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1 (18)F-FDG PET/CT in patients with suspected paraneoplastic syndrome.
2 malignancy in patients suspected of having a paraneoplastic syndrome.
3 n of the nervous system may be involved in a paraneoplastic syndrome.
4 (18)F-FDG PET/CT in patients with suspected paraneoplastic syndrome.
5 malignancy in patients suspected of having a paraneoplastic syndrome.
6 cally suspected neurologic and nonneurologic paraneoplastic syndromes.
7 ntly fatal malignancy and for the associated paraneoplastic syndromes.
8 oplasm in patients presenting with suspected paraneoplastic syndromes.
9 elevant pathways that lead to the associated paraneoplastic syndromes.
10 15 patients with cancer presented as classic paraneoplastic syndromes (5 limbic encephalitis, 1 paran
12 ytotoxic CD8+ T cell in patients with the Hu paraneoplastic syndrome and suggest that SCLC may evade
13 93 individual patients suspected of having a paraneoplastic syndrome and who underwent (18)F-FDG PET
14 93 individual patients suspected of having a paraneoplastic syndrome and who underwent (18)F-FDG PET
15 stic neurological symptoms, 96 patients with paraneoplastic syndromes and 10 patients with non-cancer
16 nd toxic molecules to the internodal axon in paraneoplastic syndromes and demyelinating diseases.
18 osition, after acute infection, as part of a paraneoplastic syndrome, and after exposure to neurotoxi
20 Melanoma-associated retinopathy (MAR) is a paraneoplastic syndrome associated with cutaneous malign
21 of this review is to define and describe the paraneoplastic syndromes associated with gynecologic neo
22 icion of a paraneoplastic condition, but any paraneoplastic syndrome can also occur in patients witho
24 ntagonist (VEGF-TRAP(R1R2)), thus defining a paraneoplastic syndrome caused by excessive VEGF activit
25 resistant, upbeat nystagmus resulting from a paraneoplastic syndrome caused by stage 2A, grade I, nod
29 rapeutic interventions for a group of visual paraneoplastic syndromes, including carcinoma-associated
31 on that their symptoms or findings reflect a paraneoplastic syndrome may allow the tumor responsible
32 toantibodies or inflammatory gene mutations, paraneoplastic syndrome mechanisms via ectopic cytokine
37 abnormalities is wide, and include cutaneous paraneoplastic syndromes such as xanthomas, acanthosis n
39 ple sclerosis, neuromyelitis optica, and the paraneoplastic syndromes where highly specific T cell re
42 melanocytic proliferation (BDUMP) is a rare paraneoplastic syndrome with characteristic findings, in
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