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1 ts increase is associated with a decrease in parathyroid hormone.
2 (receptor activator of NFkappaB ligand) and parathyroid hormone.
3 There was no effect modification by parathyroid hormone.
4 nd action of fibroblast growth factor 23 and parathyroid hormone.
5 blood pressure or arterial pressure and with parathyroid hormone.
6 ndoxyl sulfate, homocysteine, uric acid, and parathyroid hormone.
7 cluding carboxymethyllysine, cystatin C, and parathyroid hormone.
8 or inappropriately normal concentrations of parathyroid hormone.
9 er characterized by autonomous production of parathyroid hormone.
12 tolerability of once-daily recombinant human parathyroid hormone 1-84 (rhPTH[1-84]) in adults with hy
14 f 39 residues (TIP39), via its receptor, the parathyroid hormone 2 receptor (PTH2R), modulates fear m
15 Small interfering RNA-mediated silencing of parathyroid hormone 2 receptor (PTH2R), the receptor for
18 hosphorus, alkaline phosphatase, creatinine, parathyroid hormone, 25 hydroxy vitamin D3 and 1, 25 dih
19 racteristics, dietary intakes, fasting serum parathyroid hormone, 25-hydroxyvitamin D [25(OH)D], and
21 ge mean+/-SD single-pass renal extraction of parathyroid hormone (44.2%+/-10.3%) that exceeded the ex
22 hyperparathyroidism (median level of intact parathyroid hormone, 693 pg per milliliter [10th to 90th
23 0.09 mmol/L, 95% CI 0.02-0.17, p=0.009), and parathyroid hormone (+7.32 pg/mL, 3.42-11.23, p<0.0001).
27 (CV) system, including increasing levels of parathyroid hormone, activating the renin-angiotensin-al
28 signature of the arrestin pathway-selective parathyroid hormone analog [d-Trp(12), Tyr(34)]bovine PT
29 The cAMP response induced by M-PTH(1-14), a parathyroid hormone analog, terminated quickly in HEK293
33 in chronic kidney disease and the effects of parathyroid hormone and FGF23 on these associations.
34 ant role for vitamin D and related hormones, parathyroid hormone and fibroblast growth factor 23 (FGF
35 splant patients significantly reduced intact parathyroid hormone and increased fibroblast growth fact
37 -dihydroxyvitamin D (1,25(OH)2D), FGF23, and parathyroid hormone and performed quantitative echocardi
38 iates the endocrine and paracrine effects of parathyroid hormone and related peptides through the act
40 alphas also inhibited internalization of the parathyroid hormone and type 2 vasopressin receptors.
41 rations <20 ng/mL who did not also have high parathyroid hormone and women with high parathyroid horm
42 luding C-terminal fragment of FGF-23, intact parathyroid hormone, and 1,25(OH)(2)D(3), were measured
44 ulated by numerous factors, including BMP-2, parathyroid hormone, and 1alpha,25-dihydroxyvitamin D3 (
45 ium, phosphorus, 25-hydroxyvitamin D, intact parathyroid hormone, and 24,25-dihydroxyvitamin D did no
47 measured serum levels of phosphate, calcium, parathyroid hormone, and 25-hydroxyvitamin D in 1,938 Ca
49 n D, 1,25-dihydroxyvitamin D [1,25(OH)(2)D], parathyroid hormone, and calcium intake were evaluated.
50 ong hormones, including 1,25(OH)2D3 (1,25D), parathyroid hormone, and fibroblast growth factor 23 (FG
51 including serum levels of 25(OH) vitamin D, parathyroid hormone, and fibroblast growth factor 23.
52 eomalacia, hypophosphatemia, increased serum parathyroid hormone, and increased levels of the phospha
53 ar, serum osteocalcin, total calcium, intact parathyroid hormone, and increased serum C telopeptide.
55 s with dissociable agonists for thyrotropin, parathyroid hormone, and sphingosine-1-phosphate were fo
57 ffect on basal bone resorption, it inhibited parathyroid hormone- and ovariectomy-induced OC activati
58 er levels of serum calcium, phosphorous, and parathyroid hormone; and nutritional vitamin D, cinacalc
59 terminal propeptide (PINP), osteocalcin, and parathyroid hormone as well as a transient decrease in t
60 oxyvitamin D, vitamin D-binding protein, and parathyroid hormone as well as bone mineral density (BMD
61 substantial single-pass renal extraction of parathyroid hormone at a rate that exceeds glomerular fi
62 hly expressed in osteocytes, is regulated by parathyroid hormone both in vitro and in vivo, and prote
63 FGF23, despite having high concentrations of parathyroid hormone, but administration of exogenous 1,2
64 evels, serum calcium homeostasis biomarkers (parathyroid hormone, calcium, and 25-hydroxyvitamin D),
65 relationships), yet within each quintile of parathyroid hormone concentration, blacks had significan
67 whether lower 25-hydroxyvitamin D and higher parathyroid hormone concentrations are associated with i
71 intervals (CIs) for 25-hydroxyvitamin D and parathyroid hormone concentrations with incident hyperte
72 perparathyroidism, and might be predicted by parathyroid hormone concentrations, even in individuals
76 operative success, defined by intraoperative parathyroid hormone criteria, and complication rates wer
77 mg/dL (95% confidence interval, 0.28-0.64), parathyroid hormone decreased by 102 pg/mL (95% confiden
81 th the use of dual-stable isotopes, 25(OH)D, parathyroid hormone, estradiol, calcitriol, and urinary
82 a common endocrine disease characterized by parathyroid hormone excess and hypercalcemia and caused
83 etermined the single-pass renal clearance of parathyroid hormone, fibroblast growth factor 23, vitami
84 /- 10.3 ng/mL; P < 0.0001) and a decrease in parathyroid hormone (from 21.4 +/- 10.4 to 12.9 +/- 7.1
85 splantation, persistent hyperparathyroidism (parathyroid hormone > 130 ng/L) and bone turnover marker
87 Compared with parathyroid hormone <33 pg/ml, parathyroid hormone >/=65 pg/ml was associated with a si
88 th low urine output, low serum albumin, high parathyroid hormone, high ferritin, and the use of bioin
91 at 1000 IU/d increases 25(OH)D and decreases parathyroid hormone in children with average vitamin D i
94 ctions in secondary HPT, including decreased parathyroid hormone, increased vitamin D, increased seru
95 der these circumstances both agents enhanced parathyroid hormone-induced osteoblast differentiation a
98 ients with hypercalcemia and elevated intact parathyroid hormone (iPTH) concentration were eligible i
101 omes measures included serum calcium, intact parathyroid hormone (iPTH), and thyroid stimulating horm
102 ium concentration is within normal range but parathyroid hormone is elevated in the absence of any ob
103 aemia caused by autonomous overproduction of parathyroid hormone, is common, especially in postmenopa
104 x, and creatinine clearance, but with intact parathyroid hormone less than 100 pg/mL, were included a
105 ltiply by 0.25; P = .15), preoperative serum parathyroid hormone level (mean [SD], 114.5 [56.8] vs 13
106 rogression were age, baseline total or whole parathyroid hormone level greater than nine times the no
107 Regarding modifiable factors, higher average parathyroid hormone level was associated with greater ri
108 walking, and higher average log-transformed parathyroid hormone level were independently associated
109 ent risk was attenuated after adjustment for parathyroid hormone level, suggesting that parathyroid h
113 ncreased sessional dialysis dose and lowered parathyroid hormone levels (from 51 +/- 67 to 24 +/- 37
115 lerated) or nonparicalcitol therapy on serum parathyroid hormone levels (primary outcome), mineral me
116 itamin D receptor activator, decreased serum parathyroid hormone levels and proteinuria in patients w
117 6-month paricalcitol supplementation reduced parathyroid hormone levels and proteinuria, attenuated b
120 s induced by aldosteronism in which elevated parathyroid hormone levels raise the risk of adverse car
121 baseline, median (interquartile range) serum parathyroid hormone levels significantly declined on par
126 mice had significantly lower serum FGF23 and parathyroid hormone levels, and higher renal 1-alpha-hyd
127 irmed by exploratory surgery, intraoperative parathyroid hormone levels, and pathologic features.
128 by increased serum 1,25-(OH)2-vitamin D3 and parathyroid hormone levels, decreased serum calcium leve
129 response or a hormonal response to decreased parathyroid hormone levels, we subjected osteocytes to a
134 translocations caused downregulation of the parathyroid hormone-like hormone (PTHLH) gene by disrupt
136 5 (Gdf5), the transcription factor Erg, and parathyroid hormone-like hormone (Pthlh), and selection
137 orylation and TGF-beta-induced expression of parathyroid hormone-like hormone (PTHLH), leading to ost
138 stric mucosal neuroendocrine cells including parathyroid hormone-like hormone in histamine-secreting
139 , and Ihh target genes Patched 1 (Ptch1) and parathyroid hormone-like peptide (Pthlh) were down-regul
144 asive parathyroidectomy using intraoperative parathyroid hormone monitoring remains the standard appr
145 y invasive parathyroidectomy, intraoperative parathyroid hormone monitoring via a reliable protocol i
149 ccurred in serum calcium, phosphorus, intact parathyroid hormone, or C-reactive protein levels, cinac
150 not be explained by hypocalcemia, changes in parathyroid hormone, or fibroblast growth factor 23.
151 , levels of calcium, phosphorus, calcitriol, parathyroid hormone, or fibroblast growth factor-23 were
152 not explained by urine phosphate excretion, parathyroid hormone, or fibroblast growth factor-23.
153 e-colony stimulating factor, growth hormone, parathyroid hormone, or stem cell factor each stimulates
154 citol treatment significantly reduced intact parathyroid hormone (P<0.001) and alkaline phosphatase (
155 etabolic pathway (e.g., 25-hydroxyvitamin D, parathyroid hormone, phosphorus) had little impact.
156 nine, free thyroxine, free triiodothyronine, parathyroid hormone, prolactin, N-terminal pro-brain nat
157 um metabolism was defined as elevated intact parathyroid hormone (PTH) (>62 pg/mL) accompanied by a v
160 cium through processes that are regulated by parathyroid hormone (PTH) and 1alpha,25-dihydroxyvitamin
164 echanisms through which Pi intake stimulates parathyroid hormone (PTH) and fibroblast growth factor-2
165 a was associated with low FGF23 despite high parathyroid hormone (PTH) and high calcitriol levels.
169 Previously, we have shown that intermittent parathyroid hormone (PTH) bone anabolic therapy involves
170 3 patients receiving hemodialysis with serum parathyroid hormone (PTH) concentrations higher than 500
171 ravenous calcimimetic etelcalcetide on serum parathyroid hormone (PTH) concentrations in patients rec
172 ciations of 25-hydroxyvitamin D (25-OHD) and parathyroid hormone (PTH) concentrations separately and
175 ], 1,25-dihydroxyvitamin D [1,25(OH)2D], and parathyroid hormone (PTH) in maternal circulation and co
176 he PT-Dicer(-/-) mice did not increase serum parathyroid hormone (PTH) in response to acute hypocalce
185 yroidism is characterized by increased serum parathyroid hormone (PTH) level and parathyroid cell pro
186 ce and impact of HPT, defined as an elevated parathyroid hormone (PTH) level, after renal transplanta
189 ibited reduced serum inorganic phosphate and parathyroid hormone (PTH) levels and decreased bone form
190 nvestigated the association between baseline parathyroid hormone (PTH) levels and major cardiovascula
191 tionship between endothelial dysfunction and parathyroid hormone (PTH) levels in RTRs has been demons
192 pocalcemia, measurement of serum calcium and parathyroid hormone (PTH) levels, and algorithms for sup
193 l quantitative computed tomography (HRpQCT), parathyroid hormone (PTH) levels, and bone turnover mark
194 us metabolism, including serum phosphate and parathyroid hormone (PTH) levels, did not consistently a
195 ned assessment of 25-OH-D with its regulator parathyroid hormone (PTH) may be required for optimal ev
196 em cells (HSCs) is induced by treatment with parathyroid hormone (PTH) or activation of the PTH recep
199 isciplinary analytical characterization of a parathyroid hormone (PTH) peptide active pharmaceutical
202 osteoblastic cell-specific activation of the parathyroid hormone (PTH) receptor attenuates BCR-ABL1 o
203 alloproteinase-13 (MMP-13) transcription and parathyroid hormone (PTH) regulates HDAC4 to control MMP
204 Ib(delNASm)), a disorder of proximal tubular parathyroid hormone (PTH) resistance associated with los
206 aR) modulates renal calcium reabsorption and parathyroid hormone (PTH) secretion and is involved in t
207 ition of the abundant let-7 family increased parathyroid hormone (PTH) secretion in normal and uremic
208 , whether they had any functional effects on parathyroid hormone (PTH) secretion in parathyroid neopl
214 absence of ascorbate and were stimulated by parathyroid hormone (PTH) to undergo rapid osteoclast fo
218 was the primary outcome, and serum calcium, parathyroid hormone (PTH), 1,25-dihydroxyvitamin D [1,25
219 ogenesis, confluent BMSCs were cultured with parathyroid hormone (PTH), 1,25-dihydroxyvitamin D3 (1,2
222 on tubular reabsorption of phosphate (TRP), parathyroid hormone (PTH), bone alkaline phosphatase (BA
223 xamined the prospective associations between parathyroid hormone (PTH), circulating 25-hydroxyvitamin
224 parathyroid chief and oxyphil cells produce parathyroid hormone (PTH), express the calcium-sensing r
225 ercalcemic patients underwent measurement of parathyroid hormone (PTH), had documentation of hypercal
227 transferrin saturation (TSAT) concentration, parathyroid hormone (PTH), IV vitamin D dose, cinacalcet
228 ata defined by their pretransplant levels of parathyroid hormone (PTH), low PTH (>65 to </=300 pg/mL;
230 enes involved in signaling through Wnt, FSH, parathyroid hormone (PTH), oxytocin, and calcineurin/nuc
231 vels of fibroblast growth factor-23 (FGF23), parathyroid hormone (PTH), phosphate, and 25-hydroxyvita
232 nd their determinants, including circulating parathyroid hormone (PTH), total alkaline phosphatase ac
233 over and pronounced osteoblast resistance to parathyroid hormone (PTH), which is indicated by decreas
234 itory effects of TCN on lipopolysaccharide-, parathyroid hormone (PTH)-, and prostaglandin E2 (PGE2)-
235 al to Cyp27b1 that mediates unique basal and parathyroid hormone (PTH)-, fibroblast growth factor 23
236 nique role of osteal macrophages in bone and parathyroid hormone (PTH)-dependent bone anabolism using
237 urinary calcium excretion in the context of parathyroid hormone (PTH)-dependent hypercalcemia, but t
238 In the kidney, these mutations interrupt parathyroid hormone (PTH)-responsive sequestration of th
239 m baseline in blood and urine markers of the parathyroid hormone (PTH)-vitamin D-fibroblast growth fa
243 a constitutively activating mutation of the parathyroid hormone (PTH)/PTH-related protein (PTHrP) re
244 e anabolism through direct activation of the parathyroid hormone (PTH)/PTH-related protein receptor (
245 ostasis and is tightly regulated through the parathyroid hormone (PTH)/PTHrP receptor (PTH1R) signali
258 sensitive radioimmunoassay to measure serum parathyroid hormone-related peptide (PTHrP) and a mouse
261 pression in chondrocytes strictly depends on parathyroid hormone-related peptide (PTHrP) signaling pa
262 r translocation of Gli2 and transcription of parathyroid hormone-related peptide (PTHrP), a key regul
264 ATDC5 chondrogenic cells is downregulated by parathyroid hormone-related peptide through transcriptio
265 targets (plasminogen activator inhibitor-1, parathyroid hormone-related peptide, connective tissue g
266 ix protein 1, a direct targeting molecule of parathyroid hormone-related peptide, negatively regulate
268 K14-PTHrP transgenic mice [which overexpress parathyroid hormone-related protein (PTHrP) in their dev
271 Here we addressed the hypothesis that the parathyroid hormone-related protein (PTHrP) potentiates
275 egulates endochondral ossification in both a parathyroid hormone-related protein (PTHrP)-dependent an
276 ivation, increased Indian hedgehog (Ihh) and parathyroid hormone-related protein (Pthrp, also known a
277 as been made in determining the roles of the parathyroid hormone-related protein, Indian hedgehog, fi
278 or the quantification of a cancer biomarker (parathyroid hormone-related protein, PTHrP) in a real cl
280 cancer cachexia, we show that tumour-derived parathyroid-hormone-related protein (PTHrP) has an impor
282 een the groups, patients who did not mount a parathyroid hormone response to vitamin D deficiency had
284 as phosphates, fibroblast growth factor 23, parathyroid hormone, sclerostin, or vitamin D and their
285 roblast growth factor (FGF-2)-knockout mice, parathyroid hormone stimulation and myeloablative treatm
288 ndomized to receive 20 mug recombinant human parathyroid hormone (teriparatide) or placebo for 18 mon
289 ositively by 1,25(OH)2D3, retinoic acid, and parathyroid hormone through both intergenic and intronic
291 e rotation was associated with the change in parathyroid hormone values (r=0.37, P=0.02) and to the c
292 calcium, phosphate, 25-hydroxyvitamin D, and parathyroid hormone), various inflammatory markers (C-re
295 nal 25-hydroxyvitamin D [25(OH)D] and intact parathyroid hormone were measured at entry to care (mean
298 is defined by a high concentration of intact parathyroid hormone when circulating 25-hydroxyvitamin D
299 high parathyroid hormone and women with high parathyroid hormone whose 25(OH)D concentrations were >2
300 actor 23 was, on average, lower than that of parathyroid hormone with greater variability across indi
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