ライフサイエンスコーパス: papillomavirus infection

1 eled to recognize the signs of genital human papillomavirus infection.

2 Cervical cancer is caused by human papillomavirus infection.

3 ctrochemical signal corresponded to a bovine papillomavirus infection.

4 surface receptor(s) is a subsequent step of papillomavirus infection.

5 hat recapitulates the establishment phase of papillomavirus infection.

6 n papillomaviruses in the context of in vivo papillomavirus infection.

7 represents the epithelial response to human papillomavirus infection.

8 ounds that might block the initial stages of papillomavirus infection.

9 pproval of vaccines to prevent initial human papillomavirus infection.

10 year post-BMT, 71% developed chronic canine papillomavirus infection.

11 ins have been shown to protect against human papillomavirus infection.

12 n against E2 protein using a rabbit model of papillomavirus infection.

13 effective strategy for immunoprophylaxis of papillomavirus infection.

14 human anogenital carcinomas show evidence of papillomavirus infection.

15 ctions with molluscum contagiosum; and human papillomavirus infections.

16 Human papillomavirus infection accounts for APOBEC3B upregulat

17 es for the prevention and treatment of human papillomavirus infection and associated diseases.

18 a role is proposed for NK function in human papillomavirus infection and cervical neoplasia.

19 irreversible over time with persistent human papillomavirus infection and genetic damage.

20 ighly active antiretroviral therapy on human papillomavirus infection and its related anogenital abno

21 be associated with lower genital tract human papillomavirus infection and may have implications in th

22 inue to confirm the high prevalence of human papillomavirus infection and to define the different hum

23 ccine platforms for both prevention of human papillomavirus infection and treatment of associated dis

24 High-risk human papillomavirus infections and abnormalities on cytologic

25 Human papillomavirus infections and human papillomavirus-assoc

26 pendent signaling pathway in the response to papillomavirus infections and the progression of these v

27 tals, and environmental tobacco smoke, human papillomavirus infection, and inherited genetic suscepti

28 ighly active antiretroviral therapy on human papillomavirus infection; and novel human papillomavirus

29 viral therapy era, raising concerns of human papillomavirus infections as a rising health burden amon

30 third of children will have persistent human papillomavirus infection beyond 2 years.

31 Given the possible link between papillomavirus infection, bracken fern in the diet and c

32 The study furthers our understanding of papillomavirus infection dynamics and provides potential

33 n of the association of cervical cancer with papillomavirus infection encouraged development of a vac

34 .9% (95% CI: 24.3-43.5%) for high-risk human papillomavirus infection from cervical samples and 78.6%

35 n cleavage of minor capsid protein L2 during papillomavirus infection has been difficult to directly

36 evious studies examining the pathogenesis of papillomavirus infections have relied on the use of orga

37 The model incorporated states for human papillomavirus infection (HPV), low- and high-grade squa

38 maviruses are widely used as models to study papillomavirus infection in humans despite differences i

39 In addition to human papillomavirus infection, increased bcl-2 expression and

40 Persistent human papillomavirus infection is associated with squamous cel

41 Most human papillomavirus infection is harmless and clears spontane

42 Human papillomavirus infection is one of the most common sexua

43 However, productive papillomavirus infection is species- and tissue-restrict

44 The viral early-to-late switch of papillomavirus infection is tightly linked to keratinocy

45 Papillomavirus infection normally involves virion bindin

46 In this study, cottontail rabbit papillomavirus infection of domestic rabbits was used as

47 fidence interval (CI): 2.4, 23.2), any human papillomavirus infection (OR = 5.8; 95% CI: 3.3, 10.0),

48 included HIV-1 infection (p = 0.013), human papillomavirus infection (p=0.0013), lower CD4 T lymphoc

49 ytological assay were used to look for human papillomavirus infection, p53 mutations, loss of heteroz

50 his is analogous to the late-onset cutaneous papillomavirus infection recently described for human XS

51 Anogenital human papillomavirus infection remains highly prevalent and pe

52 tigated the extent to which animal models of papillomavirus infection resemble human disease by compa

53 hain reaction-based tests for cervical human papillomavirus infection, tests for gonorrhea and chlamy

54 uggest that the natural progression of human papillomavirus infection, the causal agent in all cervic

55 During papillomavirus infection, the E5 protein localizes in th

56 During persistent papillomavirus infection, the viral E2 protein tethers t

57 In papillomavirus infections, the viral genome is establish

58 differences in the natural history of human papillomavirus infections; the effect of highly active a

59 Human papillomavirus infection was the most significant risk f

60 Gastroesophageal acid reflux and human papillomavirus infection were ruled out.

61 Interventions aimed at preventing human papillomavirus infections with vaccinations need to be e