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1 ve been detected in patients with hereditary papillary renal cell carcinoma.
2 not by clear cell renal cell carcinoma or by papillary renal cell carcinomas.
3 f the MET gene have been identified in human papillary renal cell carcinomas.
4 of the proteins that become fused to TFE3 in papillary renal cell carcinomas.
5 to translocation partners on 1 and X in some papillary renal cell carcinomas.
6 e RCC samples, including both clear cell and papillary renal cell carcinomas.
7 X;1)(p11.2;q21.2) has been observed in human papillary renal cell carcinomas.
8 shown to play a role in 13% of patients with papillary renal cell carcinoma and no family history of
9 the histopathology of 103 bilateral archival papillary renal cell carcinomas and 4 metastases in 29 p
10 is known about the genetic basis of sporadic papillary renal-cell carcinoma, and no effective forms o
13 metastases in 29 patients from 6 hereditary papillary renal cell carcinoma families with germline c-
14 roliferative cells, resembling precursors of papillary renal cell carcinoma, formed in these proximal
16 ma type 1 histology, not all type 1 sporadic papillary renal cell carcinomas harbor c-met mutations.
22 of a recently proposed subclassification of papillary renal cell carcinoma (PRCC) that designates tu
27 omarkers to diagnose malignant clear cell or papillary renal cell carcinoma (RCC) in a screening para
30 translocation t(X;1)(p11.2;p34) observed in papillary renal cell carcinomas results in the fusion of
31 thermore, post hoc analysis of human class 1 papillary renal cell carcinoma revealed reduced Notch ac
32 od vessels and in the basement membrane of a papillary renal cell carcinoma, strong FGF-7 binding is
33 as observed in a distinct subgroup of type 2 papillary renal-cell carcinomas that was characterized b
35 l cell carcinomas with c-met mutations share papillary renal cell carcinoma type 1 histology, not all
37 umors with c-met genotype show a distinctive papillary renal cell carcinoma type 1 phenotype and are
38 ve molecular characterization of 161 primary papillary renal-cell carcinomas, using whole-exome seque
43 families with germline c-met mutations and 6 papillary renal cell carcinomas with c-met mutations fro
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