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1 pecially lung, ovarian and breast, can cause paraneoplastic cerebellar degeneration.
2 thophysiology were described 10 years ago in paraneoplastic cerebellar degeneration.
4 was the cause of death of 65% HuAb positive paraneoplastic cerebellar degeneration and 10% HuAb nega
7 e anti-Tr immune response is associated with paraneoplastic cerebellar degeneration and Hodgkin lymph
8 litis, 1 paraneoplastic encephalomyelitis, 1 paraneoplastic cerebellar degeneration, and 1 opsoclonus
9 rum and cerebrospinal fluid of patients with paraneoplastic cerebellar degeneration associated with c
10 rocessed HLA-A2.1 restricted epitopes of the paraneoplastic cerebellar degeneration breast/ovarian ca
11 immunomodulation did not alter the course of paraneoplastic cerebellar degeneration, but improved Lam
12 at patients with the same tumour can develop paraneoplastic cerebellar degeneration by different immu
13 patients who differed from the HuAb negative paraneoplastic cerebellar degeneration cohort, HuAb posi
14 m of 57 patients with presenting symptoms of paraneoplastic cerebellar degeneration for the presence
16 /or serum samples from 5 other patients with paraneoplastic cerebellar degeneration, HL, and anti-Tr.
17 In patients with small-cell lung cancer, paraneoplastic cerebellar degeneration may occur with or
18 and immunological developments, focusing on paraneoplastic cerebellar degeneration, opsoclonus-myocl
20 erebellar degeneration and 10% HuAb negative paraneoplastic cerebellar degeneration patients (P < 0.0
22 0% of HuAb positive and 20% of HuAb negative paraneoplastic cerebellar degeneration patients, the tum
25 among the better described autoantibodies in paraneoplastic cerebellar degeneration (PCD) combined wi
31 era from patients with Hodgkin's disease and paraneoplastic cerebellar degeneration resulted in the i
33 bodies associated with different cancers and paraneoplastic cerebellar degeneration suggests that sev
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