ライフサイエンスコーパス: parenchymal cell

1 epatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

2 rves distinct functions in immune and tissue parenchymal cells.

3 b-mediated destruction of transplanted liver parenchymal cells.

4 PDL1 exclusively on either hematopoietic or parenchymal cells.

5 hose concentration is regulated by the liver parenchymal cells.

6 manifested mainly by stimulation of genital parenchymal cells.

7 cropatterned alone and in coculture with non-parenchymal cells.

8 mitting diffuser, with no intervening viable parenchymal cells.

9 d at higher levels than by the corresponding parenchymal cells.

10 e antigens that are expressed exclusively in parenchymal cells.

11 PPARalpha-regulated gene expression in liver parenchymal cells.

12 interactions between blood vessels and brain parenchymal cells.

13 en by IL-15 produced by either BM-derived or parenchymal cells.

14 s of leukocytes, epithelial cells, and other parenchymal cells.

15 phyrin accumulation), more iron was found in parenchymal cells.

16 bral microvessels and central nervous system parenchymal cells.

17 ment, where they are ingested by neighboring parenchymal cells.

18 ng T cells and potentially tolerogenic graft parenchymal cells.

19 tence of an efflux transport system in brain parenchymal cells.

20 eas TIMP-1 RNA was detected predominantly in parenchymal cells.

21 ation can replenish large numbers of hepatic parenchymal cells.

22 the opportunity for lymphocyte attachment to parenchymal cells.

23 tion of whether HIV-1 directly infects renal parenchymal cells.

24 ized in primary cultures of mature liver non-parenchymal cells.

25 expression of this cell adhesion molecule on parenchymal cells.

26 with ET, neutrophils transmigrate and attack parenchymal cells.

27 nduced by peptide-MHC complexes expressed on parenchymal cells.

28 into mice expressing HA as a self-antigen on parenchymal cells.

29 ferate for 1 month, forming foci of dividing parenchymal cells.

30 ional NOS2 expression was localized to graft-parenchymal cells.

31 ing inflammatory cells, but not in allograft parenchymal cells.

32 ccur via a regulatory protein found in liver parenchymal cells.

33 ecialized to fulfill the particular needs of parenchymal cells.

34 ission of multiple metabolic vasodilators by parenchymal cells.

35 ire IL-15 produced by both hematopoietic and parenchymal cells.

36 anced ASO delivery to hepatocytes versus non-parenchymal cells.

37 libraries of ECs devoid of lymphatic ECs or parenchymal cells.

38 t develop in mice lacking TLR4 expression on parenchymal cells.

39 label-free identification of lipid-rich non-parenchymal cells.

40 ly in mice that had TLR4 expression on their parenchymal cells.

41 myofibroblasts (MFBs) in place of functional parenchymal cells.

42 emical analysis of interleukin (IL)-1beta in parenchymal cells.

43 delivered by intact MyD88 signaling on renal parenchymal cells.

44 erentiation into fully mature and functional parenchymal cells.

45 rating inflammatory cells, but not allograft parenchymal cells; (2) aminoguanidine ameliorated the hi

46 Non-parenchymal cells (3T3 fibroblasts) were added to hepato

47 Parenchymal cell activation via TLR4 is a key component

48 fic IRF3-KO mice, deficiency of IRF3 only in parenchymal cells aggravated alcohol-induced liver injur

49 Liver parenchymal cell allografts initiate both CD4-dependent

50 focused on inflammatory cell TLR4 responses, parenchymal cells also express TLR4.

51 LT and liver injury, and this suggested that parenchymal cell and BMDC B7-H1 expression was involved

52 To evaluate the relative contributions of parenchymal cell and bone marrow-derived cell (BMDC) B7-

53 termined the CD phenotype of normal prostate parenchymal cells and are now extending this analysis to

54 efflux transport processes at both the brain parenchymal cells and blood-brain barrier (BBB); one or

55 on transporters for the efflux of VPA at the parenchymal cells and capillary endothelium in the brain

56 of antigen that is actually transferred from parenchymal cells and cross-presented in vivo is unknown

57 ecognition receptors expressed by immune and parenchymal cells and drive innate immunity that can in

58 exokinase in pancreatic beta-cells and liver parenchymal cells and functions as a critical component

59 gy is conserved in both immune and nonimmune/parenchymal cells and is fundamental for the respective

60 3-dioxygenase (IDO) is expressed by APCs and parenchymal cells and is further inducible by inflammati

61 Heterotypic cell interaction between parenchymal cells and nonparenchymal neighbors has been

62 examination revealed that a large number of parenchymal cells and other types of cells in the liver

63 Our results indicate that IRF3 activation in parenchymal cells and resulting type I IFNs have protect

64 t dendritic cells can acquire MHC from graft parenchymal cells and simultaneously present it as intac

65 ired for CD8 T-cell allorecognition of graft parenchymal cells and suggest a mechanism by which indir

66 in part, this cytokine activation program in parenchymal cells and that liver-derived G-CSF may contr

67 t not only in immune cells but also in liver parenchymal cells and the complexity of the cell populat

68 lymphocytes and the surrounding stromal and parenchymal cells and together with the two related cyto

69 ve observed that MF6p/FhHDM-1 is produced by parenchymal cells and transported to other tissues (e.g.

70 opoietic cell-produced IL-10 acting on brain parenchymal cells and vice versa.

71 ntrifugation into two portions, hepatocytes (parenchymal cells) and LSEC (non-parenchymal cells).

72 ion between infiltrating cells, endothelium, parenchymal cells, and components of extracellular matri

73 rphogenic processes that sculpt vasculature, parenchymal cells, and mesenchyme to form the multifacet

74 SmSmad2 was localized in the subtegument, parenchymal cells, and sex organs in both male and femal

75 ysema, in which there is both increased lung parenchymal cell apoptosis and defective AC uptake by AM

76 T)-induced liver failure is characterized by parenchymal cell apoptosis and inflammation leading to l

77 The degree to which parenchymal cell apoptosis is contributing to multiple o

78 In this study, we tested the hypothesis that parenchymal cell apoptosis may induce neutrophil transen

79 We conclude that excessive parenchymal cell apoptosis represents an important signa

80 intercellular adhesion molecule-1 (sICAM-1), parenchymal cell apoptosis, and neutrophil sequestration

81 proteases is critical for the development of parenchymal cell apoptosis.

82 Parenchymal cells are closely associated with lineages o

83 a new paradigm in which parenchymal and non-parenchymal cells are continuously replaced by newly for

84 Since tissue parenchymal cells are important local producers of IL-8

85 eric mice show that both leukocytes and lung parenchymal cells are sources of profibrotic MMP-8 durin

86 sms of cellular crosstalk between immune and parenchymal cells are still elusive.

87 These findings suggest that liver parenchymal cells are the dominant source of Type I IFN

88 We have also shown that parenchymal cells are tolerogenic in vitro.

89 t alpha-tanycytes, but not beta-tanycytes or parenchymal cells, are neurospherogenic.

90 se data demonstrate an unanticipated role of parenchymal cells, as shown here for hepatocytes, in tis

91 ocytes produced neoorgans populated by these parenchymal cells, as well as by chimeric human vessels

92 with extensive infection and destruction of parenchymal cells at the margins of granulomas.

93 ent and disorganized pancreatic fibrosis and parenchymal cell atrophy occur.

94 FGFR type 4 (FGFR4) in liver parenchymal cells binds only FGF-1, whereas FGFR1 binds

95 n the regulation of innate immunity in liver parenchymal cells both in vitro and in vivo and to our k

96 The enzyme is secreted by the hepatic parenchymal cell but exists, and presumably exerts its e

97 t p62/SQSTM1, a protein upregulated in liver parenchymal cells but downregulated in HCC-associated HS

98 , the protective phenotype tracked with lung parenchymal cells but not bone marrow-derived cells.

99 Interestingly, mice with wild-type parenchymal cells but TNFRsf1a/1b(-/-) hematopoietic cel

100 d with diffuse increases in iron staining of parenchymal cells but without evidence of significant li

101 n which IFN-gammaR expression was limited to parenchymal cells, but resolution was significantly dela

102 striatal injections led to beta-gal-positive parenchymal cells, but, unlike rAAV2betagal, rAAV5betaga

103 idue assists in preferential transduction of parenchymal cells by AAV6 vectors in comparison with AAV

104 ne only in their leukocytes or only in their parenchymal cells by lethal radiation and reconstitution

105 thesis that acute damage of allogeneic liver parenchymal cells by the CD4-dependent pathway is alloan

106 ntributing to the postnatal expansion of the parenchymal cell compartment.

107 ne cells or between immune cells and hepatic parenchymal cells contribute to the exacerbation of live

108 s to show that B7-H1 expression by recipient parenchymal cells controls the second wave of alloreacti

109 cant decrease in lymphocyte infiltration and parenchymal cell damage in the submandibular salivary gl

110 osis did not correlate with graft failure or parenchymal cell damage, suggesting that cytotoxic T cel

111 l oxidant production is a critical factor in parenchymal cell death caused by alcohol.

112 diffused into hepatocytes and contributed to parenchymal cell death in vivo.

113 lay a key role in hepatocyte dysfunction and parenchymal cell death upon reperfusion.

114 Further, parenchymal cell-derived Type I IFNs increase antiinflam

115 However, the fact that hepatic parenchymal cells, despite NF-kappa B activation do not

116 We propose that parenchymal cells direct SIRS in response to LPS.

117 nt protein-1 (MCP-1) is upregulated in renal parenchymal cells during kidney disease.

118 the crosstalk between liver lymphocytes and parenchymal cells during liver regeneration after partia

119 Loss of B7-H1 expression by parenchymal cells during the course of GVHD was associat

120 In liver and pancreas, replication of mature parenchymal cells ensures the physiological turnover and

121 orce separated plasma through packed beds of parenchymal cells exhibited significant necrosis with a

122 Given that activated endothelial and tissue parenchymal cells express both class I and class II MHC

123 These studies demonstrate that parenchymal cell expression of B7-H1 is required for tol

124 beta IEL populations, depended completely on parenchymal cell expression of IL-15R alpha and IL-15 bu

125 The hepatic parenchymal cells extract mebrofenin from the blood usin

126 in the targeting of transplanted allogeneic parenchymal cells for macrophage-mediated cytotoxic immu

127 R alpha expression by bone marrow-derived or parenchymal cells for mediating lymphocyte subset develo

128 and accumulated in nonparenchymal more than parenchymal cells for prolonged periods, significantly a

129 l cells play an important role in protecting parenchymal cell from rejection.

130 Detachment of parenchymal cells from a solid matrix switches contextua

131 arenchymal cells may effectively protect the parenchymal cells from immune attack.

132 Flow cytometry analysis of hepatic non-parenchymal cells from infected mice reveals that IKKbet

133 se that T cells activated in the presence of parenchymal cells from the eye (an immune privileged sit

134 tterning and differentiation, maintenance of parenchymal cell function, and the size, shape, and inva

135 Both groups of mice with TNFR-deficient parenchymal cells had low bronchoalveolar lavage fluid t

136 Direct infection of renal parenchymal cells has been implicated in the pathogenesi

137 by bone-marrow (BM) derived versus infected parenchymal cells has on T cell exhaustion.

138 Parenchymal cells have a major role in the trans-present

139 pffer cell NADPH oxidase, and peroxisomes in parenchymal cells have been proposed as the potential so

140 The rate of consumption of NO by parenchymal cells (hepatocytes) linearly depends on both

141 mphocytes to produce IL-1beta, which induces parenchymal cell IL-8 release.

142 cKK-E12 was highly selective toward liver parenchymal cell in vivo, with orders of magnitude lower

143 by labile plasma iron, which is taken up by parenchymal cells in a dysregulated manner; in contrast,

144 at NO production from NOS2 expressed in lung parenchymal cells in a murine model of ARDS would correl

145 e detected a large number of apoptotic renal parenchymal cells in advanced nephritis and determined t

146 led patterns of increased C5aR expression in parenchymal cells in all four organs following CLP.

147 Parenchymal cells in aorta, liver, and lung bearing TLR4

148 ers and differentiated to mature, functional parenchymal cells in approximately 1 week, remaining via

149 ities, but the extent to which leukocytes vs parenchymal cells in different organs contribute to the

150 In addition to lymphocytes, parenchymal cells in ileum, colon, lung, and to a lesser

151 cells and vascularization by these cells of parenchymal cells in implants.

152 pathway that leads to regulated necrosis of parenchymal cells in ischemia-reperfusion injury (IRI),

153 nuclear cells and CCL20 induction by hepatic parenchymal cells in liver disease patients.

154 pPar1, c-kit, and alpha-fetoprotein (AFP) in parenchymal cells in massive necrosis.

155 Further, selective MyD88 deficiency in parenchymal cells in mice with wild-type BM failed to pr

156 F-alpha), and apoptotic destruction of renal parenchymal cells in MRL-Fas(lpr) autoimmune kidney dise

157 ed on a variety of renal parenchymal and non-parenchymal cells in normal kidney.

158 t mechanism of cell death in lymphocytes and parenchymal cells in sepsis and occurs systemically in m

159 These results suggest that parenchymal cells in some organs may contribute substant

160 ociated with decreased TGF-beta derived from parenchymal cells in the BAL fluid, lower nitrite levels

161 nflammatory cells and activation of resident parenchymal cells in the central nervous system.

162 r (ASGP-R), which is abundantly expressed by parenchymal cells in the liver of mammals.

163 xpressed by sinusoidal endothelial cells and parenchymal cells in the liver, respectively.

164 role of productive HIV-1 infection of renal parenchymal cells in the pathogenesis of HIV-associated

165 l contribution of TLR4 on nonparenchymal and parenchymal cells in the pathogenesis of PH as determine

166 ntly spreads to hepatocytes and unidentified parenchymal cells in the spleen.

167 states of their 'client cells': namely, the parenchymal cells in the various tissues in which macrop

168 94, which is specifically expressed in liver parenchymal cells, in preventing liver cancer cell metas

169 transduce not only 3LL cells but also brain parenchymal cells including endothelial cells, neurons,

170 an anergic T cells can impair the ability of parenchymal cells (including endothelial and epithelial

171 These therapies target several types of parenchymal cells (including neural stem cells, cerebral

172 ch is expressed broadly on hematopoietic and parenchymal cells, including pancreatic islet cells; and

173 ble expression of viral antigens even in non-parenchymal cells, indicating that infection of these ce

174 animals expressing CCL2 in the CNS promoted parenchymal cell infiltration and ascending paralysis in

175 hat the observed signaling pathways regulate parenchymal cell injury and death in CCK-induced pancrea

176 r leukocytes (neutrophils) can cause hepatic parenchymal cell injury during endotoxin (ET) shock.

177 Neutrophils can cause parenchymal cell injury in the liver during ischemia-rep

178 iver is important for the genesis of hepatic parenchymal cell injury in this model.

179 However, the molecular mechanism of parenchymal cell injury remains controversial.

180 oliferation, activation, and apoptotic graft parenchymal cell injury, but the high frequency of apopt

181 may be a cause rather than a consequence of parenchymal cell injury.

182 ailure, which involves a neutrophil-mediated parenchymal cell injury.

183 tration in the liver and severe vascular and parenchymal cell injury.

184 LPL is secreted by parenchymal cells into the interstitial spaces; it then

185 ne lupus nephritis, while apoptosis of renal parenchymal cells is a feature of advanced human lupus n

186 APC to cross-present Ag to MHC class I from parenchymal cells is essential for priming as well as to

187 tudies suggest that PD-L1 expression on host parenchymal cells is more critical than hematopoietic ce

188 kine receptor on circulating blood cells and parenchymal cells is often used to coordinate complex ti

189 -L1 expression on hemopoietic cells, and not parenchymal cells, is primarily responsible for limiting

190 DCs) cross-dressed by Ag from virus-infected parenchymal cells, it is unknown whether conditions exis

191 other DC subtypes, macrophages, B cells, and parenchymal cells lack of expression of the I-A(beta)(b)

192 Although the absence of Bim in parenchymal cells led to markedly attenuated liver damag

193 persistent expression of B7-H1 expression by parenchymal cells led to reduced proliferation of donor

194 However, HSV antigens were detected in non-parenchymal cells lining the ventricles.

195 enchymal stem cell (MSC) therapy can prevent parenchymal cell loss and promote tissue repair in model

196 Mice lacking NEMO in liver parenchymal cells (LPC) spontaneously develop steatohepa

197 While ablating either RIPK1 or RelA in liver parenchymal cells (LPCs) did not cause spontaneous liver

198 nhibition of catalytic IKK activity in liver parenchymal cells (LPCs; IKKalpha/beta(LPC-KO) ) were in

199 ed progressively to repopulate 60% to 80% of parenchymal cell mass in 60 days.

200 Therefore, activation of TLR4 on renal parenchymal cells may activate p38 MAPK pathways, leadin

201 V-induced responses, indicating that CCR1(+) parenchymal cells may also play a significant role in th

202 hat inflammatory mediators produced by renal parenchymal cells may influence the function of remote o

203 dependent on bone marrow stromal or hepatic parenchymal cell monolayers.

204 In this process, the antigen-bearing parenchymal cells must somehow transfer their antigens t

205 d heart transplants and donor-specific renal parenchymal cells (n=4); group 4 animals received heart

206 , evaluation of ischemic death mechanisms in parenchymal cells needs to be performed with caution.

207 stem can uniquely address the ability of CNS parenchymal cells (neurons, astrocytes, and microglia) t

208 The influence of irradiation on graft non-parenchymal cells (NPC) was determined by monoclonal ant

209 nd cues from surrounding environment and non-parenchymal cells (NPCs).

210 that form a barrier between blood and liver parenchymal cells, NS2(H126R) activates RNase L, which l

211 the hypothesis that oxLDL may be present in parenchymal cells of cerebrum after infarction and that

212 y stages of chemical hepatocarcinogenesis in parenchymal cells of Fischer 344 rats fed a diet supplem

213 , however, about how these chemokines affect parenchymal cells of the CNS.

214 MHC class II antigens is up-regulated on the parenchymal cells of the kidney.

215 Hepatocytes, the key parenchymal cells of the liver, are a particularly attra

216 Hepatocytes, the highly metabolic parenchymal cells of the liver, are efficient at differe

217 Furthermore, we determined that the non-parenchymal cells of the liver, i.e. endothelial, Kupffe

218 te-specific, endocytic receptor expressed by parenchymal cells of the liver.

219 sing of glucose in pancreatic beta-cells and parenchymal cells of the liver.

220 ed with an elevation of Bmp6 mRNA in the non-parenchymal cells of the liver.

221 lls to serve as precursors of differentiated parenchymal cells of the lung.

222 Fusion of donor mesenchymal stem cells with parenchymal cells of the recipient can occur in the brai

223 antigen on endothelial cells, and on tissue parenchymal cells once they have entered the tissue.

224 Mice bearing TLR4 on parenchymal cells only, marrow-derived cells only, both,

225 A selective B7-H1 deficiency on parenchymal cells or BMDCs resulted in similar levels of

226 ients chimeric liver grafts lacking B7-H1 on parenchymal cells or BMDCs.

227 rks to support engineered tissues of desired parenchymal cell origin.

228 and for their expression in macrophages and parenchymal cells outside of the tumors by immunohistoch

229 eport that targeted deletion of PBP in liver parenchymal cells (PBP(Liv-/-)) results in the abrogatio

230 Parenchymal cells (PC), Kupffer cells (KC), and liver en

231 t MyD88 in immune cells rather than in liver parenchymal cells plays an important role in inflammator

232 clei constitute only a small fraction of the parenchymal cell pool.

233 ression in crescentic GN, acting to regulate parenchymal cell populations by modulating both cell pro

234 proteins appear to be expressed in cerebral parenchymal cell populations that do not normally do so.

235 Small, extraportal, hepatic parenchymal cells, positive for biliary-type cytokeratin

236 e direct interaction between tumor cells and parenchymal cells predicted from experimental rodent met

237 after IRI, suggesting that CD47 signaling in parenchymal cells predominantly mediates renal damage.

238 otection; instead, they recognize antigen on parenchymal cells-presumably parasitized hepatocytes.

239 Whereas the initial step of parenchymal cell proliferation was not affected by acute

240 aken together, these data imply that CD47 on parenchymal cells promotes injury after renal ischemia a

241 ta provide evidence that PD-L1 expression on parenchymal cells rather than hematopoietic cells protec

242 he effects of TRPM2 are due to expression in parenchymal cells rather than hematopoietic cells.

243 Transfer of IFN-gamma into brain parenchymal cells rather than tumor is both necessary an

244 ar network must be organized so that all the parenchymal cells receive adequate nutrients.

245 e generated by continual processing of graft parenchymal cells; recognition of donor haemopoietic fra

246 long-term protection to ischemically injured parenchymal cells regardless of how effectively they can

247 n the thymus, whereas both hematopoietic and parenchymal cells regulated iNKT cell numbers in the per

248 onstrate that compartmental placement of non-parenchymal cells relative to primary or induced pluripo

249 muscle gene activation, which may facilitate parenchymal cell remodeling and impair graft function.

250 Therefore, parenchymal cells represent an effective and necessary t

251 Small, extraportal, biliary-type parenchymal cells represent cross-sections of the CoH th

252 In wild-type recipients, arterial and parenchymal cells showed increased MHC class II molecule

253 To do so, we created mice harboring liver parenchymal cell-specific deletion of HOIP (Hoip(Deltahe

254 ase of heart valves but it is not present in parenchymal cells such as hepatocytes and renal tubular

255 However, mechanisms governing its release by parenchymal cells such as hepatocytes are unknown.

256 AhR activation changes the response of lung parenchymal cells, such that regulatory pathways in the

257 emoving taurine released into the CSF during parenchymal cell swelling.

258 cated in the subendothelial space, and liver parenchymal cells, take on the roles of antigen-presenti

259 substrates in the bloodstream and the tissue parenchymal cells that require these substrates, a major

260 tigen was expressed on graft vascular and/or parenchymal cells, the outcome was acute graft destructi

261 ace between circulating cells and underlying parenchymal cells, the vascular endothelium provides sig

262 ominent GmPIP2,2 hybridization to RNA in the parenchymal cells tightly juxtaposed to the syncytium.

263 We have shown that parenchymal cell TLR4 activation drives LPS-induced syst

264 This study supports a key role of parenchymal cell TNFRs in lung injury induced by Pc and

265 alkaloid is sufficient to prime normal host parenchymal cells to be slowly replaced by transplanted

266 tegrate with host vessels and interface with parenchymal cells to form a functional tissue mimic.

267 range of stromal cells that co-develop with parenchymal cells to form tissues.

268 does not require cognate recognition of the parenchymal cells to occur.

269 n the capacity of the vasculature and/or the parenchymal cells to present Ag, the accumulation of cel

270 The inability of renal parenchymal cells to secrete IL-1beta may explain why ur

271 s sufficient to confer the capacity of islet parenchymal cells to stimulate allorejection.

272 HOIP deficiency in liver parenchymal cells triggered tumorigenesis at 18 months o

273 This suggests that a signal from parenchymal cells triggers neutrophil transmigration.

274 he intestinal epithelial cells (IEC) are the parenchymal cell type involved, a mouse model that expre

275 The parenchymal cell types whose TLR4 activation directs thi

276 ymphoid tissues and can mediate apoptosis of parenchymal cells upon injury and infiltration of inflam

277 show that targeted deletion of PBP in liver parenchymal cells, using the Cre-loxP system, results in

278 factors and individual cell types, including parenchymal cells, vascular cells, and immune cells, can

279 port here that transpresentation of IL-15 by parenchymal cells was crucial for generating normal numb

280 coprotein-receptor (ASGP-R) located on liver parenchymal cells was originally identified and characte

281 linear ubiquitination specifically in liver parenchymal cells, we investigated the physiological rol

282 Both lymphoid cells and parenchymal cells were apoptotic.

283 (0.5% w/w) for 2 wk, killed, and their liver parenchymal cells were isolated.

284 r for 1 mo, were sacrificed, and their liver parenchymal cells were isolated.

285 ry, neutrophils and macrophages, rather than parenchymal cells, were the source of T/HS lymph-induced

286 em detects viral infections and mutations in parenchymal cells when antigens from these cells are cro

287 ed TGs to FAs for their subsequent uptake by parenchymal cells, whereas intracellular lipolysis gener

288 a expression by both bone marrow-derived and parenchymal cells, whereas memory-phenotype CD8 T cells

289 tained within endosomes of hematopoietic and parenchymal cells, whereupon IgG is diverted from degrad

290 um composed by a heterogeneous population of parenchymal cells which are structurally integrated but

291 bility complex (MHC) ligand expressed on the parenchymal cell, which lacks appropriate costimulatory

292 large and progressively increasing number of parenchymal cells with aging.

293 s targeted injury in hepatic endothelial and parenchymal cells with suitable drugs will also help adv

294 (iNOS or NOS2) in cardiac myocytes and other parenchymal cells within the heart may in addition to co

295 capable of generating new blood vessels and parenchymal cells within tissues they have colonized has