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1  roles in regulating cardiac development via paracrine signaling.
2 involvement of stem cell differentiation and paracrine signaling.
3 hat this factor plays in cardiac disease and paracrine signaling.
4 ing both hypertrophic and hypoxia-stimulated paracrine signaling.
5 primed state of DC maturation mediated by DC paracrine signaling.
6 DCs to a panoply of cytokines/chemokines via paracrine signaling.
7 ifferentiated state of mammary epithelia via paracrine signaling.
8 tivation of Smads through TGFbeta1 autocrine/paracrine signaling.
9 e for the increased autocrine endogenous and paracrine signaling.
10 ses secretion of S1P, allowing for autocrine/paracrine signaling.
11 lls, it may be engaged in both autocrine and paracrine signaling.
12 implicated in metastatic progression through paracrine signaling.
13 rstood as an ATP release channel involved in paracrine signaling.
14 al systems may be affected by such limits of paracrine signaling.
15 an promote growth via KITLG autocrine and/or paracrine signaling.
16 organized in a modular network implicated in paracrine signaling.
17 asis, and angiogenesis through autocrine and paracrine signaling.
18 ate stem cell fate outcomes to autocrine and paracrine signaling.
19 liberated during cellular necrosis to effect paracrine signaling.
20 TGF-beta1, which is capable of autocrine and paracrine signaling.
21 n addition to spatial patterns of intratumor paracrine signaling, a possible cell-cycle-associated re
22                                         This paracrine signaling acts to constrain ISC proliferation
23 omote repair of the infarcted myocardium via paracrine signaling after transplantation.
24 may participate importantly in autocrine and paracrine signaling among leukocytes and vascular endoth
25 s animal models documented that simultaneous paracrine signaling and cell-to-cell surface contact reg
26 mechanisms underlying astrocytic-endothelial paracrine signaling and have found that integrin-mediate
27 creted TGF-beta1 is capable of autocrine and paracrine signaling and is dependent upon expression of
28 rotein also efficiently blocks autocrine and paracrine signaling and reduces the proliferation of MCF
29 icroColonies') to quantitatively investigate paracrine signaling and the response to external stimuli
30 ation through inhibition of cell activation, paracrine signaling, and dampened cellular proinflammato
31 issue formation by neonatal chondrocytes via paracrine signaling, and highlights the importance of co
32 ing environments, regulated by autocrine and paracrine signaling, and modulated by cell organization,
33 ude direct incorporation into blood vessels, paracrine signaling, and tunneling nanotube renewal of m
34 sues did not have similar effects, excluding paracrine signaling as a major mechanism.
35 y recruited inflammatory cells, allowing for paracrine signaling at the site of an infection.
36 s reveal a novel TGF-beta, androgen, and Wnt paracrine signaling axis that enables prostatic regressi
37 hat alphavbeta8 integrin is a component of a paracrine signaling axis that links astrocytes to blood
38 Thus, HIFs mediate complex and bidirectional paracrine signaling between BCCs and MSCs that stimulate
39 idence, however, points to a direct role for paracrine signaling between blood vessel cells and surro
40             We demonstrate that HIFs mediate paracrine signaling between breast cancer cells (BCCs) a
41 ate the latter effect is caused by disturbed paracrine signaling between endothelial and surrounding
42                                              Paracrine signaling between hepatic stellate cells (HSCs
43 est the hypothesis that sorafenib influences paracrine signaling between HSCs and LECs and thereby re
44 autoreceptor function, provide a pathway for paracrine signaling between NG neurons, and contribute t
45                                              Paracrine signaling between pancreatic islet beta-cells
46                                              Paracrine signaling between podocytes and glomerular end
47 lies upon coordination of both autocrine and paracrine signaling between the budding epithelium and a
48 ing on the nutrient type and likely involves paracrine signaling between the differentiated beta-cell
49 riction (IUGR) on pancreatic vascularity and paracrine signaling between the EC and beta-cell.
50                      These changes occur via paracrine signaling between the uterine epithelium and s
51                                 Autocrine or paracrine signaling by beta interferon (IFN-beta) is ess
52                                              Paracrine signaling by enterochromaffin cells (EC), whic
53  more slowly developing mechanisms involving paracrine signaling by extracellular peptides (C-type na
54  We conclude that HER-mediated autocrine and paracrine signaling by HB-EGF or other EGF family member
55 Survival and proliferation of hHpSCs require paracrine signaling by hepatic stellate cells and/or ang
56 s to a positive feedback loop involving auto/paracrine signaling by IL13 and the IL4/13 receptor.
57 rom of each other, a distance sufficient for paracrine signaling by leukotrienes to operate effective
58                         The concept of local paracrine signaling by nitric oxide over subcellular dis
59                 Enhancement of Hmga2-induced paracrine signaling by overexpression of androgen recept
60 ngs establish a critical functional role for paracrine signaling by tumor-derived osteopontin in repr
61                        Blockade of autocrine/paracrine signaling can aid in dissecting the contributi
62 of how quantitative control of autocrine and paracrine signaling can be integrated with spatial organ
63            While it is well established that paracrine signaling can similarly elicit diverse respons
64    Additionally, ER-stressed astrocytes, via paracrine signaling, can stimulate activation of microgl
65 ix metalloproteinase-1 (MMP1), orchestrate a paracrine signaling cascade to modulate the bone microen
66                                              Paracrine signaling cascades regulated by hypoxia initia
67 expressing oncogenic mutants of Src, whereas paracrine signaling could stimulate EGFR and ERK signali
68 tion, which effectively blocks all autocrine/paracrine signaling crucial to induction of downstream e
69 ivate valvular interstitial cells and latent paracrine signaling cytokines (eg, transforming growth f
70              Our findings help elucidate the paracrine signaling events activated by a compromised mu
71 rain) can trigger a cascade of autocrine and paracrine signaling events between ECs and SMCs critical
72    These effects occur through autocrine and paracrine signaling events initiated by interactions bet
73 poptosis during cytotoxic treatment activate paracrine signaling events that promote the growth of su
74 as ATP and ADP) and subsequent autocrine and paracrine signaling events through nucleotide receptors.
75  effect on the epithelium through additional paracrine signaling events.
76 gest that suPAR may function as an important paracrine signaling factor in EGFRvIII-positive GBMs, in
77 h is a well-established potent autocrine and paracrine signaling factor modulating a variety of cellu
78 l breast cancer: TGFbeta is an autocrine and paracrine signaling factor that drives cell invasion and
79 zation are coupled by localized secretion of paracrine signaling factors by the differentiating hMSCs
80  other nucleotides function as autocrine and paracrine signaling factors in many tissues.
81 for kinins, including those in autocrine and paracrine signaling for skeletal and cardiac muscle ener
82                               Mast cells use paracrine signaling for the transfer of chemical informa
83 omatic events may drive tumor growth through paracrine signaling fostering a tumor ecologic niche are
84 results demonstrate a directionally specific paracrine signaling from epithelial FGF9 and stromal FGF
85                    This likely resulted from paracrine signaling from hepatocytes in the peritoneal c
86                                              Paracrine signaling from infected DCs induced an antivir
87  occurred in hepatocytes and was mediated by paracrine signaling from Kupffer cells.
88                                              Paracrine signaling from lung epithelium to the surround
89 IgG-mediated changes may be a means by which paracrine signaling from neuronal activity influences mi
90 s then applied to HMECs to determine whether paracrine signaling from PELP1-cyto-activated macrophage
91                                              Paracrine signaling from SCF to KIT, between differentia
92  CFB mechanical activation and found that 1) paracrine signaling from stretched cardiomyocytes induce
93                                    Moreover, paracrine signaling from the epicardium and endocardium
94                        The expression of the paracrine signaling hormone pituitary adenylate cyclase-
95              Cilia are critical mediators of paracrine signaling; however, it is unknown whether prot
96                                              Paracrine signaling in a bacterial population ensures th
97                 Here we present evidence for paracrine signaling in bacterial populations-some cells
98 a general tool for the quantitative study of paracrine signaling in both adherent and nonadherent cel
99 nd provide a quantitative assay for studying paracrine signaling in early development.
100 ata suggest that inhibition of Ezh2 promotes paracrine signaling in osteoblasts and has bone-anabolic
101  growth factor-1 receptor (IGF-1R) autocrine/paracrine signaling in patients with renal cell carcinom
102 ivity is required in Shh-producing cells for paracrine signaling in Shh target fields, we used a ShhG
103 tive oxygen species activate NRG-1beta/erbB4 paracrine signaling in the heart and suggest that this s
104 role of the cardiac myocyte as a mediator of paracrine signaling in the heart has remained unclear.
105 ng the inflammatory and fibrotic response by paracrine signaling inducing the secretion of a variety
106 skeletal muscle hypertrophy alters autocrine/paracrine signaling, intracellular signaling, and transc
107 nterfering with homeostatic VEGFR2-dependent paracrine signaling involving interactions between hepat
108 ls in BCP-ALL cells, but we demonstrate that paracrine signaling involving prostaglandin E2-induced c
109 itive cells but rather in adjacent cells via paracrine signaling involving several local growth facto
110                     These data indicate that paracrine signaling is an important mediator of bone mar
111 atical modeling suggests that a high rate of paracrine signaling is likely to occur among DCs located
112                                              Paracrine signaling is proposed as a link that emphasize
113                          We demonstrate that paracrine signaling is sufficient to restore cellular sy
114                                Autocrine and paracrine signaling leading to stimulation of tumor cell
115                        A BDNF/TrkB autocrine/paracrine signaling loop has additionally been implicate
116 contributes to an active BDNF/TrkB autocrine/paracrine signaling loop in HTLV-1-infected T cells that
117 MCP-1 upregulation is driven by an autocrine/paracrine signaling loop in which interleukin (IL)-1alph
118 s and perivascular fibroblasts, suggesting a paracrine signaling loop.
119   We previously cataloged putative autocrine/paracrine signaling loops in pancreatic islets, includin
120 ctivity not only disrupts IL-1 autocrine and paracrine signaling loops that can alert effector cells
121  blood mononuclear cells upon coculture in a paracrine signaling manner.
122 cinoma cells, suggesting that this autocrine-paracrine signaling may be a common response to Ras/Raf
123 ddition, these findings argue that US28-CCL5 paracrine signaling may contribute to glioma progression
124 t outcome, suggesting that HCMV pp71-induced paracrine signaling may contribute to the aggressive phe
125 ion during mechanotransduction and that VEGF paracrine signaling may provide potent cross-talk among
126  a dual circulation, and that VEGF autocrine/paracrine signaling may regulate these processes.
127 evels, indicating that a TNF-alpha autocrine/paracrine signaling mechanism alone is not sufficient to
128                          We describe various paracrine signaling mechanism(s) by which the loss of st
129 ivated in sebocyte progenitors, suggesting a paracrine signaling mechanism.
130 mmune systems, possibly through autocrine or paracrine signaling mechanisms.
131 r growth and survival through autocrine- and paracrine-signaling mechanisms.
132                         However, whether the paracrine signaling mediated by angiogenin secretion is
133                                              Paracrine signaling mediated by FGF-10 and the FGF-R2III
134 enous NO in the microenvironment facilitates paracrine signaling, mediates immune responses, and trig
135 and identify fibroblast-derived miR-21* as a paracrine signaling mediator of cardiomyocyte hypertroph
136 ns; however, miRNAs have emerged recently as paracrine signaling mediators.
137 l of peptide growth factors in autocrine and paracrine signaling, mesenchymal-epithelial interactions
138 ignaling components, the logic of this auto-/paracrine signaling module in growth control remains poo
139 minobutyric acid, an important autocrine and paracrine signaling molecule and a survival factor in is
140             The agouti locus encodes a novel paracrine signaling molecule containing a signal sequenc
141 ytryptamine (5-HT) is a neurotransmitter and paracrine signaling molecule in the gut.
142                            Agouti protein, a paracrine signaling molecule normally limited to skin, i
143 (eNOS)-derived NO has long been considered a paracrine signaling molecule only capable of affecting n
144                The mouse agouti protein is a paracrine signaling molecule that causes yellow pigment
145  the Agouti coat color gene, which encodes a paracrine signaling molecule that induces a swithc in me
146 ta (LPAAT-beta), is a well-studied autocrine/paracrine signaling molecule that is secreted by ovarian
147 n ATP metabolite, which acts as an autocrine/paracrine signaling molecule through A2b adenosine recep
148 llular ATP represents an important autocrine/paracrine signaling molecule within the liver.
149 roto-oncogene Wnt3, which encodes a secreted paracrine signaling molecule, is expressed in developing
150 ties, such as converting a juxtacrine into a paracrine signaling molecule.
151 ression of agouti-signaling protein (ASP), a paracrine-signaling molecule that regulates pigment-type
152 tor parathyroid hormone-related protein is a paracrine-signaling molecule that regulates the developm
153 the family of prostaglandins (PG), autocrine/paracrine signaling molecules synthesized via the cycloo
154 AgRP) involved in energy balance, are novel, paracrine signaling molecules that act as inverse agonis
155                          This suggested that paracrine signaling molecules that induce PMN degranulat
156 e only member of the WNT family of autocrine/paracrine signaling molecules whose expression in the lu
157 etastatic growth in mice via upregulation of paracrine signaling molecules.
158 rotein and Agouti-related protein (Agrp) are paracrine-signaling molecules that normally regulate pig
159                    Here, we have uncovered a paracrine signaling network between the adaptive and inn
160 rapeutic approaches, namely the miR-mediated paracrine signaling network.
161 tal stress can therefore block autocrine and paracrine signaling of the Wnt/beta-catenin pathway and
162 nomas and other cancers, while autocrine and paracrine signaling of this receptor/ligand pair has bee
163 sent study, we identified ATP-triggered PGE2 paracrine signaling originating from beta-ICs as a mecha
164         Prostaglandins mediate autacrine and paracrine signaling over short distances.
165 over, the evidence provided suggests a novel paracrine signaling pathway for epithelia, which previou
166            These findings imply an important paracrine signaling pathway in the heart.
167                         Here we identified a paracrine signaling pathway in the kidney in which high
168 in this pathway, miR-145 seems to suppress a paracrine signaling pathway in the tumor microenvironmen
169 ound that HBZ promotes a BDNF/TrkB autocrine/paracrine signaling pathway that is known to enhance the
170          Collectively, these data identify a paracrine signaling pathway that links the neuroepitheli
171  the differential activation of an autocrine/paracrine signaling pathway.
172 ion of 5'-AMP to adenosine required for this paracrine signaling pathway.
173 at basal body proteasomal regulation governs paracrine signaling pathways and suggest that augmenting
174                        Complex autocrine and paracrine signaling pathways control the multiple cycles
175 hophysiological significance, the origin and paracrine signaling pathways that regulate epicardial ad
176  neuropeptides urocortins (Ucns) are ancient paracrine-signaling peptides secreted in both the centra
177 o prostate cancer disease recurrence through paracrine signaling processes.
178 SC-mediated heterocellular coupling (HC) and paracrine signaling (PS) on human cardiac contractility
179 fected DCs and uninfected DCs to investigate paracrine signaling responses.
180 hat Disp1 is not absolutely required for the paracrine signaling role of Ihh in the skeleton.
181 duction of an alpha-ketoglutarate (alpha-KG) paracrine signaling system.
182            Thus, CAMP is a promoter of islet paracrine signaling that enhances islet function and glu
183                The latter involves autocrine/paracrine signaling that enhances the viability and grow
184 tic colonization are most likely mediated by paracrine signaling that enhances tumor/stromal cell int
185 uggest a previously undescribed mechanism of paracrine signaling that in vivo may involve the reversi
186 entiation and behavior mediated by autocrine-paracrine signaling that instructs transcriptional proce
187 may influence PEL through VEGF autocrine and paracrine signaling that promotes PEL cell growth and ex
188                                              Paracrine signaling through receptor activator of NF-kap
189  suggest that strain may induce autocrine or paracrine signaling through TGFbeta superfamily ligands.
190           Secreted IFNs induce autocrine and paracrine signaling through the JAK-STAT pathway, leadin
191 uced progesterone resistance indicating that paracrine signaling through the stroma is essential for
192 ted therapy-induced stromal ELR(+) chemokine paracrine signaling, thus enhancing treatment response a
193 n interstitial fibrosis, and through altered paracrine signaling to cardiomyocytes, which become hype
194                                              Paracrine signaling to endogenous cells amplifies the ef
195 reveals that cells utilize optimal levels of paracrine signaling to maximize the accuracy of gradient
196  active as shown by its ability to stimulate paracrine signaling via c-Met, the cell surface receptor
197                                              Paracrine signaling via gamma-aminobutyric acid (GABA) a
198 nstrated Akt1 mediated MEC migration through paracrine signaling via induction of expression and secr
199  These results suggest that autocrine and/or paracrine signaling via locally generated SPMs in the va
200                                              Paracrine signaling via platelet-derived growth factor B
201 on of ADAM17 by Src(E378G) leads to enhanced paracrine signaling via release of EGFR ligands into the
202                       Activation of NRG/erbB paracrine signaling was also seen in the intact heart su
203                         To determine role of paracrine signaling, we cultured pure mESC-CMs within mi
204 llus subtilis biofilm formation depends upon paracrine signaling where the signal-producing and targe
205 on of cancer by mediating stromal-epithelial paracrine signaling, which can aberrantly modulate cellu
206                                        Hence paracrine signaling will generally extend beyond the syn
207 man mammary tumor model is dependent on both paracrine signaling with host macrophages as well as aut
208 onceivable that TrkB also mediates autocrine/paracrine signaling within these structures or anterogra

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