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1 nuses, with reduced abundance in the deep LN paracortex.
2 tor T cells localized exclusively within the paracortex.
3 tical ridge, poorly accessing the lymph node paracortex.
4 ecreased the speed of locomotion in the deep paracortex.
5 169(-/-) mice but penetrated deeper into the paracortex.
6 ction a small amount of LPS was found in the paracortex.
7 icients than naive T lymphocytes in the deep paracortex.
8 network regulated naive T cell access to the paracortex and also supported and defined the limits of
9 cific CD4 T cells redistributed to the outer paracortex and interacted with CD11b(+), but not CD8(+)
11 ation, infected cells were identified in the paracortex and subcapsular sinus of the draining interna
12 ompetent mangabeys but were seen in both the paracortex and the germinal center of SIV-infected macaq
13 lutaminase is expressed at low levels in the paracortex around primary follicles but is markedly up-r
14 aining lymph nodes, where they reside in the paracortex as interdigitating dendritic cells (IDCs).
16 ection, they were confined to the lymph node paracortex in immune-competent mangabeys but were seen i
17 ual expressed CXCL10 and CXCL11 mRNAs in the paracortex, including venules, as detected by in situ hy
21 Naive T lymphocyte locomotion in the deep paracortex of the LN required a perfusion rate of >13 mi
23 f soluble molecules to distinct sites in the paracortex, particularly the high endothelial venule.
24 dominately in extrafollicular regions of the paracortex that contain T-lymphocytes and macrophages.
25 ormly distributed throughout the T cell-rich paracortex, whereas CD11b(+) dendritic cells were locate
26 to follicles, whereas T cells remain in the paracortex, with each lymphocyte type showing apparently
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