ライフサイエンスコーパス: parotid

1 tivation, consistent with reports from mouse parotid.

2 tially to the deactivation of the current in parotid.

3 ts and acetylcholine-evoked secretion in the parotid.

4 uamous cell carcinoma, thyroid, adrenal, and parotid.

5 anes of secretory granules isolated from rat parotids.

6 069 +/- 0.022 mSv/MBq), and salivary glands (parotids, 0.031 +/- 0.011 mSv/MBq; submandibular, 0.061

7 ates of unstimulated whole (35%), stimulated parotid (47%), unstimulated submandibular/sublingual (23

8 es including 14 stomach, 11 lung, 7 orbit, 7 parotid, 5 thyroid, 5 lacrimal gland, 3 small intestine,

9 In parotid, a rapid global Ca(2+) signal was invariably ind

10 The P2X7R-mediated current was measured in parotid acinar and duct cells of wild type and P2X7R-/-

11 Here we used parotid acinar and duct cells to reveal the unique cell-

12 tiated apical [Ca(2+)](i) clearance in mouse parotid acinar cells and apical PMCA activity in Par-C10

13 ased the rate of oxygen consumption (QO2) of parotid acinar cells and PC12 cells.

14 rpolarization-activated chloride currents in parotid acinar cells and, as described previously, displ

15 nnels in red blood cells, T lymphocytes, and parotid acinar cells are indeed encoded by the Kcnn4 gen

16 Ca(2+)](i) clearance in the apical region of parotid acinar cells because of a dynamic translocation

17 ctifier chloride channel (Cl(ir)) from mouse parotid acinar cells by external protons (H(+)(o)) using

18 dy-state activation of BK channels in native parotid acinar cells by only 6 mV.

19 Parotid acinar cells exhibit rapid cytosolic calcium sig

20 ases in oxygen consumption) were measured in parotid acinar cells exposed to tyrosine kinase inhibito

21 Parotid acinar cells express two types of Ca(2+)-activat

22 re of normal appearance and fertility, their parotid acinar cells expressed no IK channels, and their

23 However, parotid acinar cells from Clcn2(-/-) mice recovered volu

24 anide-sensitive Cl(-) influx was observed in parotid acinar cells from mice lacking NKCC1.

25 Parotid acinar cells from these animals lacked maxi-K ch

26 The BK channels in parotid acinar cells have a much more hyperpolarized vol

27 Thus, stimulation of parotid acinar cells in Ca2+ -free medium with 0.5 micro

28 CO(3)(-) exchanger activity was increased in parotid acinar cells isolated from knockout mice suggest

29 (+) exchanger expression was examined in the parotid acinar cells of Nhe1(-/-) and Nhe2(-/-) mice, bo

30 arly, immunofluorescence of acutely isolated parotid acinar cells showed that the regulatory subunit

31 Exposure of freshly isolated parotid acinar cells to rottlerin and FCCP reduced cellu

32 These findings show that parotid acinar cells use low capacity, high sensitivity

33 To address these concerns, we profiled mouse parotid acinar cells using live-cell imaging to follow t

34 on sites on the alpha1 subunit in native rat parotid acinar cells using tandem mass spectrometry and

35 Ca(2+) signaling and cAMP pathways in human parotid acinar cells was investigated.

36 P(3) receptors, the Cl(-) current density of parotid acinar cells was more than four-fold greater tha

37 muscarinic receptor-induced acidification in parotid acinar cells was of a similar magnitude (0.25 +/

38 dly affect a variety of nuclear processes in parotid acinar cells while facilitating efficient fluid

39 In human parotid acinar cells, carbachol stimulation evoked incre

40 These data indicate that in human parotid acinar cells, in addition to modulation of Ca(2+

41 - channels previously characterized in mouse parotid acinar cells, nor is it dependent on P2 nucleoti

42 gain further insight into I(ATPCl) in mouse parotid acinar cells, we investigated the effects of ATP

43 as localized to the basolateral membranes of parotid acinar cells, whereas expression was not detecte

44 study demonstrates CICR in the non-excitable parotid acinar cells, which resembles the mechanism desc

45 ways from the maturing secretory granules in parotid acinar cells.

46 role in the storage of secretory proteins in parotid acinar cells.

47 mpaired in Kcnn4 null mice but was normal in parotid acinar cells.

48 -trisphosphate receptors (InsP(3)R) in mouse parotid acinar cells.

49 2X(4)R and P2X(7)R mRNA and protein in human parotid acinar cells.

50 temporal properties of [Ca2+]i signaling in parotid acinar cells.

51 icted [Ca2+]i signals were never observed in parotid acinar cells.

52 cAMP regulates Ca(2+) clearance pathways in parotid acinar cells.

53 oupling in a variety of cell types including parotid acinar cells.

54 This hypothesis was tested in mouse parotid acinar cells.

55 duced up-regulation of NHE activity in mouse parotid acinar cells.

56 her TMEM16A or TMEM16B as well as from mouse parotid acinar cells.

57 and P2X(7)R activation evokes exocytosis in parotid acinar cells.

58 or ligand carbachol in freshly dispersed rat parotid acinar cells.

59 MCA4 was localized to the apical membrane of parotid acinar cells.

60 TPase (PMCA) in a Ca(2+)-dependent manner in parotid acinar cells.

61 atial regulation of [Ca(2+)](i) clearance in parotid acinar cells.

62 levels is due to activation of AC8 in mouse parotid acini, and strongly support a role for AC5/6 in

63 2+) entry stimulates cAMP synthesis in mouse parotid acini, suggesting that one of the Ca(2+)-sensiti

64 coupling Ca(2+) signals to cAMP synthesis in parotid acini.

65 Water permeability decreased by 65% in parotid and 77% in sublingual acinar cells from Aqp5(-)/

66 ffects the salivary glands and in particular parotid and cervical lymph nodes.

67 he pancreas, chief cells of the stomach, and parotid and lacrimal secretory cells.

68 Ca(2+) sensitivity of the Cl(-) channels in parotid and pancreas was determined from the [Ca(2+)]-cu

69 strated the presence of ARC channels in both parotid and pancreatic acinar cells and shown that, agai

70 In parotid and pancreatic acinar cells, changes in [Ca(2+)]

71 (SMSL) saliva as well as citrate-stimulated parotid and SMSL saliva were measured in 399 subjects.

72 In contrast, the water permeability in parotid and sublingual acinar cells isolated from Aqp5(-

73 dy was to determine variations in stimulated parotid and submandibular flow rates over 6 hours and to

74 the immature acinar cells in developing rat parotid and submandibular glands and are also products o

75 imed to report a RCC case with metastasis to parotid and submandibular glands that has the same sonog

76 ulated whole and stimulated glandular (i.e., parotid and submandibular) saliva flow rate and composit

77 ve properties to blots of SDS-PAGE-separated parotid and submandibular-sublingual (SM-SL) saliva.

78 ed pattern observed for protein secretion in parotid and submandibular/sublingual glands, and that th

79 Parotid and submandibular/sublingual saliva samples and

80 tes and antimicrobial proteins in stimulated parotid and submandibular/sublingual saliva were determi

81 ns of appropriate agonists (carbachol in the parotid, and both carbachol and cholecystokinin in the p

82 reacted with the cancers (breast, colon and parotid) available from three anti-Ma patients, but not

83 Parotid biopsies from four non-pSS patients served as co

84 NA transcripts expressing IGHV3 genes in all parotid biopsies.

85 minimal effect on steady-state activation of parotid BK channels, it produced an approximate 2-fold s

86 This was not because the parotid BK isoform (parSlo) is inherently insensitive to

87 d essential part of the apoptotic program in parotid C5 cells and that specific activated isoforms of

88 roteases by all agents was also inhibited in parotid C5 cells expressing PKCdeltaKD.

89 DNA fragmentation was blocked up to 71% in parotid C5 cells infected with the PKCdeltaKD adenovirus

90 Induction of apoptosis in rat parotid C5 cells produced catalytic domain polypeptides

91 elta cleavage product does not accumulate in parotid C5 cells treated with rottlerin and etoposide to

92 Pretreatment of parotid C5 cells with rottlerin prior to the addition of

93 arotid cells from PKCdelta(-/-) cells and in parotid C5 cells, which express a dominant inhibitory mu

94 We have previously shown that parotid C5 salivary acinar cells undergo apoptosis in re

95 ree cDNA clones were isolated from a porcine parotid cDNA library.

96 uced activation of p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and

97 amino-terminal kinase is reduced in primary parotid cells from PKCdelta(-/-) cells and in parotid C5

98 Primary parotid cells from PKCdelta(-/-) mice are defective in m

99 Notably, in native parotid cells isoproterenol enhanced the carbachol-promo

100 8 in Ca(2+) stimulation of cAMP synthesis in parotid cells, acini were isolated from AC1 mutant (AC1-

101 In parotid cells, this was distinct from the effects of the

102 and studied the lymphomatous tissue from the parotid, cervical lymph node, and spleen using molecular

103 We propose that mouse parotid Cl(ir) current has a bimodal dependence on the e

104 Parotid cysts are an unrecognized sign of early HIV infe

105 NUR, 8-45 min; submandibular TNUR, 2-45 min; parotid DF, 20%-99%; and submandibular DF, 27%-98%.

106 These results suggest that in parotid duct cells apical NHE2 and NHE3 do not play a ma

107 nce, we have characterized the peptides from parotid ductal saliva collected from nine adults who hav

108 initiate fluid secretion in salivary gland (parotid) epithelial cells.

109 ve Car2.Ae2 HCO(3)(-) transport metabolon in parotid exocrine cell function.

110 he correlation between any pair of the three parotid fat content measurement methods.

111 The parotid fat contents measured with the three methods wer

112 The means of measured parotid fat contents revealed significant differences (P

113 t squares (IDEAL) method were used to derive parotid fat contents.

114 Success rates in parotid fat measurements by using T1, T2, and IDEAL meth

115 DEAL method provided a high success rate for parotid fat measurements, even in subjects with metallic

116 neity of fat saturation to determine whether parotid fat quantification was successful, with the succ

117 afe and transfer of the hAQP1 cDNA increased parotid flow and relieved symptoms in a subset of subjec

118 negative effect of a high-sucrose diet on a parotid function involved in the control of intradentina

119 play an important role in the regulation of parotid function.

120 d epidermal cyst (1), Warthin's tumor of the parotid gland (1), surgical wound inflammation (2), leio

121 mice following delivery of the vector to the parotid gland (PTG), the submandibular gland (SMG) or to

122 nd amylase secretion (16-93%) from, isolated parotid gland acinar cells.

123 IL14alphaTG mice, but they lacked the later parotid gland and lung injury.

124 ixty neutral sugars were identified from the parotid gland and many of these were additionally found

125 hly fucosylated glycans that co-exist in the parotid gland are not fully known.

126 dynamic contrast-enhanced MR imaging of the parotid gland at 1.5 T.

127 d focus of follicular lymphoma in the second parotid gland biopsy.

128 at the stimulation of amylase secretion from parotid gland cells by NaF may be mediated by an increas

129 timulate amylase secretion from isolated rat parotid gland cells.

130 The parotid gland contributes a variety of secretory protein

131 In the present work we used the parotid gland duct which expressed Slc26a4 and Slc26a6 a

132 gnosis of DILS was established in those with parotid gland enlargement by minor salivary gland biopsy

133 The fluid-secreting acinar cells of the parotid gland express both IK and BK channels, raising q

134 The similar neonatal submandibular and parotid gland expression patterns observed for these gen

135 The stimulated parotid gland fluid secretion rate was normal, but the s

136 l solute movement led us to hypothesize that parotid gland function(s) may have a role in regulating

137 A parotid gland hormone that stimulates intradentinal flui

138 A transfer to a single previously irradiated parotid gland in 11 subjects using an open label, single

139 Acetylcholine-evoked secretion from the parotid gland is substantially potentiated by cAMP-raisi

140 esting secretion of salivary proteins by the parotid gland is sustained in situ between periods of ea

141 The parotid gland is the mostly affected site among major sa

142 Based on these observations, the parotid gland kinase may be related to other Golgi-local

143 The follicular lymphoma of the parotid gland lymph node and the follicular lymphoma of

144 We propose that follicular lymphoma in the parotid gland lymph node may have resulted from coloniza

145 y and accurately differentiate epitopes from parotid gland N-glycans and milk oligosaccharides based

146 ultiple compensatory changes in the exocrine parotid gland of Nhe1(-/-) mice that together attenuate

147 ound (US) with painful swelling in the right parotid gland region.

148 hDF-EpiSC-loaded native de-cellularized rat parotid gland scaffolds into the renal capsule of nude m

149 correlated with decreased submandibular and parotid gland sizes.

150 Parotid gland swelling appeared to be the sine qua non o

151 l transcripts correlated with viral loads in parotid gland tissue and saliva.

152 systems biology approach was used to analyze parotid gland tissue samples obtained from patients with

153 Furthermore, the ability of the parotid gland to conserve NaCl was abolished in NKCC1-de

154 vation of p53 is required and sufficient for parotid gland transformation in the presence of activate

155 In contrast to the dramatic effect of p53 in parotid gland transformation, p53 loss has little effect

156 thyroid carcinoma, basal cell carcinoma, and parotid gland tumor, and 68.5% +/- 6.4% for those with n

157 [SIR] 52.3), non-Hodgkin lymphoma (SIR 8.3), parotid gland tumors (SIR 33.4), thyroid cancer (SIR 13.

158 mice heterozygous for a p53 deletion develop parotid gland tumors and loose their wild type p53 allel

159 accompanied by the down regulation of p21 in parotid gland tumors but not breast tumors.

160 the risk of meningioma, acoustic neuroma, or parotid gland tumors in relation to mobile phone use.

161 The parotid gland tumors were aneuploid and demonstrated inc

162 th the risks of brain, acoustic neuroma, and parotid gland tumors.

163 AdhAQP1 vector delivery to a single parotid gland was safe and transfer of the hAQP1 cDNA in

164 inductive site of the mucosal immune system (parotid gland) become polyclonal in piglets reared germf

165 dings included gross enlargement of the left parotid gland, a focal lesion in the right parotid gland

166 t parotid gland, a focal lesion in the right parotid gland, and cervical lymphadenopathy.

167 mRNA expression was detected in the gingiva, parotid gland, buccal mucosa, and tongue.

168 f the salivary glands, adrenal gland, colon, parotid gland, kidney, thyroid gland, thymus, or uterus

169 us, ESE-2, but not ESE-1, transactivates the parotid gland-specific PSP promoter and the prostate-spe

170 retion and regulatory volume decrease in the parotid gland.

171 rted DNase I mRNA transcript cloned from rat parotid gland.

172 t of SLPI isolated previously from the human parotid gland.

173 ed in the superficial-deep lobe of the right parotid gland.

174 iated lymphoid tissue (MALT) lymphoma in the parotid gland.

175 2 females, presented painless masses in the parotid gland.

176 andibular glands (PRP, 53%; mean SUV, 2.11), parotid glands (PRP, 51%; mean SUV, 1.90), and vocal cor

177 One or both parotid glands and at least two-thirds of the tongue wer

178 erian, lacrimal, mandibular, sublingual, and parotid glands and from liver, kidney, pancreas, testis

179 ons, BPIFA2 is expressed specifically in the parotid glands and is abundant in salivary secretions.

180 s, and lymphocytic infiltrates in the lungs, parotid glands and kidneys.

181 or bilateral nonsuppurative swelling of the parotid glands and neurological complications that can r

182 This hormone has been purified from porcine parotid glands and partially sequenced in our previous s

183 associated with high viral loads in infected parotid glands and that late viral protein expression is

184 an, lacrimal, submandibular, sublingual, and parotid glands and the liver.

185 Parotid glands appeared to exert a positive effect on de

186 IgG and IgA repertoire within pSS patients' parotid glands are distinct from those in non-pSS contro

187 ter mRNA increased dramatically in Nhe1(-/-) parotid glands but not in those of Nhe2(-/-) or Nhe3(-/-

188 Stimulation of amylase secretion from parotid glands by beta-adrenergic agonists is mediated b

189 Mumps viral antigen was detected in parotid glands by immunohistochemistry (IHC).

190 Saliva from the submandibular and parotid glands contained higher concentrations of adreno

191 Systems biology analyses of the parotid glands from patients with primary SS and those w

192 p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and PKCdelta(-/-) mice

193 on is suppressed in vivo in gamma-irradiated parotid glands from PKCdelta(-/-) mice.

194 cute time points (30 min to 2 h), irradiated parotid glands had significantly decreased levels of the

195 and to probe its specific roles, we studied parotid glands in mice with the K(Ca)1.1 gene ablated.

196 n vitro functioning of acinar cells from the parotid glands of mice with targeted disruption of Na(+)

197 Indeed, the parotid glands of Nhe1(-/-) mice expressed higher levels

198 sis is suppressed by greater than 60% in the parotid glands of PKCdelta(-/-) mice.

199 ping the Ig-producing cell repertoire in the parotid glands of primary Sjogren's syndrome (pSS) patie

200 culate that B cell hyperproliferation within parotid glands of pSS patients may result from Ag-indepe

201 ts in a robust induction of apoptosis in the parotid glands of wild type mice, whereas apoptosis is s

202 Parotid glands received higher doses (1.3 mGy/MBq) than

203 that IGF-1 promotes DNA repair in irradiated parotid glands through the maintenance and activation of

204 IK channels, activated fluid secretion from parotid glands was normal.

205 Homogenates and subfractions from macaque parotid glands were able to phosphorylate synthetic pept

206 ng demonstrated that the acinar cells of the parotid glands were the primary location for both the pa

207 Sparing the parotid glands with IMRT significantly reduces the incid

208 ritical structures were the spinal cord, the parotid glands, and the mandible.

209 Uptake in bone marrow, parotid glands, and tonsils was slightly but statistical

210 In perfused rat parotid glands, isoproterenol induced staining of both a

211 the production of type 1 IFN; injury to the parotid glands, lungs, and kidneys is seen; 3) progressi

212 ound uptake than (68)Ga-HBED-PSMA-11 (in the parotid glands, the mean SUVmax for (68)Ga-THP-PSMA was

213 om the native pancreas and submandibular and parotid glands.

214 the in vivo functions of these exchangers in parotid glands.

215 revealed that AC5/6 and AC3 are expressed in parotid glands.

216 ncluding human gingiva and submandibular and parotid glands.

217 istent uptake in the salivary, lacrimal, and parotid glands.

218 therapy (IMRT) can reduce irradiation of the parotid glands.

219 ssory protein, LRRC26, which is expressed in parotid glands: expressed parSlo + LRRC26 channels were

220 ted glycans were shown to be constituents of parotid glycoproteins for the first time.

221 G uptake in the cerebral cortex, cerebellum, parotid grand, myocardium, and bowel mostly reflected th

222 from secretion granules isolated from rabbit parotid has been shown to be contaminated by residual se

223 In addition, the 30-amino acid parotid hormone was synthesized.

224 lands were the primary location for both the parotid hormone-related mRNAs and the translation produc

225 with the amino acid sequence of the isolated parotid hormone.

226 cell lines from the submandibular (HSG) and parotid (HSY) salivary glands.

227 Our findings show that malignant parotid lesion was the stiffest lesion according to elas

228 RESENT THE ROLE OF ELASTOSONOGRAPHY IN THREE PAROTID LESIONS: a case of benign pleomorphic adenoma, a

229 By using parotid lobules from ad libitum fed rats stimulated with

230 the livers, kidneys, spleens and superficial parotid lymph nodes of the mice.

231 f male and female mouse lacrimal, harderian, parotid, mandibular, sublingual, and pancreas glands and

232 rotid PP, 22%-49%; submandibular PP, 4%-31%; parotid NUR, 2.2-16.0; submandibular NUR, 1.4-16.2; paro

233 and showed that ectopic expression in either parotid or pulmonary MEC tumor cell lines containing the

234 eristics when compared with the proteomes of parotid or submandibular/sublingual secretions.

235 ndard and high-sucrose diets, removal of the parotid or the submandibular/sublingual glands, and diet

236 e activity of the amelogenin promoter in rat parotid Pa-4 cells and Madin-Darby canine kidney cells w

237 essed by the MSCs of human major SGs, namely parotid (PAG), sublingual (SLG) and submandibular (SMG)

238 , we achieved relative quantification of the parotid peptides at four time points.

239 he thymus as a source of immature T cells in parotid (PG) and submandibular salivary glands (SMG) wer

240 secreted by the 3 major salivary glands: the parotid (PG), the submandibular (SMG), and the sublingua

241 AG10 and AG18 blocked parotid phosphorylation events only at concentrations th

242 eased abundance of Car2.Ae2 complexes in the parotid plasma membranes of Nhe1(-/-) mice.

243 (+)-K(+)-2Cl(-) cotransporter (NKCC1) in rat parotid plasma membranes was studied using the reversibl

244 nomas, myeloma, melanoma, colonic carcinoid, parotid pleomorphic adenoma, and teratoma.

245 The following ranges were observed: parotid PP, 22%-49%; submandibular PP, 4%-31%; parotid N

246 The increased potency of InsP(3) in parotid probably results from a four-fold higher number

247 hole saliva (r = 0.10, p < 0.05), stimulated parotid saliva (r = 0.13, p < 0.02), and stimulated SMSL

248 le saliva (r = 0.24, p < 0.0001), stimulated parotid saliva (r = 0.13, p < 0.03), unstimulated SMSL (

249 however, it was 37%, 18% lower than Db from parotid saliva (reported previously).

250 ons between the peptide composition of human parotid saliva and dental decay (caries) experience, we

251 Proline-rich proteins were enriched from parotid saliva and found to increase binding of anthocya

252 We collected parotid saliva as the ductal secretion from three human

253 ntified as the major iron-binding protein in parotid saliva by 59Fe autoradiography and Western blott

254 Iron binding was determined in parotid saliva by addition of nonlabeled and 59Fe labele

255 No iron was found in LF from parotid saliva in either group.

256 agglutinin blocked the inhibitory effect of parotid saliva on exogenously applied human leukocyte el

257 rnal effects on the composition of the human parotid saliva peptidome.

258 ptides in the < or =10-kDa fraction of human parotid saliva that included many novel species.

259 Whole and parotid saliva was collected from LAgP patients and matc

260 Parotid saliva was collected from subjects into separate

261 ed that the low-molecular-weight fraction of parotid saliva was peptide-rich, with novel fragments of

262 ated and stimulated whole saliva, stimulated parotid saliva, and stimulated labial minor gland saliva

263 Parotid saliva, nasal wash, and serum were collected pri

264 Parotid saliva, nasal wash, and serum were collected pri

265 When iron was added to parotid saliva, the LAgP saliva bound 20 to 30 times les

266 o prepare a low-molecular-weight fraction of parotid saliva, which was analyzed directly or after rev

267 e majority of glucan made by GTF adsorbed to parotid saliva-coated HA remained attached to the surfac

268 several other components from both SM-SL and parotid saliva.

269 l saliva, but it was essentially absent from parotid saliva.

270 uence, we isolated the agglutinin from human parotid saliva.

271 requency was 14%, similar to Db protein from parotid saliva.

272 he wild-type strains adhered better to human parotid-saliva- and amylase-coated hydroxyapatite than d

273 efficacy was evaluated with measurements of parotid salivary flow rate.

274 ly shown that the IK1 and maxi-K channels in parotid salivary gland acinar cells are encoded by the K

275 Sequential parotid salivary gland biopsies were taken before RTX, a

276 n double-stranded breaks (DSB) in the DNA of parotid salivary gland cells immediately after treatment

277 Using rat parotid salivary gland cells, we examined multiple pathw

278 ults indicate that proteolytic processing of parotid salivary proteins differs among individuals who

279 is high and these compounds localize to the parotid, salivary, and lacrimal glands as well as to the

280 n of sodium chloride to dialyzed, fungicidal parotid secretion abolished this activity, indicating th

281 genes, respectively, and in vivo stimulated parotid secretion is severely reduced in double-null mic

282 the fungicidal activities of some proteins, parotid secretion was subjected to dialysis with membran

283 Os of >=1000 promoted fungicidal activity of parotid secretion, and this activity was dose dependent.

284 Protein samples from parotid secretion, submandibular/sublingual secretion, w

285 the major candidacidal capacity of dialyzed parotid secretion.

286 Parotid secretory protein (PSP) and palate-lung-nasal ep

287 re, we report that the soluble cargo protein Parotid Secretory Protein (PSP) is bound to the membrane

288 1), anti-carbonic anhydrase 6 (CA6) and anti-parotid secretory protein (PSP), which occur early in th

289 ncluding amylase, proline-rich proteins, and parotid secretory protein (PSP)-to these functions.

290 1 (SP1), anti-carbonic anhydrase 6 (CA6) and parotid secretory protein (PSP).

291 ng family A member 2 (BPIFA2), also known as parotid secretory protein, which we identified via a mul

292 serine kinases with proteolytic activity for parotid secretory protein.

293 Parotid secretory proteins are stored in large dense-cor

294 preponderance was seen except in the case of parotid space lesions where female predominance was seen

295 In the parotid space, pleomorphic adenoma and in the prestyloid

296 We assessed the hypothesis that parotid-sparing IMRT reduces the incidence of severe xer

297 red conventional radiotherapy (control) with parotid-sparing IMRT.

298 Immunofluorescence of parotid tissue slices revealed that PMCA1 was distribute

299 NUR, 2.2-16.0; submandibular NUR, 1.4-16.2; parotid TNUR, 8-45 min; submandibular TNUR, 2-45 min; pa

300 ozygous p53 deletion have increased rates of parotid tumor onset suggesting that inactivation of p53

301 adenoma, a Wharthin's tumour and a malignant parotid tumour.

302 from the epithelial component of preexisting parotid Warthin tumors (WT).

303 InsP(3) was more effective in parotid, where [Ca(2+)](c) signals initiated with shorte