ライフサイエンスコーパス: parasite infection

1 immunological status of the lungs induced by parasite infection.

2 arvum via upregulation of survivin, favoring parasite infection.

3 ase in C57BL/6 mice with Leishmania mexicana parasite infection.

4 ignaling enhances immune responses following parasite infection.

5 sufficient to reproduce the major effects of parasite infection.

6 ective role in the innate immune response to parasite infection.

7 ll subset in the protection against systemic parasite infection.

8 ion, a defense mechanism against pathogen or parasite infection.

9 ation of the inflammatory response following parasite infection.

10 standing the biological processes underlying parasite infection.

11 t cell actin polymerization is necessary for parasite infection.

12 to wheeze in the presence of high-intensity parasite infection.

13 T cells has been shown to be important in a parasite infection.

14 oidan) whereas high concentrations inhibited parasite infection.

15 kinetes and sporozoites (CelTOS) can inhibit parasite infection.

16 received a diagnosis of blood-stage malaria parasite infection.

17 tive immune responses that depend on ongoing parasite infection.

18 n essential early step in successful malaria parasite infection.

19 nfected host cells during the early hours of parasite infection.

20 esponding effector T cells (Teff) in chronic parasite infection.

21 l initiator of the type 2 immune response to parasite infection.

22 features of nematodes are recognized during parasite infection.

23 way, altering macrophage polarization during parasite infection.

24 e host immune system to sense and respond to parasite infection.

25 these flies were thus highly susceptible to parasite infection.

26 n 1) that were associated significantly with parasite infection.

27 l active today and can be exploited to treat parasite infection.

28 ective method of biological control of plant parasite infection.

29 t an autonomous response to the challenge of parasite infection.

30 with the development of Th2 immunity during parasite infection.

31 -) mice and examined their responsiveness to parasite infection.

32 n important protective role in bacterial and parasite infections.

33 hway during immune induction by pathogen and parasite infections.

34 at levels theoretically sufficient to clear parasite infections.

35 ologic changes associated with hemoprotozoan parasite infections.

36 use, are now under investigation for various parasite infections.

37 atory method for diagnosing gastrointestinal parasite infections.

38 the doses, will efficiently clear resistant parasite infections.

39 ty, and have wide applicability for studying parasite infections.

40 ular functions and are involved in viral and parasite infections.

41 lood stage parasite growth and clear malaria parasite infections.

42 tions, Viral Syndromes, and Blood and Tissue Parasite Infections.

43 issues of allergic conditions and helminthic parasite infections.

44 trol their overall inflammatory responses to parasite infections.

45 ed significantly increased susceptibility to parasite infection accompanied by increased numbers of p

46 as p38 MAPK activating kinases revealed that parasite infection activates only MKK3.

47 velopment of T helper type 2 immunity during parasite infection and allergic inflammation.

48 localization of QTL conferring resistance to parasite infection and beetle fitness may result from th

49 abnormal amphibians are associated with both parasite infection and chemical contaminants, but that t

50 gets because of their indispensable roles in parasite infection and development, especially in the pr

51 ve trait loci (QTL) mapping of resistance to parasite infection and fitness traits using the red flou

52 from suppression of collateral damage during parasite infection and from reduced allergic, autoimmune

53 the hypothesis that 'pace-of-life' predicts parasite infection and host pathology.

54 consequences of these changes for recurrent parasite infection and infection-associated pathologies

55 hat type I IFN can be induced in response to parasite infection and influence the outcome of infectio

56 at TLR9 mediates the innate response to oral parasite infection and is involved in the development of

57 onal light microscopy in detecting low-grade parasite infection and offers an exceptional advantage f

58 tion but were overtly normal with respect to parasite infection and pathological responses.

59 section of human immune responses to malaria parasite infection and the evaluation of therapeutics an

60 bodies against P. vivax CSP strongly inhibit parasite infection and thus support the notion that thes

61 They were first described in the context of parasite infections and allergic processes.

62 contaminants, (ii) land use practices, (iii) parasite infection, and (iv) targeted interactions betwe

63 ppressor cells have been described in tumor, parasite infection, and severe trauma models.

64 TPLEELYPT211, was observed after the initial parasite infection, and the anti-iB-1 antibodies were no

65 is granulocyte, including allergic diseases, parasite infections, and tumorigenesis.

66 prove child health outcomes (diarrhea, HCGI, parasite infection, anemia, growth).

67 Helminth parasite infections are associated with a battery of imm

68 Mixed parasite infections are common in many parts of the worl

69 e findings call for further investigation of parasite infection as a cause of amphibian deformities i

70 stickleback and three ecologically relevant parasite infections as a "wild" model.

71 l host responses to a non-replicating type I parasite infection associated with development of long-l

72 arbor seals (Phoca vitulina), a weak HFC for parasite infection based on 27 microsatellites strengthe

73 the expected long-term effect of climate on parasite infections but can also shift the seasonal peak

74 evamisole is commonly used to treat nematode parasite infections but therapy is limited by resistance

75 -macroglobulin responded strongly to malaria parasite infection, but displayed little or no response

76 wheeze as an outcome measure and ascertained parasite infection by fecal examination.

77 Furthermore, demonstration that a parasite infection can counteract the deleterious effect

78 Intrapopulation differences in parasite infections can develop from specialist individu

79 However, we also suggest that parasite infections can drive this niche specialisation.

80 Chronic, low-intensity parasite infections can reduce host fitness through nega

81 roles in diverse chronic diseases, including parasite infections, cancer, and allergic responses.

82 protection against controlled human malaria parasite infection (CHMI) and natural exposure.

83 ssibility by chromatin remodeling during the parasite infection cycle.

84 Parasite infection decreased 57 (inclusive of IL-12 and

85 Parasite infections do not in general protect against as

86 Parasite infection does not prevent allergen sensitisati

87 Because parasite infections elicit a similar immunologic environ

88 rmation, the exploration of the effects that parasite infections exert on populations of commensal gu

89 Data on confirmed malarial parasite infections from health facilities in interventi

90 Trypanosomatid parasite infections have a devastating impact on human h

91 stigated in treatments of cancer, virus, and parasite infections (i.e., malaria) as well as in crop s

92 to hemocytes to differentiate and respond to parasite infection, implicating hemocytes as critical mo

93 en-related protein 1 (FREP1) is critical for parasite infection in Anopheles gambiae and facilitates

94 Second, the low incidence of parasite infection in industrial nations is cited as a f

95 monstrated that Wnt5a-mediated inhibition of parasite infection in macrophages is Rac1/Rho dependent.

96 Progression of the parasite infection in the fly depends on factors inheren

97 8 cells and observed consistent reduction of parasite infection in these knockdown cells.

98 demonstrated that 1294 significantly reduces parasite infection in vitro, with a half maximal effecti

99 damage during development and by controlling parasite infections in adults that can otherwise reduce

100 ombined field-derived estimates of trematode parasite infections in aquatic snails with measurements

101 The mechanisms of protective immunity to parasite infections in humans are still elusive.

102 CD8(+) CTLs protect against parasite infections in mice primarily by secreting inter

103 ne whether total or species-specific current parasite infection is associated with a reduced risk of

104 To determine whether host resistance to parasite infection is associated with fitness costs, we

105 rst, obligatory replication phase of malaria parasite infections is characterized by rapid expansion

106 n, the role of endogenous Gal-1 during acute parasite infections is uncertain.

107 antibodies were not readily produced during parasite infection, it may be desirable to direct antibo

108 periodic relapses of symptomatic blood stage parasite infections likely initiated by activation of do

109 urs (through anaesthesia exposure) increased parasite infection loads in isolated hosts by 62-102% re

110 e, two such proteins that antagonize malaria parasite infections, LRIM1 and APL1C, circulate in the h

111 dataset of 934 collections (without data on parasite infection), malformation frequency was best pre

112 urce availability, and seasonal variation in parasite infections may further alter ruminant body cond

113 High degrees of parasite infection might prevent asthma symptoms in atop

114 man PBMCs from HVL, similar to the wild type parasite infection, mimicking a naturally acquired prote

115 Among collections also examined for parasite infection (n = 154), average parasite load and

116 development of effective vaccines that block parasite infection of erythrocytes is identifying the pl

117 P. berghei we are able to robustly quantify parasite infection of hepatocyte cell lines by flow cyto

118 c cleavage of the CSP, a key requirement for parasite infection of hepatocytes.

119 mes play distinctive biological roles during parasite infection of mammalian cells.

120 ns most of the genetic variation for malaria parasite infection of mosquitoes in nature.

121 ity have decreased substantially, malaria, a parasite infection of red blood cells, still kills rough

122 Experimental parasite infections of advanced mosquito intercrosses de

123 onsidered for virotherapy to control certain parasite infections of man and animals.

124 Parasite infections often lead to dramatically different

125 e to confirm previously described effects of parasite infection on host dopamine either in vitro or i

126 the effects of rainfall and gastrointestinal parasite infections on springbok (Antidorcas marsupialis

127 ittle is known of the effects of concomitant parasite infections on the immune response or severity o

128 e mice were shown to protect against mucosal parasite infection (P < 0.05), demonstrating that mucosa

129 udies and case reports suggest that internal parasite infections (PI) can cause CSU.

130 isease include microbial exposures including parasite infection, pollution, diet and obesity.

131 Prior malaria parasite infection primed the production of anti-native

132 city of macrophages is evident in helminthic parasite infections, providing protection from inflammat

133 Current therapies for human parasite infections rely on a few drugs, most of which h

134 However, their role during parasite infection remains to be clarified.

135 cing fibrinogen-related protein 30 increased parasite infection significantly, whereas ablation of fi

136 er level of ITGA2 protein was present in the parasite infection sites.

137 pose an improved indicator that incorporates parasite infection status (as assessed by a rapid diagno

138 mucosal immunity to intestinal intracellular parasite infections such as Eimeria infection.

139 Women with >/=2 malarial parasite infections tended to have lower z scores than u

140 Malaria parasite infections that are only detectable by molecula

141 of 20-50 mg/kg given after establishment of parasite infection, the compounds reduced parasitemia in

142 an important and preferential target cell of parasite infection, the injection of ROP16 has multiple

143 es, daily fluctuations in temperature affect parasite infection, the rate of parasite development, an

144 ate their functions in mosquito survival and parasite infection, these genes were knocked down by RNA

145 odified course of infection, controlling its parasite infection to levels below detection by thick bl

146 Gene induction by parasite infection was associated with trimethylation of

147 sults from previous studies at local scales; parasite infection was more influential in the West and

148 Blood parasite infection was significantly different between p

149 evalence, as well as the intensity of midgut parasite infections were found to be significantly highe

150 choriomeningitis virus and Leishmania major parasite infections, which were rescued with diet supple

151 t has a complex life cycle; however, asexual parasite infection within the blood stream is responsibl