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2 ously used selective combinations of normal, parabiotic, and radioablated mice to demonstrate that bl
5 mice in which remyelination was enhanced by parabiotic coupling to a younger mouse and in multiple s
8 epair by generating chimeric animals through parabiotic joining of wild-type (wt) and diabetic (db/db
9 migration of endogenous DCs to the thymus in parabiotic mice and after painting mouse skin with fluor
11 ether with congenic bone marrow chimeras and parabiotic mice as tools to differentiate LC- and blood-
28 of blood-borne HSCs using genetically marked parabiotic mice, which are surgically conjoined and shar
31 mice, together with bone marrow chimera and parabiotic models, we found that embryonic precursor cel
32 ertility, we established transplantation and parabiotic mouse models to assess the capacity of circul
33 ralization factor(s) in circulation, we used parabiotic pairing, ie, surgical joining of two mice, to
34 tor cells from these tissues, we established parabiotic pairings (that is, a shared circulatory syste
35 the circulation into tumors was confirmed in parabiotic pairs of COL-EGFP mice and transgenic mice de
38 OL-EGFP(+) cells in tumors developing in the parabiotic partner lacking the fluorescent reporter.
39 ns of blood-derived monocytes from the young parabiotic partner, and preventing this recruitment part
40 Th2 cells recirculate and seed the lung of a parabiotic partner, conferring susceptibility to OVA cha
41 ocytes and progenitor cells derived from the parabiotic partner, suggesting splenic progenitor cells
45 eria: 1) inefficient (<20%) exchange between parabiotic partners; 2) gated importation by the thymus;
47 this historic study, Kurt R. Reissmann used parabiotic rats to demonstrate the functional existence
48 /ob) confirm hypotheses generated from early parabiotic studies that suggested the existence of a cir
50 lized a combination of genetic fate mapping, parabiotic, transcriptional, and functional analyses and
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