ライフサイエンスコーパス: parenchymal tissue

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1 (larynx, trachea and bronchus) and the lung parenchymal tissue.

2 matory cytokines systemically and within the parenchymal tissues.

3 d found predominantly in the bone marrow; in parenchymal tissues.

4 nsported across the capillary endothelium to parenchymal tissues.

5 red mainly by their counterparts residing in parenchymal tissues.

6 ion leading to migration of lymphocytes into parenchymal tissues.

7 atched, paired NSCLC tumor and non-malignant parenchymal tissues.

8 ed a marked accumulation of iron in affected parenchymal tissues, a finding consistent with early wor

9 days before such T cells can enter the lung parenchymal tissue and airway.

10 pillary beds, efferent vein, and surrounding parenchymal tissue are explanted and maintained for 24 h

11 FN-gamma expression in cognate Ag-expressing parenchymal tissues as well as death via a mechanism tha

12 ve been implicated in the repair of inflamed parenchymal tissue, but the signals that regulate their

13 l dysfunction by day 15 and little remaining parenchymal tissue by day 20.

14 Clara cell of the airway protect surrounding parenchymal tissue by inducing apoptosis of Fas(+) immun

15 However, in parenchymal tissue, CBFv oscillated with peaks at both a

16 ompensation for loss of IP-10 in lymphoid vs parenchymal tissue compartments.

17 reased apoptosis of lymphoid organs and some parenchymal tissues contributes to this immune suppressi

18 hat neutrophil infiltration was required for parenchymal tissue damage in the brain.

19 Renal parenchymal tissues express a limited repertoire of rece

20 apid response mechanism that is operative in parenchymal tissues for effective defense against tissue

21 onstitutively in phagocytes and inducibly in parenchymal, tissue-forming cells.

22 etabolic rate of the vessel wall relative to parenchymal tissue in the rat mesentery suggests that in

23 or restore expression of B7-H1 expression by parenchymal tissues in the recipient could prevent or am

24 lobase message was present in multiple human parenchymal tissues including heart, skeletal muscle, pl

25 way hyperresponsiveness to methacholine, and parenchymal tissue inflammation were also dramatically r

26 tion of host hematopoietic cells but minimal parenchymal tissue injury.

27 contrast, infiltration of immune cells into parenchymal tissue is significantly delayed.

28 In chronic disease, failure to regenerate parenchymal tissue leads to the replacement of lost cell

29 The results suggest that parenchymal tissues may retain the capacity to directly

30 ng enzyme (ACE) in pulmonary artery and lung parenchymal tissues (obtained at the time of resection f

31 ome fibroblasts in various organs, including parenchymal tissue of the gut, lung, skin, and liver.

32 ted KT inhibitor associated with the gut and parenchymal tissues of the infective juvenile stage of F

33 the lung, unable to populate either the lung parenchymal tissue or the airway under homeostatic condi

34 lorative single-center clinical trial, renal parenchymal tissue perfusion of 32 stable kidney allogra

35 8(+) effector cells, and PD-L1 expression on parenchymal tissues protected against effector OT-I T ce

36 histologic evidence of ongoing vascular and parenchymal tissue remodeling, including interstitial fi

37 (TNF) superfamily is a conserved response of parenchymal tissues to injury and inflammation that comm

38 ve immunity, dendritic cells (DCs) move from parenchymal tissues to lymphoid organs by migrating alon

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