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1 astic hypertrophy and hyperplasia of the rat parotid gland.
2 ed in the superficial-deep lobe of the right parotid gland.
3 2 females, presented painless masses in the parotid gland.
4 retion and regulatory volume decrease in the parotid gland.
5 rted DNase I mRNA transcript cloned from rat parotid gland.
6 t of SLPI isolated previously from the human parotid gland.
7 iated lymphoid tissue (MALT) lymphoma in the parotid gland.
8 in the submandibular glands, but not in the parotid glands.
9 submandibular glands and rat sublingual and parotid glands.
10 istent uptake in the salivary, lacrimal, and parotid glands.
11 therapy (IMRT) can reduce irradiation of the parotid glands.
12 om the native pancreas and submandibular and parotid glands.
13 the in vivo functions of these exchangers in parotid glands.
14 revealed that AC5/6 and AC3 are expressed in parotid glands.
15 ncluding human gingiva and submandibular and parotid glands.
16 d epidermal cyst (1), Warthin's tumor of the parotid gland (1), surgical wound inflammation (2), leio
17 dings included gross enlargement of the left parotid gland, a focal lesion in the right parotid gland
20 ixty neutral sugars were identified from the parotid gland and many of these were additionally found
22 erian, lacrimal, mandibular, sublingual, and parotid glands and from liver, kidney, pancreas, testis
23 ons, BPIFA2 is expressed specifically in the parotid glands and is abundant in salivary secretions.
25 y in secretory endpieces isolated from sheep parotid glands and loaded with the pH-sensitive fluoropr
26 or bilateral nonsuppurative swelling of the parotid glands and neurological complications that can r
27 This hormone has been purified from porcine parotid glands and partially sequenced in our previous s
28 associated with high viral loads in infected parotid glands and that late viral protein expression is
36 IgG and IgA repertoire within pSS patients' parotid glands are distinct from those in non-pSS contro
38 inductive site of the mucosal immune system (parotid gland) become polyclonal in piglets reared germf
41 ter mRNA increased dramatically in Nhe1(-/-) parotid glands but not in those of Nhe2(-/-) or Nhe3(-/-
44 volumes for head and neck tumors as well as parotid glands can be well-defined on cross-sectional CT
45 at the stimulation of amylase secretion from parotid gland cells by NaF may be mediated by an increas
47 y against bands of 140-190 and 120 kd in the parotid gland, colon, and stomach, sites where the secre
50 that ACh-evoked HCO3- secretion in the sheep parotid gland differs from that in many other salivary g
54 gnosis of DILS was established in those with parotid gland enlargement by minor salivary gland biopsy
55 that Ca++ and calmodulin may play a role in parotid gland exocytosis by modulating the interaction b
57 ssory protein, LRRC26, which is expressed in parotid glands: expressed parSlo + LRRC26 channels were
61 p53 is similar in primary parotid cells and parotid glands from PKCdelta(+/+) and PKCdelta(-/-) mice
63 st that with the use of 3-DTP, contralateral parotid gland function can be partially preserved for at
64 l solute movement led us to hypothesize that parotid gland function(s) may have a role in regulating
65 cute time points (30 min to 2 h), irradiated parotid glands had significantly decreased levels of the
68 A transfer to a single previously irradiated parotid gland in 11 subjects using an open label, single
69 and to probe its specific roles, we studied parotid glands in mice with the K(Ca)1.1 gene ablated.
70 salivary gland dysfunction to contralateral parotid glands in patients with head and neck cancers.
72 esting secretion of salivary proteins by the parotid gland is sustained in situ between periods of ea
75 f the salivary glands, adrenal gland, colon, parotid gland, kidney, thyroid gland, thymus, or uterus
77 the production of type 1 IFN; injury to the parotid glands, lungs, and kidneys is seen; 3) progressi
79 We propose that follicular lymphoma in the parotid gland lymph node may have resulted from coloniza
80 y and accurately differentiate epitopes from parotid gland N-glycans and milk oligosaccharides based
82 ultiple compensatory changes in the exocrine parotid gland of Nhe1(-/-) mice that together attenuate
83 n vitro functioning of acinar cells from the parotid glands of mice with targeted disruption of Na(+)
86 ping the Ig-producing cell repertoire in the parotid glands of primary Sjogren's syndrome (pSS) patie
87 culate that B cell hyperproliferation within parotid glands of pSS patients may result from Ag-indepe
88 ts in a robust induction of apoptosis in the parotid glands of wild type mice, whereas apoptosis is s
89 andibular glands (PRP, 53%; mean SUV, 2.11), parotid glands (PRP, 51%; mean SUV, 1.90), and vocal cor
90 mice following delivery of the vector to the parotid gland (PTG), the submandibular gland (SMG) or to
94 hDF-EpiSC-loaded native de-cellularized rat parotid gland scaffolds into the renal capsule of nude m
96 us, ESE-2, but not ESE-1, transactivates the parotid gland-specific PSP promoter and the prostate-spe
98 ound uptake than (68)Ga-HBED-PSMA-11 (in the parotid glands, the mean SUVmax for (68)Ga-THP-PSMA was
99 that IGF-1 promotes DNA repair in irradiated parotid glands through the maintenance and activation of
101 systems biology approach was used to analyze parotid gland tissue samples obtained from patients with
103 vation of p53 is required and sufficient for parotid gland transformation in the presence of activate
104 In contrast to the dramatic effect of p53 in parotid gland transformation, p53 loss has little effect
105 thyroid carcinoma, basal cell carcinoma, and parotid gland tumor, and 68.5% +/- 6.4% for those with n
107 [SIR] 52.3), non-Hodgkin lymphoma (SIR 8.3), parotid gland tumors (SIR 33.4), thyroid cancer (SIR 13.
108 mice heterozygous for a p53 deletion develop parotid gland tumors and loose their wild type p53 allel
110 the risk of meningioma, acoustic neuroma, or parotid gland tumors in relation to mobile phone use.
114 t Raf-1 protein kinase activity in the mouse parotid glands was induced by chronic isoproterenol admi
116 Homogenates and subfractions from macaque parotid glands were able to phosphorylate synthetic pept
117 ng demonstrated that the acinar cells of the parotid glands were the primary location for both the pa
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