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1 ression, associated with a decrease in serum paraprotein.
2 was positively correlated with the level of paraprotein.
3 ecreted by the tumor cells and the IgG serum paraprotein.
4 monoclonal antibodies, immune complexes and paraproteins.
5 c method to identify the cognate antigens of paraproteins.
6 rden of clonal plasma cells that produce the paraproteins.
7 s from all patients with HSP90-SUMO1-binding paraproteins.
8 s (1.9 years) than patients with IgA and IgG paraproteins (2.3 and 2.5 years, respectively) (P < .001
9 tinuum that requires the presence of a serum paraprotein and differs in severity of skin lesions, ext
12 experienced a greater than 50% reduction in paraprotein, and four (17%) of 24 patients entered compl
13 of 18 cases, the serum contained benign IgM paraproteins, and in four of these cases at least two Ig
15 increase in FLC both alone and with a whole paraprotein, as a result of a significantly shorter surv
20 onic ataxic neuropathy, ophthalmoplegia, IgM paraprotein, cold agglutinins and disialosyl antibodies.
24 ls in SCID-hu mice produced human monoclonal paraprotein (immunoglobulin M [IgM] and/or kappa or lamb
25 resulted in a greater than 25% reduction in paraprotein in 67% of patients, 13 patients (54%) experi
29 in bands (APB), distinct from the presenting paraprotein, in 550 patients receiving high-dose therapy
31 was one complete response and a reduction in paraprotein levels and/or marrow plasmacytosis in eight
33 TGM1 cells, reduced serum myeloma-associated paraprotein levels, and better preservation of bone mine
34 th IgM AL amyloidosis have a significant IgM paraprotein (median, 10 g/L), less frequent lambda light
36 Best responses of at least 25% reduction in paraprotein occurred in 17 (71%) of 24 patients (90% con
37 ented with a whole paraprotein relapsed with paraprotein only (PO) and "FLC escape," respectively.
38 ectrophoresis (IFE), could be due to altered paraprotein production by the malignant plasma cell clon
40 4 of 520 patients who presented with a whole paraprotein relapsed with paraprotein only (PO) and "FLC
41 lantation in the management of patients with paraprotein-related kidney disease continues to evolve.
44 SUMO1 carriers produced HSP90-SUMO1-specific paraproteins, suggesting that sumoylated HSP90 promotes
45 we identified a posttranslationally modified paraprotein target (paratargs) in monoclonal gammopathie
46 es reduced tumor load assessed by monoclonal paraprotein titers, prevented splenomegaly, limited deve
47 Protein and nucleotide sequencing of the paraproteins' variable regions revealed 2 predominant Vl
50 Stable disease (less than 25% reduction in paraprotein) was observed in an additional 2 (8%) patien
51 and reduction in levels of circulating human paraprotein were observed in mice treated with B-B4-DM1.
54 vious efforts to reconstruct the epitopes of paraproteins, with the significant difference that it pr
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