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1 ads to different outcomes than heterochronic parabiosis.
2 ession patterns, genetic lineage tracing and parabiosis.
3 molecules into the brains of mice following parabiosis.
4 ges, we joined the circulation of mice using parabiosis.
5 uthful systemic milieu through heterochronic parabiosis.
6 s controls were phenotypically unaffected by parabiosis.
7 and Abeta plaques after a 12-month period of parabiosis.
8 T iso-mRNA switching and was not affected by parabiosis.
9 e of circulating factors using heterochronic parabiosis, a surgical technique in which joining of ani
12 ne translation, we tested their validity via parabiosis and quantitative immunofluorescence microscop
13 youthful levels recapitulated the effects of parabiosis and reversed age-related hypertrophy, reveali
14 SF) and stem cell factor at days 17 to 20 of parabiosis and were studied 3 weeks later; 10.1% of marr
15 he negative effects of B2M and heterochronic parabiosis are, in part, mitigated in the hippocampus of
19 established a chimeric blood circulation by parabiosis between fetal chicks and quails to determine
20 in-expressing haematopoietic stem cells, and parabiosis between genetically marked mice, confirmed th
23 ell and tissue transplantation, apheresis or parabiosis.Clarifying the source of proteins in mixed bi
25 ed a naturally occurring process of vascular parabiosis coupled with intravascular microinjection of
27 poietic and endothelial cell chimerism after parabiosis demonstrates that circulating cells can give
34 tivating gene 2 GFP reporter mice along with parabiosis experiments, we demonstrate that the vast maj
35 g lymphocytes into lymph nodes and long-term parabiosis experiments, we have found that, contrary to
36 d not circulate or emigrate from the lung in parabiosis experiments, were protected from in vivo Ab l
38 Here we provide evidence that mice joined by parabiosis gradually recover much physiology relevant to
39 animals using a surgical procedure known as parabiosis has created a wealth of information towards o
40 between mice even after prolonged joining by parabiosis have suggested that DCs are derived from self
41 e conclude that, compared with heterochronic parabiosis, heterochronic blood exchange in small animal
50 total body irradiation, we employed a murine parabiosis model with tie-2-LacZ FvB/N mice connected to
51 enhanced ILC2 trafficking to the lungs in a parabiosis mouse model of tissue disruption and repopula
52 ither exogenous or endogenous clusterin, and parabiosis of db clusterin(-/-) double-mutant to WT mice
55 normally decline with age, by heterochronic parabiosis or systemic delivery of recombinant protein,
56 shared blood circulation between 2 mice (aka parabiosis) or repeated injections of young blood plasma
57 providing a shared blood circulation through parabiosis, or through repeated injections of plasma fro
60 equal exchange of DCs between mice joined by parabiosis reflected uneven distribution of DC precursor
65 ure to youthful circulation by heterochronic parabiosis reverses the aged fracture repair phenotype a
70 6/INK4A mRNA did not change in heterochronic parabiosis, suggesting the involvement of other pathways
72 al proteins after exposure to young blood in parabiosis (synaptophysin P = .02; calbindin P = .02) or
75 at the appetite suppression in WT mice after parabiosis to db mutants is the result of induced hyperl
78 ineage tracing, transplantation studies, and parabiosis to show that most adult cardiac fibroblasts d
82 set out to explain the differing effects of parabiosis with genetically diabetic (db) mice versus ad
83 is impaired in Chrm1(--) mice and rescued by parabiosis with wild-type mice, suggesting a relay by a
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