ライフサイエンスコーパス: papule

1 d with asymptomatic red to brown macules and papules.

2 the pathogenesis and resistance of Gottron's papules.

3 so greatly increased and similar to those in papules.

4 hough levels were not equivalent to those in papules.

5 areas were significantly smaller than parent papules.

6 condition characterized by widespread warty papules.

7 er had begun as an asymptomatic erythematous papule 1 week previously.

8 the ciprofloxacin-treated subjects developed papules 1 week after the initial treatment, and the nint

9 patients with only patches and/or follicular papules (10-year OS, 71% vs 80%), while the survival rat

10 he 9 azithromycin-treated subjects developed papules 4-10 weeks (mean, 6.8) after the initial treatme

11 biopsies were obtained from an inflammatory papule and from normal skin in six patients with acne.

12 Overall, the papule and pustule formation rates for 35000HP and FX533

13 The papule and pustule formation rates were similar to those

14 5000HP and 35000HP-sodC-cat and observed for papule and pustule formation.

15 fected with 35000 and FX517 and observed for papule and pustule formation.

16 and among those who started treatment in the papule and vesicle lesion stages.

17 Classical features include beaded eyelid papules and laryngeal infiltration leading to hoarseness

18 Follicular hyperkeratotic papules and marked pruritus were also prominent clinical

19 the epidermis consisting of verrucoid scaly papules and plaques following Blaschko's lines.

20 On examination, there were scattered pink papules and plaques on the trunk, thighs, and buttocks a

21 4, he was seen at our clinic with new dermal papules and plaques.

22 Sites inoculated with the mutant produced papules and pustules at rates similar to the rates obser

23 culation experiments, the neuA mutant formed papules and pustules at rates that were no different tha

24 Papules and pustules contained a predominant T cell infi

25 Both papules and pustules contained mixed or T helper 1 type

26 that is impaired in its ability to form both papules and pustules in humans.

27 Subsequently, there were numerous papules and pustules--similar to the rash seen in patien

28 /=10 mixed-type lesions such as inflammatory papules and ulcers, located in >/=2 body parts.

29 expression was increased to levels found in papules and vascular adhesion molecule 1 levels were upr

30 All subjects developed papules and were randomized to treatment with a single d

31 8% progressed to papillomas, 9% persisted as papules, and 22% completely regressed.

32 ions were characterized by purpuric macules, papules, and confluent plaques predominantly on the lowe

33 y finger pad inflammation, palmar violaceous papules, and digital ulcerations.

34 y finger pad inflammation, palmar violaceous papules, and digital ulcerations.

35 aracterized clinically by multiple keratotic papules, and histologically by focal loss of adhesion be

36 of acne vulgaris lesions such as comedones, papules, and pustules.

37 Gottron's papules are a particularly resistant manifestation of DM

38 Such papules are histologically analogous to angiofibromas; t

39 ulation experiments, 35000HPDeltacpxA formed papules at a rate and size that were significantly less

40 om histological sections and 7 of 7 of whole papules by mutation-specific PCR analysis.

41 eated and untreated skin for changes in mean papule counts (-4.2 vs -2.2; P =.08), mean pustule count

42 ons including the heliotrope rash, Gottron's papules, cuticular changes including periungual telangie

43 Gottron's papule dermis contained more C4S and CD44v7 than non-Got

44 Papules developed at 92.8% (95% confidence interval [CI]

45 Papules developed at 95% of 40 sites infected with live

46 Papules developed at similar rates at sites inoculated w

47 Papules developed at similar rates at sites inoculated w

48 Papules developed at similar rates at sites inoculated w

49 Papules developed at similar rates at sites inoculated w

50 Papules developed at similar rates at sites inoculated w

51 When papules developed, the subjects were treated with antibi

52 Sixty-nine percent (95% CI, 36.8%-90.9%) of papules evolved into pustules in the reinfection group,

53 and extended over the whole body, with some papules evolving into vesiculobullous lesions.

54 tules but did not detect bacteria in the one papule examined.

55 There was a relationship between papule formation and estimated delivered dose.

56 The papule formation rates were 88% (95% confidence interval

57 tory portions were dilated, and blisters and papules formed on the skin surface in the knockout mice.

58 atments in 2 patients and a reduction of the papules in 2 patients.

59 Here we present a case of atrichia with papules in a patient with a normal HAIRLESS gene but wit

60 he leg and foot in case 1 and small perioral papules in case 2.

61 of the Molluscipoxvirus genus, causes benign papules in healthy people but disfiguring lesions in imm

62 us (MCV) is a poxvirus that causes localized papules in healthy persons.

63 disease characterized by multiple keratotic papules in the seborrheic regions of the body.

64 Generalized atrichia with papules is a rare disorder characterized by loss of hair

65 On May 29, their infant daughter developed a papule on her philtrum.

66 tiple skin lesions, including a 6-mm crusted papule on the left eyebrow, which was resected and, on p

67 led numerous small dome-shaped flesh-colored papules on the head and neck, as well as multiple palpab

68 in his 60s presented with chronic verrucous papules on the lips, tongue, and buccal mucosa refractor

69 white man with widespread, crusted, pruritic papules on the scalp, ears, and face and a purpuric and

70 thema lesion stage) and when initiated late (papule or vesicle stage).

71 ted with multiple erythematous nonfollicular papules partially confluent to plaques on his breast and

72 , indurated, nonscarring, pink to violaceous papules, plaques, or nodules, devoid of surface changes

73 Pearly penile papules (PPPs) are benign, dome-shaped lesions found aro

74 om open and closed comedones to inflammatory papules, pustules, cysts, and nodules, and scarring may

75 Rubbery papules reflected an intense cellular immune reaction do

76 n 24 sites followed to end point, 27% of the papules resolved, 69% (95% CI, 47.1%-86.6%) evolved into

77 on of serial sections of 69 DMBA/TPA-induced papules revealed that they were focal hyperplastic lesio

78 mined non-Gottron's DM lesions and Gottron's papule skin biopsies for C4S, CD44 variant 7 (CD44v7), a

79 where papillomas subsequently developed were papules, slightly raised areas of skin ranging in diamet

80 rom biopsies of 12 of 15 pustules and 1 of 7 papules, suggesting that H. ducreyi replicates between t

81 However, mutant papule surface areas were significantly smaller than par

82 hree molecules uniquely present in Gottron's papules that contribute to inflammation individually and

83 Although both strains caused papules to form at similar rates, the pustule formation

84 Of 78 DMBA/TPA-induced papules tracked during 15 weeks of TPA treatments, 68% p

85 On biopsy, this papule was an invasive acantholytic squamous cell carcin

86 In each patient, the appearance of the papules was either completely diminished or significantl

87 Complete clearance of the papules was noted after 2 to 3 treatments in 2 patients

88 Papules were first detected in DMBA-initiated mice 21 da

89 Sandy patches and rubbery papules were found in 41 of 118 women (35%).

90 However, papules were significantly smaller at mutant sites (mean

91 Papules were significantly smaller at mutant sites than

92 stinguishable from generalized atrichia with papules, which was previously thought to be caused only

93 ficant decrease in the prevalence of rubbery papules with age, even after adjustment for urinary ova

94 inical morphology consisting of erythematous papules with scale in a variety of distributions.

95 sponsive to vismodegib but with an enlarging papule within the tumor bed.