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1 utrient phospholipid transfer in the malaria-parasitized erythrocyte.
2 of different membrane components within the parasitized erythrocyte.
3 lar beds are susceptible to sequestration of parasitized erythrocytes.
4 al cytoadhesion with sequestration of mature parasitized erythrocytes.
5 ppropriate membrane localization of KAHRP in parasitized erythrocytes.
6 ic digestion of streptolysin O-permeabilized parasitized erythrocytes.
7 therosclerosis, and sequestration of malaria-parasitized erythrocytes.
8 als were challenged with 10(4) P. falciparum-parasitized erythrocytes.
9 traperitoneally with various passages of WA1-parasitized erythrocytes.
10 tes but not on either uninfected or B. bovis-parasitized erythrocytes.
11 pholipids, and Plasmodium falciparum malaria parasitized erythrocytes.
12 ding overall was the marked sequestration of parasitized erythrocytes across most organs in patients
13 and four other genes, results in the loss of parasitized erythrocytes adhering to chondroitin 4-sulfa
14 site killing, we studied the distribution of parasitized erythrocytes and bacteria in the spleens of
15 identified as one of the host receptors for parasitized erythrocytes and has been implicated as bein
16 RNA in bovine macrophages by either B. bovis-parasitized erythrocytes and IFN-gamma or CM was also ob
19 advantageous for large-scale preparation of parasitized erythrocytes (and potentially immunogens the
20 how impaired rosetting interactions with non-parasitized erythrocytes, and reduced agglutination in t
21 levels and parasite densities suggests that parasitized erythrocytes are one possible source of exce
22 of visualized microvascular sequestration of parasitized erythrocytes at both time points (rs=0.55; p
23 adjuvants produced antibodies that recognize parasitized erythrocytes by IFA and native PyP140/RON4 b
24 ytes, because it is by these properties that parasitized erythrocytes can sequester in postcapillary
25 e interplay among capillary sequestration of parasitized erythrocytes, deregulated inflammatory respo
26 roteins and their ability to directly attack parasitized erythrocytes further explain their anti-Plas
28 ne responses, margination of leukocytes, and parasitized erythrocytes in cerebral vessels leading to
29 hological assessment of the sequestration of parasitized erythrocytes in multiple organs obtained dur
30 placentas with P. vivax monoinfection showed parasitized erythrocytes in the intervillous space but n
32 parum malaria--results from sequestration of parasitized erythrocytes in the microcirculation, not fr
33 aria results primarily from sequestration of parasitized erythrocytes in the microvasculature rather
34 hesis, obstruction of blood flow by adherent parasitized erythrocytes is the cause of tissue hypoxia
36 y replication in blood, surface molecules on parasitized erythrocytes, or merozoites) activate platel
38 ucing activity associated with P. falciparum-parasitized erythrocytes (PE) appear to be lost after ce
42 P1), present on the surface of P. falciparum-parasitized erythrocytes (PE), plays a central role in n
46 membrane protein-1-mediated sequestration of parasitized erythrocytes plays a central role in malaria
47 m falciparum merozoites and cytoadherence of parasitized erythrocytes (PRBC) to endothelial cells.
48 Adherence of mature Plasmodium falciparum parasitized erythrocytes (PRBCs) to microvascular endoth
51 Increased levels of eCAMs result in further parasitized-erythrocyte sequestration and marked local i
52 The common feature of IgM binding to the parasitized erythrocyte surface among otherwise genetica
53 rocyte membrane protein-1 is exported to the parasitized erythrocyte surface have recently been eluci
54 ia by reducing PfEMP-1-mediated adherence of parasitized erythrocytes, thereby mitigating the effects
55 d (ii). CD41 is not an adhesion molecule for parasitized erythrocytes, these findings support the hyp
57 a imply that no change occurs in the flow of parasitized erythrocytes through the spleen during the t
58 Adhesion of mature Plasmodium falciparum parasitized erythrocytes to microvascular endothelial ce
60 ferences between the two lines in binding of parasitized erythrocytes to the variant ICAM-1 proteins.
61 in the mammalian host, from sequestration of parasitized erythrocytes, to antigenic variation and hos
64 ls of the mechanisms behind sequestration of parasitized erythrocytes would "become increasingly more
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