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1 eptor kinase that undergoes rearrangement in papillary thyroid cancer).
2 ancer, and 29 with the follicular variant of papillary thyroid cancer).
3 st 4 decades, driven largely by increases in papillary thyroid cancer.
4 r (95% CI, 1.1%-4.7%) for SEER distant stage papillary thyroid cancer.
5 oid cancer mortality rate for advanced-stage papillary thyroid cancer.
6 afenib in patients with BRAF(V600E)-positive papillary thyroid cancer.
7 The most frequent association is noted with papillary thyroid cancer.
8 od markedly increases the risk of developing papillary thyroid cancer.
9 ificance of Delphian node (DN) metastasis in papillary thyroid cancer.
10 istent disease in patients with conventional papillary thyroid cancer.
11 istent disease in patients with conventional papillary thyroid cancer.
12 ve of disease-free survival in patients with papillary thyroid cancer.
13 utation is primarily present in conventional papillary thyroid cancer.
14 ay result in a transient increase in risk of papillary thyroid cancer.
15 ary thyroid cancers (MTC) and of a subset of papillary thyroid cancers.
16 e, 3 (20%) were malignant, all of which were papillary thyroid cancers.
17 e lesions were malignant, the majority being papillary thyroid cancers.
18 al rearrangements in a majority of childhood papillary thyroid cancers.
19 action and found frequent CpG methylation in papillary thyroid cancer (23 of 39 patients; 59%) and fo
20 cancer (51.0%) than in follicular variant of papillary thyroid cancer (24.1%) and follicular thyroid
21 AF V600E mutation was higher in conventional papillary thyroid cancer (51.0%) than in follicular vari
22 s, 46% of follicular thyroid cancers, 71% of papillary thyroid cancers, 71% of anaplastic thyroid can
23 s with thyroid cancer (245 with conventional papillary thyroid cancer, 73 with follicular thyroid can
24 ne (BRAF V600E), which is primarily found in papillary thyroid cancer and is associated with more agg
25 's thyroiditis is frequently associated with papillary thyroid cancer and may indeed be a risk factor
26 ns of BRAF are found in approximately 45% of papillary thyroid cancers and are enriched in tumors wit
27 urgeons and pathologists in the treatment of papillary thyroid cancer, and especially intermediate-ri
28 erwent thyroid surgery from 2007 to 2009 for papillary thyroid cancer, and had their DN harvested ab
29 010-2013), primarily related to increases in papillary thyroid cancer (annual percent change, 4.4% [9
31 l disease risk in patients after surgery for papillary thyroid cancer as a function of primary tumor
35 overshadowed by its more common counterpart-papillary thyroid cancer-despite its unique biological b
36 that prophylactic CLND may be performed for papillary thyroid cancer, especially for advanced tumors
40 unclear whether the increasing incidence of papillary thyroid cancer has been related to thyroid can
43 ing for an 11-y-old girl with differentiated papillary thyroid cancer, heavy lung involvement, and ce
44 ho were aged 18-64 years when diagnosed with papillary thyroid cancer in 1988-1994 and 574 controls t
47 he key role of TSH signaling in Braf-induced papillary thyroid cancer initiation and provide experime
50 ed in G(1) arrest in two well differentiated papillary thyroid cancer lines (PTCs) and both G(1) arre
51 of a cohort of 262 patients (66% women, 93% papillary thyroid cancer; median dose, 5,217 MBq [141 mC
52 noted in a gastrointestinal stromal tumour, papillary thyroid cancers, non-small-cell lung cancer, o
53 idual tumor foci in patients with multifocal papillary thyroid cancer often arise as independent tumo
58 mutated gene in malignant melanoma (MM) and papillary thyroid cancer (PTC) and is causally involved
59 d most frequent target region for metastatic papillary thyroid cancer (PTC) and medullary thyroid can
60 PURPOSE OF REVIEW: Aggressive variants of papillary thyroid cancer (PTC) have been recognized with
61 ctive iodine (RAI) treatment for early-stage papillary thyroid cancer (PTC) is complex because of unc
64 shown to be highly associated with increased papillary thyroid cancer (PTC) risk with an odds ratio o
66 lysed a multi-generation CS-like family with papillary thyroid cancer (PTC), applying a combined link
68 ty risk factor in the risk stratification of papillary thyroid cancer (PTC), but whether this is gene
69 vascular endothelial growth factor (VEGF) in papillary thyroid cancer (PTC), we conducted a phase II
70 nsidered to play a unique prognostic role in papillary thyroid cancer (PTC), with a distinct staging
77 only in patients age 45 years or older with papillary thyroid cancer (PTC); patients younger than ag
78 rs including 15 follicular adenomas (FA), 13 papillary thyroid cancers (PTC) and 14 follicular thyroi
81 ten (Braf(V600E)/Pten(-/-)/TPO-Cre) leads to papillary thyroid cancers (PTC) that rapidly progress to
84 pression of oncogenic BRAF (Tg-Braf) develop papillary thyroid cancers (PTCs) that are locally invasi
86 ients with progressive, BRAF(V600E)-positive papillary thyroid cancer refractory to radioactive iodin
87 ologically confirmed recurrent or metastatic papillary thyroid cancer refractory to radioactive iodin
92 ent, and a long noncoding RNA (lncRNA) gene, papillary thyroid cancer susceptibility candidate 2 (PTC
93 lexity was observed from well-differentiated papillary thyroid cancer to poorly differentiated and an
94 sence of MSCs with a fibrotic fingerprint in papillary thyroid cancer tumors and the autocrine-paracr
95 iagnosed with thyroid cancer from 1974-2013, papillary thyroid cancer was the most common histologic
97 attributable to an increase in incidence of papillary thyroid cancer, which increased from 2.7 to 7.
98 t patients diagnosed with localized >/= 1-cm papillary thyroid cancer who underwent thyroidectomy wit
100 hyroiditis report an increased prevalence of papillary thyroid cancer, with a favorable disease profi
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