ライフサイエンスコーパス: panic attack

1 in both twins, with one twin experiencing a panic attack.

2 icipation of the next uncued (unpredictable) panic attack.

3 f internal and external cues predictive of a panic attack.

4 d periods of increased anxiety or tension or panic attack.

5 esponse to a conditioned fear stimulus and a panic attack.

6 ically to suppress excessive arousal such as panic attacks.

7 ptoms and least correlated with reduction in panic attacks.

8 the pattern observed during the 34 reported panic attacks.

9 rs to be a genuine risk factor for secondary panic attacks.

10 est for the families of the probands without panic attacks.

11 AS predicts panic attacks.

12 asing the number of full and limited-symptom panic attacks.

13 development of neurological symptoms during panic attacks.

14 son subjects, and this increase preceded the panic attacks.

15 cur in different patients from m-CPP-induced panic attacks.

16 vestibular symptoms between, but not during, panic attacks.

17 substance abuse or substance dependence, or panic attacks.

18 nxiety disorder with recurrent, debilitating panic attacks.

19 condition caused by the experience of uncued panic attacks.

20 n implicated in the pathophysiology of acute panic attacks.

21 mpared with young persons with no history of panic attacks.

22 with a significantly greater risk of current panic attacks.

23 ence respiration-related difficulties during panic attacks.

24 r the co-occurrence of cigarette smoking and panic attacks.

25 f the co-occurrence of cigarette smoking and panic attacks.

26 inent depersonalization/derealization during panic attacks.

27 icide attempts than were adolescents without panic attacks.

28 be one of the mechanisms linking smoking to panic attacks.

29 , 27 patients with anxiety but no history of panic attacks, 13 patients with depressive disorders but

30 higher rate than seen among subjects without panic attacks (69.4%).

31 ic ethnicity, two or more prior stressors, a panic attack, a low level of social support, the death o

32 During the panic attack, a strong association with the surrounding

33 two were characterized by past or concurrent panic attacks, a rate that was not significantly differe

34 Although panic attacks account for only a portion of the morbidit

35 , neutral conditioned stimuli present during panic attacks acquire panicogenic properties.

36 Patients with panic disorder experience panic attacks after intravenous sodium lactate infusions

37 The associations between panic attack and depression were attenuated in models th

38 ed an increased risk for first occurrence of panic attack and disorder; the risk was higher in active

39 order or for the severity of lactate-induced panic attack and the quantified PET abnormality.

40 Of the 115 patients, 41% (N = 47) reported panic attacks and 17% (N = 20) met screening criteria fo

41 Measures of anxiety included history of panic attacks and a composite variable reflecting curren

42 re used to determine the association between panic attacks and cigarette smoking and to determine whe

43 ents with PTSD experienced yohimbine-induced panic attacks and had significantly greater increases co

44 ts (31%) with PTSD experienced m-CPP-induced panic attacks and had significantly greater increases co

45 ed anxiety and sympathetic drive seen during panic attacks and in hypercapnic states such as COPD.

46 provocation on the subsequent development of panic attacks and panic disorder in nonclinical subjects

47 Evidence from a family study suggests that panic attacks and panic disorder may be related genetica

48 (N = 115) were screened for the presence of panic attacks and panic disorder with a self-report ques

49 sought to determine the association between panic attacks and psychoticism among young adults in the

50 Much of the relationship between panic attacks and psychoticism appears to be explained b

51 The symptoms of panic attacks and pulmonary disease overlap, so that pan

52 rs is the sudden shift in affect observed in panic attacks and some rapid cycling states.

53 s were used to estimate associations between panic attacks and suicidal ideation and suicide attempts

54 ds to the evidence of an association between panic attacks and suicide attempts during the middle yea

55 study was to investigate the association of panic attacks and suicide attempts in a community-based

56 direction (i.e., from prior smoking to first panic attack) and the possibility of a higher risk in ac

57 ysthymia, generalized anxiety disorders, and panic attacks) and drug use during the previous 12 month

58 ionnaire-2 [PHQ-2], GAD-2, and an item about panic attacks), and a diagnostic evaluation using PHQ-9

59 ssive disorders but no history of anxiety or panic attacks, and 45 normal comparison subjects.

60 gonists acting at this receptor can suppress panic attacks, and both inverse agonists and antagonists

61 rder are highly susceptible to CO(2)-induced panic attacks, and depressed patients appear to be insen

62 xcept PMDD), the physiological features of a panic attack appear similar across groups.

63 Panic attacks are a common complication of affective dis

64 Panic attacks are a hallmark in panic disorder (PAND).

65 h previous findings, these data suggest that panic attacks are associated with greater risk of cigare

66 Panic attacks are relatively common among postmenopausal

67 Individuals with panic disorder perceive panic attacks as unpredictable.

68 ienced significantly more panic symptoms and panic attacks, as well as elevated respiratory rates.

69 were used to estimate the risk for onset of panic attacks associated with prior smoking and vice ver

70 The role of lung disease in the smoking-panic attacks association was explored.

71 scored highly for PTSD-related symptoms and panic attacks at 8 wks (p = .023 and .014, respectively)

72 No subject had a panic attack before hyperventilation.

73 sorder, with anxiety disorders (particularly panic attacks) being the most common comorbid condition.

74 -occurrence of regular cigarette smoking and panic attacks but did not predict either panic attacks o

75 ession predicted a first onset of subsequent panic attacks but not of panic disorder.

76 That primary depression predicts panic attacks but not panic disorder suggests that secon

77 hors sought to determine the relationship of panic attacks, cigarette smoking, and neuroticism.

78 o Pavlovian conditioning, failure to predict panic attacks could be due to a basic deficit in conditi

79 Specific symptoms occurring during panic attacks differ by gender.

80 cantly lower in those who subsequently had a panic attack during 5% CO(2) breathing than those who di

81 ic ethnicity, two or more prior stressors, a panic attack during or shortly after the events, residen

82 Panic attacks during adolescence are associated with sig

83 in minute ventilation at baseline predicted panic attacks during CO2 inhalation.

84 ensitivity to CO(2) and in the threshold for panic attacks during hypoxic and hypercapnic states.

85 e placebo-treated patients were free of full panic attacks during the 2 weeks ending at week 10.

86 Despite a mean of 9.5 to 11.6 full panic attacks during the screening period, 86.0% of the

87 A 6-month history of full-blown panic attacks, endorsed by 10% of postmenopausal women i

88 ty Survey respondents with panic disorder or panic attacks, female respondents were more likely than

89 Primary outcome measures were change in panic attack frequency and clinician-rated Clinical Glob

90 ic disorder have generally used reduction in panic attack frequency as the primary measure of improve

91 rtion of the morbidity of panic disorder and panic attack frequency assessments are unreliable, studi

92 Reductions in panic attack frequency in subjects given either fluoxeti

93 easures, including global improvement, total panic attack frequency, phobic symptoms, and functional

94 th placebo for the primary outcome variable, panic attack frequency.

95 The majority of individuals with panic attacks had been regular smokers during their life

96 th panic disorder who had a low frequency of panic attacks had elevated daytime corticotropin levels

97 Subjects with CO(2)-induced panic attacks had similarly high ratings on the behavior

98 ' goal was to determine whether treatment of panic attacks has a protective effect on the risk of maj

99 The symptoms of CO(2)-induced panic attacks have a similar intensity regardless of the

100 Having a panic attack in the preceding 3 years was associated wit

101 re used to determine the association between panic attacks in adolescence (age 15-21) and psychoticis

102 idence of an independent association between panic attacks in adolescence and psychoticism during you

103 ion between A2AR polymorphisms and phobia or panic attacks in humans and prompts a therapeutic intere

104 oradrenergic agent yohimbine reliably induce panic attacks in humans with panic disorder but not in h

105 fusion has induced flashbacks accompanied by panic attacks in male combat veterans with posttraumatic

106 Panic attacks in panic disorder may be explained by inef

107 ough CO(2) breathing causes a higher rate of panic attacks in patients with PD than other groups (exc

108 r in panic disorder, evokes intense fear and panic attacks in susceptible individuals.

109 Proportion of subjects remitted (no panic attacks in the past month, minimal anticipatory an

110 ompleted a questionnaire about occurrence of panic attacks in the previous 6 months.

111 tory of panic disorder, panic attacks, or no panic attacks in the probands.

112 of 35% CO(2) evoked not only fear, but also panic attacks, in three rare patients with bilateral amy

113 ent in frequency of full and limited-symptom panic attacks, intensity of full panic attacks, phobic f

114 Other assessments included a panic attack inventory, clinician-rated and patient-rate

115 -response relationship suggests that primary panic attack is a marker, rather than a causal risk fact

116 , the exact mechanism of lactate eliciting a panic attack is still unknown.

117 Once a panic attack is triggered, minute ventilation and respir

118 Panic attacks occurred in five patients with generalized

119 of the respiratory dysfunction manifested in panic attacks occurring in panic disorder.

120 set and was less likely to be complicated by panic attacks or alcoholism.

121 and panic attacks but did not predict either panic attacks or cigarette smoking in the absence of the

122 onset of daily smoking in persons with prior panic attacks or disorder.

123 tic anxiety ratings, but not the presence of panic attacks or of any lifetime anxiety disorder, added

124 der if the proband with bipolar disorder had panic attacks or panic disorder was calculated with logi

125 s increased significantly if the proband had panic attacks or panic disorder.

126 any panic, which included panic disorder and panic attacks (OR = 2.1; 95% CI, 1.1, 4.5).

127 any anxiety disorder [OR = 1.3 (1.1, 1.6)], panic attacks [OR = 1.6 (1.1, 2.1)], panic disorder [OR

128 groups based on a history of panic disorder, panic attacks, or no panic attacks in the probands.

129 tion exists about the epidemiology of DSM-IV panic attacks (PAs) and panic disorder (PD).

130 who completed the study, the mean number of panic attacks per week dropped by 88% in the sertraline-

131 ted-symptom panic attacks, intensity of full panic attacks, phobic fear, anxiety, and depressive symp

132 d safety of fluoxetine treatment in reducing panic attacks, phobic symptoms, anxiety, and depressive

133 History of panic attacks proved to be a significant correlate of no

134 The findings suggest that the perception of panic attacks reflects central rather than peripheral re

135 suggest a causal hypothesis for the smoking-panic attacks relationship.

136 accompanying a PTSD flashback and those in a panic attack remains unclear.

137 site variable and patients with a history of panic attacks reported more severe medication side effec

138 common in women and are not associated with panic attacks, suggesting a late-life subtype.

139 n other men to evidence major depression and panic attack syndromes.

140 Yohimbine-induced panic attacks tended to occur in different patients from

141 patients had a higher rate of CO(2)-induced panic attacks than depressed patients and normal subject

142 More patients with PD and PMDD had panic attacks than did controls or patients with major d

143 subsyndromal panic disorder characterized by panic attacks that failed to meet either the criterion o

144 adjustment for confounding factors, having a panic attack was still associated with an increased rate

145 that depersonalization/derealization during panic attacks was associated with childhood trauma.

146 Occurrence of panic attacks was judged with DSM-IV criteria by a blind

147 met DSM-III-R criteria for panic disorder or panic attacks were analyzed to test for gender differenc

148 uency of certain symptoms between and during panic attacks were obtained.

149 Panic symptoms and panic attacks were rated with the Acute Panic Inventory

150 However, patients with panic attacks were significantly more likely to report d

151 ugs, the authors found that adolescents with panic attacks were three times more likely to have expre

152 umed alcohol experienced significantly fewer panic attacks when applying liberal panic criteria; howe

153 major depressive episodes (odds ratios: for panic attacks with depression, 6.2; for panic disorder w

154 Temporally primary panic attacks, with or without panic disorder and whethe

155 ncing depersonalization/derealization during panic attacks would be more likely to have a history of