ライフサイエンスコーパス: parathyroid tumor

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1 nally inactivated in hereditary and sporadic parathyroid tumors.

2 r understanding, diagnosis, and treatment of parathyroid tumors.

3 o HPT-JT and in development of some sporadic parathyroid tumors.

4 rmed with a large group of uremia-associated parathyroid tumors.

5 might account for false-negative studies in parathyroid tumors.

6 nally inactivated in hereditary and sporadic parathyroid tumors.

7 from those involved in mutant MEN 1-related parathyroid tumors.

8 s to better define the molecular genetics of parathyroid tumors.

9 Parathyroid tumors are heterogeneous, and diagnosis is o

10 Parathyroid tumors are virtually always benign with an e

11 ncreased in the transfectants expressing the parathyroid tumor-derived receptor.

12 Parathyroid tumor infiltrating lymphocytes are T cells b

13 nctional and discussion of the management of parathyroid tumors is tantamount to the discussion of pr

14 The parathyroid tumors may arise as diffuse areas of hyperpl

15 nesis not only of cancer, but also in benign parathyroid tumors occurring in HPT.

16 Four tumors (two parathyroid tumors, one gastrinoma, and one lung carcino

17 Two tumors (one gastrinoma and one parathyroid tumor) showed an LOH boundary that placed th

18 ned occurrence of pituitary, pancreatic, and parathyroid tumors showing loss of heterozygosity in the

19 ished role in parathyroid tumorigenesis, and parathyroid tumor suppressor genes on chromosome arms 1p

20 sion profiling of 53 hereditary and sporadic parathyroid tumors to better define the molecular geneti

21 men, 14 women; age range, 24-72 years) with parathyroid tumors undergoing parathyroid arteriography

22 ions, was the most striking, suggesting that parathyroid tumors with somatic HRPT2 mutation or tumors

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