ライフサイエンスコーパス: parietal region

1 he P400cz has its major source in the medial parietal region.

2 in the left and right amygdala and the right parietal region.

3 dent on computations implemented within this parietal region.

4 periventricular regions of interest and the parietal region.

5 usions about the evolution of this posterior parietal region.

6 x that most severely affects the frontal and parietal regions.

7 ortex and in the right premotor and inferior parietal regions.

8 r tracts that connect frontal, temporal, and parietal regions.

9 nt ERP or fMRI retinotopic memory effects in parietal regions.

10 glia, but not in the frontal, occipital, and parietal regions.

11 related to a loss of deactivation in medial parietal regions.

12 and midbrain as well as select temporal and parietal regions.

13 ctivation (FE>NE) in the midline frontal and parietal regions.

14 entral midline region, with a later onset in parietal regions.

15 yrification in women than men in frontal and parietal regions.

16 ntrolled' system depends upon prefrontal and parietal regions.

17 edial and inferior temporal, prefrontal, and parietal regions.

18 from the phonological store in temporal and parietal regions.

19 ty in the lateral premotor and inferolateral parietal regions.

20 ter hyperintensity growth in the frontal and parietal regions.

21 the left-temporal, right-temporal, and right-parietal regions.

22 l, precuneus, left postcentral, and inferior parietal regions.

23 cortex, dorsolateral prefrontal cortex, and parietal regions.

24 l inference process with specific visual and parietal regions.

25 may indicate the functional role of specific parietal regions.

26 o-parietal cortex and more posterior temporo-parietal regions.

27 teral temporal, lateral parietal, and medial parietal regions.

28 iated with cortical thinning in temporal and parietal regions.

29 e of posture on primary motor, premotor, and parietal regions.

30 ct category decoding in occipitotemporal and parietal regions.

31 bal fluency scores to atrophy in frontal and parietal regions.

32 dent of the integrity of areas MT/V5, V3A or parietal regions.

33 x, posterior basal ganglia, and thalamic and parietal regions.

34 the motor regions, including prefrontal and parietal regions.

35 e CMRglc of the conscious brain (e.g., right parietal region, 99.6 +/- 10.2 mumol/100 g/min; n = 6) w

36 In inferior temporal, frontal, and parietal regions, a gradual buildup in activity peaking

37 ew effect-related activations in frontal and parietal regions, a pattern not seen in control subjects

38 wo different pathways connecting frontal and parietal regions: a corticocortical pathway and a subcor

39 correction), and less grey matter in several parietal regions (all p<0.002 uncorrected and corrected

40 lvian (pMS) sulcus in the posterior inferior parietal region also results in an equally severe impair

41 al, working memory in ventral prefrontal and parietal regions, although they showed less verbal super

42 activation in the left prefrontal and right parietal regions, an effect not observed at any time poi

43 in particular, the activity in the inferior parietal region and in the anterior-medial cerebellum wa

44 with white matter volume in the left temporo-parietal region and that white matter volume influenced

45 on at the perceptual level occurs within the parietal region and the interaction at categorical decis

46 riod involved direction-specific activity in parietal regions and both dorsal and ventral sensory reg

47 groups showed reduced glucose metabolism in parietal regions and in middle and superior temporal reg

48 ficantly reduced connectivity in the PCC and parietal regions and increased frontal connectivity arou

49 mporal regions and some thinning in inferior parietal regions and the posterior cingulate (P < 0.001)

50 bjects between activation in the frontal and parietal regions and the rate of correct responses on th

51 UVR (P < .05 for medial temporal and lateral parietal regions) and gray matter volumes (all 4 ROIs; P

52 edial temporal, lateral temporal, and medial parietal regions) and gray matter volumes (P < .05 for m

53 he first trimester (eyes, midface, chin, and parietal region), and binge-level exposure in the first

54 ted with retinotopic activity in frontal and parietal regions, and assessed whether retinotopic activ

55 ral prefrontal cortex and medial and lateral parietal regions, and between the medial prefrontal cort

56 as increased gamma band power in frontal and parietal regions, and decreased spectral power in theta,

57 ral activity in hippocampal, prefrontal, and parietal regions, and that age-related memory impairment

58 nd neighboring motor, premotor, and inferior parietal regions, and tonic components, centered on oper

59 ncy of around 14 Hz, occurring in the centro-parietal region; and slow spindles, with a frequency of

60 trophy rates in the left medial-temporal and parietal regions; and (ii) in contrast, increased amyloi

61 orienting, which may be mediated by lateral parietal regions; and the experience of joint attention

62 Dorsal striatum, insula, posterior parietal regions, anterior and posterior cingulate, and

63 dings are discussed in the context that left parietal regions are critical for the modification of st

64 us, our results show that left but not right parietal regions are critical for visuomotor adaptation.

65 The frontal, temporal, and parietal regions are heteromodal association cortices th

66 If certain parietal regions are involved in action understanding, t

67 These results suggest that parietal regions are part of a network of brain areas th

68 information content, a number of frontal and parietal regions are thought to be domain- and process-g

69 In the parietal region, area 45 is connected with the angular g

70 ness, delta waves propagated from frontal to parietal regions as a traveling wave.

71 further indicated several common frontal and parietal regions as being involved in the control of bot

72 m, extending laterally into adjacent temporo-parietal regions as well as splenium and fornix.

73 vely with activity in several prefrontal and parietal regions (as measured by fMRI).

74 n the right parietal and left prefrontal and parietal regions, as early as acute phase I.

75 pattern similarity in lateral prefrontal and parietal regions, as well as in anterior and posterior m

76 ing working memory in the medial frontal and parietal regions bilaterally and in the right dorsolater

77 not only adjoining prefrontal, temporal and parietal regions but also bilateral caudate and left put

78 fects in posterior cingulate, precuneus, and parietal regions but also significant positive associati

79 se in activation (right prefrontal and right parietal regions) but less in regions that showed a satu

80 ckout mice, particularly in both rostral and parietal regions, but not caudal cortex.

81 Cortical thickness in the parietal regions by risk status.

82 several areas of the human brain, including parietal regions, can respond multimodally.

83 edicted fear included superior-occipital and parietal regions, cerebellum and prefrontal cortex.

84 orsomedial frontal, and insular and inferior parietal regions closely similar to the human counterpar

85 wed reduced activation in bilateral temporal-parietal regions compared to the other groups, which did

86 us and widespread frontal, frontolimbic, and parietal regions compared with HC subjects.

87 Fronto-parietal regions connected by this tract were activated

88 , the intraparietal sulcus, and the inferior parietal region), consistent with neural circuits that h

89 where distinct pathways between frontal and parietal regions contribute to multiple spatial represen

90 We addressed how human fronto-parietal regions control visuospatial attention on a fin

91 First, visual and parietal regions coordinated with sensorimotor and premo

92 Lower FA in the parietal region correlated significantly with higher Yal

93 o the subgenual cingulate and prefrontal and parietal regions correlated with BDI scores and [(18)F]P

94 Although a superior parietal region demonstrated the retrieval success patte

95 These results suggest that DMN parietal regions directly supported memory retrieval, wh

96 d with enhancement of gamma power in a right parietal region during movement execution as well as gam

97 increase of theta power over that same right parietal region during movement planning, which is corre

98 ateralized network of frontal, temporal, and parietal regions during a variety of semantic processing

99 k-induced decrease in BOLD signal) in medial parietal regions during successful compared with failed

100 teral activation of prefrontal and posterior parietal regions during successful identification of old

101 y the task (prefrontal, medial temporal, and parietal regions) during encoding were similar to those

102 vision of trigeminal nerve, neck, nose, jaw, parietal region, ear, teeth, eyebrow, shoulder (ipsilate

103 memory encoding task-related deactivation in parietal regions (eg, mean [SD] parameter estimates for

104 ivation in the dyslexic group, only the left parietal region exhibited reduced gray matter volume rel

105 oinsular, thalamus, cerebellum and bilateral parietal regions for C9P FTLD relative to C9N FTLD, and

106 of memory-related activations in frontal and parietal regions for retrieval of scenes and the absence

107 In bilateral posterior parietal regions, greater activation was associated with

108 Whereas some parietal regions have specific motor functions, others a

109 Evidence from lesion patients suggests that parietal regions house supramodal representations of spa

110 s located predominantly in left temporal and parietal regions (i.e. the superior temporal sulcus, inf

111 differential connectivity with occipital and parietal regions implicated in the processing of low-lev

112 i, high attentional demands suppress ventral parietal regions important for veridical remembering.

113 n imaging studies have implicated a specific parietal region in symbolic number processing, leading t

114 These findings indicate a critical role for parietal regions in action planning when there is respon

115 xtension of cortical thinning in the temporo-parietal regions in addition to frontal atrophy could be

116 EG theta activity over frontal, temporal and parietal regions in response to negative stimuli in SAD

117 nently in the inferior temporal and inferior parietal regions in the full cohort, with florbetapir po

118 l temporal areas as well as lateral temporal-parietal regions in the left hemisphere.

119 oke patients with focal damage to frontal or parietal regions in the left or right brain hemispheres

120 atter in left superior temporal and inferior parietal regions in the PPA group.

121 ed at recording locations over the posterior parietal regions in the vertical direction.

122 nnectivity was reduced between S1 and fronto-parietal regions, in both the TMSR and non-TMSR patients

123 one proceeds from area 30 toward the medial parietal regions, including areas 3, 1, 2, 5, 31, and th

124 re associated with activation in frontal and parietal regions, including bilateral dorsal premotor co

125 to pop-out) were observed in several fronto-parietal regions, including IPS, FEF, MFG and IFG, in ad

126 es depend on a shared network of frontal and parietal regions, including white matter association tra

127 y a shared network of frontal, temporal, and parietal regions, including white matter association tra

128 the topography and response profile of human parietal regions inside and outside the DMN, independent

129 related to recollection and a more superior parietal region involved in familiarity.

130 That grey matter loss in parietal regions is a part of healthy aging suggests tha

131 ional and structural connectivity in temporo-parietal regions known to have high MET expression, part

132 and S1 and of both these regions with fronto-parietal regions, known to be important for multisensory

133 ained by event-related potentials around the parietal regions: low performers showed increased wavefo

134 mi-spatial neglect suggest that the inferior parietal region may lie at the junction of the visual an

135 The medial parietal region may, then, be conceived of as a nodal st

136 The aim of this work was to demonstrate that parietal regions may mediate selective attention to moti

137 alyses indicated that lateral prefrontal and parietal regions may mediate the relation between abilit

138 task, and they thus suggest that frontal and parietal regions may play a general role in visual aware

139 c resonance (ER-fMRI), we show that distinct parietal regions mediated these different attentional pr

140 e matter diffusion anisotropy in the temporo-parietal region of the left hemisphere was significantly

141 matter was thinner in frontal, temporal and parietal regions of both brain hemispheres.

142 dominance based on either frontal or temporo-parietal regions of interest (ROIs) defined for the enti

143 re significantly higher in the occipital and parietal regions of the adults with Down's syndrome than

144 production are depressed in the temporal and parietal regions of the cortex in patients with AD, we d

145 nectivity between frontoparietal systems and parietal regions of the dorsal attention network involve

146 elation between the default mode network and parietal regions of the dorsal attention network is cons

147 ng metabolism in the prefrontal and inferior parietal regions (P < 0.001).

148 eas connections originating from frontal and parietal regions peaked at beta frequency.

149 antic enhancement effects within frontal and parietal regions, perhaps reflecting downstream attempts

150 tion of the posterior cingulate and inferior parietal regions persisted in the remitted subjects, sug

151 absent, whereas activation in occipital and parietal regions persisted.

152 udies suggests that a network of frontal and parietal regions plays a crucial role.

153 ferior-frontal, middle-frontal, and inferior-parietal regions preceded by high-gamma attenuation in t

154 that interactions between primary visual and parietal regions predominantly influenced activity in fr

155 higher MPL precursor levels in the inferior parietal region (primarily right side) were noted in the

156 ts with damage to either the temporal or the parietal regions provide support for this functional dis

157 th the largest effects in frontotemporal and parietal regions; psychotic bipolar probands had small r

158 e effect on the form of BOLD response in the parietal region reflecting imagined transformations to t

159 The left hemisphere temporal and parietal regions remained significant when converters we

160 that a set of superior temporal and inferior parietal regions respond more strongly to conditions con

161 representational similarity analysis in this parietal region revealed that beneficial recurrent retri

162 related to alterations of neural activity in parietal regions seen over the course of MCI and AD.

163 ly, many cells in association and motor-like parietal regions show increasingly regular spike trains

164 Furthermore, the inferior parietal region showed nonspecific tactile and motor res

165 The parietal region showed significantly greater left latera

166 Whereas beta-band activity (18-30 Hz) in parietal regions showed body-centered spatial selectivit

167 All three parietal regions showed comparable delay-period response

168 The two sets of parietal regions showed different resting-state function

169 r cingulate, posterior inferior frontal, and parietal regions showed extended activation for all type

170 maging data with prespecified prefrontal and parietal regions showed that, although both regions were

171 these results suggest that the left inferior parietal region subserves subword orthographic-to-phonol

172 s represented within both ventral-stream and parietal regions, suggesting that communication between

173 matter revealed lower FA bilaterally in the parietal region (supramarginal gyri), right posterior ci

174 d the prefrontal and between the SMA and the parietal regions tended to decrease after training.

175 as with spread to contralateral temporal and parietal regions than in simple partial seizures.

176 Two seeds were used: a medial parietal region that contributes to the default network

177 nses relative to non-switch trials in fronto-parietal regions that appeared to be left lateralized, i

178 These clusters are linked by prefrontal and parietal regions that are hub nodes in the underlying st

179 rted by a distributed network of frontal and parietal regions that enable complex, goal-directed beha

180 to increased activity in medial and lateral parietal regions that have been implicated in attention

181 and the uncinate fasciculus and in the left parietal regions that include the fiber bundle of the su

182 Paradoxically, damage to specific parietal regions that lack spatial maps can cause patien

183 -temporal regions and decreases in posterior-parietal regions that largely recovered by two weeks pos

184 on of structurally intact dorsal and ventral parietal regions that mediate related attentional operat

185 right inferior parietal and bilateral medial parietal regions that support egocentric movement throug

186 lar gyrus, but also more anterior and dorsal parietal regions that were anatomically separate from th

187 components I, II and III; from the occipital-parietal region, the fronto-occipital fasciculus; from t

188 In the human brain, a network of frontal and parietal regions, the "multiple demand" (MD) system, is

189 vation in a network including prefrontal and parietal regions, the amygdala, caudate, and mid-brain.

190 Similarly, in medial parietal regions, the posterior cingulate was related to

191 l magnetic resonance imaging, allowing these parietal regions to be routinely and reliably identified

192 ound increasing involvement of occipital and parietal regions together with caudal-rostral recruitmen

193 he hypothesis that a single, domain-specific parietal region underlies both symbolic and nonsymbolic

194 in dorsal premotor cortex, with no effect in parietal regions until 150 ms post-perturbation.

195 ) and no retrieval success-sensitive lateral parietal region was insensitive to cueing.

196 ollowing damage to the right fronto-temporal-parietal region was slow and sometimes unable to find ta

197 eta burden in bilateral precuneus/cuneus and parietal regions was associated with increased brain atr

198 Deactivation in lateral parietal regions was equivalent across groups; in medial

199 ilk servings (P </= 0.013), and those in the parietal region were also correlated with cheese serving

200 fferences in the topological organization of parietal regions were found between phenotypically diffe

201 Precuneus and lateral parietal regions were more activated for True and False

202 sial temporal, middle temporal, and inferior parietal regions were more active in the patients during

203 supported memory retrieval, whereas non-DMN parietal regions were more involved in postretrieval pro

204 ocampus, along with cerebellar, temporal and parietal regions were more substantial in major depressi

205 ases in the BOLD signal in right frontal and parietal regions when compared with valid trials.

206 tion in the circuitry connecting frontal and parietal regions, where direct frontoparietal connection

207 ressed for IC sources in central midline and parietal regions, whereas mean beta band power increased

208 al gyrus is not connected with the posterior parietal region, which lies outside the confines of the

209 showed vasogenic edema in the right temporal parietal region, which prompted her transfer to our inst

210 In SD patients only, this inferior parietal region, which was not atrophied, was also activ

211 ities (hubs) in the primary sensorimotor and parietal regions, which formed a commonly shared core hu

212 ided into functionally distinct temporal and parietal regions, which have been implicated in feature-

213 l cortex and decreased in the right inferior parietal region with decreasing self-reference.

214 rior and middle temporal gyri, left inferior parietal region with postcentral gyrus, and right superi

215 y-related deactivation in medial and lateral parietal regions with greater deactivation in less-impai

216 the skull itself (e.g., 47% mismatch for the parietal region), with decreased performance in the base

217 up x age interaction in the PFC and inferior parietal region, with relatively older psychostimulant-n