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1 s a genetic predisposition to many childhood parasomnias.
2 ructive sleep apnea syndrome (OSAS) (0.06%), parasomnia (0.7%), restless leg syndrome (0.9%), or psyc
4 tic challenges between nonrapid eye movement parasomnia and nocturnal frontal lobe epilepsy remain.
5 te the strong familial aggregation for the 2 parasomnias and lend support to the notion that sleepwal
6 ly, symptoms of some sleep disorders such as parasomnias and narcolepsy can be confused with those of
7 bility of a common pathogenic background for parasomnias and nocturnal seizures that is summarized in
10 ehavior disorder is distinguished from other parasomnias by clinical features and the demonstration o
17 t (REM) sleep behaviour disorder, which is a parasomnia manifested by vivid dreams associated with dr
18 nto the functional mechanisms of this common parasomnia: sleepwalkers exhibited improved movement aut
21 azepine receptor agonist hypnotics can cause parasomnias, which in rare cases may lead to suicidal id
22 antibodies identify a unique non-REM and REM parasomnia with sleep breathing dysfunction and patholog
23 ildhood sleepwalking and sleep terrors are 2 parasomnias with a risk of serious injury for which fami
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