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1 be useful as a model stimulus for studies of paresthesia.
2 nderstood conditions such as hyperpathia and paresthesias.
3 OX4 was associated with more neutropenia and paresthesias.
4 ation was commonly associated with transient paresthesias.
5 man presented with blurred vision and distal paresthesias.
6 ofacial paresthesia (4 events [20%]), finger paresthesia (1 event [5%]), and ataxia (1 event [5%]).
9 (25% vs. 12%) in the amitriptyline group and paresthesia (31% vs. 8%) and weight loss (8% vs. 0%) in
10 che (46.8%), fatigue (44.2%), malaise (39%), paresthesias (32.5%), peripheral facial palsy (PFP) (36.
11 ted symptoms, most commonly dizziness (59%), paresthesia (34%), euphoria (30%), and hypoesthesia (30%
12 her persistent adverse events were orofacial paresthesia (4 events [20%]), finger paresthesia (1 even
13 opiramate vs placebo, respectively, included paresthesia (50.8% vs 10.6%), taste perversion (23.0% vs
16 05), and incidences of peripheral neuropathy/paresthesia and alopecia were significantly higher in pa
20 latin neuropathy is characterized by sensory paresthesias and muscle cramps that are notably exacerba
21 hich began with vomiting and upper extremity paresthesias and progressed to fever, seizures, dysphagi
23 difficulties, 4 (9%) a sensation of left arm paresthesia, and 3 (7%) sympathetic flight/fright respon
24 norexia, dysgeusia, diarrhea, fatigue, pain, paresthesia, and dyspnea were translated into Italian an
30 erum creatinine level, insomnia, leg cramps, paresthesias, and tremor, were managed with dose reducti
31 Raynaud's phenomenon as persistent pain and paresthesia are common in the hands and arms and occur i
32 Sensory abnormalities such as numbness and paresthesias are often the earliest symptoms in neuroinf
33 lications assessed included axillary seroma, paresthesia, arm morbidity and range of motion, and lymp
34 nausea) and neurological symptoms (extremity paresthesia, arthralgia, myalgia, malaise, pruritus, hea
35 rmation on wound infection, axillary seroma, paresthesia, brachial plexus injury (BPI), and lymphedem
36 ly ash plant family, induces robust tingling paresthesia by activating a subset of somatosensory neur
37 thy in humans that is accompanied by painful paresthesias, dysesthesias, and occasionally hypoesthesi
44 al anesthesia/analgesia range from transient paresthesias (<10%) to potentially devastating epidural
46 frequency of blepharoptosis, skin tightness, paresthesias, neck stiffness, muscle weakness, and neck
47 Self-resolving perineal abrasion and focal paresthesia of the glans penis each occurred in one pati
48 fifty-three (76.5%) patients had numbness or paresthesias of the medial arm and/or axilla after surge
50 sinophilia, particularly in association with paresthesias or hyperesthesias, should alert clinicians
52 (P <or= .0016), seromas (P <or= .0001), and paresthesias (P <or= .0001) than those in the SLND-alone
53 or no diagnostic value, including nocturnal paresthesias; Phalen and Tinel signs; thenar atrophy; an
54 e fatigue, difficulty lifting, and extremity paresthesias) represented strongly clustered symptoms; w
55 tient satisfaction level were discomfort and paresthesia, satisfaction with appearance, and ability t
57 tallic taste, visual disturbance, circumoral paresthesia, temperature reversal, or toothache) or syst
59 and molecular mechanisms underlying tingling paresthesia that accompanies peripheral neuropathy and i
61 ted in both treatment groups, but tremor and paresthesia were more frequent in the tacrolimus group.
64 an increased incidence of hypertrichosis and paresthesia, were observed in the patients treated with
65 ) presented with episodic diplopia or facial paresthesias with subsequent brainstem and occasionally
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