ライフサイエンスコーパス: parenteral nutrition

1 nutritional deficit for 1 week in ICU (late parenteral nutrition).

2 U) is clinically superior to providing early parenteral nutrition.

3 t length of stay in comparison with standard parenteral nutrition.

4 63% of the extra nitrogen intake from early parenteral nutrition.

5 ring intensive insulin therapy than is total parenteral nutrition.

6 nal and liver failure were gastroschisis and parenteral nutrition.

7 al and special population patients receiving parenteral nutrition.

8 Fluoride toxicity may be a concern in parenteral nutrition.

9 ented parenteral nutrition is better than no parenteral nutrition.

10 n insulin, intravenous amino acids, and full parenteral nutrition.

11 quality of life for patients living on home parenteral nutrition.

12 e typically administered with standard total parenteral nutrition.

13 ion for any patient permanently dependent on parenteral nutrition.

14 nal failure and 15% to 40% of adults on home parenteral nutrition.

15 more efficacious and poses lower risks than parenteral nutrition.

16 rrier function that is associated with total parenteral nutrition.

17 y increased with the administration of total parenteral nutrition.

18 de in neonatal intensive care and the use of parenteral nutrition.

19 lower than in wild-type mice receiving total parenteral nutrition.

20 oblastic MDS, even in patients not requiring parenteral nutrition.

21 U was clinically superior to providing early parenteral nutrition.

22 c dependence on central venous catheters for parenteral nutrition.

23 e a promising therapy to allow autonomy from parenteral nutrition.

24 to acquire intestinal failure requiring home parenteral nutrition.

25 d weight loss while receiving long term home parenteral nutrition.

26 y ill neonates, and patients receiving total parenteral nutrition.

27 enal replacement therapy observed with early parenteral nutrition.

28 odds ratio [95% confidence interval]): total parenteral nutrition (2.79 [1.26-6.17]), dialysis depend

29 P<0.001), and the incidence of use of total parenteral nutrition (31 percent vs. 55 percent, P<0.001

30 ar portion of the patients require long-term parenteral nutrition (86% vs. 84%) or have undergone int

31 Secondary outcomes included dependence on parenteral nutrition 90 days postoperatively and length

32 ared with 18.5% in the group receiving early parenteral nutrition (adjusted odds ratio, 0.48; 95% con

33 interval, 1.8 to 148.1) and receipt of total parenteral nutrition (adjusted odds ratio, 9.2; 95% conf

34 Total parenteral nutrition administration was associated with

35 s received olive oil-based lipid emulsion in parenteral nutrition (age 46 +/- 19 yrs, body mass index

36 al of 49 patients received soybean oil-based parenteral nutrition (age 51 +/- 15 yrs, body mass index

37 Early parenteral nutrition also increased the volume of adipos

38 s indicate that copper requirements in total parenteral nutrition amount to 0.3 mg/day in the adult.

39 Parenteral nutrition and elemental diets both cause bact

40 inct physiological differences between total parenteral nutrition and enteral nutrition that are more

41 Survivors are off parenteral nutrition and have demonstrated significant g

42 eatment included application of colchicines, parenteral nutrition and magnesium substitution, antiper

43 e's use in critically ill patients requiring parenteral nutrition and new data reveal safety and effi

44 do not, and why some patients tolerate total parenteral nutrition and others develop liver dysfunctio

45 el syndrome (SBS) is now possible because of parenteral nutrition and small bowel transplantation.

46 d over the past few decades, especially with parenteral nutrition and surgical repair.

47 illus atrophy by the administration of total parenteral nutrition, and a model of villus hypertrophy

48 tidisciplinary approach to the management of parenteral nutrition, and aseptic catheter techniques to

49 eservation of venous access, "hepatosparing" parenteral nutrition, and avoidance of liver sepsis are

50 mined include patient survival, weaning from parenteral nutrition, and need for intestinal transplant

51 itive function, decreased functional status, parenteral nutrition, and pressure ulcers.

52 iglycerides, therapeutic paracentesis, total parenteral nutrition, and somatostatins.

53 functional compromise induced by fasting and parenteral nutrition, and the enhanced adaptive capacity

54 support various beliefs about the utility of parenteral nutrition, and then to critically evaluate th

55 However, amino acids in parenteral nutrition are effective for increasing protei

56 ection was 10.7% in the group receiving late parenteral nutrition, as compared with 18.5% in the grou

57 s 6.5+/-0.4 days in the group receiving late parenteral nutrition, as compared with 9.2+/-0.8 days in

58 ations of portal hypertension resulting from parenteral nutrition associated cholestasis.

59 Parenteral nutrition-associated liver disease (PNALD) is

60 Parenteral nutrition-associated liver disease (PNALD) is

61 ous lipid emulsion (ILE) in the treatment of parenteral nutrition-associated liver disease (PNALD).

62 Parenteral nutrition-associated liver disease is reversi

63 If high survival could be achieved and if parenteral nutrition-associated liver disease were rever

64 titis, primary sclerosing cholangitis, total parenteral nutrition-associated liver disease, and cysti

65 ts with short-gut syndrome may develop total parenteral nutrition-associated liver disease, which may

66 ns, phytosterols may promote liver injury in parenteral nutrition-associated liver disease.

67 All surviving patients weaned-off total parenteral nutrition at a median time of 32 days and 90%

68 ose into the systemic circulation with total parenteral nutrition at rates that approximate usual pos

69 estine and from 13 SBS patients dependent on parenteral nutrition because of chronic malabsorption.

70 Enteral or parenteral nutrition before, during, and after CABG may

71 tation of these amino acids with enteral and parenteral nutrition before, during, and after surgery m

72 All children were weaned from parenteral nutrition between 31 and 85 d posttransplanta

73 ailure and life-threatening complications of parenteral nutrition, but it is still plagued by high le

74 se receiving omega-3 fatty acid supplemented parenteral nutrition, but results were strongly influenc

75 The cost of parenteral nutrition compared to intestinal transplantat

76 pecified analysis from this trial, the Early Parenteral Nutrition Completing Enteral Nutrition in Adu

77 tudy of a randomized controlled trial (Early Parenteral Nutrition Completing Enteral Nutrition in Adu

78 with insulin did not lower glucagon, whereas parenteral nutrition containing amino acids increased gl

79 Parenteral nutrition containing soybean oil-based (Intra

80 The administration of parenteral nutrition containing soybean oil-based and ol

81 n = 61) or early parenteral nutrition (early parenteral nutrition, control) by jugular vein catheter

82 ature infants, patients with long-term total parenteral nutrition, Crohn's disease, cystic fibrosis,

83 are important adjuncts to the elimination of parenteral nutrition dependence and need for intestinal

84 once biochemical cholestasis is detected in parenteral nutrition-dependent patients is recommended.

85 ajor cause of morbidity and mortality in the parenteral nutrition-dependent population.

86 sed for augmentation of energy absorption in parenteral nutrition-dependent subjects with short bowel

87 Parenteral nutrition depressed the selective transport i

88 tral line, and had 1 additional risk factor (parenteral nutrition, dialysis, surgery, pancreatitis, s

89 The timing of parenteral nutrition did not affect the incidence of AKI

90 Early parenteral nutrition did not affect the time course of c

91 Early parenteral nutrition did not prevent the pronounced wast

92 d that omega-3 fatty acid supplementation of parenteral nutrition does not improve mortality, infecti

93 sed pulmonary risk, routine total enteral or parenteral nutrition does not reduce risk (1 meta-analys

94 In conclusion, early parenteral nutrition does not seem to impact AKI inciden

95 mental changes in the nature and practice of parenteral nutrition during the review period, there hav

96 ntion) by nasojejunal tube (n = 61) or early parenteral nutrition (early parenteral nutrition, contro

97 However, energy provision with parenteral nutrition, either instead of or supplemental

98 atic review assessed 37 trials that compared parenteral nutrition, enteral nutrition, or nutritional

99 e randomly assigned to EEN (n = 61) or early parenteral nutrition (EPN, n = 62) in addition to an ora

100 attributable to increasing complications of parenteral nutrition, especially infectious complication

101 efine the parameters that best predict total parenteral nutrition failure and the unique mechanisms t

102 complicated by short gut syndrome and total parenteral nutrition failure.

103 ing an active infection, seven of nine total parenteral nutrition-fed animals continued to have viral

104 children to investigate whether withholding parenteral nutrition for 1 week (i.e., providing late pa

105 In critically ill children, withholding parenteral nutrition for 1 week in the ICU was clinicall

106 placement therapy, compared with withholding parenteral nutrition for 1 week.

107 an or equal to 13, expected to require total parenteral nutrition for at least 5 days.

108 r a recipient of CLDILTx, currently on total parenteral nutrition for late fistula.

109 ntilated within 48 hours, received exclusive parenteral nutrition for more than or equal to 5 days, a

110 with type 2 diabetes and a child on chronic parenteral nutrition for short bowel syndrome.

111 ased parenteral nutrition or olive oil-based parenteral nutrition for up to 28 days.

112 35%) of those who were alive at 28 days were parenteral nutrition free.

113 High patient survival and parenteral nutrition-free survival can be achieved after

114 76%, p = .03), and a decrease in patients on parenteral nutrition (from 26% to 21%, p = .04).

115 rbachol and glucose were higher in the total parenteral nutrition group compared with the control gro

116 Ion transport in the total parenteral nutrition group was significantly increased.

117 scharge from the ICU at any time in the late-parenteral-nutrition group (adjusted hazard ratio, 1.23;

118 d with soybean oil-based and olive oil-based parenteral nutrition had a similar length of stay (47 +/

119 ndomized trials have found that supplemental parenteral nutrition has a deleterious effect in compari

120 Parenteral nutrition has achieved extended success for t

121 Parenteral nutrition has been associated with metabolic

122 Total parenteral nutrition has significant effects on intestin

123 The indications for parenteral nutrition have been examined over the past ye

124 Home parenteral nutrition (HPN) and intestinal transplantatio

125 intestinal macronutrient absorption and home parenteral nutrition (HPN) dependence in SBS patients wi

126 We aimed to review the indications for home parenteral nutrition (HPN) in children and describe the

127 testinal failure (IF) and dependency on home parenteral nutrition (HPN).

128 eling are usually ineffective, intradialytic parenteral nutrition (IDPN) has been proposed as a poten

129 A recent study showed that intradialytic parenteral nutrition (IDPN) improves whole-body protein

130 Intradialytic parenteral nutrition (IDPN) reverses the net negative wh

131 Intradialytic parenteral nutrition (IDPN), with or without exercise, h

132 Whether early versus late parenteral nutrition impacts the incidence and recovery

133 -abdominal abscess in 23 patients, and total parenteral nutrition in 74 (36%).

134 tinal absorption at the time of weaning from parenteral nutrition in a series of children after intes

135 ntration greater than 7 g/dl; (3) do not use parenteral nutrition in adequately nourished critically

136 trials have questioned the benefit of early parenteral nutrition in adults.

137 ed trials of omega-3 fatty acid supplemented parenteral nutrition in critically ill adult patients ad

138 Use of early parenteral nutrition in critically ill patients in whom

139 GLP-2R signaling reduces the requirement for parenteral nutrition in human subjects with short-bowel

140 Enteral nutrition may be more effective than parenteral nutrition in limiting proteolysis and produci

141 guidelines recommend the use of enteral over parenteral nutrition in patients undergoing gastrointest

142 a specialized diet will reduce the need for parenteral nutrition in patients with short bowel syndro

143 gonlike peptide 2 that reduces dependence on parenteral nutrition in patients with short bowel syndro

144 l nutrition for 1 week (i.e., providing late parenteral nutrition) in the pediatric intensive care un

145 l measure in humans (such as those receiving parenteral nutrition) in whom choline deficiency is susp

146 The administration of parenteral nutrition, including lipid emulsion (LE), to

147 to B (stable in C), and days on combined and parenteral nutrition increased progressively.

148 Furthermore, early parenteral nutrition increased the amount of adipose tis

149 cholangitis, cholestasis of pregnancy, total parenteral nutrition-induced cholestasis, and drug-induc

150 ss the role of interferon-gamma on the total parenteral nutrition-induced loss of epithelial barrier

151 In the group that received late parenteral nutrition, infusing amino acids after the fir

152 meliorates hepatic steatosis associated with parenteral nutrition infusion.

153 There are no convincing data that parenteral nutrition is beneficial in severely malnouris

154 but it is unknown if glutamine-supplemented parenteral nutrition is better than no parenteral nutrit

155 Parenteral nutrition is central to the care of very imma

156 not uniformly been able to demonstrate that parenteral nutrition is efficacious in acute pancreatiti

157 ong-term survival of patients with continued parenteral nutrition is higher than after intestinal tra

158 The relative safety of long-term parenteral nutrition is reconfirmed.

159 Total parenteral nutrition led to a loss of EBF, and this was

160 Parenteral nutrition may help to meet many of the nutrit

161 o popular belief, appropriately administered parenteral nutrition may provide similar or more benefit

162 bile salts, as occurs during starvation and parenteral nutrition, may have a detrimental effect on m

163 meability in interferon-gamma knockout total parenteral nutrition mice was significantly lower than i

164 ough there is evidence to suggest that total parenteral nutrition more effectively spares protein in

165 These factors may make total parenteral nutrition more efficacious, at least initiall

166 Parenteral nutrition needs attention to detail if it is

167 teral nutrition produces fewer problems than parenteral nutrition; no data suggest that either modali

168 ortality was higher in patients who received parenteral nutrition (odds ratio 2.61 [1.3, 5.3], p = .0

169 : odds ratio, 2.65; 95% CI, 1.93-3.63; total parenteral nutrition: odds ratio, 3.27; 95% CI, 2.24-4.7

170 The effect of early parenteral nutrition on clinical outcomes in critically

171 To investigate the effect of total parenteral nutrition on intestinal ion transport, we use

172 assess the effect of early administration of parenteral nutrition on muscle volume and composition by

173 Wild-type mice received total parenteral nutrition or enteral diet (control group) for

174 were randomized to either soybean oil-based parenteral nutrition or olive oil-based parenteral nutri

175 fluoride parenterally would prevent or treat parenteral nutrition osteopenia.

176 considerable amount of work in the field of parenteral nutrition over the last year.

177 The use of total parenteral nutrition (p = 0.03), longer duration of anti

178 rase and alkaline phosphatase than was early parenteral nutrition (P=0.001 and P=0.04, respectively),

179 echanical ventilatory support than was early parenteral nutrition (P=0.001), as well as a smaller pro

180 For the 723 patients receiving early parenteral nutrition, parenteral nutrition was initiated

181 whereas for the 717 patients receiving late parenteral nutrition, parenteral nutrition was not provi

182 Use of alternative IV fat emulsions in parenteral nutrition, particularly olive and fish oil, w

183 When compared with lipid-free parenteral nutrition, patients who received fish oil had

184 Seven were put on total parenteral nutrition plus octreotide.

185 Sixteen patients were on parenteral nutrition (PN) after 74 PN months (range, 2.5

186 Discontinuation of parenteral nutrition (PN) after reSTEP was achieved in 6

187 Compounding pharmacies often prepare parenteral nutrition (PN) and must adhere to rigorous st

188 me (SBS) who were "permanently" dependent on parenteral nutrition (PN) and to identify possible progn

189 Long-term parenteral nutrition (PN) carries the risk of progressiv

190 Experimentally, parenteral nutrition (PN) decreases GALT cell mass and m

191 The impact of dilatation on parenteral nutrition (PN) dependence and survival has no

192 not routinely added that should be part of a parenteral nutrition (PN) formula?

193 Standard trace element-supplemented neonatal parenteral nutrition (PN) has a high manganese content a

194 lant sterols, including stigmasterol, during parenteral nutrition (PN) have been linked with serum bi

195 ded and the means of delivery in patients on parenteral nutrition (PN) have been unclear.

196 Parenteral nutrition (PN) impairs mucosal immunity and i

197 Efforts to optimize early parenteral nutrition (PN) in extremely low-birth-weight

198 Parenteral nutrition (PN) increases risks of infections

199 The use of early parenteral nutrition (PN) is one potential strategy to a

200 Parenteral nutrition (PN) is still widely preferred to e

201 Parenteral nutrition (PN) is the main treatment for inte

202 ILE based on soybean oil administered with parenteral nutrition (PN) may contribute to its etiology

203 testinal failure (IF) treated with long-term parenteral nutrition (PN) may present with low bone mine

204 arly enteral nutrition (EN) may benefit from parenteral nutrition (PN) provided within 24 hours of IC

205 her (a) 0.9% sodium chloride, (b) a standard parenteral nutrition (PN) solution without glutamine, or

206 For children and adults on parenteral nutrition (PN) the main mortality risk factor

207 em cell transplantation (HSCT) often require parenteral nutrition (PN) to optimize caloric intake.

208 rol concentrations are an untoward effect of parenteral nutrition (PN) with vegetable oil-based lipid

209 Of this group, 41% were receiving parenteral nutrition (PN), 41% were receiving enteral fe

210 Total parenteral nutrition (PN), including fat administered as

211 raphy or ultrasonography), laboratory tests, parenteral nutrition (PN), peripherally inserted central

212 Infants with intestinal failure who are parenteral nutrition (PN)-dependent may develop cholesta

213 ldren with intestinal failure (IF) depend on parenteral nutrition (PN).

214 ed intravenously to all patients who require parenteral nutrition (PN).

215 ection for mesenteric infarction may require parenteral nutrition (PN).

216 with short bowel syndrome (SBS) who require parenteral nutrition (PN).

217 ical illness and are allegedly aggravated by parenteral nutrition (PN).

218 Patients received total parenteral nutrition prepared either with a lipid emulsi

219 Late parenteral nutrition prevented infections and accelerate

220 ne therapy/apnea of prematurity, duration of parenteral nutrition, pulmonary hemorrhage, and white ma

221 Twenty-six patients who had failed parenteral nutrition received 28 isolated intestinal tra

222 Early parenteral nutrition reduced the quality of the muscle t

223 t multivisceral transplantation due to total parenteral nutrition-related liver disease.

224 omography severity index score at admission, parenteral nutrition requirement before or after radiolo

225 lutamine and diet in hopes of reducing their parenteral nutrition requirements.

226 The use of parenteral nutrition should be limited within the first

227 tory liver dysfunction in patients receiving parenteral nutrition should prompt consideration for iso

228 Total parenteral nutrition significantly increased small-bowel

229 Parenteral nutrition significantly reduced virus-specifi

230 Glutamine supplementation of parenteral nutrition solutions may reduce the infectious

231 rtage of injectable zinc available for total parenteral nutrition supplementation over the last 2 yea

232 Severe malabsorption required parenteral nutrition support for longer than 1.5 years;

233 they represent a major population requiring parenteral nutrition support for survival.

234 ea and failure to thrive, required prolonged parenteral nutrition support, and had high mortality.

235 ld promise as aids in restoring freedom from parenteral nutrition support; however, their long-term b

236 Patients receiving long-term home parenteral nutrition tend to fall under the care of adul

237 much higher serum insulin responses to total parenteral nutrition than with enteral nutrition that ap

238 ctors influence survival of patients on home parenteral nutrition, the costs related to this therapy,

239 accepted therapy for intestinal failure when parenteral nutrition therapy cannot be tolerated.

240 that hepatic steatosis, which occurs during parenteral nutrition therapy, develops as a result of ch

241 e most devastating complication of long-term parenteral nutrition therapy.

242 9.2+/-0.8 days in the group receiving early parenteral nutrition; there was also a higher likelihood

243 d a few days after the child had weaned from parenteral nutrition to exclusive enteral tube feeding.

244 To assess the recent literature regarding parenteral nutrition to identify publications that have

245 There is no role for supplemental parenteral nutrition to increase caloric delivery in the

246 Copper supplementation is essential in parenteral nutrition to prevent an adverse effect of def

247 be in part responsible for the inability of parenteral nutrition to reduce proteolysis in preterm in

248 randomized controlled trial found that early parenteral nutrition to supplement insufficient enteral

249 al failure and require total or supplemental parenteral nutrition (TPN or PN, respectively).

250 Enteral nutrient deprivation via total parenteral nutrition (TPN) administration leads to local

251 ons in hospitalized patients beginning total parenteral nutrition (TPN) and whether a 3-d regimen of

252 o 60% of infants who require long-term total parenteral nutrition (TPN) for intestinal failure and 15

253 Total parenteral nutrition (TPN) is an invasive and advanced r

254 Total parenteral nutrition (TPN) is commonly used clinically t

255 Total parenteral nutrition (TPN) leads a loss of epithelial ba

256 d that enteral nutrient deprivation or total parenteral nutrition (TPN) led to a loss of intestinal e

257 roplasty [STEP]) in terms of survival, total parenteral nutrition (TPN) weaning, and complications.

258 l early enteral nutrition (NJEEN) with total parenteral nutrition (TPN), after pancreaticoduodenectom

259 al interest, with special reference to total parenteral nutrition (TPN), an area in which I have been

260 d antifungals, and ICU factors such as total parenteral nutrition (TPN), blood product transfusions,

261 n several models, including rats given total parenteral nutrition (TPN), IGF-I more potently stimulat

262 We utilized a model of total parenteral nutrition (TPN), or enteral nutrient deprivat

263 el of enteral nutrient deprivation, or total parenteral nutrition (TPN), resulting in intestinal muco

264 Mice that received total parenteral nutrition (TPN), which deprives the animals o

265 ailure patients do well with long-term total parenteral nutrition (TPN), while others develop life-th

266 Total parenteral nutrition (TPN), with the complete removal of

267 He recovered from PTLD but developed total parenteral nutrition (TPN)-induced liver failure.

268 nto the veins of rats by the method of total parenteral nutrition (TPN).

269 flammation are common complications of total parenteral nutrition (TPN).

270 of gut failure (GF) with the need for total parenteral nutrition (TPN).

271 as provided to rats for 7 days by oral total parenteral nutrition (TPN; elemental diet) 307 kcal/kg/d

272 itation with either jejunal tube feedings or parenteral nutrition until weight gain results in relief

273 judicious jejunostomy tube feeding, or total parenteral nutrition usage may reduce morbidity.

274 d with improved 60-day survival; conversely, parenteral nutrition use was associated with higher mort

275 Is parenteral nutrition via peripherally inserted central c

276 Compared with short peripheral cannulas, parenteral nutrition via PICCs is associated with better

277 hospital blood glucose concentration during parenteral nutrition was 129 +/- 14 mg/dL, without diffe

278 ars, and time from ITx to cessation of total parenteral nutrition was 31 days.

279 ent in protein and calories when appropriate parenteral nutrition was added to enteral sources.

280 Late parenteral nutrition was also associated with lower plas

281 Late parenteral nutrition was associated with a shorter durat

282 inued, diarrhea had completely resolved, and parenteral nutrition was discontinued.

283 Early parenteral nutrition was given as control nutrition to o

284 tients receiving early parenteral nutrition, parenteral nutrition was initiated within 24 hours after

285 atients receiving late parenteral nutrition, parenteral nutrition was not provided until the morning

286 Parenteral nutrition was slowly tapered while increasing

287 se as survival, macronutrient absorption and parenteral nutrition weaning are improved after autologo

288 ety and efficacy of teduglutide as an aid to parenteral nutrition weaning.

289 percentages of infants who depended on total parenteral nutrition were 17 of 36 (47.2 percent) in the

290 -chain triglyceride, olive, and fish oils in parenteral nutrition were compared using an adjusted Cox

291 ic use in patients also receiving enteral or parenteral nutrition were included in the review.

292 r size larger than 10 cm, and need for total parenteral nutrition were shown to further define surviv

293 EPaNIC]), which compared early initiation of parenteral nutrition when enteral nutrition was insuffic

294 rd therapy for short bowel syndrome is total parenteral nutrition, which is expensive and associated

295 Patients received total parenteral nutrition with standard (1.5 g . kg(-1) . day

296 en enteral nutrition was insufficient (early parenteral nutrition) with tolerating a pronounced nutri

297 and a need for opioid analgesic and enteral/parenteral nutrition, with an effect on patient survival

298 alue (hypocaloric) via enteral tube feeds or parenteral nutrition, with an equal protein allocation i

299 ddition to their routine care, compared with parenteral nutrition without omega-3 fatty acid suppleme

300 ur because of poor dietary intake, long-term parenteral nutrition without supplementation, and entera