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1 rogenital tract, pancreas, liver, brain, and parathyroid gland.
2 thymus hypoplasia and mislocalization of the parathyroid gland.
3 ur parathyroid glands and resecting enlarged parathyroid glands.
4 best tool available for imaging of abnormal parathyroid glands.
5 d hypercalcemia and caused by hypersecreting parathyroid glands.
6 ion of calcium homeostasis in the absence of parathyroid glands.
7 factor whose expression is restricted to the parathyroid glands.
8 cule exists in zebrafish, species which lack parathyroid glands.
9 ction that is expressed in the pituitary and parathyroid glands.
10 thyroid glands in one patient, and 51 normal parathyroid glands.
11 he likelihood of additional hyperfunctioning parathyroid glands.
12 ized by precise preoperative localization of parathyroid glands.
13 , the coreceptor for FGF23 in the kidney and parathyroid glands.
14 retion of parathyroid hormone (PTH) from the parathyroid glands.
15 continue to guide surgical resection of the parathyroid glands.
16 ng endodermal progenitors of both thymus and parathyroid glands.
17 CD44/CD24 population was 10.93% for enlarged parathyroid glands.
18 by varying defects of the heart, thymus, and parathyroid glands.
19 rathyroid imaging except in locating ectopic parathyroid glands.
20 ied 89% of the surgically confirmed diseased parathyroid glands.
21 metabolism and a potent hormone made by the parathyroid glands.
22 ide counts > background to localize abnormal parathyroid glands.
24 c-sestamibi scan demonstrated a hyperplastic parathyroid gland, a large anterior mediastinal mass and
26 o vitamin D2-treated animals with suppressed parathyroid gland activity produced marked elevation in
27 regulate overactivity and hyperplasia of the parathyroid gland after the onset of renal insufficiency
29 receptor proteins (CaRs) enable cells in the parathyroid gland and kidney thick ascending limb of Hen
31 ardiac outflow defects and hypoplasia of the parathyroid gland and thymus due to haploinsufficiency o
32 g CD44 antibody was performed on 27 abnormal parathyroid glands and 7 normal parathyroid gland biopsi
33 ent mice in association with ablation of the parathyroid glands and correction of the severe hyperpar
35 Here we show that Gcm2-deficient mice lack parathyroid glands and exhibit a biological hypoparathyr
36 the white matter of uremic dogs with intact parathyroid glands and in normal dogs and TPTX uremic do
37 binding protein-beta, in human hyperplastic parathyroid glands and in the human epidermoid carcinoma
38 roidism is due to enlargement of one or more parathyroid glands and is most often treated by surgical
39 he likelihood of additional hyperfunctioning parathyroid glands and let the surgeon determine whether
40 eleton, we used a genetic approach to ablate parathyroid glands and remove the confounding effects of
41 the neck with the intent of visualizing four parathyroid glands and resecting enlarged parathyroid gl
43 ry conservation of abundant miRNAs in normal parathyroid glands and the regulation of these miRNAs in
45 n protein predominantly expressed in kidney, parathyroid glands, and choroids plexus of the brain.
46 receptor were present in normal and diseased parathyroid glands, and if so, whether they had any func
47 rior lobes of the pituitary, the thyroid and parathyroid glands, and the adrenal medulla within the f
49 al pharyngeal pouch derivatives (the thymus, parathyroid glands, and thyroid gland), heart, and gut.
53 ccompanied by an increase in the size of the parathyroid gland as well as an increase in PTH mRNA lev
54 the effects of extracellular calcium in the parathyroid gland as well as other tissues has been iden
56 n vertebrate-specific tissues, placenta, and parathyroid glands, begging questions on the evolutionar
58 tivating the calcium-sensing receptor in the parathyroid glands, but clinical experience with them is
60 ures, including hypoplasia of the thymus and parathyroid glands, cardiac outflow tract abnormalities,
61 m (PHPT), discovering a minimally "enlarged" parathyroid gland creates a dilemma for the surgeon rega
62 nd laser confocal microscopy of normal human parathyroid gland demonstrated expression of parafibromi
63 tions of the aortic arch, heart, thymus, and parathyroid glands described as DiGeorge syndrome (DGS).
64 showed that this technique is able to detect parathyroid gland devascularization before it is visuall
66 not reverse the arrest in tooth, thymus, and parathyroid gland development, suggesting that the relat
70 scularized (n = 32) and compromised (n = 27) parathyroid glands during thyroid surgery with an accura
71 yroidism is due to increased activity of the parathyroid glands, either from an intrinsic abnormal ch
72 of function of the GCMB gene impairs normal parathyroid gland embryology and is responsible for isol
73 cterized primarily by multiple tumors in the parathyroid glands, endocrine pancreas, and anterior pit
76 PT may result from reduced expression of the parathyroid gland extracellular Ca(2+)-sensing receptor
77 ne-associated antigen of 120-140 kD in human parathyroid gland extracts using immunoblot analysis.
80 obtained for FACS analysis from 25 enlarged parathyroid glands from 20 patients, 17 with primary HPT
83 ivation analyses have demonstrated that most parathyroid glands from patients with uremic refractory
87 n bone mineralization, vitamin D metabolism, parathyroid gland function, and renal phosphate handling
89 , intravenous calcitriol appears to decrease parathyroid gland functional mass, as reflected by decre
91 Radioguided resection of hyperfunctioning parathyroid glands has been shown to be technically feas
92 l phenotype, which were manifested by larger parathyroid glands, higher serum parathyroid hormone lev
94 lume group were more likely to have a missed parathyroid gland in a normal anatomic location (89% vs.
95 se, Met-PET/CT identified 2 hyperfunctioning parathyroid glands in 1 patient, 1 gland in 3 individual
97 ative failure can be due to hyperfunctioning parathyroid glands in ectopic locations, less experience
100 adenomas in one patient; and 3 hyperplastic parathyroid glands in one patient, and 51 normal parathy
102 th SPECT/CT for the localization of abnormal parathyroid glands in patients with primary hyperparathy
103 nt this complication include preservation of parathyroid glands in situ and autotransplantation of pa
105 e thyroidectomies attempting to preserve the parathyroid glands in situ with an intact vascular pedic
107 ion computed tomography to localize enlarged parathyroid glands in three dimensions, limited explorat
109 t the secretion of PTH by chief cells in the parathyroid gland is regulated by extracellular ionized
110 ing receptor (CaSR), first identified in the parathyroid gland, is expressed in several tissues and c
111 autonomous growth in two, three, or all four parathyroid glands, is a distinct molecular entity and d
113 wed specific immunoreactivity in adrenal and parathyroid glands, kidney, heart, and skeletal muscle.
114 emic level causes promiscuous effects in the parathyroid glands, kidneys, and other tissues, and the
115 rain regions, coronary arteries, thyroid and parathyroid glands, large intestine, colon, bladder, tes
116 n the general population, blacks have higher parathyroid gland mass and circulating parathyroid hormo
117 s known to be required for normal thymus and parathyroid gland morphogenesis, whereas Pax1, Hoxa3, Ey
118 show that loss of Men1 gene function in the parathyroid glands of mice results in histological chang
119 on, with rates >fourfold higher than that in parathyroid glands of wild-type littermates (P<0.0001).
120 arathyroidism can be cured by removal of the parathyroid gland or glands but identification of patien
122 e brain, musculoskeletal system, thyroid and parathyroid glands, pancreas, kidney, lung, and breast;
123 Despite the sustained stimulation to the parathyroid gland, parathyroid cells did not undergo hyp
124 cts of dietary P on serum PTH, PTH mRNA, and parathyroid gland (PTG) hyperplasia in uremic rats.
125 iple Endocrine Neoplasia type 2A should have parathyroid glands resected at the time of thyroidectomy
127 id glands in situ and autotransplantation of parathyroid glands resected or devascularized during thy
128 d by hypoplasia or atresia of the thymus and parathyroid glands resulting in T cell-mediated deficien
129 racellular calcium ions (Ca2+o), cloned from parathyroid gland, serves a critical function in Ca2+o h
131 hyroidism and enlargement of only one or two parathyroid glands that the resection be limited to thes
132 , but does not affect the sensitivity of the parathyroid gland to changes in iCa, as set point and no
133 ]o) in the physiological range, allowing the parathyroid gland to regulate serum [Ca2+]o; however, th
134 within the physiological range, allowing the parathyroid gland to regulate serum Ca(o); however, the
135 The accuracy for localizing a pathologic parathyroid gland to the correct side of the neck was 59
138 upled receptor for external Ca2+ cloned from parathyroid gland, was shown to be expressed in PF cells
139 cytometric analysis of resected adenomatous parathyroid glands, we have isolated and characterized c
140 am, consisting of the combination of WIN and parathyroid gland weight, accurately predicted the likel
141 xcision, radionuclide counts of each ex vivo parathyroid gland were determined and expressed as a per
145 dental disruption of blood supply to healthy parathyroid glands, which are responsible for regulating
146 ts from the calcium-sensing receptors on the parathyroid glands, which detect changes in calcium conc
147 arathyroid adenomas are benign tumors in the parathyroid glands, whose pathogenesis is largely unknow
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