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1 nent component of this group was Haemophilus parainfluenzae.
2 sthmatic airway macrophages with Haemophilus parainfluenzae, a uniquely expanded potential pathogen f
3 gy for the identification of 103 Haemophilus parainfluenzae, Aggregatibacter aphrophilus, Aggregatiba
4  examine the possible interaction between H. parainfluenzae and its close relative Haemophilus influe
5 rial persistence and biofilm formation by H. parainfluenzae and knowledge about the survival defects
6 cilis, Capnocytophaga granulosa, Haemophilus parainfluenzae, and Lautropia mirabilis were most abunda
7 81.8% similarity to the H. influenzae and H. parainfluenzae copper (Cu), zinc (Zn)-superoxide dismuta
8 d knowledge about the survival defects of H. parainfluenzae during coinfection with H. influenzae are
9 and 66% identity with E. coli and Hemophilus parainfluenzae endonuclease III, respectively.
10 m in vitro biofilm studies confirmed that H. parainfluenzae formed biofilm communities within which t
11  OM infection model, we demonstrated that H. parainfluenzae formed surface-associated biofilm communi
12 udy addresses the primary hypothesis that H. parainfluenzae forms biofilm communities that are import
13  with H. influenzae promoted clearance of H. parainfluenzae from biofilm communities during OM infect
14                                  Haemophilus parainfluenzae is a nutritionally fastidious, Gram-negat
15 es of Haemophilus influenzae and Haemophilus parainfluenzae isolates were determined with three comme
16 um hominis, Gemella haemolysans, Haemophilus parainfluenzae, Kingella oralis, Lautropia mirabilis, Ne
17 s mitis, Rothia mucilaginosa and Haemophilus parainfluenzae were the most significantly abundant in t

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