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1 receptor 2 (PAR2) on colonocytes to increase paracellular permeability.
2  (TJs) regulate epithelial cell polarity and paracellular permeability.
3 pproach, ConA labels only blood vessels with paracellular permeability.
4 alpha(12) in MDCK cells reversibly increased paracellular permeability.
5  claudin-1, and is associated with increased paracellular permeability.
6 ntal or mutant E. faecalis strains indicated paracellular permeability.
7 ansepithelial resistance (TER) and decreased paracellular permeability.
8 BB tight junctional disruption and increased paracellular permeability.
9 on of intracellular energetics and increased paracellular permeability.
10 1 likely contribute to regulated endothelial paracellular permeability.
11 ite, adenosine, in modulation of endothelial paracellular permeability.
12 may be the unifying mechanism for regulating paracellular permeability.
13 erved a PMN-mediated decrease in endothelial paracellular permeability.
14 resistance is attributable to an increase in paracellular permeability.
15 exhibited TER levels > 150 omega cm2 and low paracellular permeability.
16 nce and reduced the ratio of sodium/chloride paracellular permeability.
17 ght junctions (TJs), structures that control paracellular permeability.
18 audin-1) that critically regulate epithelial paracellular permeability.
19 s an important role in regulating epithelial paracellular permeability.
20 human microvascular endothelium and measured paracellular permeability.
21 tion of tight and adherens junctions and BEC paracellular permeability.
22  We studied the role of claudin-1 in hepatic paracellular permeability.
23 ns at junctions, wound healing dynamics, and paracellular permeability.
24 ability of junctional proteins and increased paracellular permeability.
25 VE-cadherin pathway responses which increase paracellular permeability.
26 EC adherens junctions, resulting in enhanced paracellular permeability.
27 nction protein internalization and increased paracellular permeability.
28 uption of endothelial cell-cell contacts and paracellular permeability.
29 id not alter the effect of occludin siRNA on paracellular permeability.
30 sed ZO-1 expression and increased epithelial paracellular permeability.
31 phorylation of VE-cadherin and p120(ctn) and paracellular permeability.
32 audin isoforms may play a role in regulating paracellular permeability.
33 CFA, resulting in significant changes in BBB paracellular permeability.
34 wn about the specific molecules that mediate paracellular permeabilities.
35 ng PrP(c) knockdown; the cells had increased paracellular permeability (1.5-fold over 48 hours; P < .
36 methasone significantly reduced [14C]sucrose paracellular permeability (-231% of controls).
37 tent with the autophagy-induced reduction in paracellular permeability, a marked decrease in the leve
38 elationship was developed for enhancement of paracellular permeability across Caco-2 cell monolayers.
39 elial cell determines the characteristics of paracellular permeability across epithelium.
40 n brain endothelial cells leads to increased paracellular permeability, allowing leukocyte entry into
41 ions as detected by a 3-fold decrease in the paracellular permeability and a 2-3-fold increase in the
42 l adhesion kinase mediates TGF-beta1-induced paracellular permeability and actin cytoskeleton dynamic
43             TGF-beta1 induces an increase in paracellular permeability and actin stress fiber formati
44 ntially deleterious increases in endothelial paracellular permeability and could serve as a basic mec
45                     RalA reduction increased paracellular permeability and decreased incorporation of
46 sms by which they contribute to the enhanced paracellular permeability and disease pathophysiology of
47 s into TJs, whereas RalB reduction decreased paracellular permeability and increased incorporation of
48 d by Claudin-1 absence, leading to increased paracellular permeability and liver injuries secondary t
49 thelial occludin may play a role in enhanced paracellular permeability and PMN transmigration that is
50                                  To test for paracellular permeability and size exclusion, FITC-label
51 rectly responsible for increasing epithelial paracellular permeability and that mice deficient in a m
52 ency of alkylphosphocholines as enhancers of paracellular permeability and the inhibitors of phosphol
53                                         Both paracellular permeability and the localization of TJ pro
54 TJ proteins, and TJ damage that may increase paracellular permeability and thereby contribute to the
55 mutation within ECs prevented VEGF-initiated paracellular permeability and tumor cell transmigration
56 oxin induces actin reorganization, increased paracellular permeability, and endothelial cell detachme
57 l cells in junction formation, regulation of paracellular permeability, and epithelial morphogenesis.
58 , and resulted in cell retraction, increased paracellular permeability, and facilitated eosinophil tr
59 VE-cadherin and beta-catenin, increased BMEC paracellular permeability, and facilitated the ability o
60 ion of claudins, the primary determinants of paracellular permeability, and measured transepithelial
61 , alters intestinal ion transport, increases paracellular permeability, and stimulates inflammation.
62  Our findings indicate that transcytosis and paracellular permeability are co-regulated through a sig
63          Claudins regulate cell adhesion and paracellular permeability as fundamental components of n
64 determined by reduced electrical resistance, paracellular permeability assays, and cell surface E-cad
65 eir basolateral side significantly decreased paracellular permeability; astrocyte-conditioned media d
66   Tight junctions (TJs) create ion-selective paracellular permeability barriers between extracellular
67 on of apical F-actin structures and enhanced paracellular permeability but did not alter the distribu
68 fore, phosphocholines are likely to increase paracellular permeability by modulating the signal trans
69 s, occludin S408 dephosphorylation regulates paracellular permeability by remodeling tight junction p
70 nslocation appears to be due to increases in paracellular permeability caused by migrating PMNs.
71 hat PKC signaling regulates toxin A-mediated paracellular permeability changes and ZO-1 translocation
72            SU6656 reduced TNF-alpha-mediated paracellular permeability changes, restored occludin, p1
73  The PAMPA data were modified to include the paracellular permeability component found in cellular mo
74                                              Paracellular permeabilities conferred by claudin-2 are c
75  thereby providing a mechanism for increased paracellular permeability during helminth infection.
76                                              Paracellular permeability enhancers have been used to im
77 olines as inhibitors of PLC and enhancers of paracellular permeability fit well into this correlation
78        With the use of cultured podocytes, a paracellular permeability flux assay was established to
79          We observed significant increase in paracellular permeability following siRNA-mediated suppr
80 ithelial electrical resistance by decreasing paracellular permeability for cations.
81 resistance (TEER), indicating an increase of paracellular permeability for ions.
82 ovide the molecular basis for regulating the paracellular permeability for water, solutes, and immune
83      In conclusion, VEGF induces a sustained paracellular permeability in capillary endothelial cells
84      Clostridium difficile toxin A increases paracellular permeability in colonic epithelial T84 cell
85 n-regulation of Sgpp2 attenuated LPS-induced paracellular permeability in cultured cells and enhanced
86 blishment of cell polarity and regulation of paracellular permeability in epithelia.
87 , knockdown of JAM1 was observed to increase paracellular permeability in epithelial monolayers.
88 ve previously shown that IFN-gamma increases paracellular permeability in model T84 epithelial cells
89        Similarly, we observed an increase of paracellular permeability in NRC cells silenced for clau
90 altering junctional complexes and increasing paracellular permeability in polarized ARPE-19 cells cul
91 alizes to tight junctions and contributes to paracellular permeability in polarized epithelia.
92     Defect in claudin-1 expression increases paracellular permeability in polarized hepatic cell line
93 ertain the mechanism by which VEGF regulates paracellular permeability in rats.
94 d to study cerebrovascular transcellular and paracellular permeability in vivo.
95 artially able to prevent the increase in BEC paracellular permeability induced by cytokines.
96 lts show that HMTBA prevents the increase in paracellular permeability induced by H2O2 or tumour necr
97       K8 silencing abolished the decrease in paracellular permeability induced by IL-6.
98                              The increase in paracellular permeability induced by TcdA and TcdB was a
99       As a morphological marker of increased paracellular permeability, its presence in patients with
100 tercellular junctional distance, and induced paracellular permeability, loss of apico-basal polarity
101                          We show that murine paracellular permeability markedly decreases during post
102 ma-induced increase in intestinal epithelial paracellular permeability may be an important mechanism
103                            We speculate that paracellular permeability may have evolved as a general
104                Using an in vitro endothelial paracellular permeability model, cell-free supernatants
105                Using an in vitro endothelial paracellular permeability model, we observed a PMN-media
106 luten-sensitized mice, P(HEMA-co-SS) reduced paracellular permeability, normalized anti-gliadin immun
107                        Decreased endothelial paracellular permeability occurred through adenosine A2B
108  cells, while the endocochlear potential and paracellular permeability of a biotin-based tracer in th
109  miRNA functions contributes to the enhanced paracellular permeability of ANDV-infected ECs and that
110                Consistent with this finding, paracellular permeability of AQP1(s) AV-infected TM mono
111                                          The paracellular permeability of bovine retinal endothelial
112                                              Paracellular permeability of cell monolayers to fluoresc
113 lonocytes, mast cell degranulation increased paracellular permeability of colonocytes.
114                                          The paracellular permeability of endothelial cells is unique
115 esistance was associated with an increase in paracellular permeability of glucose.
116  cofilin-1 by RNA interference increased the paracellular permeability of human colonic epithelial ce
117 ot stimulate Cl- secretion but increases the paracellular permeability of intestinal epithelia.
118 tructural changes may selectively affect the paracellular permeability of ions or small molecules, re
119 e gate functions to transiently regulate the paracellular permeability of large solutes and ions coul
120 eins and results in a 2-fold increase in the paracellular permeability of MDCK cell monolayers.
121  of apically applied occludin peptide on the paracellular permeability of molecular tracers and viral
122 ght junction membrane proteins that regulate paracellular permeability of renal epithelia to small io
123 akdown of TJs but in a selective increase in paracellular permeability of small molecules.
124              Nutrient starvation reduced the paracellular permeability of small-sized urea but not la
125 ed TAL tubules of claudin-10-deficient mice, paracellular permeability of sodium is decreased, and th
126           This coincided with an increase of paracellular permeability of the BBB to the small tracer
127 d decreases the effect of swainsonine on the paracellular permeability of the cell monolayer and also
128 promoting contraction of BBEC and increasing paracellular permeability of the CNS vasculature.
129 cytes also induced a twofold increase in the paracellular permeability of the endothelial monolayer.
130 uences resting intracellular volume and thus paracellular permeability of TM cell monolayers in vitro
131 tinal epithelium to dynamically regulate its paracellular permeability properties and better define t
132 s for the molecular components that regulate paracellular permeability properties in epithelial tissu
133 as been demonstrated to transiently increase paracellular permeability properties to provide an addit
134 opose these patterns underlie differences in paracellular permeability properties, such as electrical
135          Epithelial tight junctions regulate paracellular permeability, restrict apical/basolateral i
136 cture and function, although the increase of paracellular permeability returned to baseline after 24
137 ed alterations of the epithelial barrier and paracellular permeability suggest that common mechanisms
138 tine tissues from PrP(c-/-) mice had greater paracellular permeability than from wild-type mice (105.
139 ng the "loosening" of TJs and an increase in paracellular permeability, the BBB is able to "bend with
140                        Despite the increased paracellular permeability, there were no changes in gros
141 s cell polarity, cytoskeleton integrity, and paracellular permeability through inhibition of the smal
142       Occludin depletion increased diffusive paracellular permeability to 467 Da TAMRA by 15%, and pe
143 ell death and resulted in an increase in the paracellular permeability to [(3)H]inulin as a function
144 cing of Claudin-1 in Can 10 clones increased paracellular permeability to a level similar to that of
145 sepithelial resistance, a marked decrease in paracellular permeability to fluorescence isothiocyanate
146 l TER, these monolayers do exhibit increased paracellular permeability to fluorescent dextrans.
147 rties, including transepithelial resistance, paracellular permeability to hydrophilic solutes, and th
148 ltured epithelial cells demonstrate enhanced paracellular permeability to large molecules, revealing
149 rier-forming Caco-2 monolayers and increases paracellular permeability to macromolecular FITC-dextran
150 es, claudin-8 expression was found to reduce paracellular permeability to monovalent inorganic and or
151                     This indicates decreased paracellular permeability to NaCl but increased permeabi
152 rrent, and pH stat measurements, to estimate paracellular permeability to protons, ammonium and bicar
153 84 monolayers and demonstrated that enhanced paracellular permeability to small solutes occurred in t
154 aired the development of TEER, and increased paracellular permeability to sodium fluorescein in airwa
155 t junction (TJ) has a key role in regulating paracellular permeability to water and solutes in the ki
156                                              Paracellular permeability was assessed by apical-to-basa
157                                              Paracellular permeability was assessed by fluorescein is
158 ion and hyperalgesia were confirmed, and BBB paracellular permeability was assessed by in situ brain
159   The role of autophagy in the modulation of paracellular permeability was confirmed by pharmacologic
160 lones, Claudin-1 was localized at the TJ and paracellular permeability was decreased, compared to par
161                                              Paracellular permeability was determined by quantifying
162 -alkoxypropylphosphocholines as enhancers of paracellular permeability was not dependent on their exi
163 ays a role in mediating the shear effects on paracellular permeability, we overexpressed hAQP5 in 16H
164 fects on ER stress activation and epithelial paracellular permeability were examined in vitro as well
165     Changes in barrier function and abnormal paracellular permeability were found in both interfollic
166    In this study, highly potent enhancers of paracellular permeability were identified in the 3-alkyl
167            Zonulin production and changes in paracellular permeability were monitored in Ussing chamb
168 ell cultures from a VEGF-induced increase in paracellular permeability, whereas recombinant OCLN expr
169 role in regulating the maintenance of TJ and paracellular permeability, which may explain how various
170 and interferon-gamma significantly increased paracellular permeability, which was blocked by cotreatm
171 ronic IP, we demonstrated alterations in BBB paracellular permeability with correlating changes in ti

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