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1 Nipah virus (NiV) is the deadliest known paramyxovirus.
2 ) is a highly pathogenic and deadly zoonotic paramyxovirus.
3 attenuated vaccines for hMPV and other human paramyxoviruses.
4 bone" appearance of helical nucleocapsids in paramyxoviruses.
5 he most promising vaccine strategy for human paramyxoviruses.
6 be possible to apply these findings to other paramyxoviruses.
7 ds with diameters of 10 to 14 nm, typical of paramyxoviruses.
8 osure model of membrane fusion triggering by paramyxoviruses.
9 tenuated vaccines for aMPV and perhaps other paramyxoviruses.
10 nal receipt mechanism may be conserved among paramyxoviruses.
11 sm for the targeting of the RIG-I pathway by paramyxoviruses.
12 d is understood neither for MV nor for other paramyxoviruses.
13 membrane fusion triggering and cell entry by Paramyxoviruses.
14 t Sendai virus or human metapneumovirus, two paramyxoviruses.
15 potential utility of inhibitory peptides for paramyxoviruses.
16 a new structural-biological finding for the paramyxoviruses.
17 may be involved in the replication of these paramyxoviruses.
18 ering broad spectrum antivirals for emerging paramyxoviruses.
19 of F, which has not been reported for other paramyxoviruses.
20 ng mechanism(s) of the henipaviruses and the paramyxoviruses.
21 se are poorly understood compared with other paramyxoviruses.
22 rix gene assay, designed to detect all avian paramyxovirus 1, did not detect four of the isolates.
27 matrix protein of Newcastle disease virus, a paramyxovirus and relative of measles virus, forms dimer
28 that is a highly effective inhibitor of both paramyxoviruses and a set of criteria to be used for eng
29 ld enhance intrinsic cross-immunity to these paramyxoviruses and approaches to controlling recurring
34 tly proposed "stalk exposure model" to other paramyxoviruses and propose an "induced fit" hypothesis
35 proposed for parainfluenza virus 5 to other paramyxoviruses and propose an "induced fit" hypothesis
37 ajor animal populations capable of harboring paramyxoviruses, and host shifting between these animals
39 rus (NDV) belongs to serotype 1 of the avian paramyxoviruses (APMV-1) and causes severe disease in ch
40 n the replication and pathogenicity of avian paramyxoviruses (APMVs), we constructed a reverse geneti
48 amental architecture appears conserved among paramyxovirus attachment protein stalk domains, we predi
49 It is unknown how receptor binding by the paramyxovirus attachment proteins (HN, H, or G) triggers
51 genomes (DVGs) can facilitate persistence of paramyxoviruses, but the underlying mechanisms are uncle
52 gglutinin (HA)-neuraminidase protein (HN) of paramyxoviruses carries out three discrete activities, e
53 hemagglutinin-neuraminidase (HN) protein of paramyxoviruses carries out three distinct activities co
54 VI) of the large (L) polymerase proteins of paramyxoviruses catalyzes methyltransferase (MTase) acti
55 VI) of the large (L) polymerase proteins of paramyxoviruses catalyzes methyltransferase (MTase) acti
62 iral RNA-dependent RNA polymerase (vRdRp) of paramyxovirus consists of the large (L) protein and the
67 ignificant zoonotic spillover of chiropteran paramyxoviruses could be missed throughout much of Afric
69 endra virus (HeV) and Nipah virus (NiV), are paramyxoviruses discovered in the mid- to late 1990s tha
70 vated infection levels as well as widespread paramyxovirus dispersal and frequent host exchange of a
71 parainfluenza virus 5 (PIV5), a prototypical paramyxovirus, encodes a V protein that inhibits viral R
77 We identified a small molecule that inhibits paramyxovirus entry into target cells and prevents infec
78 te closure of the fusion core does not drive paramyxovirus entry may aid the design of strategies for
79 unctionally varies conserved elements of the paramyxovirus entry pathway, providing a possible explan
81 This first evidence that activation of a paramyxovirus F can be specifically induced before the v
82 ship between enhanced fusion activity in the paramyxovirus F protein and increased susceptibility to
83 ermine factors driving this association, 140 paramyxovirus F protein TM domain sequences were analyze
85 protein linked to the TM segments from three paramyxovirus F proteins was analyzed by sedimentation e
88 ilarity between postfusion coronavirus S and paramyxovirus F structures demonstrates that a conserved
89 ding of the mechanism(s) of receptor-induced paramyxovirus F triggering during viral entry and cell-c
90 terminal helix is a necessary early step for paramyxovirus F-protein refolding and presents a novel t
93 loped viruses such as HIV and members of the paramyxovirus family use metastable, proteinaceous fusio
94 exes may differ among diverse members of the paramyxovirus family, central elements of the triggering
95 sles virus (MeV), a morbillivirus within the paramyxovirus family, expresses two envelope glycoprotei
96 The measles virus (MeV), a member of the paramyxovirus family, is an important cause of pediatric
100 ent fusion protein classes by example of the paramyxovirus, flavivirus and rhabdovirus families.
102 es PP1 antagonism as a mechanism employed by paramyxoviruses for evading innate immune recognition.
103 can be applicable to other animal and human paramyxoviruses for rationally designing live attenuated
105 nsive structure-function relationship of any paramyxovirus FP and demonstrate that the HeV F FP and p
106 rate that the HeV F FP and potentially other paramyxovirus FPs likely require an alpha-helical struct
109 recently shown that isolated TMDs from three paramyxovirus fusion (F) proteins interact as trimers us
113 has broad implications for the mechanism of paramyxovirus fusion and for strategies to prevent viral
114 multiple critical parameters that govern the paramyxovirus fusion cascade, and our assays should help
123 e results advance our basic understanding of paramyxovirus genome packaging interactions and also hav
126 iC) reveals that a high-affinity receptor-to-paramyxovirus H monomer stoichiometry below parity is su
127 phosphorylation site within the P protein in paramyxovirus has been found to play a positive role in
128 phosphorylation site within the P protein in paramyxovirus has been identified as playing a positive
129 is is the first time that the P protein of a paramyxovirus has been systematically analyzed for S/T r
130 he small hydrophobic (SH) protein of certain paramyxoviruses has been found to result in attenuation,
131 redness, because non-human coronaviruses and paramyxoviruses have been listed as priority concerns in
137 N stalk domain, and properties of a chimeric paramyxovirus HN protein, we propose a simple model for
138 By extensive study of properties of multiple paramyxovirus HN proteins, we show that key features of
142 he HN protein, which is conserved in several paramyxoviruses.IMPORTANCE Oncolytic Newcastle disease v
143 nd mRNA editing experiments revealed a novel paramyxovirus in the genus Ferlavirus, named anaconda pa
145 cent studies have shown a great diversity of paramyxoviruses in an urban-roosting population of straw
154 aturally occurring cleavage sites of several paramyxoviruses, including neurovirulent and avirulent s
157 Phylogenetic analyses revealed that all paramyxoviruses infecting Malagasy bats are UMRVs and sh
159 portance of innate responses in DC following paramyxovirus infection and their consequences for the a
160 ation that H2S also has a protective role in paramyxovirus infection by modulating inflammatory respo
168 etapneumovirus (HMPV), a recently discovered paramyxovirus, infects nearly 100% of the world populati
172 Human parainfluenza virus type 3 (HPIV3), a paramyxovirus, is a major viral cause of severe lower re
173 Newcastle disease virus (NDV), an avian paramyxovirus, is a promising OV and is inherently tumor
175 Newcastle disease virus (NDV), an avian paramyxovirus, is inherently tumor selective and is curr
176 ycoprotein G of Hendra virus (HeV), a deadly paramyxovirus, is N-glycosylated at six sites (G2 to G7)
181 s to Newcastle disease virus (NDV), an avian paramyxovirus known to elicit a strong innate immune res
182 ain organization of phylogenetically diverse Paramyxovirus L proteins derived from measles virus (MeV
183 Here we describe four approaches by which paramyxoviruses limit IFN induction: by limiting synthes
186 and Hendra virus (recently emerged zoonotic paramyxoviruses) M (matrix) protein-derived virus-like p
190 he cytoplasmic tail of the F proteins of the paramyxoviruses measles virus, mumps virus, Newcastle di
191 s, including orthomyxoviruses (influenza A), paramyxoviruses (measles), and hepadnaviruses (hepatitis
196 V proteins of measles virus and the related paramyxovirus Nipah virus interact with PP1alpha/gamma,
200 defined regions near the C-terminal ends of paramyxovirus nucleocapsid proteins that are important f
201 the F protein of Newcastle disease virus, a paramyxovirus of a different genus, suggesting a conserv
205 odies (sAbs) against multiple domains of the paramyxovirus parainfluenza 5 (PIV5) pre- and postfusion
206 region (MPSR) (HN, residues 37 to 56) of the paramyxovirus parainfluenza virus (PIV5), a region of th
207 We show that Cav-1 colocalizes with the paramyxovirus parainfluenza virus 5 (PIV-5) nucleocapsid
209 ure of the F protein (prefusion form) of the paramyxovirus parainfluenza virus 5 (PIV5) WR isolate wa
211 receptor-binding globular head domain of the paramyxovirus parainfluenza virus 5 HN protein is entire
218 sion states of the fusion proteins including paramyxovirus PIV5 F and influenza virus hemagglutinin s
219 exploited the well-characterized ability of paramyxovirus (PMV) V proteins to counteract both IFN in
226 rological, and genomic characterization of a paramyxovirus recently isolated from rockhopper penguins
227 factor may be involved in the regulation of paramyxovirus replication and could be a target for broa
228 highlights a critical role of 2'-O MTase in paramyxovirus replication and pathogenesis and a new ave
233 The promotion of membrane fusion by most paramyxoviruses requires an interaction between the vira
235 annual and biennial pattern of three common paramyxoviruses, Respiratory Syncytial Virus (RSV), Huma
237 with the F protein TM domains from two other paramyxoviruses, Sendai virus (SV) and measles virus (MV
238 stle disease virus (NDV), representing avian paramyxovirus serotype 1 (APMV-1), cause respiratory and
240 structed a reverse genetics system for avian paramyxovirus serotype 7 (APMV-7) to investigate the rol
242 simultaneous identification of IAV-specific, paramyxovirus-specific, and broad-spectrum inhibitors.
246 inhibited the synthesis of the RNA of other paramyxoviruses, such as Nipah virus (NiV), human parain
247 ding hemagglutinin-neuraminidases of certain paramyxoviruses suggest that fusion triggering is preced
248 studies of the triggering mechanism of other paramyxoviruses suggest that receptor binding to their h
250 .IMPORTANCE Nipah virus (NiV) is an emerging paramyxovirus that can cause a lethal respiratory and ne
251 Nipah virus (NiV) is a zoonotic emerging paramyxovirus that can cause fatal respiratory illness o
253 s (hMPV) is a relatively recently identified paramyxovirus that causes acute upper and lower respirat
259 uman metapneumovirus (hMPV) is a respiratory paramyxovirus that is distributed worldwide and induces
261 h virus (NiV) is a deadly emerging enveloped paramyxovirus that primarily targets human endothelial c
263 (HeV) are closely related, recently emerged paramyxoviruses that are capable of causing considerable
264 and Nipah virus (NiV), are emerging zoonotic paramyxoviruses that can cause severe and often lethal n
265 CE Hendra virus and Nipah virus are zoonotic paramyxoviruses that cause lethal infections in humans.
266 (NiV) are closely related, recently emerged paramyxoviruses that form Henipavirus genus and are capa
276 te we measured the interbilayer spacing of a paramyxovirus trapped in the process of fusing with soli
279 ed by Newcastle disease virus (NDV) or avian paramyxovirus type 1 (APMV-1), a negative-sense single-s
284 within infected hosts, the vast majority of paramyxoviruses utilize two viral envelope glycoproteins
285 onsequences of MDA5 and LGP2 interference by paramyxovirus V proteins and help resolve the distinct r
289 of generating an experimental Nipah virus (a paramyxovirus) vaccine, we generated two defective VSVDe
290 the crystal structures of HN from different paramyxoviruses, varying energy requirements for fusion
292 scription (RT)-PCR results were positive for paramyxovirus (viral loads of 2.33 x 10(4) to 1.05 x 10(
296 bserved with parainfluenza virus 5 (PIV5), a paramyxovirus, when neutralizing antibody was used to bl
297 ribed models, including the one proposed for paramyxovirus, where following random movement efficienc
298 otypical members of the Henipavirus genus of paramyxoviruses, which are designated biosafety level 4
300 irus in the genus Ferlavirus, named anaconda paramyxovirus, with a typical Ferlavirus genomic organiz
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