ライフサイエンスコーパス: papillary carcinoma

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1 ies that included follicular hyperplasia and papillary carcinoma.

2 s in the alemtuzumab group developed thyroid papillary carcinoma.

3 n reported in patients with hereditary renal papillary carcinomas.

4 in the development of post-Chernobyl thyroid papillary carcinomas.

5 5-22.3) years after HCT and included 13 with papillary carcinoma and 5 with benign adenomas.

6 oidectomy was done in 85.3% of patients with papillary carcinoma and 71.3% of patients with follicula

7 ents with high-risk thyroid cancer (303 with papillary carcinoma and 82 with follicular carcinoma).

8 ations of c-Met have been described in renal papillary carcinoma and gastrointestinal cancers includi

9 thyroid hyperplasia, solid tumor variants of papillary carcinoma and metastatic cancer.

10 and spontaneously progressed to both cystic papillary carcinoma and metastatic PDAC.

11 It is possible to distinguish between papillary carcinomas and other lesions with the thyroid

12 linkage between chronic pancreatitis, cystic papillary carcinoma, and PDAC.

13 e VHL gene and clear cell carcinoma, MET and papillary carcinoma, and the families of genes that they

14 as, 1 of 30 Hurthle cell carcinomas, 1 of 90 papillary carcinomas, and 0 of 10 nodular goiters had 3p

15 inomas, grade I and II from grade III serous papillary carcinomas, and ovarian from breast carcinomas

16 NA was overexpressed in three of six thyroid papillary carcinomas as compared with paired normal thyr

17 ally positive p53 staining was found in four papillary carcinomas being primarily confined to solid a

18 ype p53-overexpressing derivative of the NPA papillary carcinoma cell line was obtained, and these ce

19 ase 8 construct effectively killed the SW579 papillary carcinoma cell line, arguing that the action o

20 0 thyroid tumor cell lines, including 2 of 2 papillary carcinoma cell lines, 4 of 5 anaplastic carcin

21 as preferentially found in radiation-induced papillary carcinomas developing after a short latency, w

22 errant expression in mice is associated with papillary carcinoma development.

23 high prevalence of ret/PTC3, whereas typical papillary carcinomas do not, suggesting that the differe

24 by well-differentiated carcinomas, including papillary carcinoma, follicular carcinoma, and medullary

25 ry carcinoma) or papillary (any component of papillary carcinoma); for papillary tumors, the proporti

26 Papillary carcinomas formed a tightly clustered group, a

27 characteristics of 38 post-Chernobyl thyroid papillary carcinomas from Belarussian children 5-18 year

28 rangements are highly prevalent in pediatric papillary carcinomas from children exposed to radiation

29 ren 5-18 years old with those of 23 sporadic papillary carcinomas from the same age children without

30 vating mutations in cancers other than renal papillary carcinoma has not been well defined.

31 Osteosarcomas in juvenile Wwox(-/-) and lung papillary carcinoma in adult Wwox(+/-) mice occurred spo

32 lial tumours of the bladder are diagnosed as papillary carcinomas in accordance with the WHO classifi

33 The presence of a component of papillary carcinoma is more common than previous reports

34 In thyroid papillary carcinomas, mutations of genes coding for effe

35 situ (n=2), ductal carcinoma in situ (n=7), papillary carcinoma (n=2), and invasive ductal carcinoma

36 was effective in helping distinguish between papillary carcinomas (n = 10) and other lesions (n = 43)

37 re as follows: clear cell carcinomas (n=23), papillary carcinomas (n=6), and chromophobe carcinomas (

38 that produced false-positive PET scans were papillary carcinoma of the thyroid (1), bronchogenic car

39 radiation-induced thyroid tumorigenesis, 33 papillary carcinomas, one follicular carcinoma and 22 be

40 ified as nodular-sclerosing (no component of papillary carcinoma) or papillary (any component of papi

41 inomas but not in 20 follicular adenomas, 10 papillary carcinomas, or 10 multinodular hyperplasias.

42 onal profiles of four normal thyroids and 51 papillary carcinomas (PCs) were generated using DNA micr

43 Five of the N-terminal genes found in papillary carcinoma rearrangements are also involved in

44 The morphological variants of papillary carcinoma showed different prevalence of the s

45 Thirty-two clear cell carcinomas, 10 papillary carcinomas, six chromophobe carcinomas, four o

46 ts with hyperplasia and Group B of rats with papillary carcinoma (stages Ta and T1).

47 expressed in radiation-induced and childhood papillary carcinomas than other members of the fusion on

48 lary lesions suspicious at SCNB proved to be papillary carcinomas; the fourth had no residual carcino

49 ase types that range from indolent localised papillary carcinomas to the fulminant and lethal anaplas

50 s (WDCs; two [6.7%] of 30 of the tumors were papillary carcinomas, two [10.5%] of 19 of them were fol

51 g radiation-induced tumors, solid variant of papillary carcinoma was found in 37%, follicular in 29%,

52 (n = 10) and other lesions (n = 43) because papillary carcinomas were stiffer than other lesions (P

53 alphavbeta3-positive vessels in the group of papillary carcinomas whereas it correlated with integrin

54 Solid papillary carcinoma with reverse polarity (SPCRP) is a r

55 nes, severe cystic pathology, and ultimately papillary carcinomas with hepatic metastases.

56 and RET genes in 12 post-Chernobyl pediatric papillary carcinomas with known RET/PTC3 rearrangement.

57 the cases were histologically classified as papillary carcinomas, with 2 of them exhibiting follicul

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