ライフサイエンスコーパス: paracentral

1 defined by type of VF defect (peripheral or paracentral).

2 reference between central (0-3 degrees ) and paracentral (4-8 degrees ) V1 in the prosimian bush baby

3 face OCT appearance may be used to classify paracentral acute maculopathy into distinct subtypes.

4 cent retinal vascular occlusion illustrating paracentral acute middle maculopathy (PAMM) in a periven

5 cal coherence tomography (SD-OCT) finding of paracentral acute middle maculopathy (PAMM) that can be

6 4 mm(-1) vs 14.7 +/- 3.5 mm(-1) in eyes with paracentral acute middle maculopathy (reduction -19.4%,

7 8 mm(-1) vs 12.1 +/- 1.9 mm(-1) in eyes with paracentral acute middle maculopathy (reduction -6.0%, P

8 ciations leading to retinal vasculopathy and paracentral acute middle maculopathy include eye compres

9 of 9 patients (10 eyes) from 5 centers with paracentral acute middle maculopathy lesions and previou

10 Paracentral acute middle maculopathy lesions correspond

11 Follow-up SD OCT analysis of these paracentral acute middle maculopathy lesions demonstrate

12 Paracentral acute middle maculopathy lesions may develop

13 Paracentral acute middle maculopathy may be best evaluat

14 Paracentral acute middle maculopathy may represent a nov

15 Paracentral acute middle maculopathy refers to character

16 , and en face OCT images of 16 patients with paracentral acute middle maculopathy were evaluated.

17 imodal imaging findings from 8 patients with paracentral acute middle maculopathy were reviewed and a

18 d type 1 SD-OCT lesions, also referred to as paracentral acute middle maculopathy, and 5 eyes (4 pati

19 he middle retinal layers, otherwise known as paracentral acute middle maculopathy, were observed in a

20 ere evaluated for the presence of coexisting paracentral acute middle maculopathy.

21 resembling other acute maculopathies such as paracentral acute middle maculopathy.

22 5 (5.2%) demonstrated evidence of concurrent paracentral acute middle maculopathy.

23 hinal cortex or the mediodorsal (MDn) or the paracentral and centrolateral (PC-CL) thalamic nuclei di

24 ticipants, with regional decreases involving paracentral and occipital regions in both PD + VH and PD

25 scicular, oval paracentral, central lateral, paracentral, and central medial nuclei), as well as the

26 erodorsal, paraventricular, central lateral, paracentral, and central medial nuclei.

27 scicular, central lateral, paracentral, oval paracentral, and central medial nuclei; in the midline t

28 d into 5 clusters: nasal, temporal, central, paracentral, and peripheral.

29 nferior parietal lobule, and also cingulate, paracentral, and precentral gyri, compared with the non-

30 terior medial parvicellular, caudal PC, oval paracentral, and reticular thalamic nuclei.

31 In the central and paracentral areas, however, significant numbers of CD11c

32 While central and paracentral axial curvatures correlated with birth weigh

33 ate functional connectivity of posterior and paracentral brain regions, whereas the presence of VH is

34 parafascicular, medial parafascicular, oval paracentral, central lateral, paracentral, and central m

35 ostral intralaminar nuclei (central lateral, paracentral, central medial nuclei) as well as to the ve

36 and lateral parafascicular, central lateral, paracentral, central medial, rhomboid, reuniens, and sub

37 epithelial opacifications in the central and paracentral cornea.

38 BOLD reduction in the right insula and left paracentral cortex in response to SS.

39 One graft had a linear paracentral fixed area of interface separation correspon

40 tion produces central corneal steepening and paracentral flattening in the central 3-mm diameter.

41 neal stromal tissue samples from central and paracentral hypocellular primitive stromal interface sca

42 emporal gyrus, superior parietal lobule, and paracentral, lateral orbitofrontal, and lateral occipita

43 scotoma and demonstrated a classic dark gray paracentral lesion with near-infrared imaging.

44 The paracentral lobule and cuneus had the highest resting me

45 Cortical thickness in the pars orbitalis, paracentral lobule, fusiform gyrus and inferior temporal

46 parietal lobes, temporoparietal junction and paracentral lobule, right superior temporal and parietal

47 of the precentral and postcentral gyrus, the paracentral lobule, the superior temporal gyrus, the mid

48 ght superior frontal and parietal lobes, and paracentral lobule.

49 Finally, voxelwise GM loss in the right paracentral lobulus correlated with bowel and bladder di

50 at diagnosis (peripheral loss only or early paracentral loss).

51 at diagnosis (peripheral loss only or early paracentral loss).

52 Paracentral, mediodorsal, lateral posterior, and medial

53 glia, thalamus, hippocampus, frontal and the paracentral motor cortex and cerebellar vermis.

54 obex was activated and projected to the oval paracentral nucleus (OPC) of the intralaminar thalamic n

55 t to the central canal, corresponding to the paracentral nucleus of Herrick, and in the lateral funic

56 cular formation, lateral geniculate nucleus, paracentral nucleus, central medial nucleus, lateral hyp

57 ation of initial visual field defect (either paracentral or peripheral).

58 ascicular, central medial, paracentral, oval paracentral, or central lateral nucleus.

59 lar, medial parafascicular, central lateral, paracentral, oval paracentral, and central medial nuclei

60 lar, lateral parafascicular, central medial, paracentral, oval paracentral, or central lateral nucleu

61 parabrachial subnuclei projected to the oval paracentral, parafascicular, and rhomboid thalamic nucle

62 eus (MD1), VL, and the central lateral (CL), paracentral (PC), central medial, rhomboid and ventromed

63 medial parafascicular, central lateral (CL), paracentral (PC), or central medial nucleus-or one of th

64 Central, paracentral, pericentral, and the thinnest corneal thick

65 ptor loss with a ring of minimally preserved paracentral photoreceptor nuclear layer.

66 The most important factor associated with paracentral progression among eyes that reached a progre

67 ery and have comparable sensitivities in the paracentral region of the cornea.

68 ery and have comparable sensitivities in the paracentral region of the cornea.

69 e, new techniques that enlist measurement of paracentral regions are discussed, and the ability of ea

70 ients with early visual field defects in the paracentral regions compared with those in the periphera

71 d with metabolism in the anterior cingulate, paracentral, right orbitofrontal, and left thalamic regi

72 patients corresponding to the diameter of a paracentral ring of increased fundus autofluorescence.

73 All patients presented with an acute paracentral scotoma and demonstrated a classic dark gray

74 d to our centre because of a deep unilateral paracentral scotoma with the presumptive diagnosis of a

75 e defects (n = 12), nasal steps (n = 11) and paracentral scotomas (n = 16).

76 atrophy of the INL, resulting in persistent paracentral scotomas for the patient.

77 in young healthy females with acute onset of paracentral scotomas.

78 ears of age and presented with complaints of paracentral scotomas.

79 The paracentral thalamic nucleus received an input only from

80 elevated at fixation, but there were greater paracentral than central abnormalities.

81 s were overrepresented in central but not in paracentral V1 and that isoorientation domain size tende

82 selectivity or magnitude between central and paracentral V1.

83 ory devoted to higher SFs in central than in paracentral V1.

84 V2 SNPs were associated with POAG with early paracentral VF (top SNP, rs17588172; pooled P = 1.07 x 1

85 ciation between CAV1/CAV2 SNPs and POAG with paracentral VF defects.

86 ogenesis in women and in patients with early paracentral VF defects.

87 for heterogeneity = .01) for POAG with early paracentral VF loss (433 cases; quintile 5 vs quintile 1

88 7; P for trend = .02) and 0.52 for POAG with paracentral VF loss (95% CI, 0.29-0.96; P for trend < .0

89 Hg (MVRR, 1.93; 95% CI, 1.09-3.43) and early paracentral VF loss (MVRR, 2.27; 95% CI, 1.32-3.88).

90 ower POAG risk, particularly POAG with early paracentral VF loss at diagnosis.

91 Progression within the paracentral VF was more common in the NTG group (75% vs.

92 dorsal representation of foveal vision, with paracentral vision represented more caudally.

93 atients are visually impaired by symptomatic paracentral visual field defects despite a normal VA.

94 enotype is potentially associated with early paracentral visual field defects in primary open-angle g

95 Central/paracentral visual field defects were seen in 9 eyes.