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1     Nipah virus (NiV) is the deadliest known paramyxovirus.
2 ) is a highly pathogenic and deadly zoonotic paramyxovirus.
3 attenuated vaccines for hMPV and other human paramyxoviruses.
4 bone" appearance of helical nucleocapsids in paramyxoviruses.
5 he most promising vaccine strategy for human paramyxoviruses.
6 be possible to apply these findings to other paramyxoviruses.
7 ds with diameters of 10 to 14 nm, typical of paramyxoviruses.
8 osure model of membrane fusion triggering by paramyxoviruses.
9 tenuated vaccines for aMPV and perhaps other paramyxoviruses.
10 nal receipt mechanism may be conserved among paramyxoviruses.
11 sm for the targeting of the RIG-I pathway by paramyxoviruses.
12 d is understood neither for MV nor for other paramyxoviruses.
13 membrane fusion triggering and cell entry by Paramyxoviruses.
14 t Sendai virus or human metapneumovirus, two paramyxoviruses.
15 potential utility of inhibitory peptides for paramyxoviruses.
16  a new structural-biological finding for the paramyxoviruses.
17  may be involved in the replication of these paramyxoviruses.
18 ering broad spectrum antivirals for emerging paramyxoviruses.
19  of F, which has not been reported for other paramyxoviruses.
20 ng mechanism(s) of the henipaviruses and the paramyxoviruses.
21 se are poorly understood compared with other paramyxoviruses.
22 rix gene assay, designed to detect all avian paramyxovirus 1, did not detect four of the isolates.
23                       This review focuses on paramyxovirus activation of innate immune pathways, the
24                         Unlike that for most paramyxoviruses, activation of the henipavirus fusion pr
25                    The V proteins from other paramyxoviruses also bind LGP2 and demonstrate LGP2-depe
26   In fact, the analogous mechanisms in other paramyxoviruses also remain undetermined.
27 matrix protein of Newcastle disease virus, a paramyxovirus and relative of measles virus, forms dimer
28 that is a highly effective inhibitor of both paramyxoviruses and a set of criteria to be used for eng
29 ld enhance intrinsic cross-immunity to these paramyxoviruses and approaches to controlling recurring
30 f these findings extends beyond NiV to other paramyxoviruses and enveloped viruses.
31 iral entry field extends beyond NiV to other paramyxoviruses and enveloped viruses.
32 s a restriction factor in the replication of paramyxoviruses and orthomyxoviruses.
33                                              Paramyxoviruses and other negative-strand RNA viruses en
34 tly proposed "stalk exposure model" to other paramyxoviruses and propose an "induced fit" hypothesis
35  proposed for parainfluenza virus 5 to other paramyxoviruses and propose an "induced fit" hypothesis
36 shares sequence homology with members of the paramyxoviruses and the filoviruses.
37 ajor animal populations capable of harboring paramyxoviruses, and host shifting between these animals
38 sted that this virus represented a new avian paramyxovirus (APMV) group, APMV10.
39 rus (NDV) belongs to serotype 1 of the avian paramyxoviruses (APMV-1) and causes severe disease in ch
40 n the replication and pathogenicity of avian paramyxoviruses (APMVs), we constructed a reverse geneti
41                                              Paramyxoviruses are a large family of membrane-enveloped
42                                              Paramyxoviruses are enveloped negative-strand RNA viruse
43                                              Paramyxoviruses are potent activators of extracellular c
44                                              Paramyxoviruses are responsible for a wide range of dise
45 terplay between glycoprotein trafficking and paramyxovirus assembly.
46         Nipah virus and Hendra virus are two paramyxoviruses associated with high mortality rates in
47                                              Paramyxovirus attachment and fusion (F) envelope glycopr
48 amental architecture appears conserved among paramyxovirus attachment protein stalk domains, we predi
49    It is unknown how receptor binding by the paramyxovirus attachment proteins (HN, H, or G) triggers
50       The multifunctional HN protein of some paramyxoviruses, besides functioning as the receptor (si
51 genomes (DVGs) can facilitate persistence of paramyxoviruses, but the underlying mechanisms are uncle
52 gglutinin (HA)-neuraminidase protein (HN) of paramyxoviruses carries out three discrete activities, e
53  hemagglutinin-neuraminidase (HN) protein of paramyxoviruses carries out three distinct activities co
54  VI) of the large (L) polymerase proteins of paramyxoviruses catalyzes methyltransferase (MTase) acti
55  VI) of the large (L) polymerase proteins of paramyxoviruses catalyzes methyltransferase (MTase) acti
56                                              Paramyxoviruses cause a wide variety of diseases, and ye
57 ion that determines the type of disease that paramyxoviruses cause is relatively small.
58               Infection of cultured cells by paramyxoviruses causes cell death, mediated by a newly d
59                                              Paramyxovirus cell entry is mediated by the fusion prote
60                        Here, we have studied paramyxovirus circulation within populations of endemic
61          Hendra virus is a highly pathogenic paramyxovirus classified as a biosafety level four agent
62 iral RNA-dependent RNA polymerase (vRdRp) of paramyxovirus consists of the large (L) protein and the
63                      The fusion apparatus of paramyxoviruses consists of a receptor binding tetramer
64                                              Paramyxoviruses contain glycoprotein fusion machineries
65                         Mumps virus (MuV), a paramyxovirus containing a negative-sense nonsegmented R
66 ruses, including bunyaviruses, arenaviruses, paramyxoviruses, coronaviruses and flaviviruses.
67 ignificant zoonotic spillover of chiropteran paramyxoviruses could be missed throughout much of Afric
68 ious RNA viruses, including influenza virus, paramyxovirus, dengue virus, and picornavirus.
69 endra virus (HeV) and Nipah virus (NiV), are paramyxoviruses discovered in the mid- to late 1990s tha
70 vated infection levels as well as widespread paramyxovirus dispersal and frequent host exchange of a
71 parainfluenza virus 5 (PIV5), a prototypical paramyxovirus, encodes a V protein that inhibits viral R
72                                              Paramyxoviruses enter host cells by fusing the viral env
73 fects the design of effective broad-spectrum paramyxovirus entry inhibitors.
74                                       During paramyxovirus entry into a host cell, receptor engagemen
75                                              Paramyxovirus entry into cells requires the fusion prote
76             Membrane fusion is essential for paramyxovirus entry into target cells and for the cell-c
77 We identified a small molecule that inhibits paramyxovirus entry into target cells and prevents infec
78 te closure of the fusion core does not drive paramyxovirus entry may aid the design of strategies for
79 unctionally varies conserved elements of the paramyxovirus entry pathway, providing a possible explan
80                                              Paramyxovirus entry requires the coordinated action of t
81     This first evidence that activation of a paramyxovirus F can be specifically induced before the v
82 ship between enhanced fusion activity in the paramyxovirus F protein and increased susceptibility to
83 ermine factors driving this association, 140 paramyxovirus F protein TM domain sequences were analyze
84 brane-proximal external region (MPER) of the paramyxovirus F protein.
85 protein linked to the TM segments from three paramyxovirus F proteins was analyzed by sedimentation e
86      The structure shares a common core with paramyxovirus F proteins, implicating mechanistic simila
87 nding to just the TM region of two different paramyxovirus F proteins.
88 ilarity between postfusion coronavirus S and paramyxovirus F structures demonstrates that a conserved
89 ding of the mechanism(s) of receptor-induced paramyxovirus F triggering during viral entry and cell-c
90 terminal helix is a necessary early step for paramyxovirus F-protein refolding and presents a novel t
91 ct that these motions may act as a universal paramyxovirus F-triggering mechanism.
92                              Importance: The paramyxovirus family of negative-strand RNA viruses caus
93 loped viruses such as HIV and members of the paramyxovirus family use metastable, proteinaceous fusio
94 exes may differ among diverse members of the paramyxovirus family, central elements of the triggering
95 sles virus (MeV), a morbillivirus within the paramyxovirus family, expresses two envelope glycoprotei
96     The measles virus (MeV), a member of the paramyxovirus family, is an important cause of pediatric
97 ed stabilizing structural element within the paramyxovirus family.
98 G interact with NiV F, a new finding for the paramyxovirus family.
99 tic distance in the phylogenetic tree of the paramyxovirus family.
100 ent fusion protein classes by example of the paramyxovirus, flavivirus and rhabdovirus families.
101 ses, poxviruses, arenaviruses, bunyaviruses, paramyxoviruses, flaviviruses, and HIV-1.
102 es PP1 antagonism as a mechanism employed by paramyxoviruses for evading innate immune recognition.
103  can be applicable to other animal and human paramyxoviruses for rationally designing live attenuated
104                                         Many paramyxoviruses for which bats and rodents serve as majo
105 nsive structure-function relationship of any paramyxovirus FP and demonstrate that the HeV F FP and p
106 rate that the HeV F FP and potentially other paramyxovirus FPs likely require an alpha-helical struct
107                                          The paramyxovirus fusion (F) glycoprotein is anchored in the
108                                      Using a paramyxovirus fusion (F) protein, we tested this paradig
109 recently shown that isolated TMDs from three paramyxovirus fusion (F) proteins interact as trimers us
110                                              Paramyxovirus fusion (F) proteins promote membrane fusio
111               We suggest a general model for paramyxovirus fusion activation in which receptor engage
112               We suggest a general model for paramyxovirus fusion activation in which receptor engage
113  has broad implications for the mechanism of paramyxovirus fusion and for strategies to prevent viral
114 multiple critical parameters that govern the paramyxovirus fusion cascade, and our assays should help
115             Moreover, F(RSV) is unique among paramyxovirus fusion proteins since F(RSV) possesses two
116                                              Paramyxovirus fusion with the host cell plasma membrane
117 propose a simple model for the activation of paramyxovirus fusion.
118  with the stalk-mediated activation model of paramyxovirus fusion.
119 favor of the HN stalk-mediated activation of paramyxovirus fusion.
120                          In common with most paramyxoviruses, fusion mediated by Sendai virus F prote
121       Receptor binding by members of related paramyxovirus genera rearranges the head domains of the
122               To study the function of novel paramyxovirus genes in JPV, a plasmid containing a full-
123 e results advance our basic understanding of paramyxovirus genome packaging interactions and also hav
124                                              Paramyxovirus genomes are ribonucleoprotein (RNP) comple
125                                 N-glycans on paramyxovirus glycoproteins are generally required for p
126 iC) reveals that a high-affinity receptor-to-paramyxovirus H monomer stoichiometry below parity is su
127 phosphorylation site within the P protein in paramyxovirus has been found to play a positive role in
128 phosphorylation site within the P protein in paramyxovirus has been identified as playing a positive
129 is is the first time that the P protein of a paramyxovirus has been systematically analyzed for S/T r
130 he small hydrophobic (SH) protein of certain paramyxoviruses has been found to result in attenuation,
131 redness, because non-human coronaviruses and paramyxoviruses have been listed as priority concerns in
132                                 In addition, paramyxoviruses have evolved diverse mechanisms to disru
133                                              Paramyxoviruses have previously been shown to block prod
134                                         Most paramyxoviruses have two integral membrane proteins: fus
135       Negative-strand RNA viruses, including paramyxoviruses, have been shown to alter autophagy, but
136                                          The paramyxovirus hemagglutinin-neuraminidase (HN) protein p
137 N stalk domain, and properties of a chimeric paramyxovirus HN protein, we propose a simple model for
138 By extensive study of properties of multiple paramyxovirus HN proteins, we show that key features of
139                                              Paramyxovirus HN, H, and G form a dimer-of-dimers consis
140  targeting, and broaden our understanding of paramyxovirus-host interactions.
141                                          The paramyxoviruses human respiratory syncytial virus (hRSV)
142 he HN protein, which is conserved in several paramyxoviruses.IMPORTANCE Oncolytic Newcastle disease v
143 nd mRNA editing experiments revealed a novel paramyxovirus in the genus Ferlavirus, named anaconda pa
144                         Nipah virus (NiV), a paramyxovirus in the genus Henipavirus, has a mortality
145 cent studies have shown a great diversity of paramyxoviruses in an urban-roosting population of straw
146           Entry of hMPV is unusual among the paramyxoviruses, in that fusion is accomplished by the f
147                                        Human paramyxoviruses include global causes of lower respirato
148                                              Paramyxoviruses include many economically and agricultur
149                                              Paramyxoviruses include many important animal and human
150                                              Paramyxoviruses include several insidious and ubiquitous
151                                        Human paramyxoviruses, including hRSV, hMPV, and hPIV3, cause
152                                              Paramyxoviruses, including human metapneumovirus (HMPV),
153                                          The paramyxoviruses, including human parainfluenza viruses (
154 aturally occurring cleavage sites of several paramyxoviruses, including neurovirulent and avirulent s
155                                              Paramyxoviruses, including the childhood pathogen human
156                                         Most paramyxoviruses infect host cells via the concerted acti
157      Phylogenetic analyses revealed that all paramyxoviruses infecting Malagasy bats are UMRVs and sh
158                                              Paramyxovirus infection also gives rise to the formation
159 portance of innate responses in DC following paramyxovirus infection and their consequences for the a
160 ation that H2S also has a protective role in paramyxovirus infection by modulating inflammatory respo
161 t these pathways, and the innate response to paramyxovirus infection of dendritic cells (DC).
162 ally varies conserved motifs of the proposed paramyxovirus infection pathway.
163 ay indicate a new paradigm for understanding Paramyxovirus infection.
164                               For nearly all paramyxoviruses, infection is initiated by fusion of the
165 otentially be targeted for the inhibition of paramyxovirus infections.
166 uld represent a novel treatment strategy for paramyxovirus infections.
167 ggesting a broad inhibitory effect of H2S on paramyxovirus infections.
168 etapneumovirus (HMPV), a recently discovered paramyxovirus, infects nearly 100% of the world populati
169                                              Paramyxoviruses initiate entry through the concerted act
170                  Here we show that different paramyxoviruses interact in distinct ways with cells in
171            The fusion process for nearly all paramyxoviruses involves the mixing of the host cell pla
172  Human parainfluenza virus type 3 (HPIV3), a paramyxovirus, is a major viral cause of severe lower re
173      Newcastle disease virus (NDV), an avian paramyxovirus, is a promising OV and is inherently tumor
174                                       MuV, a paramyxovirus, is an important human pathogen.
175      Newcastle disease virus (NDV), an avian paramyxovirus, is inherently tumor selective and is curr
176 ycoprotein G of Hendra virus (HeV), a deadly paramyxovirus, is N-glycosylated at six sites (G2 to G7)
177              Parainfluenza virus 5 (PIV5), a paramyxovirus, is not known to cause any illness in huma
178                                            J paramyxovirus (JPV) encodes four integral membrane prote
179                 At 18,954 nucleotides, the J paramyxovirus (JPV) genome is one of the largest in the
180                                            J paramyxovirus (JPV) was first isolated from moribund mic
181 s to Newcastle disease virus (NDV), an avian paramyxovirus known to elicit a strong innate immune res
182 ain organization of phylogenetically diverse Paramyxovirus L proteins derived from measles virus (MeV
183    Here we describe four approaches by which paramyxoviruses limit IFN induction: by limiting synthes
184                            The V proteins of paramyxoviruses limit interferon induction by binding md
185                                        Other paramyxovirus M proteins have been shown to dimerize, an
186  and Hendra virus (recently emerged zoonotic paramyxoviruses) M (matrix) protein-derived virus-like p
187                                     For many paramyxoviruses, M proteins drive viral assembly and egr
188                                              Paramyxovirus matrix (M) proteins organize virus assembl
189 e and sequence conservation imply that other paramyxovirus matrix proteins function similarly.
190 he cytoplasmic tail of the F proteins of the paramyxoviruses measles virus, mumps virus, Newcastle di
191 s, including orthomyxoviruses (influenza A), paramyxoviruses (measles), and hepadnaviruses (hepatitis
192 interactions as conserved core mechanisms of paramyxovirus-mediated membrane fusion.
193 interactions as conserved core mechanisms of paramyxovirus-mediated membrane fusion.
194        According to the existing paradigm of paramyxovirus membrane fusion, the initial activation of
195                                   The deadly paramyxovirus Nipah virus (NiV) contains a fusion glycop
196  V proteins of measles virus and the related paramyxovirus Nipah virus interact with PP1alpha/gamma,
197 g partner for the M proteins of the zoonotic paramyxoviruses Nipah virus and Hendra virus.
198 espiratory syncytial virus, and the zoonotic paramyxoviruses Nipah virus and Hendra virus.
199       In contrast to our findings with other paramyxoviruses, normal human serum (NHS) alone did not
200  defined regions near the C-terminal ends of paramyxovirus nucleocapsid proteins that are important f
201  the F protein of Newcastle disease virus, a paramyxovirus of a different genus, suggesting a conserv
202 uman metapneumovirus (hMPV) is a respiratory paramyxovirus of global clinical relevance.
203                                              Paramyxoviruses of the morbillivirus genus, such as meas
204                   A quantitative approach to paramyxovirus packaging revealed a majority of infectiou
205 odies (sAbs) against multiple domains of the paramyxovirus parainfluenza 5 (PIV5) pre- and postfusion
206 region (MPSR) (HN, residues 37 to 56) of the paramyxovirus parainfluenza virus (PIV5), a region of th
207      We show that Cav-1 colocalizes with the paramyxovirus parainfluenza virus 5 (PIV-5) nucleocapsid
208                                          The paramyxovirus parainfluenza virus 5 (PIV5) enters cells
209 ure of the F protein (prefusion form) of the paramyxovirus parainfluenza virus 5 (PIV5) WR isolate wa
210           Here, we show that the FP from the paramyxovirus parainfluenza virus 5 fusogenic protein, F
211 receptor-binding globular head domain of the paramyxovirus parainfluenza virus 5 HN protein is entire
212                                      For the paramyxoviruses parainfluenza virus 5 (PIV5) and mumps v
213 teractions of the FP of the F protein of the paramyxovirus, parainfluenza virus 5 (PIV5).
214 biquitination of the matrix (M) protein of a paramyxovirus, parainfluenza virus 5 (PIV5).
215                                              Paramyxovirus particles are formed by a budding process
216                               Infectivity of paramyxovirus particles depends on matrix-nucleocapsid p
217                                              Paramyxovirus pathogens include measles virus, mumps vir
218 sion states of the fusion proteins including paramyxovirus PIV5 F and influenza virus hemagglutinin s
219  exploited the well-characterized ability of paramyxovirus (PMV) V proteins to counteract both IFN in
220                                          The paramyxovirus pneumonia virus of mice (PVM) is a model o
221                                          The paramyxovirus pneumonia virus of mice (PVM) is a rodent
222                                              Paramyxovirus polymerases are composed of at least two t
223                        During transcription, paramyxoviruses produce capped, methylated, and polyaden
224                                              Paramyxoviruses produce pleiomorphic particles containin
225                               However, other paramyxovirus proteins, including glycoproteins, nucleoc
226 rological, and genomic characterization of a paramyxovirus recently isolated from rockhopper penguins
227  factor may be involved in the regulation of paramyxovirus replication and could be a target for broa
228  highlights a critical role of 2'-O MTase in paramyxovirus replication and pathogenesis and a new ave
229              Understanding the regulation of paramyxovirus replication will enable the rational desig
230                                              Paramyxoviruses represent a remarkably diverse family of
231                                         Most paramyxoviruses require the interaction of two viral pro
232                                              Paramyxoviruses require two viral membrane glycoproteins
233     The promotion of membrane fusion by most paramyxoviruses requires an interaction between the vira
234                                Cell entry by paramyxoviruses requires fusion between viral and cellul
235  annual and biennial pattern of three common paramyxoviruses, Respiratory Syncytial Virus (RSV), Huma
236 magglutinin-neuraminidase protein of another Paramyxovirus revealed a four-helix bundle stalk.
237 with the F protein TM domains from two other paramyxoviruses, Sendai virus (SV) and measles virus (MV
238 stle disease virus (NDV), representing avian paramyxovirus serotype 1 (APMV-1), cause respiratory and
239                                        Avian paramyxovirus serotype 2 (APMV-2) is one of the nine ser
240 structed a reverse genetics system for avian paramyxovirus serotype 7 (APMV-7) to investigate the rol
241            Our data suggest a model in which paramyxoviruses share an overall common strategy for dir
242 simultaneous identification of IAV-specific, paramyxovirus-specific, and broad-spectrum inhibitors.
243                         The V protein of the paramyxovirus subfamily Paramyxovirinae is an important
244                                  Respiratory paramyxoviruses such as respiratory syncytial virus (RSV
245 tenuated vaccines for hMPV and perhaps other paramyxoviruses, such as hRSV and hPIV3.
246  inhibited the synthesis of the RNA of other paramyxoviruses, such as Nipah virus (NiV), human parain
247 ding hemagglutinin-neuraminidases of certain paramyxoviruses suggest that fusion triggering is preced
248 studies of the triggering mechanism of other paramyxoviruses suggest that receptor binding to their h
249 l action of ISG56/IFIT1, while all the other paramyxoviruses tested were resistant.
250 .IMPORTANCE Nipah virus (NiV) is an emerging paramyxovirus that can cause a lethal respiratory and ne
251     Nipah virus (NiV) is a zoonotic emerging paramyxovirus that can cause fatal respiratory illness o
252            Mumps virus (MuV) is a reemerging paramyxovirus that caused large outbreaks in the United
253 s (hMPV) is a relatively recently identified paramyxovirus that causes acute upper and lower respirat
254                   Nipah virus is an emergent paramyxovirus that causes deadly encephalitis and respir
255             Hendra virus (HeV) is a zoonotic paramyxovirus that causes deadly illness in horses and h
256              Nipah virus (NiV) is a zoonotic paramyxovirus that causes high mortality rates in humans
257            Human metapneumovirus (HMPV) is a paramyxovirus that causes respiratory disease worldwide.
258                       Nipah virus (NiV) is a paramyxovirus that causes severe disease in humans and a
259 uman metapneumovirus (hMPV) is a respiratory paramyxovirus that is distributed worldwide and induces
260                         Nipah is an emerging paramyxovirus that is of serious concern to human health
261 h virus (NiV) is a deadly emerging enveloped paramyxovirus that primarily targets human endothelial c
262         Nipah virus (NiV) is a highly lethal paramyxovirus that recently emerged as a causative agent
263  (HeV) are closely related, recently emerged paramyxoviruses that are capable of causing considerable
264 and Nipah virus (NiV), are emerging zoonotic paramyxoviruses that can cause severe and often lethal n
265 CE Hendra virus and Nipah virus are zoonotic paramyxoviruses that cause lethal infections in humans.
266  (NiV) are closely related, recently emerged paramyxoviruses that form Henipavirus genus and are capa
267                      Bats carry a variety of paramyxoviruses that impact human and domestic animal he
268                 This is in contrast to other paramyxoviruses that require attachment protein function
269 and fusion could differ mechanistically from paramyxoviruses that use glycan-based receptors.
270                                          For paramyxoviruses, the activities of two membrane proteins
271                     Unlike the case for most paramyxoviruses, the fusion proteins (F) of a number of
272                       We have shown that for paramyxoviruses, the inhibitory efficacy of HR peptides
273                            Unlike most other paramyxoviruses, the respiratory syncytial virus (RSV) F
274                                   Across the paramyxoviruses, these domains share little sequence ide
275                                     In other paramyxoviruses, these residues were shown to affect fus
276 te we measured the interbilayer spacing of a paramyxovirus trapped in the process of fusing with soli
277                             We conclude that paramyxoviruses trigger the DNA damage response, a pathw
278                                As with other paramyxoviruses, two major RSV surface viral glycoprotei
279 ed by Newcastle disease virus (NDV) or avian paramyxovirus type 1 (APMV-1), a negative-sense single-s
280                                        Avian paramyxovirus type 1, NDV, has been an attractive oncoly
281 referred to as Unclassified Morbilli-related paramyxoviruses (UMRVs).
282                 These findings indicate that paramyxoviruses use a single amino acid to distinguish M
283                                              Paramyxoviruses use a specialized fusion protein to merg
284  within infected hosts, the vast majority of paramyxoviruses utilize two viral envelope glycoproteins
285 onsequences of MDA5 and LGP2 interference by paramyxovirus V proteins and help resolve the distinct r
286                                              Paramyxovirus V proteins are interferon antagonists that
287                                              Paramyxovirus V proteins bind to MDA5 (melanoma differen
288                                  Importance: Paramyxovirus V proteins interact with two innate immune
289 of generating an experimental Nipah virus (a paramyxovirus) vaccine, we generated two defective VSVDe
290  the crystal structures of HN from different paramyxoviruses, varying energy requirements for fusion
291                                              Paramyxovirus viral fusion proteins (F) insert into the
292 scription (RT)-PCR results were positive for paramyxovirus (viral loads of 2.33 x 10(4) to 1.05 x 10(
293                                           In paramyxoviruses, viral attachment and membrane fusion ar
294                                          For paramyxoviruses, viral matrix (M) proteins are key drive
295 omain of PIV5 F with the C terminus of other paramyxoviruses were unable to cause cell fusion.
296 bserved with parainfluenza virus 5 (PIV5), a paramyxovirus, when neutralizing antibody was used to bl
297 ribed models, including the one proposed for paramyxovirus, where following random movement efficienc
298 otypical members of the Henipavirus genus of paramyxoviruses, which are designated biosafety level 4
299 ncing of JPV (JPV-LW) confirms that JPV is a paramyxovirus with several unique features.
300 irus in the genus Ferlavirus, named anaconda paramyxovirus, with a typical Ferlavirus genomic organiz

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