroendocrine tumors.
3 n the diagnosis, prognosis, and treatment of pancreatic neuroendocrine tumors.
4 able tool in the diagnosis and management of pancreatic neuroendocrine tumors.
5 er the last year in EUS in the evaluation of pancreatic neuroendocrine tumors.
6 peutic EUS interventions in the treatment of pancreatic neuroendocrine tumors.
7 utility of EUS in the clinical management of pancreatic neuroendocrine tumors.
8 trinomas comprise the majority of functional pancreatic neuroendocrine tumors.
9 future multi-institutional investigations on pancreatic neuroendocrine tumors.
10 iagnosis, imaging, treatment, and staging of pancreatic neuroendocrine tumors.
11 f rhEndostatin in patients with carcinoid or pancreatic neuroendocrine tumors.
12 h metastatic carcinoid, pheochromocytoma, or pancreatic neuroendocrine tumors.
15 stations include renal carcinomas and cysts, pancreatic neuroendocrine tumors and cysts, pheochromocy
16 val among patients with progressive advanced pancreatic neuroendocrine tumors and was associated with
20 Intraductal papillary mucinous neoplasm and pancreatic neuroendocrine tumor biology affect patient o
21 ther targeting IFNgamma and/or TNFalpha into pancreatic neuroendocrine tumors can alleviate immune su
22 itro accumulation experiments with the human pancreatic neuroendocrine tumor cell line BON-1 were per
23 cent information and advancements concerning pancreatic neuroendocrine tumor diagnosis, imaging chara
26 tatic spread among patients with early-stage pancreatic neuroendocrine tumors has not been well estab
27 y altered in human tumors, including cognate pancreatic neuroendocrine tumors, implying a generality.
28 ided by both cancer cells and macrophages in pancreatic neuroendocrine tumors in humans and mice.
29 -Met is upregulated in lymphatics in or near pancreatic neuroendocrine tumors in RIP-Tag2 transgenic
30 antitumor activity in patients with advanced pancreatic neuroendocrine tumors, in two phase 2 studies
31 gnostic significance of specific proteins in pancreatic neuroendocrine tumors including insulinoma.
32 Although rare, a thorough understanding of pancreatic neuroendocrine tumors is essential for all ph
36 ords of patients with carcinoid (n = 84) and pancreatic neuroendocrine tumors (n = 69) managed at our
38 PURPOSE No established treatment exists for pancreatic neuroendocrine tumor (NET) progression after
39 ival by 6.4 months in patients with advanced pancreatic neuroendocrine tumors (NET) compared with pla
41 ced its first TNM staging classification for pancreatic neuroendocrine tumors (NETs) derived from the
42 Patients with inoperable or unresectable pancreatic neuroendocrine tumors (NETs) have limited tre
44 ntributing to development and maintenance of pancreatic neuroendocrine tumors (NETs), with special re
45 anding of the pathogenesis of gastrinoma and pancreatic neuroendocrine tumors, new prognostic classif
48 udopapillary neoplasms, cystic variations of pancreatic neuroendocrine tumors, pancreatic ductal aden
50 rs; however, its specific functions in human pancreatic neuroendocrine tumors (PanNETs) and spontaneo
55 mutations of these genes are common in human pancreatic neuroendocrine tumors (PanNETs), we examined
56 time to tumor progression was 7.7 months in pancreatic neuroendocrine tumor patients and 10.2 months
59 xysterol 24-hydroxycholesterol (24S-HC) in a pancreatic neuroendocrine tumor (pNET) model commonly us
60 AC), whereas tumor burden was reduced in the pancreatic neuroendocrine tumor (PNET) model, the latter
62 L/6 (B6) background develop both noninvasive pancreatic neuroendocrine tumors (PNET) and invasive car
69 olecule tyrosine inhibitor sunitinib in both pancreatic neuroendocrine tumors (PNETs) in RIP-Tag2 mic
76 sent study, 12 gastrinoma and nonfunctioning pancreatic neuroendocrine tumor specimens were evaluated
77 re examined in RIP-Tag2 transgenic mice with pancreatic neuroendocrine tumors that developed spontane
78 ors) are the most common type of functioning pancreatic neuroendocrine tumors that occur sporadically
79 evaluate the dose-response relationship for pancreatic neuroendocrine tumors treated with PRRT using
80 24 lesions in 24 patients with metastasized pancreatic neuroendocrine tumors treated with repeated c
81 expression of UCH-L1 and alpha-internexin in pancreatic neuroendocrine tumors was significantly assoc
82 linger et al reported 2 female patients with pancreatic neuroendocrine tumors, WDS, and achlorhydria.
83 ad advanced, low-grade or intermediate-grade pancreatic neuroendocrine tumors with radiologic progres
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