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1             Of the 93 patients, 57 (61%) had papillary thyroid carcinoma.
2 by the active RET mutant, RET/PTC1, found in papillary thyroid carcinoma.
3 tic factor for the metastatic progression of papillary thyroid carcinoma.
4 BRAF gene mutation is frequently detected in papillary thyroid carcinoma.
5 osomal rearrangements found in radio-induced papillary thyroid carcinoma.
6 iration of the mass suggested a diagnosis of papillary thyroid carcinoma.
7 thrombosis is an extremely rare condition in papillary thyroid carcinoma.
8 stologic and cytologic similarities to human papillary thyroid carcinoma.
9 uced iodide uptake ability observed in human papillary thyroid carcinoma.
10 he peripheral blood of only one patient with papillary thyroid carcinoma.
11 his oncogene has been detected only in human papillary thyroid carcinomas.
12 ngements are one of the genetic hallmarks of papillary thyroid carcinomas.
13  of the RET tyrosine kinase commonly seen in papillary thyroid carcinomas.
14 re believed to be tumor-initiating events in papillary thyroid carcinomas.
15  of the RET proto-oncogene detected in human papillary thyroid carcinomas.
16 d three different somatic mutations (23%) in papillary thyroid carcinomas.
17 s, are frequently found in radiation-induced papillary thyroid carcinomas.
18 as been reported to be associated with human papillary thyroid carcinomas.
19 revalent in radiation-induced post-Chernobyl papillary thyroid carcinomas.
20 pothesis that RET/PTC is sufficient to cause papillary thyroid carcinomas.
21 is aberrant methylation is evident in 83% of papillary thyroid carcinoma, 11% of follicular thyroid c
22                     Molecular profiles of 11 papillary thyroid carcinomas, 13 follicular variant of p
23 thyroid carcinomas, 13 follicular variant of papillary thyroid carcinomas, 9 follicular thyroid carci
24  of cutaneous melanomas, cutaneous nevi, and papillary thyroid carcinoma and in a small fraction of o
25 e length were found in follicular variant of papillary thyroid carcinomas and follicular adenomas.
26 ion protein which is frequently expressed in papillary thyroid carcinomas and has been detected in th
27 active protein, not only in FTCs but also in papillary thyroid carcinomas and Hurthle cell carcinomas
28 (55 FA, 27 follicular thyroid carcinomas, 35 papillary thyroid carcinomas, and 22 undifferentiated th
29 reviously been implicated in pathogenesis of papillary thyroid carcinoma as a fusion partner of RET.
30 st commonly detected BRAF mutations in human papillary thyroid carcinomas (BRAF(V600E)) in thyroid fo
31 ll lines and samples of human follicular and papillary thyroid carcinoma by reverse transcriptase-pol
32 region shared by FOXE1 and PTCSC2 in a human papillary thyroid carcinoma cell line (KTC-1) and unaffe
33                             Furthermore, the papillary thyroid carcinoma cell line could be induced t
34 ay in primary thyroid follicular cells and a papillary thyroid carcinoma cell line in vitro.
35 ransforming growth factor-beta1 (TGF-beta1), papillary thyroid carcinoma cells acquired increased can
36 pecificity of 96.2% (3 follicular variant of papillary thyroid carcinomas clustered with the benign l
37 mphoma, chronic myelomonocytic leukaemia and papillary thyroid carcinoma described similar rearrangem
38 n, and that PRRX1 plays an important role in papillary thyroid carcinoma EMT and disease progression.
39              We demonstrated previously that papillary thyroid carcinomas exhibit aberrant patterns o
40 d tumor DNA from 17 pediatric post-Chernobyl papillary thyroid carcinomas for mutations at three diff
41                               Like the other papillary thyroid carcinoma forms of Ret, Ret/ptc2 is ac
42                               A patient with papillary thyroid carcinoma had stable disease for more
43 d a remarkable increase in radiation-induced papillary thyroid carcinoma in children and young adults
44                                              Papillary thyroid carcinomas in humans are associated wi
45 yroid carcinoma oncogene (RET/PTC) generates papillary thyroid carcinomas in one genetic step.
46  thyroidectomy caused by local recurrence of papillary thyroid carcinoma is extremely rare.
47                                              Papillary thyroid carcinoma is frequently multifocal.
48 d demonstrate that alternative exon usage in papillary thyroid carcinomas is restricted primarily to
49  patients thyroidectomized for follicular or papillary thyroid carcinoma may represent the thymus.
50 ovel strategies to prevent or to treat human papillary thyroid carcinomas, MEN 2 disease, as well as
51 MDS, and both solid tumors (osteosarcoma and papillary thyroid carcinoma) occurred in recipients of D
52                Current evidence suggests the papillary thyroid carcinoma oncogene (RET/PTC) generates
53 epithelial cells were microinjected with the papillary thyroid carcinoma oncogene (RET/PTC1 short iso
54                                              Papillary thyroid carcinoma overexpress transforming gro
55                    A substantial increase in papillary thyroid carcinoma (PTC) among children exposed
56 ological examination of the mass confirmed a papillary thyroid carcinoma (PTC) and enlarged metastati
57 es of nonmedullary thyroid carcinoma, namely papillary thyroid carcinoma (PTC) and follicular thyroid
58  protein RET/PTC3 (RP3) that is expressed in papillary thyroid carcinoma (PTC) and thyroid epithelia
59  observed that 63 of 110 (57%) human primary papillary thyroid carcinoma (PTC) cases expressed nuclea
60  MTC and follicular tumours resembling human papillary thyroid carcinoma (PTC) depending on the found
61                                              Papillary thyroid carcinoma (PTC) displays higher herita
62                                              Papillary thyroid carcinoma (PTC) displays strong but so
63                                     Although papillary thyroid carcinoma (PTC) displays strong herita
64   Many patients undergoing thyroidectomy for papillary thyroid carcinoma (PTC) have subclinical nodal
65                                              Papillary thyroid carcinoma (PTC) is clinically heteroge
66                                              Papillary thyroid carcinoma (PTC) is the most common typ
67                                Biomarkers of papillary thyroid carcinoma (PTC) metastasis can accurat
68           A genome-wide association study of papillary thyroid carcinoma (PTC) pinpointed two indepen
69                                              Papillary thyroid carcinoma (PTC) remained to be the mos
70    Approximately 40 percent of patients with papillary thyroid carcinoma (PTC) typically have either
71 600E) is the most common somatic mutation in papillary thyroid carcinoma (PTC), how it induces tumor
72                            One such disease, papillary thyroid carcinoma (PTC), is the leading endocr
73                                              Papillary thyroid carcinoma (PTC), the most frequent thy
74 NP (rs965513) firmly associated with risk of papillary thyroid carcinoma (PTC).
75 rticularly those associated with a precursor papillary thyroid carcinoma (PTC).
76 earrangements, which are frequently found in papillary thyroid carcinoma (PTC).
77 young age at exposure and risk of developing papillary thyroid carcinoma (PTC).
78 tively little is known about the genetics of papillary thyroid carcinoma (PTC).
79 ral soluble factors that were induced by RET/papillary thyroid carcinoma (PTC)3 gene expression inclu
80                    Genetic analysis of human papillary thyroid carcinomas (PTC) has revealed unique c
81                                         Most papillary thyroid carcinomas (PTC) have an isozyme-speci
82  sequencing and expression analysis of eight papillary thyroid carcinomas (PTC) to comprehensively ch
83 ted BRAF is detected in approximately 45% of papillary thyroid carcinomas (PTC).
84 he number of genetic alterations detected in papillary thyroid carcinomas (PTC).
85 ion is the most common genetic alteration in papillary thyroid carcinomas (PTC).
86  is the most prevalent genetic alteration in papillary thyroid carcinomas (PTC).
87  prognostication and treatment selection for papillary thyroid carcinomas (PTCs) do not uniformly pre
88                                              Papillary thyroid carcinomas (PTCs) that invade into loc
89 95 sporadic thyroid tumors, of which 39 were papillary thyroid carcinomas (PTCs), 12 were follicular
90  occurs in various thyroid neoplasms such as papillary thyroid carcinomas (PTCs), follicular thyroid
91 is the most common endocrine malignancy, and papillary thyroid carcinoma represents the most common t
92 tive activation of the RET proto-oncogene in papillary thyroid carcinomas results from rearrangements
93 embers of the rearranged during transfection/papillary thyroid carcinoma (RET/PTC) fusion oncogene fa
94 ing RET chimeric oncoproteins found in human papillary thyroid carcinomas (RET/PTC) as well as RET on
95 tergenic, noncoding RNA gene (lincRNA) named Papillary Thyroid Carcinoma Susceptibility Candidate 3 (
96                                              Papillary thyroid carcinomas that invade locally or meta
97 id-targeted expression of ret/PTC1 developed papillary thyroid carcinomas that were minimally invasiv
98                      Combi-TA mice developed papillary thyroid carcinomas, the incidence of which was
99 s strongly correlated with the generation of papillary thyroid carcinomas, the most prevalent maligna
100 ssed cDNA fragments were isolated from human papillary thyroid carcinoma tissues, among them a cDNA w
101     To gain insight into the pathogenesis of papillary thyroid carcinoma, transcriptional profiles of
102                             We conclude that papillary thyroid carcinoma tumor cells exhibit increase
103                         None of the cases of papillary thyroid carcinoma was positive for ras oncogen
104 sidual neck thyroid tissues of patients with papillary thyroid carcinoma were quantified.
105 nscripts were associated with follicular and papillary thyroid carcinomas, whereas long telomeres and
106                          We report a case of papillary thyroid carcinoma which invades IJV with hyper
107 HB/SDHD mutation carriers have renal cell or papillary thyroid carcinomas, which are also CS-related
108  mutations in p53 were found by SSCP in 2/33 papillary thyroid carcinomas, with one missense mutation

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