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1 rus, rhinovirus, and influenza virus but not parainfluenza virus.
2 p of viruses that includes measles virus and parainfluenza viruses.
3 an respiratory syncytial virus and the human parainfluenza viruses.
4 tein subunits play the cell entry concert of parainfluenza viruses.
5 detects influenza A virus (Flu-A) and Flu-B, parainfluenza virus 1 (PIV-1), PIV-2, and PIV-3, and res
6 l virus (RSV), human metapneumovirus (HMPV), parainfluenza virus 1 to 3 (PIV1, PIV2, and PIV3), and a
7                   FilmArray RP detected more parainfluenza viruses 1 and 3 than ProParaflu+ (18 versu
8  viruses, respiratory syncytial virus (RSV), parainfluenza viruses 1 to 3, and adenovirus (DAKO Diagn
9 virus, influenza A virus, influenza B virus, parainfluenza viruses 1 to 3, and respiratory syncytial
10 nfluenza A virus H1-2009, influenza B virus, parainfluenza viruses 1 to 4, respiratory syncytial viru
11  syncytial virus; influenza A and B viruses; parainfluenza viruses 1, 2, 3, and 4; human metapneumovi
12 an respiratory syncytial virus (HRSV); human parainfluenza viruses 1, 2, and 3 (HPIV1, -2, and -3, re
13 piratory syncytial virus, influenza A and B, parainfluenza viruses 1-3, and adenovirus.
14 picornaviruses, coronaviruses 229E and OC43, parainfluenza viruses 1-3, influenza viruses AH1, AH3, a
15 in reaction for respiratory syncytial virus, parainfluenza viruses 1-4, influenza A and B, human meta
16 ramyxoviruses simian virus 5 (SV5) and human parainfluenza virus 2 (HPIV2) overcome IFN-alpha/beta re
17 hese are encoded by mumps virus (MuV), human parainfluenza virus 2 (hPIV2), and parainfluenza virus 5
18    Simian virus 5 (SV5) targets STAT1, human parainfluenza virus 2 targets STAT2, and mumps virus tar
19 ith respiratory syncytial virus (RSV), human parainfluenza virus 3 (HPIV-3), and influenza virus on t
20          We previously reported that a human parainfluenza virus 3 (HPIV3) F peptide effectively inhi
21 described for a cluster of 12 cases of human parainfluenza virus 3 (HPIV3) infection that occurred am
22 titatively influence fusion promotion, human parainfluenza virus 3 (HPIV3) variants with alterations
23 yxoviruses, such as Nipah virus (NiV), human parainfluenza virus 3 (HPIV3), measles virus (MeV), mump
24                                              Parainfluenza virus 3 (PIV-3) infection is highly restri
25         The majority of this decrease was in parainfluenza virus 3 (PIV3) (8.3% to 2.2%, P < .001).
26 c fibrosis patients; however, its use during parainfluenza virus 3 (PIV3) infection has not been eval
27                           The secreted human parainfluenza virus 3 F forms a trimer with distinct hea
28               However, we found that a human parainfluenza virus 3 F-peptide is more effective at inh
29 eaved ectodomain of the paramyxovirus, human parainfluenza virus 3 fusion (F) protein, a member of th
30  homologous HN protein, as well as NDV-human parainfluenza virus 3 HN chimeras.
31 ection but similar to that for influenza and parainfluenza virus 3 infection in all age groups.
32  mumps virus, Newcastle disease virus, human parainfluenza virus 3, and Nipah virus.
33                Antibody titers to RSV, human parainfluenza virus 3, measles, and influenza H1N1, H3N2
34  cell lines with Sendai virus (SeV) or human parainfluenza virus 3, two prototypic paramyxoviruses, c
35 dence similar to that of influenza virus and parainfluenza virus 3.
36 on of 274 of 279 influenza viruses, 33 of 38 parainfluenza viruses, 35 of 51 adenoviruses, and 52 of
37      Putative viral pathogens included human parainfluenza virus 4 (aOR 9.3, P = .12), human bocaviru
38 rus [EV], 118; bocavirus, 8; coronavirus, 7; parainfluenza virus 4, 4; Mycoplasma pneumoniae, 1).
39 lture (metapneumovirus, coronaviruses [CoV], parainfluenza viruses 4a and 4b, and rhinoviruses) and t
40 hat Cav-1 colocalizes with the paramyxovirus parainfluenza virus 5 (PIV-5) nucleocapsid (NP), matrix
41                                              Parainfluenza virus 5 (PIV5) activates and is neutralize
42                      For the paramyxoviruses parainfluenza virus 5 (PIV5) and mumps virus, M-NP inter
43 Proline substitution in this region of HN of parainfluenza virus 5 (PIV5) and Newcastle disease virus
44     In this work, we generated a recombinant parainfluenza virus 5 (PIV5) containing NP from H5N1 (A/
45                            The paramyxovirus parainfluenza virus 5 (PIV5) enters cells by fusion of t
46 igh similarity to the structure of prefusion parainfluenza virus 5 (PIV5) F, with the main structural
47  Because only the prefusion structure of the parainfluenza virus 5 (PIV5) F-trimer is available, to s
48 MR spectroscopy, we show that the TMD of the parainfluenza virus 5 (PIV5) fusion protein adopts lipid
49                                              Parainfluenza virus 5 (PIV5) HN exists as a noncovalent
50                                              Parainfluenza virus 5 (PIV5) is a member of the Paramyxo
51                                              Parainfluenza virus 5 (PIV5) is a promising viral vector
52                                              Parainfluenza virus 5 (PIV5) is a prototypical paramyxov
53                                              Parainfluenza virus 5 (PIV5) is an appealing vector for
54                                              Parainfluenza virus 5 (PIV5) is an enveloped, single-str
55                             The P protein of parainfluenza virus 5 (PIV5) is an essential cofactor of
56                                              Parainfluenza virus 5 (PIV5) is thought to contribute to
57     To investigate the role of NP protein in parainfluenza virus 5 (PIV5) particle formation, NP prot
58        In this work, we tested a recombinant parainfluenza virus 5 (PIV5) strain expressing the glyco
59 serendipitously identified a viral mRNA from parainfluenza virus 5 (PIV5) that activates IFN expressi
60                              Preparations of parainfluenza virus 5 (PIV5) that are potent activators
61 quence variation of 16 different isolates of parainfluenza virus 5 (PIV5) that were isolated from a n
62 unable to be recognized by measles virus and parainfluenza virus 5 (PIV5) V proteins were tested in s
63                    We tested the recombinant parainfluenza virus 5 (PIV5) vectors expressing RSV glyc
64 he threonine residue at position 286 of P of parainfluenza virus 5 (PIV5) was found phosphorylated.
65     The V proteins of measles virus (MV) and parainfluenza virus 5 (PIV5) were introduced into HFLC u
66 rotein (prefusion form) of the paramyxovirus parainfluenza virus 5 (PIV5) WR isolate was determined.
67                                              Parainfluenza virus 5 (PIV5), a paramyxovirus, is not kn
68      Similar results were also observed with parainfluenza virus 5 (PIV5), a paramyxovirus, when neut
69           Here we show that vaccination with parainfluenza virus 5 (PIV5), a promising live viral vec
70                                 Infection by parainfluenza virus 5 (PIV5), a prototypical member of t
71                                              Parainfluenza virus 5 (PIV5), a prototypical paramyxovir
72                                The genome of parainfluenza virus 5 (PIV5), a prototypical paramyxovir
73 V), human parainfluenza virus 2 (hPIV2), and parainfluenza virus 5 (PIV5), all members of the genus R
74                                              Parainfluenza virus 5 (PIV5), formerly known as simian v
75  that a porcine isolate of the paramyxovirus parainfluenza virus 5 (PIV5), known as SER, requires a l
76       The strain diversity of a rubulavirus, parainfluenza virus 5 (PIV5), was investigated by compar
77                 Previously, we developed two parainfluenza virus 5 (PIV5)-based RSV vaccine candidate
78                    We previously generated a parainfluenza virus 5 (PIV5)-vectored vaccine candidate
79              In this study, we evaluated two parainfluenza virus 5 (PIV5)-vectored vaccines expressin
80 f the matrix (M) protein of a paramyxovirus, parainfluenza virus 5 (PIV5).
81 -3 as a binding partner for the M protein of parainfluenza virus 5 (PIV5).
82      Similar results were also observed with parainfluenza virus 5 (PIV5).
83 he FP of the F protein of the paramyxovirus, parainfluenza virus 5 (PIV5).
84 sion (F) protein of the paramxyovirus simian parainfluenza virus 5 (SV5) promotes virus-cell and cell
85 on (F) protein from the paramyxovirus simian parainfluenza virus 5 (SV5) resulted in mutant F protein
86 ined the ability of the paramyxovirus simian parainfluenza virus 5 (SV5) to affect cell cycle progres
87 NASEK was dispensable for viruses, including parainfluenza virus 5 and Coxsackie B virus, that enter
88 ructed chimeras containing the ectodomain of parainfluenza virus 5 F (PIV5 F) and either the MPER, th
89  Here we report the crystal structure of the parainfluenza virus 5 F protein in its prefusion conform
90 ess the functional role of the paramyxovirus parainfluenza virus 5 F protein TM domain, alanine scann
91                      Here, a soluble form of parainfluenza virus 5 F was triggered to refold using te
92 rystal structure of a fragment of the simian parainfluenza virus 5 fusion protein (SV5 F), revealing
93 , we show that the FP from the paramyxovirus parainfluenza virus 5 fusogenic protein, F, forms an N-t
94 ng globular head domain of the paramyxovirus parainfluenza virus 5 HN protein is entirely dispensable
95 usion activation, F activation involving the parainfluenza virus 5 HN stalk domain, and properties of
96         By modeling the crystal structure of parainfluenza virus 5 into the density, it is shown that
97                            The paramyxovirus parainfluenza virus 5 mediates membrane merger through i
98 t robust maturation following infection with parainfluenza virus 5 or influenza virus.
99  this study, we show that vaccination with a parainfluenza virus 5 recombinant vaccine candidate expr
100 ed "stalk exposure model" first proposed for parainfluenza virus 5 to other paramyxoviruses and propo
101 athogens: human respiratory syncytial virus, parainfluenza virus 5, and Sendai virus.
102                                            A parainfluenza virus 5-vectored vaccine expressing the na
103 sequence, FPIV, important for the budding of parainfluenza virus 5.
104 recently published prefusogenic structure of parainfluenza virus 5/SV5 F places CBF(2) in direct cont
105 pproach is further demonstrated here for the parainfluenza virus, a virus which can be life threateni
106 or detecting respiratory viruses, especially parainfluenza virus and adenovirus.
107 d five in which the PLx-RVP failed to detect parainfluenza virus and one in which the detection of in
108 ow fever virus, Japanese encephalitis virus, parainfluenza virus and Sendai virus.
109 ple viruses (respiratory syncytial virus and parainfluenza virus) and multiple phenotypes.
110 tory syncytial virus, human metapneumovirus, parainfluenza virus, and influenza virus) by reverse-tra
111 nfluenza A virus, two influenza B virus, one parainfluenza virus, and six adenovirus).
112 y 90% vaccinated each year), picornaviruses, parainfluenza viruses, and coronaviruses were most commo
113 etapneumovirus, respiratory syncytial virus, parainfluenza viruses, and Haemophilus influenzae being
114                                              Parainfluenza viruses are a common cause of seasonal res
115                                              Parainfluenza viruses are known to inhibit type I interf
116 causes of lower respiratory disease like the parainfluenza viruses, as well as agents of lethal encep
117 protein and nucleocapsid sustain assembly of parainfluenza viruses at the plasma membrane.
118 ke (S) protein from a recombinant attenuated parainfluenza virus (BHPIV3) that is being developed as
119                                  Three human parainfluenza viruses bind to glycans terminating with N
120  (RSV), adenoviruses, influenza viruses, and parainfluenza viruses by use of nested polymerase chain
121                                        Human parainfluenza viruses cause several serious respiratory
122                       Although influenza and parainfluenza viruses commonly cause respiratory tract i
123 , which include respiratory syncytial virus, parainfluenza viruses, coronavirus, rhinovirus, and huma
124 canine adenovirus type 2 (CAV-2), and canine parainfluenza virus (CPIV), respiratory disease was ende
125 sociation with age; especially rhinovirus or parainfluenza virus detection showed positive associatio
126             The tissue culture prevalence of parainfluenza virus during this period of time was low (
127  Our results illustrate how the particles of parainfluenza viruses efficiently accommodate cargoes of
128                                              Parainfluenza viruses enter host cells by fusing the vir
129 V-respiratory syncytial virus (RSV) or human parainfluenza virus (HPIV) coinfections had wheezing tha
130                                    The human parainfluenza virus (hPIV) hemagglutinin-neuraminidase (
131 presence of the second binding site on human parainfluenza virus (hPIV) type 1, 2, and 3 and Sendai v
132 es, Respiratory Syncytial Virus (RSV), Human Parainfluenza Virus (HPIV), and Human Metapneumovirus (h
133 (23), human herpesvirus (HHV)-6B (10), human parainfluenza virus (HPIV)-2 (3), HPIV-3 (1), and human
134 he hemagglutinin-neuraminidase (HN) of human parainfluenza viruses (hPIV) in vitro and protected mice
135                                        Human parainfluenza viruses (HPIVs) are a common cause of acut
136                                        Human parainfluenza viruses (HPIVs) are among the most common
137 glutinin-neuraminidase (HN) protein of human parainfluenza viruses (hPIVs) both binds (H) and cleaves
138                                        Human parainfluenza viruses (HPIVs) cause widespread respirato
139         The first step in infection by human parainfluenza viruses (HPIVs) is binding to the surface
140         The paramyxoviruses, including human parainfluenza viruses (HPIVs), cause a large share of th
141 ndications that the pleomorphic particles of parainfluenza viruses incorporate multiple genomes.
142 infected cells (Wake Forest strain of Canine parainfluenza virus) induced IL-8 secretion by a mechani
143 tudies we tested the role of CD8+ T cells in parainfluenza virus-induced hyperreactivity and M2R dysf
144                          While type II human parainfluenza virus induces STAT2 degradation, simian vi
145                                  Analysis of parainfluenza virus infection in mice revealed an unexpe
146        We used mouse models of influenza and parainfluenza virus infection to show that intranasally
147 uenza virus, respiratory syncytial virus, or parainfluenza virus infection.
148  in mixed bone marrow chimeric mice during a parainfluenza virus infection.
149  central memory subpopulations to intranasal parainfluenza virus infection.
150 ccine nor clinically effective treatment for parainfluenza virus infection.
151                                              Parainfluenza virus infections did not differentially af
152  to pneumonitis and/or mortality of treating parainfluenza virus infections with available (ribavirin
153 ools elicited by nonpersistent influenza and parainfluenza virus infections.
154 iratory syncytial virus, 2 adenovirus, and 1 parainfluenza virus infections.
155 ents with respiratory syncytial virus (RSV), parainfluenza virus, influenza virus, metapneumovirus (M
156                                              Parainfluenza viruses initiate infection by binding to c
157 t for lower-respiratory-tract infection with parainfluenza virus; it stabilized during the months aft
158  mouse model in which infection with a mouse parainfluenza virus known as Sendai virus (SeV) leads to
159 for RSV (n = 35), 2.6 x 10(6) copies/mL; for parainfluenza virus (n = 35), 4.9 x 10(7) copies/mL; for
160 rs mutations in the P/V gene from the canine parainfluenza virus (P/V-CPI(-)) is a potent inducer of
161        Respiratory syncytial virus (RSV) and parainfluenza virus (PIV) are two respiratory pathogens
162                                              Parainfluenza virus (PIV) commonly infects patients foll
163                            RSV F and related parainfluenza virus (PIV) F proteins are cleaved by furi
164                                              Parainfluenza virus (PIV) in humans is associated with b
165      Data on characteristics and outcomes of parainfluenza virus (PIV) infections in these patients a
166                                              Parainfluenza virus (PIV) infections may be significant
167                                              Parainfluenza virus (PIV) is a negative-sense single-str
168 ually and in combinations from a recombinant parainfluenza virus (PIV) type 3 vector called BHPIV3.
169 mens), followed by human rhinovirus (17.8%); parainfluenza virus (PIV) types 1-4 (7.5%); enterovirus
170 sting for respiratory syncytial virus (RSV), parainfluenza virus (PIV), and influenza A and B, and by
171 us (HRV), respiratory syncytial virus (RSV), parainfluenza virus (PIV), influenza virus (InfV), metap
172 (HN, residues 37 to 56) of the paramyxovirus parainfluenza virus (PIV5), a region of the HN stalk tha
173                                              Parainfluenza viruses (PIVs) are one of the most common
174  significance of membrane fusion activity in parainfluenza virus replication and pathogenesis in vivo
175 e (HN) glycoprotein plays a critical role in parainfluenza virus replication.
176 ruses, including measles virus, mumps virus, parainfluenza viruses, respiratory syncytial virus, huma
177 roviruses, influenza virus, metapneumovirus, parainfluenza virus, rhinovirus, and respiratory syncyti
178                                          The parainfluenza virus simian virus 5 (SV5) is a poor induc
179     Human epithelial cells infected with the parainfluenza virus simian virus 5 (SV5) show minimal ac
180 aring the sequence of MV F with those of the parainfluenza virus SV5 and Newcastle disease virus (NDV
181                        SER virus is a type 5 parainfluenza virus that does not exhibit syncytium form
182 eviously described heterotypic peptides from parainfluenza virus that potently inhibit Nipah virus in
183                Paramyxoviruses such as human parainfluenza viruses that bear inserts encoding protect
184 e protein or whole virus digests enables the parainfluenza virus to be identified and typed and for i
185 ew evidence regarding strategies employed by parainfluenza viruses to effectively circumvent respirat
186 ontact transmission, the predominant mode of parainfluenza virus transmission, was modeled accurately
187 n to the catalytic binding site, HN of human parainfluenza virus type 1 (hPIV-1) may have a second re
188                                        Human parainfluenza virus type 1 (HPIV1) also causes severe pe
189 irions in two closely related viruses, human parainfluenza virus type 1 (hPIV1) and Sendai virus (SV)
190                 Sendai virus (SeV) and human parainfluenza virus type 1 (hPIV1) are highly similar in
191              We evaluated a version of human parainfluenza virus type 1 (HPIV1) bearing a stabilized
192                  Sendai virus (SV) and human parainfluenza virus type 1 (hPIV1) have genomes consisti
193                                        Human parainfluenza virus type 1 (HPIV1) is a significant caus
194                                        Human parainfluenza virus type 1 (HPIV1) is a significant caus
195                                        Human parainfluenza virus type 1 (HPIV1) is an important respi
196                                        Human parainfluenza virus type 1 (HPIV1) is an important respi
197  respiratory syncytial virus (RSV) and human parainfluenza virus type 1 (HPIV1) to HPIV4 infect virtu
198 onkeys from challenge with the related human parainfluenza virus type 1 (hPIV1), and SV has advanced
199 ned M, as was previously observed with human parainfluenza virus type 1 (hPIV1).
200 valuation of an attenuated recombinant human parainfluenza virus type 1 (rHPIV1) expressing the membr
201            Live attenuated recombinant human parainfluenza virus type 1 (rHPIV1) was investigated as
202                            Recombinant human parainfluenza virus type 1 (rHPIV1) was modified to crea
203 ons of the L polymerase of recombinant human parainfluenza virus type 1 (rHPIV1).
204  live virus vaccine, we have used the murine parainfluenza virus type 1 (Sendai virus [SV]) as a xeno
205  Hamsters immunized with a recombinant human parainfluenza virus type 1 expressing the fusion F prote
206                                        Human parainfluenza virus type 1 is the major cause of croup i
207 secreted from A549 cells infected with Human parainfluenza virus type 2 (HPIV-2) but not from cells i
208                                        Human parainfluenza virus type 2 (HPIV-2), an important pediat
209                                        Human parainfluenza virus type 2 (HPIV2) is the only member of
210       We sought to develop a live attenuated parainfluenza virus type 2 (PIV2) vaccine strain for use
211 t for association with V proteins from human parainfluenza virus type 2, parainfluenza virus type 5,
212 d by the V proteins of mumps virus and human parainfluenza virus type 2.
213 fever (SF) and Kansas (Ka) strains of bovine parainfluenza virus type 3 (BPIV3) are restricted in the
214                  The Kansas strain of bovine parainfluenza virus type 3 (BPIV3) is 100- to 1,000-fold
215                                       Bovine parainfluenza virus type 3 (bPIV3) is being evaluated as
216 raminidase (HN) glycoprotein genes of bovine parainfluenza virus type 3 (BPIV3) to its restricted rep
217 to evaluate the antibody responses to bovine parainfluenza virus type 3 (bPIV3) vaccination in young
218  doses of an intranasal vaccine using bovine parainfluenza virus type 3 (bPIV3).
219                        The envelope of human parainfluenza virus type 3 (HPF3) contains two viral gly
220                        The envelope of human parainfluenza virus type 3 (HPF3) contains two viral gly
221                    Entry and fusion of human parainfluenza virus type 3 (HPF3) require the interactio
222                    Entry and fusion of human parainfluenza virus type 3 (HPF3) requires interaction o
223 erence have not been characterized for human parainfluenza virus type 3 (HPF3), and the possible role
224  inhibit the neuraminidase activity of human parainfluenza virus type 3 (HPF3).
225 ur previous observation on the role of human parainfluenza virus type 3 (HPIV 3) C protein in the tra
226          The RNA polymerase complex of human parainfluenza virus type 3 (HPIV 3), a member of the fam
227                                        Human parainfluenza virus type 3 (hPIV-3) is a clinically sign
228                                        Human parainfluenza virus type 3 (HPIV-3) is an airborne patho
229                                        Human parainfluenza virus type 3 (HPIV-3) is an airborne patho
230 on factor, with the cis-acting RNAs of human parainfluenza virus type 3 (HPIV3) and packaging of thes
231 valent live attenuated vaccine against human parainfluenza virus type 3 (HPIV3) and respiratory syncy
232  respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are major pediatric r
233  respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are major viral agent
234 SV), human metapneumovirus (hMPV), and human parainfluenza virus type 3 (hPIV3) are responsible for t
235  Respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are the first and sec
236  Respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are two major causes
237                The genomic promoter of human parainfluenza virus type 3 (HPIV3) contains multiple cis
238 genes, of a gene cassette encoding the human parainfluenza virus type 3 (HPIV3) hemagglutinin-neurami
239 t also the receptor interaction of the human parainfluenza virus type 3 (HPIV3) hemagglutinin-neurami
240 plementation to follow the dynamics of human parainfluenza virus type 3 (HPIV3) HN/F pairs in living
241                    We demonstrate that human parainfluenza virus type 3 (HPIV3) induces incomplete au
242                                        Human parainfluenza virus type 3 (HPIV3) is one of the major c
243        It was previously reported that human parainfluenza virus type 3 (HPIV3) multiplication was in
244 onnected to the stalk region of either human parainfluenza virus type 3 (HPIV3) or Nipah virus recept
245                   We have analyzed the human parainfluenza virus type 3 (HPIV3) P protein and deletio
246 binant virus rHPIV3-N(B), a version of human parainfluenza virus type 3 (HPIV3) that is attenuated du
247  against Ebola virus (EV), recombinant human parainfluenza virus type 3 (HPIV3) was modified to expre
248 rt here that for three paramyxoviruses-human parainfluenza virus type 3 (HPIV3), a major cause of low
249                                        Human parainfluenza virus type 3 (HPIV3), a paramyxovirus, is
250 monkeys 100- to 1,000-fold compared to human parainfluenza virus type 3 (HPIV3), and the Ka strain al
251 common pediatric respiratory pathogen, human parainfluenza virus type 3 (HPIV3), as a vaccine vector
252  encephalomyocarditis virus (EMCV) and human parainfluenza virus type 3 (HPIV3), induced down-regulat
253 ing the childhood respiratory pathogen human parainfluenza virus type 3 (HPIV3), possess an envelope
254                                    For human parainfluenza virus type 3 (HPIV3), the effects of speci
255                                    For human parainfluenza virus type 3 (HPIV3), the receptor binding
256  intranasal paediatric vaccine against human parainfluenza virus type 3 (HPIV3).
257 s), Newcastle disease virus (NDV), and human parainfluenza virus type 3 (HPIV3).
258 aminidase (HN) surface glycoprotein of human parainfluenza virus type 3 (HPIV3).
259 tory syncytial virus (RSV) and one strain of parainfluenza virus type 3 (PIV-3) was determined.
260 In this study, a chimeric bovine/human (b/h) parainfluenza virus type 3 (PIV3) expressing the human P
261                                              Parainfluenza virus type 3 (PIV3) infection led to laryn
262                                              Parainfluenza virus type 3 (PIV3) infections are a major
263                                              Parainfluenza virus type 3 (PIV3) is major pathogen of c
264                    During a phase 2 trial of parainfluenza virus type 3 (PIV3) vaccine, sequential se
265                            Recombinant human parainfluenza virus type 3 (PIV3) was used as a vector t
266 r the ability to inhibit the growth of human parainfluenza virus type 3 (PIV3), a nonsegmented negati
267                     A live attenuated bovine parainfluenza virus type 3 (PIV3), harboring the fusion
268      A live attenuated chimeric bovine/human parainfluenza virus type 3 (rB/HPIV3) was developed prev
269                     Recombinant bovine/human parainfluenza virus type 3 (rB/HPIV3), a recombinant bov
270           We constructed a human recombinant parainfluenza virus type 3 (rPIV3) that expresses enhanc
271                                    For human parainfluenza virus type 3 and many other paramyxoviruse
272 tial innate antiviral response against human parainfluenza virus type 3 and respiratory syncytial vir
273 s virus of the Arenaviridae family and human parainfluenza virus type 3 of the Paramyxoviridae family
274                                        Human parainfluenza virus type 3, a mildly cytopathic virus th
275 s of a live-attenuated vaccine candidate for parainfluenza virus type 3, an enveloped RNA virus that
276 yncytial virus, human metapneumovirus, human parainfluenza virus type 3, and measles virus, and highl
277 uses, including the childhood pathogen human parainfluenza virus type 3, enter host cells by fusion o
278 explores the binding and entry into cells of parainfluenza virus type 3, focusing on how the receptor
279 minidase abolished infection of HAE by human parainfluenza virus type 3, this treatment did not signi
280  vaccine for respiratory syncytial virus and parainfluenza virus type 3, two major causes of severe r
281  viruses, including the human pathogen human parainfluenza virus type 3, yet these compounds by thems
282  protective efficacy of an aerosolized human parainfluenza virus type 3-vectored vaccine that express
283 lymphocytic choriomeningitis virus and human parainfluenza virus type 3.
284 ion, which was 40-fold lower than that of WT parainfluenza virus type 3.
285 ction of interferon (IFN) alpha/beta against parainfluenza virus type 5 (PIV5), selectively inhibitin
286 teins from human parainfluenza virus type 2, parainfluenza virus type 5, measles virus, mumps virus,
287         We investigated the binding of human parainfluenza virus types 1 and 3 (hPIV1 and hPIV3, resp
288 luding influenza virus A, influenza virus B, parainfluenza virus types 1 and 3, respiratory syncytial
289 tial virus [RSV], influenza A and B viruses, parainfluenza virus types 1 to 3, and adenovirus) was co
290 A and B and human metapneumovirus, and (iii) parainfluenza virus types 1 to 4.
291  virus (RSV), influenza virus type A (FluA), parainfluenza virus types 1, 2, and 3 (PIV1, PIV2, and P
292 criptase (RT)-PCR assay for the detection of parainfluenza virus types 1, 2, and 3, respiratory syncy
293 iratory syncytial viruses A and B; and human parainfluenza virus types 1, 2, and 3.
294 iruses, including influenza A and B viruses, parainfluenza virus types 1-3, respiratory syncytial vir
295 ed negative for respiratory syncytial virus, parainfluenza viruses (types 1-3), influenza A and B vir
296 act of respiratory syncytial virus (RSV) and parainfluenza virus URIs on the frequency of AOM caused
297                         C28a HN is the first parainfluenza virus variant found so far to be specifica
298 pared to tissue culture for the detection of parainfluenza virus were 100, 95.8, 19.0, and 100%, resp
299  naturally occurring SV5 variant Wake Forest parainfluenza virus (WF-PIV) activates the synthesis of
300 so showed that extraction will be needed for parainfluenza virus, which was only identified correctly

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