ライフサイエンスコーパス: parasitic infection

1 ease globally in parallel with a decrease in parasitic infection.

2 omponents of the innate immune system during parasitic infection.

3 egral link in the chronic immune response to parasitic infection.

4 e important in Th2 cell-mediated immunity to parasitic infection.

5 tripartite model for nematode parasitism and parasitic infection.

6 beta induction and other markers of viral or parasitic infection.

7 the CD8(+) T cells during both a viral and a parasitic infection.

8 -49 receptor-expressing NK cell subsets in a parasitic infection.

9 -helper cells may be modulated by concurrent parasitic infection.

10 arthenogenetic Daphnia magna with respect to parasitic infection.

11 diated immune responses for an intracellular parasitic infection.

12 facilitate recognition and treatment of this parasitic infection.

13 tic doses to animals to selectively arrest a parasitic infection.

14 tion of the IgE response to allergens and to parasitic infection.

15 kk-1) upon allergen challenge or non-healing parasitic infection.

16 allergic inflammation and protection against parasitic infection.

17 the liver that can determine the outcome of parasitic infection.

18 igher background rate of eosinophilia due to parasitic infection.

19 expression was up-regulated in response to a parasitic infection.

20 to humans and an important blood-borne human parasitic infection.

21 the drug discovery pipeline for this deadly parasitic infection.

22 uring Th2-mediated immune responses, such as parasitic infections.

23 llular infiltrate associated with asthma and parasitic infections.

24 ty-adjusted life year (DALY), is unsuited to parasitic infections.

25 reatment of cancer, heart disease, AIDS, and parasitic infections.

26 both human and veterinary medicine to treat parasitic infections.

27 reatment of cancer, heart disease, AIDS, and parasitic infections.

28 tokine production and protective immunity to parasitic infections.

29 in parasite epidemiology and the control of parasitic infections.

30 therapeutics for the treatment of cancer and parasitic infections.

31 plain many immune features characteristic of parasitic infections.

32 n allergic diseases and the host response to parasitic infections.

33 model for the study of human and veterinary parasitic infections.

34 novel drug targets and vaccine candidates in parasitic infections.

35 tion of eosinophils in allergic diseases and parasitic infections.

36 eripheral blood T cells during bacterial and parasitic infections.

37 ment of eosinophils in allergic diseases and parasitic infections.

38 e and may confer physiological protection in parasitic infections.

39 cancers associated with bacterial, viral, or parasitic infections.

40 an attractive drug target for the control of parasitic infections.

41 diated disorders such as allergy, atopy, and parasitic infections.

42 in the initial suppression of bacterial and parasitic infections.

43 kers, is a common occurrence in HIV and some parasitic infections.

44 eactive protein levels and low prevalence of parasitic infections.

45 rfusion injury, enteroinvasive bacterial and parasitic infections.

46 s a possible source for symptom disparity in parasitic infections.

47 lt in flare-ups of many bacterial, viral and parasitic infections.

48 ron metabolism and the etiology of anemia in parasitic infections.

49 odulatory functions in allergic diseases and parasitic infections.

50 mmune responses during viral, bacterial, and parasitic infections.

51 n diverse diseases from cancer to eukaryotic parasitic infections.

52 rological diseases and disorders, as well as parasitic infections.

53 including aggressive cancer, arthritis, and parasitic infections.

54 region occurred for select diagnoses such as parasitic infections.

55 eir contribution to enhance host exposure to parasitic infections.

56 ify new treatments for asthma, allergies and parasitic infections.

57 with high peanut consumption and widespread parasitic infections.

58 skin reactivity), consumption patterns, and parasitic infections.

59 ses, IL-17E is essential for defense against parasitic infections.

60 reatment of cancer, heart disease, AIDs, and parasitic infections.

61 ulating intracellular bacterial, fungal, and parasitic infections.

62 herpes viruses, mycobacteria, and fungal and parasitic infections, a single arm of the immune respons

63 in the past year fatal viral, fungal, and/or parasitic infections accounted for 16% and 34% of the mo

64 ted for centuries as chronically disfiguring parasitic infections affecting millions of people across

65 ory states arising from viral, bacterial and parasitic infection, allergic and asthmatic reactions, a

66 The pathological process was recognised as a parasitic infection, although its features were differen

67 particularly Th2-related responses including parasitic infection and allergic inflammation.

68 complexity of the interrelationship between parasitic infection and allergy.

69 response able to clear bacterial, viral, and parasitic infection and an ability to efficiently store

70 ation of IFN-gamma-mediated immunity to this parasitic infection and identify a novel function for th

71 How host cells sense intestinal parasitic infection and initiate the appropriate immune

72 complexity of the murine host response to a parasitic infection and suggest that an outbred human po

73 rinary and human medicine for the control of parasitic infection and was the joint focus of the 2015

74 s that regulate both protective responses in parasitic infections and allergic airway inflammation.

75 The inverse association between parasitic infections and allergy at an ecological level

76 at high levels in the T cells of people with parasitic infections and asthma.

77 imarily destructive and are only relevant in parasitic infections and asthma.

78 icle, we review the current understanding of parasitic infections and basophils and discuss their put

79 Chronic inflammatory conditions such as parasitic infections and cancer trigger a metabolic repr

80 ological conditions, including bacterial and parasitic infections and cancer.

81 have been shown to function in recovery from parasitic infections and in antibody responses.

82 4-dependent, Th2-mediated responses in other parasitic infections and in models of allergic asthma, t

83 reports on the exact roles for IL-17 during parasitic infections and limited knowledge about cellula

84 Eosinophil recruitment is a hallmark of parasitic infections and many forms of cancer, and eosin

85 This approach may find useful application in parasitic infections and more broadly in any disease sta

86 potential, particularly for the treatment of parasitic infections and tuberculosis.

87 rnivore scavenging strategies under risks of parasitic infection, and (iii) conducting a literature-r

88 d into tissues during allergic inflammation, parasitic infection, and certain malignancies.

89 te that CD4+ TRM cells form in response to a parasitic infection, and indicate that optimal protectiv

90 ied a novel inhibitory function for IL-17 in parasitic infection, and our results demonstrate importa

91 52 days) elapsed before the first documented parasitic infection, and the median intervals from arriv

92 y recruited into the CNS in response to this parasitic infection, and they were able to target the in

93 g, 34 serum samples from patients with other parasitic infections, and 15 normal human serum samples.

94 , atopic dermatitis, allergic rhinitis, some parasitic infections, and cancer.

95 ector functions during bacterial, viral, and parasitic infections, and contributed to innate response

96 admission included malnutrition and wasting, parasitic infections, and haematological disorders in th

97 eptors have not been investigated to date in parasitic infections, and little is known about the role

98 loped because they are already used to treat parasitic infections, and there is interest in their use

99 ly credible gastrointestinal illness [HCGI], parasitic infections, anemia, growth).

100 tion of drugs against bacterial, fungal, and parasitic infections, antineoplastic agents against canc

101 The pathological processes resulting from parasitic infection are known to have important impacts

102 ch anti-viral innate immune responses affect parasitic infection are largely unknown.

103 Helminth parasitic infections are a major global health and socia

104 Chronic parasitic infections are a major risk factor for cancer

105 Parasitic infections are a potent stimulus for the gener

106 The majority of parasitic infections are caused by Giardia duodenalis, E

107 capable of preventing viral, bacterial, and parasitic infections are currently under development.

108 Moreover, parasitic infections are important causes of anemia in t

109 better management of malaria and intestinal parasitic infections are likely to reduce the incidence

110 d optimal host defence against bacterial and parasitic infections are mediated by mast cells.

111 It most commonly occurs in parasitic infection as focal eosinophilic myositis but c

112 s review focuses on studies that use enteral parasitic infections as a tool to investigate the functi

113 ignature appear during viral, bacterial, and parasitic infections, but also arise during humoral auto

114 number of enteric infections, including many parasitic infections, but also likely provide protection

115 Hosts are likely to respond to parasitic infections by a combination of resistance (exp

116 Hydatid disease is a parasitic infection caused by Echinococcus granulosus an

117 Cysticercosis is a parasitic infection caused by the larval stages of the p

118 Schistosomiasis or snail fever is an endemic parasitic infection caused by various trematodes of the

119 nological rationale underpinning therapeutic parasitic infection, describe recently initiated trials,

120 atopoietic growth factors or emerging during parasitic infections, display tolerogenic properties.

121 effect of stunting, diarrhoeal disease, and parasitic infections during infancy on cognitive functio

122 incidence and natural history of intestinal parasitic infections during the PCVs' >2-year overseas s

123 mass drug administration (MDA) against these parasitic infections emphasise treatment of school-aged

124 The current studies suggest acute parasitic infections exacerbate allergic symptoms, where

125 Parasitic infections frequently result in highly polariz

126 eosinophils was decreased in the presence of parasitic infection (GM = 0.9 vs. 5.7%, p = 0.02).

127 In response to parasitic infection, hosts may evolve defences that redu

128 ective innate immunity against bacterial and parasitic infections; however, its role in host immunity

129 adpoles leads to increased susceptibility to parasitic infection in adult frogs, in the absence of su

130 Malaria is the most important parasitic infection in people, accounting for more than

131 rovide possible explanations for the role of parasitic infection in susceptibility and resistance to

132 to treat Chagas disease, the most important parasitic infection in the Americas.

133 ted to the development of host resistance to parasitic infection in the resistant breed.

134 Giardiasis, the most common enteric parasitic infection in the United States, causes an esti

135 -1+ monocytes in the control of disseminated parasitic infection in vivo.

136 n response to Toxoplasma gondii profilin and parasitic infection in vivo.

137 This is the first evidence of a parasitic infection in which down-regulation of CD8+ T c

138 across age, sex, anthropometric indexes, and parasitic infections in a representative sample of 3595

139 chronic infection is one of the most common parasitic infections in humans and can be reactivated to

140 Toxoplasma gondii is one of the most common parasitic infections in humans.

141 T cell differentiation and susceptibility to parasitic infections in murine models, we compared T cel

142 tervention addressing 2 of the most virulent parasitic infections in pregnant women in sub-Saharan Af

143 osomiasis are the world's two most important parasitic infections in terms of distribution, morbidity

144 risk of common bacterial, viral, fungal, and parasitic infections in the general population of indivi

145 faces and serves to protect against helminth parasitic infections in the intestinal tract.

146 tudies toxoplasmosis, one of the most common parasitic infections in the world, caused by Toxoplasma

147 It is one of the most widespread human parasitic infections in tropical and subtropical regions

148 at can protect against bacterial, viral, and parasitic infections, including lethal influenza and mal

149 th a broad-spectrum activity against various parasitic infections, including malaria, toxoplasmosis a

150 vaccines are available for human use for any parasitic infections, including the helminthic disease s

151 Many parasitic infections, including those of humans, are cau

152 o Leishmania major (Lm) or Toxoplasma gondii parasitic infections, indicating that exogenously delive

153 The direct benefit to females of avoiding parasitic infection is proposed to lead to the linkage b

154 uation of patients with suspected intestinal parasitic infection is proposed.

155 Parasitic infection is uncommon in the United States, bu

156 Although parasitic infection is usually detrimental, it can be be

157 information on DC-NK cell interaction during parasitic infections is available.

158 Serologic testing for chronic parasitic infections is often necessary.

159 An important feature of many chronic parasitic infections is the ability of the invading path

160 s and those chronic and debilitating (mostly parasitic) infections known as neglected tropical diseas

161 d neoplastic (chronic lymphocytic leukemia), parasitic infection (Leishmania major), and infectious d

162 At entry persons with parasitic infections may be asymptomatic, and stool exam

163 This review discusses T cell exhaustion in parasitic infection, mechanisms of development, and a po

164 etion in autoimmune, transplant disease, and parasitic infection models.

165 r regulation of immune responses, such as in parasitic infection, necessitate further characterizatio

166 Toxoplasma gondii, the most common parasitic infection of human brain and eye, persists acr

167 Repeated parasitic infection of TCR beta-/- mice, but not of T ce

168 Neurocysticercosis, a parasitic infection of the human central nervous system

169 or immune intervention in ocular allergy and parasitic infections of the eye.

170 tion problems, particularly the influence of parasitic infections on child health and nutrition.

171 munity that can either be protective against parasitic infection or cause tissue damage in allergy an

172 patients affected by Th2 pathologies such as parasitic infections or atopic dermatitis often suffer f

173 ffector cells useful in the immunotherapy of parasitic infections or neoplasms.

174 A systemic bacterial, viral, or parasitic infection other than malaria or typhoid fever

175 esults are negative, O&P can be performed if parasitic infections other than G. lamblia, E. histolyti

176 ietary factors such as chronic inflammation, parasitic infections, overweight, and genetic hemoglobin

177 l established risk factors for CC, including parasitic infections, primary sclerosing cholangitis, bi

178 sed by granulomatous disorders, DNA viruses, parasitic infections, pulmonary amyloidosis, vascular ca

179 ationship was mostly explained by fungal and parasitic infections rather than by viral and bacterial

180 Surviving a parasitic infection requires the generation of a control

181 disruptions in mucosal interfaces induced by parasitic infection, respectively.

182 vitamin D insufficiency, and eradication of parasitic infections) risk factors.

183 wever, the role of NK cells in extracellular parasitic infections such as filarial infections is not

184 Parasitic infections such as filariasis, schistosomiasis

185 The incidence of parasitic infections such as malaria, leishmaniasis, and

186 otic enzyme and an attractive drug target in parasitic infections such as malaria.

187 Schistosomiasis is a parasitic infection that is widespread in sub-Saharan Af

188 l burden of high E4 prevalence combined with parasitic infections that can also reduce cognitive perf

189 e habitat for intermediate hosts of tropical parasitic infections that cause disease and suffering.

190 e been proposed by which hosts can withstand parasitic infections, the role of multiple infections an

191 rheal episodes, the continued acquisition of parasitic infections throughout the PCVs' >2-year stay i

192 rol patients who had other parasitic and non-parasitic infections to a weak cation exchange chip, and

193 major human diseases ranging from viral and parasitic infections to autoimmunity and cancer.

194 Murine neurocysticercosis is a parasitic infection transmitted through the direct inges

195 In several parasitic infections, Treg maintain equilibrium to ensur

196 lations with a high prevalence of intestinal parasitic infections, two independently collected stool

197 d mortality associated with these intestinal parasitic infections warrant the development of rapid an

198 ed modelling showed that only positivity for parasitic infections was a significant predictor of redu

199 Susceptibility to having 1 or more parasitic infections was significantly associated with p

200 dazole, an oral drug currently used to treat parasitic infections, was used as a positive control.

201 y intake, nutritional status, and intestinal parasitic infection were collected.

202 n thus offers an example of an intracellular parasitic infection where CD8(+) T-cell immunity can be

203 s suggests an innate immune response against parasitic infections, whereas the other gene expression

204 r without Schistosoma mansoni coinfection, a parasitic infection with T helper (Th) 2 immune bias.

205 In parasitic infection with Trichinella spiralis, the immun

206 The physiological effects of such events on parasitic infections within endotherms are poorly studie