1 inistration reduced the number of scratching paroxysms by 68% (P < 0.01).

2                                  The rate of paroxysms could be reduced by functional segregation of

3 sider it to be a disconnection syndrome with paroxysms driven by a loss of inhibitory control over ex

4 vere left-sided orbital and temporal pain in paroxysms lasting 60 to 90 seconds, and accompanied by i

5 subgroup of 114 patients, deterioration from paroxysm of AF to (long-standing) persistent/permanent A

6 ty in patients with epilepsy is not a simple paroxysm of hypersynchronous excitatory activity, but ra

7  ion channel disorder characterised by brief paroxysms of cerebellar dysfunction and persistent neuro

8 ness lasts 4-8 weeks and is characterized by paroxysms of coughing with posttussive vomiting and whoo

9 g cardiac action potential prolongation with paroxysms of life-threatening arrhythmias.

10 ihood that external events evoke the painful paroxysm typical of this condition.

11 ellular components of the gouty inflammatory paroxysm, with new material on pathogenesis.