ライフサイエンスコーパス: parenchymal

1 ative compartments: ventricular, midline and parenchymal.

2 eno-associated virus (AAV) vector, decreased parenchymal Abeta amyloid deposition in TgCRND8 mice and

3 aducanumab is shown to enter the brain, bind parenchymal Abeta, and reduce soluble and insoluble Abet

4 Parenchymal abnormalities are nonspecific and occur in p

5 measure, count, and describe the airway and parenchymal abnormalities in end-stage CF lungs.

6 d may often result from initially unapparent parenchymal abnormalities.

7 ed protection required hematopoietic but not parenchymal alpha7nAChRs, as shown by experiments in bon

8 cular emphysema, is characterized by loss of parenchymal alveolar tissue and impaired tissue repair.

9 ted with cortical tissue loss independent of parenchymal Alzheimer's disease pathology.

10 The larger parenchymal amyloid deposits were associated with a high

11 r amyloid, in association with low levels of parenchymal amyloid deposits.

12 Less diffuse parenchymal amyloid pathology in persons with severe CAA

13 s of CAA exist in the absence of appreciable parenchymal amyloid pathology.

14 The molecular interplay between parenchymal amyloid plaques and CAA is unclear.

15 Alzheimer disease, can occur in the form of parenchymal amyloid plaques and cerebral amyloid angiopa

16 These findings indicate that early-onset parenchymal amyloid plaques can serve as a scaffold to c

17 hology of Tg-SwDI mice, but exhibited larger parenchymal amyloid plaques compared with Tg-5xFAD mice.

18 Here we investigated how early-onset parenchymal amyloid plaques impact the development of mi

19 accumulated on and adjacent to pre-existing parenchymal amyloid plaques in Tg-5xFAD mice.

20 The periphery of parenchymal amyloid plaques was largely composed of CAA

21 -mutated human Abeta and develop early-onset parenchymal amyloid plaques, were bred to Tg-SwDI mice,

22 ase (AD) is characterized by the presence of parenchymal amyloid-beta (Abeta) plaques, cerebral amylo

23 presurgical evaluation of renal vascular and parenchymal anatomy.

24 oking and occupational endotoxin exposure to parenchymal and airway remodeling as defined by quantita

25 e detection of, and differentiation between, parenchymal and extraparenchymal disease.

26 ALTCs were engineered with human parenchymal and non-parenchymal liver cell lines (HepG2

27 Both parenchymal and non-parenchymal liver cells grown in ALT

28 ALD develops via a complex process involving parenchymal and nonparenchymal cells, as well as recruit

29 eceptor (ManR) are expressed in the liver by parenchymal and sinusoidal endothelial cells, respective

30 The marked deposition of parenchymal and vascular Abeta in these relatively young

31 SWiTCH neuroimaging data document severe parenchymal and vascular abnormalities in children with

32 r astrocytes contribute to the regulation of parenchymal arteriole (PA) tone in response to hemodynam

33 NVC was modeled by measuring parenchymal arteriole (PA) vasodilation in response to n

34 he other hand, flow/pressure decreases evoke parenchymal arteriole dilation and increased resting pyr

35 On the other hand, decreasing parenchymal arteriole tone increased resting cortical py

36 l-GCaMP3 mice, we demonstrate that increased parenchymal arteriole tone significantly increased intra

37 We showed that, in response to increases in parenchymal arteriole tone, astrocyte intracellular Ca(2

38 During increases in parenchymal arteriole tone, the pyramidal neuron respons

39 ow/pressure-evoked increases or decreases in parenchymal arteriole vascular tone, which result in art

40 Here, using parenchymal arterioles (PAs) from within the brain, we d

41 Here we show that parenchymal arterioles are responsible for 50% of the ex

42 responses, increases in flow/pressure within parenchymal arterioles increased the firing activity of

43 Flow/pressure increases within parenchymal arterioles increased vascular tone and simul

44 K(+)) from perivascular endfeet surrounding parenchymal arterioles.

45 ans blue extravasation, and 5) appearance of parenchymal astrogliosis.

46 mmon findings were the reverse halo (48.4%), parenchymal bands (54.8%) and subpleural bands (32.3%).

47 Encephalitis is parenchymal brain inflammation, commonly due to herpes s

48 These parenchymal brain lesions are considered key contributor

49 No parenchymal brain lesions were evident on imaging, but t

50 SHV infected neurons and oligodendrocytes in parenchymal brain tissues.

51 arrow chimera studies suggest that pulmonary parenchymal, but not hematopoietic, RAGE has a central r

52 revealed an almost agyric brain with diffuse parenchymal calcifications, hydrocephalus ex vacuo, and

53 ease in F4/80(+) macrophages, a reduction in parenchymal CD11c(+)CD11b(+)CD103(-) DCs, but no effect

54 M. tuberculosis-specific parenchymal CD4 T cells migrate rapidly back into the lu

55 and immunohistology revealed the presence of parenchymal CD8(+) T cells in several regions of the bra

56 cKK-E12 was highly selective toward liver parenchymal cell in vivo, with orders of magnitude lower

57 may be a cause rather than a consequence of parenchymal cell injury.

58 Whereas the initial step of parenchymal cell proliferation was not affected by acute

59 To do so, we created mice harboring liver parenchymal cell-specific deletion of HOIP (Hoip(Deltahe

60 Mice lacking NEMO in liver parenchymal cells (LPC) spontaneously develop steatohepa

61 While ablating either RIPK1 or RelA in liver parenchymal cells (LPCs) did not cause spontaneous liver

62 nhibition of catalytic IKK activity in liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) were in

63 stem can uniquely address the ability of CNS parenchymal cells (neurons, astrocytes, and microglia) t

64 nd cues from surrounding environment and non-parenchymal cells (NPCs).

65 ecognition receptors expressed by immune and parenchymal cells and drive innate immunity that can in

66 gy is conserved in both immune and nonimmune/parenchymal cells and is fundamental for the respective

67 t dendritic cells can acquire MHC from graft parenchymal cells and simultaneously present it as intac

68 ired for CD8 T-cell allorecognition of graft parenchymal cells and suggest a mechanism by which indir

69 ve observed that MF6p/FhHDM-1 is produced by parenchymal cells and transported to other tissues (e.g.

70 sms of cellular crosstalk between immune and parenchymal cells are still elusive.

71 n the regulation of innate immunity in liver parenchymal cells both in vitro and in vivo and to our k

72 t p62/SQSTM1, a protein upregulated in liver parenchymal cells but downregulated in HCC-associated HS

73 , the protective phenotype tracked with lung parenchymal cells but not bone marrow-derived cells.

74 ne cells or between immune cells and hepatic parenchymal cells contribute to the exacerbation of live

75 the crosstalk between liver lymphocytes and parenchymal cells during liver regeneration after partia

76 In liver and pancreas, replication of mature parenchymal cells ensures the physiological turnover and

77 and accumulated in nonparenchymal more than parenchymal cells for prolonged periods, significantly a

78 pathway that leads to regulated necrosis of parenchymal cells in ischemia-reperfusion injury (IRI),

79 nuclear cells and CCL20 induction by hepatic parenchymal cells in liver disease patients.

80 xpressed by sinusoidal endothelial cells and parenchymal cells in the liver, respectively.

81 l contribution of TLR4 on nonparenchymal and parenchymal cells in the pathogenesis of PH as determine

82 states of their 'client cells': namely, the parenchymal cells in the various tissues in which macrop

83 hat inflammatory mediators produced by renal parenchymal cells may influence the function of remote o

84 Hepatocytes, the highly metabolic parenchymal cells of the liver, are efficient at differe

85 Fusion of donor mesenchymal stem cells with parenchymal cells of the recipient can occur in the brai

86 he effects of TRPM2 are due to expression in parenchymal cells rather than hematopoietic cells.

87 range of stromal cells that co-develop with parenchymal cells to form tissues.

88 HOIP deficiency in liver parenchymal cells triggered tumorigenesis at 18 months o

89 ntrifugation into two portions, hepatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

90 epatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

91 se data demonstrate an unanticipated role of parenchymal cells, as shown here for hepatocytes, in tis

92 that form a barrier between blood and liver parenchymal cells, NS2(H126R) activates RNase L, which l

93 factors and individual cell types, including parenchymal cells, vascular cells, and immune cells, can

94 linear ubiquitination specifically in liver parenchymal cells, we investigated the physiological rol

95 tained within endosomes of hematopoietic and parenchymal cells, whereupon IgG is diverted from degrad

96 mitting diffuser, with no intervening viable parenchymal cells.

97 ecialized to fulfill the particular needs of parenchymal cells.

98 ission of multiple metabolic vasodilators by parenchymal cells.

99 ire IL-15 produced by both hematopoietic and parenchymal cells.

100 anced ASO delivery to hepatocytes versus non-parenchymal cells.

101 myofibroblasts (MFBs) in place of functional parenchymal cells.

102 rves distinct functions in immune and tissue parenchymal cells.

103 e generated by continual processing of graft parenchymal cells; recognition of donor haemopoietic fra

104 Parenchymal changes were also seen, mostly in a patchy a

105 und to be valuable parameters in determining parenchymal changes.

106 PbO2 was measured hourly by a parenchymal Clark electrode, and cerebral metabolism was

107 urological symptoms in DLBCL and BL and with parenchymal CNS lymphoma in DLBCL; sCD19 emerged as a po

108 TP preferentially to the basolateral ("brain parenchymal") compartment.

109 Evidence of vessels and parenchymal compression with no source of bleeding was f

110 Available data indicate that employing parenchymal cooling can mitigate the deleterious effects

111 arterial damage (p = 0.04) and in those with parenchymal damage (p = 0.05).

112 rcadian gene Clock, renal fibrosis and renal parenchymal damage were significantly worse after ureter

113 trophils recruited to the airways, more lung parenchymal damage, and increased pulmonary consolidatio

114 mpaired hepatic microcirculation, aggravated parenchymal damage, decelerated recovery, and impaired t

115 ure to reduce ischemic cerebral vascular and parenchymal damages.

116 Crystalline nephropathy refers to renal parenchymal deposition of crystals leading to kidney dam

117 PP colocalized with amyloid plaques in brain parenchymal deposits, suggesting that these peptides may

118 ation of airways disease (bronchiolitis) and parenchymal destruction (emphysema), whose relative prop

119 iability in the severity and distribution of parenchymal destruction throughout the lungs.

120 and functional residual capacity for severe parenchymal destruction), predicted better than single m

121 riate functional variables (FEV1/VC for mild parenchymal destruction, Dlco% and functional residual c

122 mphysema with severe hyperinflation and less parenchymal destruction.

123 tional small airways disease (PRM(fSAD)) and parenchymal disease (PRM(PD)) were compared between bila

124 No patients were identified with diffuse parenchymal disease on the basis of computed tomography

125 The diagnosis of thyroid cancer and diffuse parenchymal disorders is generally based on clinical man

126 dren with known or suspected airway and lung parenchymal disorders.

127 ese mice also showed significantly decreased parenchymal EB levels.

128 ial fibrosis are associated with lower renal parenchymal elasticity.

129 = 80, 18.3%), moderate or marked background parenchymal enhancement (BPE) (n = 91, 20.9%), posttreat

130 Purpose To assess the extent of background parenchymal enhancement (BPE) at contrast material-enhan

131 utomated quantitative measures of background parenchymal enhancement (BPE) derived from an early vers

132 Purpose To evaluate the effect of background parenchymal enhancement (BPE) on breast magnetic resonan

133 Background parenchymal enhancement (BPE), and the amount of fibrogl

134 argest diameter on MR images (P = .049), and parenchymal enhancement (P = .011) were significant.

135 tion) and normal tissue features (background parenchymal enhancement [BPE] volume, mean BPE) were qua

136 The effects of tamoxifen and AI on benign parenchymal enhancement differ.

137 olinium administration = 1 x dysmorphy + 1 x parenchymal enhancement heterogeneity).

138 Results suggest that parenchymal enhancement in the contralateral breast of p

139 tio of lesion enhancement rate to background parenchymal enhancement rate are more likely to be lumin

140 s who underwent endocrine therapy (n = 174), parenchymal enhancement was the only significant covaria

141 alitative and quantitative degrees of benign parenchymal enhancement were investigated before treatme

142 ession was used to test associations between parenchymal enhancement, patient and tumor characteristi

143 ip between lesion enhancement and background parenchymal enhancement.

144 rotective CNS inflammation by regulating the parenchymal entry of CXCR4(+) virus-specific T cells dur

145 transport; (ii) transport of dextrans in the parenchymal extracellular space, measured by 2-photon fl

146 e first time, this study demonstrates that a parenchymal factor, RAGE, mediates lung-specific accumul

147 nd behavioral impairment, but never develops parenchymal fibrillar amyloid deposits.

148 ry endpoint, PC, was defined by new abnormal parenchymal findings on chest imaging in the setting of

149 ide eliciting dilation and preserving normal parenchymal function by inhibiting inflammation and prol

150 g of POMC neurons was reversed ex vivo or by parenchymal glucose administration.

151 the systemic circulation, resolution of the parenchymal "ground glass" opacity and absence of furthe

152 s accumulate lipids before the major wave of parenchymal growth.

153 Mortality at 90 days and risk of parenchymal haematoma and symptomatic intracranial haemo

154 Alteplase increased the odds of type 2 parenchymal haemorrhage (occurring in 231 [6.8%] of 3391

155 62 were haemorrhagic infarction and 26 were parenchymal haemorrhage (PH).

156 symptomatic intracranial haemorrhage (type 2 parenchymal haemorrhage definition 231 [6.8%] of 3391 vs

157 cluded patients aged 18 years and older with parenchymal haemorrhage on the first CT scan.

158 ITS-MOST) haemorrhage within 24-36 h (type 2 parenchymal haemorrhage with a deterioration of at least

159 intracranial haemorrhage (defined by type 2 parenchymal haemorrhage within 7 days and, separately, b

160 cations of intracerebral haemorrhage: type 2 parenchymal haemorrhage within 7 days; Safe Implementati

161 cteplase, 7; alteplase, 2; P=0.018) and less parenchymal hematoma (2 of 75 versus 10 of 71; P=0.02).

162 Parenchymal hematoma (PH) following intravenous thrombol

163 67-1.97; RD, 0.00; 95% CI, -0.02 to 0.03) or parenchymal hematoma (RR, 1.18; 95% CI, 0.71-1.94; RD, 0

164 ause mortality, intracranial hemorrhage, and parenchymal hematoma at 90 days were also assessed.

165 %) and 2 treatment patients (7%) developed a parenchymal hematoma type 2 (P > .99).

166 nitoring Study (SITS-MOST), which included a parenchymal hematoma type 2 and at least a 4-point incre

167 t growth at 24 hours and occurrence of large parenchymal hematoma.

168 No sICH or parenchymal hematomas occurred.

169 tients had hemorrhagic infarction and 15 had parenchymal hemorrhage.

170 matic intracranial hemorrhage (1% vs 4%) and parenchymal hemorrhages type 1 (1% vs 3%) or type 2 (1%

171 n individual cell populations, specifically, parenchymal hepatocytes (HCs), myeloid cells, including

172 ments were evaluated visually before PEA for parenchymal hypoperfused segments.

173 itic cells, are known, whereas the source of parenchymal IL-15 remains elusive.

174 l15(-/-) mice, we identified adipocytes as a parenchymal IL-15 source that supported NK cell developm

175 level of the trachea on mediastinal and lung parenchymal images (P < .001) and no significant differe

176 y allergic immune response, whereas the lung parenchymal immune system has been largely neglected.

177 ll pleural infections can be related to lung parenchymal infection.

178 Chest radiograph revealed a parenchymal infiltrate in 188 patients.

179 CT scan revealed a parenchymal infiltrate in 40 (33%) of the patients witho

180 d excluded CAP in 56 (29.8%) of the 188 with parenchymal infiltrate on radiograph.

181 perivascular predominance as well as diffuse parenchymal infiltration (14/14), present in meninges, w

182 ve oxygen species mediates both dilation and parenchymal inflammation leading to cellular dysfunction

183 s (CFIMs), as possible surrogate markers for parenchymal inflammation.

184 ytic anaemia, even when subclinical, lead to parenchymal injury and chronic organ damage, causing sub

185 Baseline parenchymal injury was prevalent (85%/79% subcortical, 5

186 /8 alternative arm subjects) had substantial parenchymal injury/vessel stenosis.

187 nal model reproduced this behavior only when parenchymal interdependence between neighboring acini wa

188 f 17; P = .016), and lesions with indistinct parenchymal interface (nine of 17; P < .001) were associ

189 reviewed to identify the frequency of liver parenchymal invasion (LPI) from perihepatic peritoneal m

190 The overall extent of parenchymal involvement was 50 to 75% in 80% of patients

191 ingeal and choroid plexus cells, and limited parenchymal involvement.

192 mice, with increased plasma iron and massive parenchymal iron accumulation.

193 transporter ferroportin (Fpn), resulting in parenchymal iron overload.

194 group of genetic disorders characterized by parenchymal iron overload.

195 FD also resulted in immediate enhancement of parenchymal labeling with the fluorescent dye Hoechst 33

196 ging showed enlargement of ventricles but no parenchymal lesion.

197 Additional imaging studies showed subtle parenchymal lesions in the posterior fossa.

198 yndromes (both acute and progressive), brain parenchymal lesions seen on neuroimaging and a set of di

199 ls in bronchoalveolar lavage fluid, ii) lung parenchymal leukocyte counts and lymphoid aggregates, ii

200 re engineered with human parenchymal and non-parenchymal liver cell lines (HepG2 and LX2 cells, respe

201 Both parenchymal and non-parenchymal liver cells grown in ALTCs exhibited markedl

202 o Tlr3-dependent interferon responses in non-parenchymal liver cells.

203 ifferent LC for (18)F-FDGal in patients with parenchymal liver disease.

204 spiratory tract infections in the absence of parenchymal lung abnormalities, retained primary teeth,

205 ugal, and the UK) evaluated cases of diffuse parenchymal lung disease in a two-stage process between

206 A consistent diagnostic approach to diffuse parenchymal lung disease is crucial if clinical trial da

207 ic pulmonary fibrosis (IPF) is a progressive parenchymal lung disease of complex cause.

208 ibrosis is a progressive and generally fatal parenchymal lung disease of unknown etiology with no cur

209 essive dyspnea and cough with the absence of parenchymal lung disease on radiographic studies.

210 Diffuse parenchymal lung disease represents a diverse and challe

211 team agreement for the diagnosis of diffuse parenchymal lung disease.

212 ystitis, renal failure, and interstitial and parenchymal lung diseases.

213 Influenza induced persistent parenchymal lung inflammation, alveolar epithelial metap

214 Chest radiograph is key in establishing parenchymal lung involvement.

215 e in the lungs under the instruction of lung parenchymal lymphocytes.

216 eta8 and IL-1beta with CCL20 protein in lung parenchymal lysates of a large cohort of COPD patients.

217 ngeal and choroid plexus macrophages are non-parenchymal macrophages that mediate immune responses at

218 may have been related to closer attention to parenchymal margins by surgeons participating in this st

219 sistent Notch signalling after injury led to parenchymal 'micro-honeycombing' (alveolar cysts), indic

220 a diverse myeloid compartment that includes parenchymal microglia and perivascular macrophages, as w

221 Although the origin of parenchymal microglia has recently been elucidated, much

222 )Ly6C(+) myeloid cells, but not on P2RY12(+) parenchymal microglia.

223 d with perivascular inflammatory cuffing and parenchymal microglial activation but precede the arriva

224 ions and benign versus malignant tumors were parenchymal microlithiasis (26 of 86 patients with tumor

225 For parenchymal NCC, the new criteria had a sensitivity of 8

226 atic vein ligation (RMHV-L) caused confluent parenchymal necrosis interspersed with viable portal tra

227 eous course of recovery in terms of enlarged parenchymal necrosis, delayed regeneration, and the abse

228 tic fluid along with a limited area of intra-parenchymal necrosis, indicating necrotizing pancreatiti

229 alanine aminotransferase) and areas of liver parenchymal necrosis.

230 Portal fibrosis, bridging, parenchymal nodules, portal inflammation, hepatocellular

231 Parenchymal oligodendrocyte progenitor cells (pOPCs) are

232 Both parenchymal oligodendrocyte progenitor cells and endogen

233 proaches that account for the involvement of parenchymal or extraparenchymal spaces, the number and f

234 is that they consider parasite location (ie, parenchymal or extraparenchymal), which is an important

235 e EXE treatment did not induce pancreatitis, parenchymal or periductal inflammatory cell accumulation

236 (POPH) may occur as a consequence of hepatic parenchymal or vascular abnormalities.

237 abolic/catabolic activities ongoing in every parenchymal organ as part of tissue growth, remodeling,

238 trates, both in exocrine glands and in other parenchymal organs (kidney, lung, and liver), are the hi

239 coefficient (ADC) values of normal abdominal parenchymal organs and signal-to-noise ratio (SNR) measu

240 f periepithelial infiltrates in exocrine and parenchymal organs or resulting from immunocomplex depos

241 Fourteen different vessel and parenchymal pathologies were assessed in 13 brain region

242 e computed tomographic examination showed no parenchymal pathology, but a isolated transverse fractur

243 lationships among pulmonary ossification and parenchymal patterns, clinical parameters, and multidisc

244 cted IL-1R1(-/-) mice also display increased parenchymal penetration of CD8(+) T cells despite lack o

245 Catheter navigation times, renal parenchymal perfusion, and renal artery flow rates were

246 ontribute to homeostatic regulation of brain parenchymal pH and control of breathing.

247 phocreatine concentrations, as well as brain parenchymal pH, were normal.

248 ified: 1) enhancement peak during pancreatic parenchymal phase (PPP) followed by a rapid decline on p

249 scular drainage pathways, resulting in fewer parenchymal plaques but more CAA because of loss of CLU

250 ate its ability to penetrate brain and label parenchymal plaques in transgenic mice.

251 are produced in a previously uncharacterized parenchymal population of cells.

252 , which persists throughout childhood, and a parenchymal population that diminishes by 7 months and w

253 producible technique that enables anatomical parenchymal preserving liver resections for selected cen

254 Nearly all parenchymal progenitors at P4 are Sox2(+)Olig2(+), but b

255 However, parenchymal proinflammatory chemokine expression, includ

256 nning with lissencephalic aspect to moderate parenchymal rarefaction, severe to mild ventriculomegaly

257 luate the contribution of non-hepatocytes to parenchymal regeneration.

258 ar regions of the cortex were larger than in parenchymal regions, demonstrating size-dependent subcel

259 Evidence of lung parenchymal remodeling and epithelial dysfunction was id

260 Finally, we determined that the parenchymal rEos found in nonasthmatic human lungs (Sigl

261 hnique was applied since 1991 for anatomical parenchymal resections including central hepatectomy (re

262 roduce the S1P egress signal, whereas thymic parenchymal S1P levels are kept low through S1P lyase (S

263 anterior approach without mobilization, and parenchymal section with ultrasonic dissector.

264 m (CNS) depend critically on the activity of parenchymal sentinels referred to as microglia.

265 nses when acting from the circulation or the parenchymal side at blood-neural barriers.

266 dy-state pulmonary rEos are IL-5-independent parenchymal Siglec-FintCD62L+CD101lo cells with a ring-s

267 e current definition of positive result (any parenchymal, solid or part-solid, noncalcified nodule >/

268 g the definition of a positive result of any parenchymal, solid or part-solid, noncalcified nodule of

269 native to extensive surgery by assuring both parenchymal sparing and suitable oncologic resection.

270 These results suggests that non-neurogenic, parenchymal structural plasticity might be more importan

271 NOX4 is necessary to maintain parenchymal structures, increase cell-cell and cell-to-m

272 Importantly, parenchymal T cells displayed greater control of infecti

273 not readily distinguish between normal (ie, parenchymal tau) and pathological tau species and showed

274 alopathy, but did not label nonpathological, parenchymal tau.

275 onal cerebral blood flow was measured with a parenchymal thermal diffusion probe.

276 nderdevelopment, ranging from major cerebral parenchymal thinning with lissencephalic aspect to moder

277 days before such T cells can enter the lung parenchymal tissue and airway.

278 hat neutrophil infiltration was required for parenchymal tissue damage in the brain.

279 the lung, unable to populate either the lung parenchymal tissue or the airway under homeostatic condi

280 However, in parenchymal tissue, CBFv oscillated with peaks at both a

281 onstitutively in phagocytes and inducibly in parenchymal, tissue-forming cells.

282 ted KT inhibitor associated with the gut and parenchymal tissues of the infective juvenile stage of F

283 (TNF) superfamily is a conserved response of parenchymal tissues to injury and inflammation that comm

284 red mainly by their counterparts residing in parenchymal tissues.

285 atched, paired NSCLC tumor and non-malignant parenchymal tissues.

286 nsported across the capillary endothelium to parenchymal tissues.

287 of FLR hypertrophy between total or partial parenchymal transection (P = 0.45).

288 is a 2-stage hepatectomy, which incorporates parenchymal transection at stage 1 enabling resection of

289 Partial parenchymal transection seems to reduce morbidity withou

290 Total parenchymal transection was identified as an independent

291 Optimal grading and treatment of pineal parenchymal tumours of intermediate differentiation (PPT

292 , separately, by the SITS-MOST definition of parenchymal type 2 haemorrhage within 36 h), fatal intra

293 Affected individuals present with cerebral parenchymal underdevelopment, ranging from major cerebra

294 is study was to quantitatively assess breast parenchymal uptake (BPU) on (18)F-FDG PET/CT as another

295 a nanoparticle, mimics a ligand for hepatic parenchymal uptake transporters resulting in hepatobilia

296 ths, and the cysticerci were observed in the parenchymal, ventricle, or submeningeal brain tissue.

297 aptic or spiking responses around individual parenchymal vessels in cats and established that the vas

298 responses for the first time and found that parenchymal vessels in cortical layer 2/3 were orientati

299 bronchoscopic intervention inducing regional parenchymal volume reduction and restoring lung recoil.

300 he olfactory bulb, and in a newly identified parenchymal zone of the telencephalon indeed declines as