コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 rus, rhinovirus, and influenza virus but not parainfluenza virus.
2 p of viruses that includes measles virus and parainfluenza viruses.
3 an respiratory syncytial virus and the human parainfluenza viruses.
4 tein subunits play the cell entry concert of parainfluenza viruses.
5 detects influenza A virus (Flu-A) and Flu-B, parainfluenza virus 1 (PIV-1), PIV-2, and PIV-3, and res
6 l virus (RSV), human metapneumovirus (HMPV), parainfluenza virus 1 to 3 (PIV1, PIV2, and PIV3), and a
8 viruses, respiratory syncytial virus (RSV), parainfluenza viruses 1 to 3, and adenovirus (DAKO Diagn
9 virus, influenza A virus, influenza B virus, parainfluenza viruses 1 to 3, and respiratory syncytial
10 nfluenza A virus H1-2009, influenza B virus, parainfluenza viruses 1 to 4, respiratory syncytial viru
11 syncytial virus; influenza A and B viruses; parainfluenza viruses 1, 2, 3, and 4; human metapneumovi
12 an respiratory syncytial virus (HRSV); human parainfluenza viruses 1, 2, and 3 (HPIV1, -2, and -3, re
14 picornaviruses, coronaviruses 229E and OC43, parainfluenza viruses 1-3, influenza viruses AH1, AH3, a
15 in reaction for respiratory syncytial virus, parainfluenza viruses 1-4, influenza A and B, human meta
16 ramyxoviruses simian virus 5 (SV5) and human parainfluenza virus 2 (HPIV2) overcome IFN-alpha/beta re
17 hese are encoded by mumps virus (MuV), human parainfluenza virus 2 (hPIV2), and parainfluenza virus 5
18 Simian virus 5 (SV5) targets STAT1, human parainfluenza virus 2 targets STAT2, and mumps virus tar
19 ith respiratory syncytial virus (RSV), human parainfluenza virus 3 (HPIV-3), and influenza virus on t
21 described for a cluster of 12 cases of human parainfluenza virus 3 (HPIV3) infection that occurred am
22 titatively influence fusion promotion, human parainfluenza virus 3 (HPIV3) variants with alterations
23 yxoviruses, such as Nipah virus (NiV), human parainfluenza virus 3 (HPIV3), measles virus (MeV), mump
26 c fibrosis patients; however, its use during parainfluenza virus 3 (PIV3) infection has not been eval
29 eaved ectodomain of the paramyxovirus, human parainfluenza virus 3 fusion (F) protein, a member of th
34 cell lines with Sendai virus (SeV) or human parainfluenza virus 3, two prototypic paramyxoviruses, c
36 on of 274 of 279 influenza viruses, 33 of 38 parainfluenza viruses, 35 of 51 adenoviruses, and 52 of
38 rus [EV], 118; bocavirus, 8; coronavirus, 7; parainfluenza virus 4, 4; Mycoplasma pneumoniae, 1).
39 lture (metapneumovirus, coronaviruses [CoV], parainfluenza viruses 4a and 4b, and rhinoviruses) and t
40 hat Cav-1 colocalizes with the paramyxovirus parainfluenza virus 5 (PIV-5) nucleocapsid (NP), matrix
43 Proline substitution in this region of HN of parainfluenza virus 5 (PIV5) and Newcastle disease virus
44 In this work, we generated a recombinant parainfluenza virus 5 (PIV5) containing NP from H5N1 (A/
46 igh similarity to the structure of prefusion parainfluenza virus 5 (PIV5) F, with the main structural
47 Because only the prefusion structure of the parainfluenza virus 5 (PIV5) F-trimer is available, to s
48 MR spectroscopy, we show that the TMD of the parainfluenza virus 5 (PIV5) fusion protein adopts lipid
57 To investigate the role of NP protein in parainfluenza virus 5 (PIV5) particle formation, NP prot
59 serendipitously identified a viral mRNA from parainfluenza virus 5 (PIV5) that activates IFN expressi
61 quence variation of 16 different isolates of parainfluenza virus 5 (PIV5) that were isolated from a n
62 unable to be recognized by measles virus and parainfluenza virus 5 (PIV5) V proteins were tested in s
64 he threonine residue at position 286 of P of parainfluenza virus 5 (PIV5) was found phosphorylated.
65 The V proteins of measles virus (MV) and parainfluenza virus 5 (PIV5) were introduced into HFLC u
66 rotein (prefusion form) of the paramyxovirus parainfluenza virus 5 (PIV5) WR isolate was determined.
73 V), human parainfluenza virus 2 (hPIV2), and parainfluenza virus 5 (PIV5), all members of the genus R
75 that a porcine isolate of the paramyxovirus parainfluenza virus 5 (PIV5), known as SER, requires a l
84 sion (F) protein of the paramxyovirus simian parainfluenza virus 5 (SV5) promotes virus-cell and cell
85 on (F) protein from the paramyxovirus simian parainfluenza virus 5 (SV5) resulted in mutant F protein
86 ined the ability of the paramyxovirus simian parainfluenza virus 5 (SV5) to affect cell cycle progres
87 NASEK was dispensable for viruses, including parainfluenza virus 5 and Coxsackie B virus, that enter
88 ructed chimeras containing the ectodomain of parainfluenza virus 5 F (PIV5 F) and either the MPER, th
89 Here we report the crystal structure of the parainfluenza virus 5 F protein in its prefusion conform
90 ess the functional role of the paramyxovirus parainfluenza virus 5 F protein TM domain, alanine scann
92 rystal structure of a fragment of the simian parainfluenza virus 5 fusion protein (SV5 F), revealing
93 , we show that the FP from the paramyxovirus parainfluenza virus 5 fusogenic protein, F, forms an N-t
94 ng globular head domain of the paramyxovirus parainfluenza virus 5 HN protein is entirely dispensable
95 usion activation, F activation involving the parainfluenza virus 5 HN stalk domain, and properties of
99 this study, we show that vaccination with a parainfluenza virus 5 recombinant vaccine candidate expr
100 ed "stalk exposure model" first proposed for parainfluenza virus 5 to other paramyxoviruses and propo
104 recently published prefusogenic structure of parainfluenza virus 5/SV5 F places CBF(2) in direct cont
105 pproach is further demonstrated here for the parainfluenza virus, a virus which can be life threateni
107 d five in which the PLx-RVP failed to detect parainfluenza virus and one in which the detection of in
110 tory syncytial virus, human metapneumovirus, parainfluenza virus, and influenza virus) by reverse-tra
112 y 90% vaccinated each year), picornaviruses, parainfluenza viruses, and coronaviruses were most commo
113 etapneumovirus, respiratory syncytial virus, parainfluenza viruses, and Haemophilus influenzae being
116 causes of lower respiratory disease like the parainfluenza viruses, as well as agents of lethal encep
118 ke (S) protein from a recombinant attenuated parainfluenza virus (BHPIV3) that is being developed as
120 (RSV), adenoviruses, influenza viruses, and parainfluenza viruses by use of nested polymerase chain
123 , which include respiratory syncytial virus, parainfluenza viruses, coronavirus, rhinovirus, and huma
124 canine adenovirus type 2 (CAV-2), and canine parainfluenza virus (CPIV), respiratory disease was ende
125 sociation with age; especially rhinovirus or parainfluenza virus detection showed positive associatio
127 Our results illustrate how the particles of parainfluenza viruses efficiently accommodate cargoes of
129 V-respiratory syncytial virus (RSV) or human parainfluenza virus (HPIV) coinfections had wheezing tha
131 presence of the second binding site on human parainfluenza virus (hPIV) type 1, 2, and 3 and Sendai v
132 es, Respiratory Syncytial Virus (RSV), Human Parainfluenza Virus (HPIV), and Human Metapneumovirus (h
133 (23), human herpesvirus (HHV)-6B (10), human parainfluenza virus (HPIV)-2 (3), HPIV-3 (1), and human
134 he hemagglutinin-neuraminidase (HN) of human parainfluenza viruses (hPIV) in vitro and protected mice
137 glutinin-neuraminidase (HN) protein of human parainfluenza viruses (hPIVs) both binds (H) and cleaves
141 ndications that the pleomorphic particles of parainfluenza viruses incorporate multiple genomes.
142 infected cells (Wake Forest strain of Canine parainfluenza virus) induced IL-8 secretion by a mechani
143 tudies we tested the role of CD8+ T cells in parainfluenza virus-induced hyperreactivity and M2R dysf
152 to pneumonitis and/or mortality of treating parainfluenza virus infections with available (ribavirin
155 ents with respiratory syncytial virus (RSV), parainfluenza virus, influenza virus, metapneumovirus (M
157 t for lower-respiratory-tract infection with parainfluenza virus; it stabilized during the months aft
158 mouse model in which infection with a mouse parainfluenza virus known as Sendai virus (SeV) leads to
159 for RSV (n = 35), 2.6 x 10(6) copies/mL; for parainfluenza virus (n = 35), 4.9 x 10(7) copies/mL; for
160 rs mutations in the P/V gene from the canine parainfluenza virus (P/V-CPI(-)) is a potent inducer of
165 Data on characteristics and outcomes of parainfluenza virus (PIV) infections in these patients a
168 ually and in combinations from a recombinant parainfluenza virus (PIV) type 3 vector called BHPIV3.
169 mens), followed by human rhinovirus (17.8%); parainfluenza virus (PIV) types 1-4 (7.5%); enterovirus
170 sting for respiratory syncytial virus (RSV), parainfluenza virus (PIV), and influenza A and B, and by
171 us (HRV), respiratory syncytial virus (RSV), parainfluenza virus (PIV), influenza virus (InfV), metap
172 (HN, residues 37 to 56) of the paramyxovirus parainfluenza virus (PIV5), a region of the HN stalk tha
174 significance of membrane fusion activity in parainfluenza virus replication and pathogenesis in vivo
176 ruses, including measles virus, mumps virus, parainfluenza viruses, respiratory syncytial virus, huma
177 roviruses, influenza virus, metapneumovirus, parainfluenza virus, rhinovirus, and respiratory syncyti
179 Human epithelial cells infected with the parainfluenza virus simian virus 5 (SV5) show minimal ac
180 aring the sequence of MV F with those of the parainfluenza virus SV5 and Newcastle disease virus (NDV
182 eviously described heterotypic peptides from parainfluenza virus that potently inhibit Nipah virus in
184 e protein or whole virus digests enables the parainfluenza virus to be identified and typed and for i
185 ew evidence regarding strategies employed by parainfluenza viruses to effectively circumvent respirat
186 ontact transmission, the predominant mode of parainfluenza virus transmission, was modeled accurately
187 n to the catalytic binding site, HN of human parainfluenza virus type 1 (hPIV-1) may have a second re
189 irions in two closely related viruses, human parainfluenza virus type 1 (hPIV1) and Sendai virus (SV)
197 respiratory syncytial virus (RSV) and human parainfluenza virus type 1 (HPIV1) to HPIV4 infect virtu
198 onkeys from challenge with the related human parainfluenza virus type 1 (hPIV1), and SV has advanced
200 valuation of an attenuated recombinant human parainfluenza virus type 1 (rHPIV1) expressing the membr
204 live virus vaccine, we have used the murine parainfluenza virus type 1 (Sendai virus [SV]) as a xeno
205 Hamsters immunized with a recombinant human parainfluenza virus type 1 expressing the fusion F prote
207 secreted from A549 cells infected with Human parainfluenza virus type 2 (HPIV-2) but not from cells i
211 t for association with V proteins from human parainfluenza virus type 2, parainfluenza virus type 5,
213 fever (SF) and Kansas (Ka) strains of bovine parainfluenza virus type 3 (BPIV3) are restricted in the
216 raminidase (HN) glycoprotein genes of bovine parainfluenza virus type 3 (BPIV3) to its restricted rep
217 to evaluate the antibody responses to bovine parainfluenza virus type 3 (bPIV3) vaccination in young
223 erence have not been characterized for human parainfluenza virus type 3 (HPF3), and the possible role
225 ur previous observation on the role of human parainfluenza virus type 3 (HPIV 3) C protein in the tra
230 on factor, with the cis-acting RNAs of human parainfluenza virus type 3 (HPIV3) and packaging of thes
231 valent live attenuated vaccine against human parainfluenza virus type 3 (HPIV3) and respiratory syncy
232 respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are major pediatric r
233 respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are major viral agent
234 SV), human metapneumovirus (hMPV), and human parainfluenza virus type 3 (hPIV3) are responsible for t
235 Respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are the first and sec
236 Respiratory syncytial virus (RSV) and human parainfluenza virus type 3 (HPIV3) are two major causes
238 genes, of a gene cassette encoding the human parainfluenza virus type 3 (HPIV3) hemagglutinin-neurami
239 t also the receptor interaction of the human parainfluenza virus type 3 (HPIV3) hemagglutinin-neurami
240 plementation to follow the dynamics of human parainfluenza virus type 3 (HPIV3) HN/F pairs in living
244 onnected to the stalk region of either human parainfluenza virus type 3 (HPIV3) or Nipah virus recept
246 binant virus rHPIV3-N(B), a version of human parainfluenza virus type 3 (HPIV3) that is attenuated du
247 against Ebola virus (EV), recombinant human parainfluenza virus type 3 (HPIV3) was modified to expre
248 rt here that for three paramyxoviruses-human parainfluenza virus type 3 (HPIV3), a major cause of low
250 monkeys 100- to 1,000-fold compared to human parainfluenza virus type 3 (HPIV3), and the Ka strain al
251 common pediatric respiratory pathogen, human parainfluenza virus type 3 (HPIV3), as a vaccine vector
252 encephalomyocarditis virus (EMCV) and human parainfluenza virus type 3 (HPIV3), induced down-regulat
253 ing the childhood respiratory pathogen human parainfluenza virus type 3 (HPIV3), possess an envelope
260 In this study, a chimeric bovine/human (b/h) parainfluenza virus type 3 (PIV3) expressing the human P
266 r the ability to inhibit the growth of human parainfluenza virus type 3 (PIV3), a nonsegmented negati
268 A live attenuated chimeric bovine/human parainfluenza virus type 3 (rB/HPIV3) was developed prev
272 tial innate antiviral response against human parainfluenza virus type 3 and respiratory syncytial vir
273 s virus of the Arenaviridae family and human parainfluenza virus type 3 of the Paramyxoviridae family
275 s of a live-attenuated vaccine candidate for parainfluenza virus type 3, an enveloped RNA virus that
276 yncytial virus, human metapneumovirus, human parainfluenza virus type 3, and measles virus, and highl
277 uses, including the childhood pathogen human parainfluenza virus type 3, enter host cells by fusion o
278 explores the binding and entry into cells of parainfluenza virus type 3, focusing on how the receptor
279 minidase abolished infection of HAE by human parainfluenza virus type 3, this treatment did not signi
280 vaccine for respiratory syncytial virus and parainfluenza virus type 3, two major causes of severe r
281 viruses, including the human pathogen human parainfluenza virus type 3, yet these compounds by thems
282 protective efficacy of an aerosolized human parainfluenza virus type 3-vectored vaccine that express
285 ction of interferon (IFN) alpha/beta against parainfluenza virus type 5 (PIV5), selectively inhibitin
286 teins from human parainfluenza virus type 2, parainfluenza virus type 5, measles virus, mumps virus,
288 luding influenza virus A, influenza virus B, parainfluenza virus types 1 and 3, respiratory syncytial
289 tial virus [RSV], influenza A and B viruses, parainfluenza virus types 1 to 3, and adenovirus) was co
291 virus (RSV), influenza virus type A (FluA), parainfluenza virus types 1, 2, and 3 (PIV1, PIV2, and P
292 criptase (RT)-PCR assay for the detection of parainfluenza virus types 1, 2, and 3, respiratory syncy
294 iruses, including influenza A and B viruses, parainfluenza virus types 1-3, respiratory syncytial vir
295 ed negative for respiratory syncytial virus, parainfluenza viruses (types 1-3), influenza A and B vir
296 act of respiratory syncytial virus (RSV) and parainfluenza virus URIs on the frequency of AOM caused
298 pared to tissue culture for the detection of parainfluenza virus were 100, 95.8, 19.0, and 100%, resp
299 naturally occurring SV5 variant Wake Forest parainfluenza virus (WF-PIV) activates the synthesis of
300 so showed that extraction will be needed for parainfluenza virus, which was only identified correctly
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。