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1 pothalamic nuclei (dorsomedial, lateral, and paraventricular nuclei).
2 ons in the hypothalamic supraoptic (SON) and paraventricular nuclei.
3 l amygdala but had normal c-fos responses in paraventricular nuclei.
4 bral cortex, and hypothalamic supraoptic and paraventricular nuclei.
5 ted in the hypothalamic supraoptic (SON) and paraventricular nuclei.
6 lar cells (MNCs) in the supraoptic (SON) and paraventricular nuclei.
7 ons including the arcuate, ventromedial, and paraventricular nuclei.
8 amin D regulates glucose homeostasis via the paraventricular nuclei and energy homeostasis via the ar
10 amic arcuate, ventromedial, dorsomedial, and paraventricular nuclei and the area postrema and the nuc
12 showed an intense phosphoCREB signal in the paraventricular nuclei and ventromedial hypothalamus in
13 ary, neurons in the hypothalamic arcuate and paraventricular nuclei, and catecholaminergic neurons in
14 terior hypothalamic nuclei, locus coeruleus, paraventricular nuclei, and the rostral ventral medulla,
15 ceptor (MC-R) populations in the arcuate and paraventricular nuclei are considered probable sites of
16 ates hypothalamic neurons of the arcuate and paraventricular nuclei are consistent with the results r
17 including the perifornical, dorsomedial, and paraventricular nuclei, as well as in the paraventricula
19 ed GPCR101 mRNA expression in supraoptic and paraventricular nuclei from late pregnancy to late lacta
20 num vasculosum of the lamina terminalis, and paraventricular nuclei in the forebrain, and the tractus
21 urocortin-ir perikarya in the supraoptic and paraventricular nuclei, in the central and periaqueducta
22 halamic NPY, particularly in the arcuate and paraventricular nuclei, may contribute to the altered fo
23 ntromedial nuclei, thalamic centromedial and paraventricular nuclei of dietary obese rats versus cont
25 found a vasopressinergic projection from the paraventricular nuclei of the hypothalamus (PVN) to the
26 ic sites, including the central amygdala and paraventricular nuclei of the hypothalamus and thalamus.
27 n of Insr protein product in the arcuate and paraventricular nuclei of the hypothalamus, which was as
30 sely in the agranular insular cortex and the paraventricular nuclei of the thalamus and hypothalamus
31 ncipal circadian clock, and the reuniens and paraventricular nuclei of the thalamus, each independent
33 ys (P<0.01) and c-fos mRNA expression in the paraventricular nuclei (P<0.01), but not in the suprachi
34 neurons were present in the infundibular and paraventricular nuclei, paraolfactory gyrus, posterior h
35 uclei (SON), and magnocellular region of the paraventricular nuclei (PVN) - areas previously shown to
37 cleus (VMN) and parvocellular portion of the paraventricular nuclei (PVN) of glucose-infused rats sho
39 ed (P<0.05) in both dorsal and median raphe, paraventricular nuclei (PVN), ventromedial nuclei (VMH),
41 SFO), median preoptic (MnPO), supraoptic and paraventricular nuclei (SON and PVN), area postrema (AP)
42 n preoptic nucleus (MPN), the supraoptic and paraventricular nuclei (SON and PVN), but did not influe
45 cluding the olfactory epithelium, bulb, peri/paraventricular nuclei, suprachiasmatic nucleus, arcuate
46 olfactory bulb, hypothalamic supraoptic and paraventricular nuclei, the medial habenula, and certain
47 s, including the hypothalamic supraoptic and paraventricular nuclei, the thalamic paraventricular nuc
48 L-R in the lateroanterior, ventrolateral and paraventricular nuclei was significantly decreased in th
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