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1 secretion and responses to feeding, we used pancreaticobiliary and intestinal cannula to divert bile
3 igation of the genetic alterations common to pancreaticobiliary cancers and aid the understanding of
6 nt prognosis; two, those with histomolecular pancreaticobiliary carcinoma with LN metastases who had
7 tients with renal cell carcinoma (7 months), pancreaticobiliary carcinomas (3, 5, and 5 months), mela
8 was performed in 265 patients with suspected pancreaticobiliary disease and in 35 control patients wi
9 ier RARE MRCP enables accurate evaluation of pancreaticobiliary disease and obviates ERCP in some pat
12 tion of hospitalization for the treatment of pancreaticobiliary disease was longer for patients who r
13 Risk factors assessed at presentation of pancreaticobiliary disease were weight change after tran
14 uracy of 100% in determining the presence of pancreaticobiliary disease, the presence and level of bi
16 ompare immune cell infiltration of different pancreaticobiliary diseased tissues (PDAC, ampullary car
19 ients (nonpancreaticobiliary, LN positive or pancreaticobiliary, LN negative) who had an intermediate
21 vided histopathologically into intestinal or pancreaticobiliary (PB) types, which may more accurately
24 olymeric nutrients, potential stimulation of pancreaticobiliary secretions, secretion of humoral medi
28 s a non-surgical approach to diseases of the pancreaticobiliary system that dates back to the late 19
29 e found at significantly higher frequency in pancreaticobiliary than intestinal, gastric or oncocytic
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