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1 idus, the thalamus, and the substantia nigra pars compacta.
2 and ventral tegmental area/substantia nigra pars compacta.
3 t remarkably, little to the substantia nigra pars compacta.
4 dopaminergic neurons of the substantia nigra pars compacta.
5 opamine (DA) neurons in the substantia nigra pars compacta.
6 opamine (DA) neurons in the substantia nigra pars compacta.
7 cortex, caudate-putamen and substantia nigra pars compacta.
8 dopaminergic neurons in the substantia nigra pars compacta.
9 ral tegmental area (VTA) or substantia nigra pars compacta.
10 the substantia nigra pars reticulata and/or pars compacta.
11 ence of degeneration of the substantia nigra pars compacta.
12 dopaminergic neurons in the substantia nigra pars compacta.
13 sis and inflammation in the substantia nigra pars compacta.
14 dopaminergic neurons in the substantia nigra pars compacta.
15 expression is noted in the substantia nigra pars compacta.
16 of dopaminergic neurons in substantia nigra pars compacta.
17 dopaminergic neurons in the substantia nigra pars compacta.
18 dopaminergic neurons in the substantia nigra pars compacta.
19 to a lesser extent, in the substantia nigra pars compacta.
20 the dopamine neurons of the substantia nigra pars compacta.
21 olfactory tract, and within substantia nigra pars compacta.
22 ral tegmental area, and the substantia nigra pars compacta.
23 dopaminergic neurons of the substantia nigra pars compacta.
24 l fields as hippocampus and substantia nigra pars compacta.
25 dopaminergic neurons of the substantia nigra pars compacta.
26 ventral tegmental area and substantia nigra pars compacta.
27 of dopamine neurons in the substantia nigra pars compacta.
28 mine neuron activity in the substantia nigra pars compacta.
29 in dopamine neurons of the substantia nigra pars compacta.
30 dopaminergic neurons in the substantia nigra pars compacta.
31 dopamine neurons from mouse substantia nigra pars compacta.
32 e dorsal GL or in the right substantia nigra pars compacta.
33 nergic interneurons and the substantia nigra pars compacta.
34 of LRRK2 expression in the substantia nigra pars compacta.
35 droxylase expression in the substantia nigra pars compacta.
36 rol vs. 36+/-2.6 O.D. BMP-7-treated, p<0.05; pars compacta: 29.0+/-4.9 O.D. control vs. 64.4+/-6.9 O.
38 of dopamine neurons of the substantia nigra pars compacta, a deficit in dopamine neurotransmission a
39 ource of serotonin, and the substantia nigra pars compacta, a major source of dopamine, while monkeys
40 s are most numerous in the substantia nigra, pars compacta, a region badly affected in homozygous wea
42 al tegmental area (VTA) and substantia nigra pars compacta activates inhibitory postsynaptic D2-recep
43 jections of 6-OHDA into the substantia nigra pars compacta and 1 week later were tested for startle a
44 opaminergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-sensitive motor d
45 ne-producing neurons in the substantia nigra pars compacta and decreased striatal dopamine levels are
46 the dopaminergic neurons of substantia nigra pars compacta and in the CA1/2 pyramidal cells of hippoc
47 dopaminergic neurons in the substantia nigra pars compacta and intracellular inclusion called Lewy bo
48 dopaminergic neurons in the substantia nigra pars compacta and locus ceruleus, without Lewy body path
49 in pigmented neurons of the substantia nigra pars compacta and locus coeruleus in all four DYT1 dysto
50 holaminergic neurons of the substantia nigra pars compacta and locus coeruleus, among other regions.
51 paminergic neurons from the substantia nigra pars compacta and noradrenergic neurons from the locus c
53 nsmission, particularly the substantia nigra pars compacta and other dopamine-synthesizing cell group
54 ed dopamine (DA) neurons in substantia nigra pars compacta and restored DA levels in striatum using t
55 essed in neurons within the substantia nigra pars compacta and striatum, two regions critically invol
56 dopaminergic neurons in the Substantia Nigra pars compacta and terminal dopamine fiber density in the
57 dopaminergic neurons in the substantia nigra pars compacta and the accumulation of the protein alpha-
58 rons at middle and caudal levels of both the pars compacta and the pars reticulata, with little label
59 paminergic neurons from the substantia nigra pars compacta and the presence, in affected brain region
60 trafficking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl-4-phenyl-1,2,3,6
61 nergic projections from the substantia nigra pars compacta and the ventral tegmental area to the basa
62 mine neurons located in the substantia nigra pars compacta and the ventral tegmental area, which form
63 dopamine by neurons in the substantia nigra pars compacta and ventral tegmental also influences basa
64 rter (DAT) mRNA in both the substantia nigra pars compacta and ventral tegmental area as shown by in
66 Dopamine neurons in the substantia nigra pars compacta and ventral tegmental area regulate behavi
67 inergic (DA) neurons in the substantia nigra pars compacta and ventral tegmental area regulate extrap
70 homologous to the mammalian substantia nigra pars compacta and was renamed accordingly; a group of ga
71 ulum (PT; homologous to the substantia nigra pars compacta and/or ventral tegmental area of mammals)
72 n dopaminergic neurons, A9 (substantia nigra pars compacta) and A10 (ventral tegmental area), have di
73 lated within neurons of the substantia nigra pars compacta, and dual labeling and confocal imaging co
76 in the ventral tier of the substantia nigra pars compacta, and the lowest levels in the ventral tegm
77 n of Mn accumulation in the substantia nigra pars compacta, and thus, can represent a link between Mn
78 sion was upregulated in the substantia nigra pars compacta, and trkB was elevated in the striatum in
79 ns, subthalamic nucleus and substantia nigra pars compacta, and was present at lower levels in the gl
81 Dopaminergic neurons of the substantia nigra pars compacta are defective in Parkinson's disease, but
83 thalamic nucleus (STN), and substantia nigra pars compacta, are conserved throughout vertebrate phylo
84 he A9 and A10 region of the substantia nigra pars compacta as well as the technical and surgical step
86 was a robust gliosis in the substantia nigra pars compacta associated with significant upregulation o
88 munoreactive neurons in the substantia nigra pars compacta (both in total and in subregions) were per
90 f brain neurons not only in substantia nigra pars compacta but also in other extrastriatal areas of t
91 of dopamine neurons in the substantia nigra pars compacta, but not in the adjacent ventral tegmental
92 ctive, was observed in the substantia nigra, pars compacta, but not in the ventral tegmental area.
93 munoreactive neurons in the substantia nigra pars compacta by 2-fold (p < 0.05) in animals lesioned w
97 minergic neurons within the substantia nigra pars compacta coupled with depletion of striatal dopamin
98 dopaminergic neurons in the substantia nigra pars compacta coupled with intracytoplasmic inclusions k
99 of dopamine neurons in the substantia nigra pars compacta, culminating in severe motor symptoms, inc
100 ning neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an end
101 vo recordings of identified substantia nigra pars compacta dopamine neurons in R1441C LRRK2 transgeni
102 ], 6-hydroxydopamine lesions of the adjacent pars compacta dopamine neurons were found not to abolish
103 sion of torsinA mRNA in the substantia nigra pars compacta dopamine neurons, the locus ceruleus, the
104 d thereby properly maintain substantia nigra pars compacta dopaminergic neurons and their innervation
107 PAR1 is expressed in human substantia nigra pars compacta glia as well as tyrosine hydroxylase-posit
108 els, forming an ultra-short substantia nigra pars compacta --> SNr dopamine pathway that regulates th
109 piriform cortex (layer II), substantia nigra pars compacta, hippocampal CA3 pyramidal cells, thalamic
112 or the vulnerability of the substantia nigra pars compacta in PD, why the disorder is age related, an
113 ctivity (TH-ir) in the substantia nigra (SN) pars compacta, in the lesioned hemisphere [31.7+/-5.2 (o
114 dopaminergic neurons of the substantia nigra pars compacta is a cardinal feature of Parkinson's disea
115 generation of DA neurons in substantia nigra pars compacta is a key neuropathological feature in Park
116 dopaminergic neurons in the substantia nigra pars compacta is the primary cause for motor symptoms ob
117 dopaminergic neurons of the substantia nigra pars compacta leading to abnormal activity within the ba
118 dopaminergic neurons in the substantia nigra pars compacta, leading to nigrostriatal degeneration.
119 ted nucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopaminergic neuron subs
121 B (trkB) receptor occurs in substantia nigra pars compacta neurons and is required for neuroprotectio
124 g the spontaneous firing of substantia nigra pars compacta neurons, isolated from transgenic mice in
125 able dopaminergic (DAergic) substantia nigra pars compacta neurons, only select downregulation of the
127 t is characterised by the presence of severe pars-compacta nigral-cell loss, and accumulation of aggr
129 ive cell body counts in the substantia nigra pars compacta of 6-OHDA- and free-3NT-treated mice (inje
130 single DA neurons from the substantia nigra pars compacta of controls and subjects with idiopathic P
131 in dopamine neurons of the substantia nigra pars compacta of mice following systemic administration
132 of dopamine neurons in the substantia nigra pars compacta of monkeys (Macaca mulatta) during a reach
136 aB was activated within the substantia nigra pars compacta of PD patients and MPTP-intoxicated mice.
137 re also up-regulated in the substantia nigra pars compacta of post-mortem PD brains as compared with
140 erikarya and primary dendrites, while in the pars compacta of the PPN (PPNc) axosomatic synapses were
143 se-positive neurons in the substantial nigra pars compacta one week after the first dose of MPTP.
145 the dopamine neurons of the substantia nigra pars compacta, other regions of the midbrain, and also t
146 ilar in injured IL-6 (+/+) and IL-6 (-/-) SN pars compacta (pc), microgliosis was severely compromise
147 ioris amygdalopallii pars basalis (PoAb) and pars compacta (PoAc), the nucleus taeniae amgygdalae (Tn
150 dopaminergic neurons of the substantia nigra pars compacta preferentially terminate in patch-like reg
151 ne-releasing neurons of the substantia nigra pars compacta produce an extraordinarily dense and expan
152 ergic (DA) neurons in their substantia nigra pars compacta, progressing to bilateral degeneration of
153 ateralis, r. subcuneiformis, r. peduncularis pars compacta, r. subcoeruleus pars alpha, raphe obscuru
154 n, thalamus, hypothalamus, substantia nigra, pars compacta, raphe, and pontine parabrachial nuclei.
155 ells of the cerebellum, the substantia nigra pars compacta, red nucleus, dorsal motor nucleus of X cr
157 dopaminergic neurons in the substantia nigra pars compacta, reproducing an important pathological fea
159 ogen receptor-bearing cell in the VTA and SN pars compacta, roughly half in the SN pars lateralis, an
160 yrosine hydroxylase mRNA in substantia nigra pars compacta (SC) (93%; P<0.05) compared with control r
162 f cell proliferation in the substantia nigra pars compacta (SN(C)) with a time-dependent adoption of
163 dopaminergic neurons in the substantia nigra pars compacta (SN) leads to debilitating motor dysfuncti
164 eration was observed in the substantia nigra pars compacta (SN), ventral tegmental area (VTA) and ret
166 dopaminergic neurons in the substantia nigra pars compacta (SNc) and consequent depletion of striatal
167 e lacks the majority of the substantia nigra pars compacta (SNc) and experiences striatal denervation
168 opaminergic neurones in the substantia nigra pars compacta (SNc) and may contribute to excitotoxic ce
171 the dopamine neurons of the substantia nigra pars compacta (SNc) and the importance of protein aggreg
172 dopaminergic neurons in the substantia nigra pars compacta (SNc) and the presence of intracytoplasmat
173 Dopamine neurons from the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA) are
174 brain dopamine centers, the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA), de
175 of dopamine neurons in the substantia nigra pars compacta (SNC) and ventral tegmental area (VTA).
176 neurons that project to the substantia nigra pars compacta (SNc) are activated by a visual CS for foo
177 al tegmental area (VTA) and substantia nigra pars compacta (SNc) are not significantly modulated by a
178 al tegmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine neurons intermixed
181 udies have established that substantia nigra pars compacta (SNc) dopamine neurons are a key node in t
182 ctor underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neurons in Parkinson's
184 layed increased activity of substantia nigra pars compacta (SNc) dopaminergic neurons, elevated basel
185 hat depend on the firing of substantia nigra pars compacta (SNc) dopaminergic neurons, we identified
186 homologous to the mammalian substantia nigra pars compacta (SNc) evokes increasing activation of MLR
187 Dopaminergic neurons of the substantia nigra pars compacta (SNc) exhibit functional heterogeneity tha
188 inergic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements requires a detailed
190 h a loss of dopamine in the substantia nigra pars compacta (SNc) leads to increased reflex blink exci
192 this receptor in regulating substantia nigra pars compacta (SNc) neuron physiology in both mice and r
193 e recordings were made from substantia nigra pars compacta (SNC) neurons in horizontal brain slice pr
194 eurons differs from that in substantia nigra pars compacta (SNc) neurons, where subthreshold calcium
195 g was much stronger in monkey than in rat SN pars compacta (SNc) neurons, whereas a moderate level of
198 TH) positive neurons in the substantia nigra pars compacta (SNc) of C57bl/6J mice following MPTP admi
199 with rAAV2/5 vectors in the substantia nigra pars compacta (SNc) on one side of the brain; the other
200 ion was seen in neighboring substantia nigra pars compacta (SNC) or substantia nigra pars reticulata.
201 and have been implicated in substantia nigra pars compacta (SNc) pathology in Parkinson's disease.
204 , dopaminergic cells in the substantia nigra pars compacta (SNc) respond immediately to unexpected co
205 opamine (DA) release in the substantia nigra pars compacta (SNc) shows a limited dependence on extrac
206 dDA) neurons, including the substantia nigra pars compacta (SNc) subpopulation that preferentially de
207 pamine neuron in the monkey substantia nigra pars compacta (SNc) that retains past learned reward val
208 on of viral vector into the substantia nigra pars compacta (SNc) to investigate its influence on nigr
209 al tegmental area (VTA) and substantia nigra pars compacta (SNc) to that of axonal dopamine release i
210 ostriatal pathway, from the substantia nigra pars compacta (SNc) to the dorsal striatum, and on relat
211 injections of rAAV into the substantia nigra pars compacta (SNc) transduced both dopaminergic and non
212 o Yellow or Fluorogold into substantia nigra pars compacta (SNc) were combined with larger injections
213 itive neuron numbers in the substantia nigra pars compacta (SNC) were estimated using stereological m
214 opamine (DA) neurons of the substantia nigra pars compacta (SNc) were found to exhibit sustained resp
215 ior colliculus (SC), to the substantia nigra pars compacta (SNc) where direct synaptic contacts are m
216 dopaminergic neurons in the substantia nigra pars compacta (SNc), but not in ventral tegmental area o
218 re first established in the substantia nigra pars compacta (SNc), but their validity in the VTA is un
219 dopaminergic neurons of the substantia nigra pars compacta (SNc), in addition to many other regions,
220 been suggested that, in the substantia nigra pars compacta (SNc), the pacemaking relies more on Ca(2+
221 ject to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neurons receive sens
235 sterior, a homologue of the substantia nigra pars compacta (SNc)/ventral tegmental area (VTA) is pres
237 opaminergic neurons of the substantia nigra, pars compacta (SNpc) akin to what is observed in Parkins
238 inergic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenergic neurons in the
239 ificantly diminished in the substantia nigra pars compacta (SNpc) and striatum, regions most affected
240 to these neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmental area (VTA), a
241 of dopamine neurons in the substantia nigra pars compacta (SNpc) and widespread aggregates of the pr
242 r centers, compromising the substantia nigra pars compacta (SNpc) and, later, the cerebral cortex.
243 ore dopamine neurons in the substantia nigra pars compacta (SNpc) are greatly needed to effectively c
244 dopaminergic neurons in the substantia nigra pars compacta (SNpc) as seen in Parkinson's disease.
245 for dopamine neurons of the substantia nigra pars compacta (SNpc) by regulating the magnitude of the
246 ctive (THir) neurons in the substantia nigra pars compacta (SNpc) compared with saline treatment.
247 rosine hydroxylase-immunoreactive (TH-ir) SN pars compacta (SNpc) DA neurons are immunoreactive for e
248 gic (DA) neurons within the substantia nigra pars compacta (SNpc) display a differential vulnerabilit
249 on/degeneration of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons contribut
250 characterized by a loss of substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons, and can
251 neurons of the ventral tier substantia nigra pars compacta (SNpc) failed to demonstrate a preponderan
253 degeneration occurs in the substantia nigra pars compacta (SNpc) of patients with Parkinson's diseas
254 ation has been shown in the substantia nigra pars compacta (SNpc) of PD models when there has been a
255 transfer of RGS10 into the substantia nigra pars compacta (SNpc) of rats reduced microgliosis and pr
256 (DA) neurons at the ventral substantia nigra pars compacta (SNpc) preferentially degenerate in Parkin
257 Dopaminergic neurons in the substantia nigra pars compacta (SNpc) undergo natural cell death during d
258 eurons containing CR in the substantia nigra pars compacta (SNpc) were relatively spared compared to
259 Here, we show that the substantia nigra pars compacta (SNpc), a brain region where dopamine (DA)
260 of dopaminergic neurons in substantia nigra pars compacta (SNpc), and there was no loss of dopaminer
261 had more TH-ir cells in the substantia nigra pars compacta (SNpc), but this difference was significan
262 sive degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striatal dopamine levels
263 rgic neurons located in the substantia nigra pars compacta (SNpc), expression of monoamines and indol
264 l intensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus, and ventral tegme
265 ecting GDF5 into either the substantia nigra pars compacta (SNpc), the lateral ventricle (LV) or the
266 f dopaminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental area (VTA), and
271 rmation was detected in the substantia nigra pars compacta, striatum, hippocampus, or selected region
272 ralis of Tsai, n. tegmenti pedunculopontinus pars compacta (substantia nigra), intercollicular n., ce
273 ein accumulation within the substantia nigra pars compacta, suggesting that nigrostriatal dopamine dy
274 ell accumulation within the substantia nigra pars compacta, suppression of microglial activation, and
275 ral level, dorsal raphe terminals in the PPT pars compacta synapse mainly with dendrites and axosomat
277 dopaminergic neurons in the substantia nigra pars compacta, the exact mechanism involved is still poo
279 bus pallidus, thalamus, and substantia nigra pars compacta to various environmental or genetic insult
280 nigra (nucleus tegmenti pedunculo-pontinus, pars compacta, TPc), and the locus coeruleus in the brai
281 eeks and a 42% reduction in substantia nigra pars compacta tyrosine hydroxylase-positive neurons at 8
282 y-associated disorders, the substantia nigra pars compacta undergoes degeneration, but the mechanism
283 e-containing neurons in the substantia nigra pars compacta use pacemaking mechanisms common to neuron
284 bgroups of neurons, such as substantia nigra pars compacta, ventral tegmental area and retrorubal fie
285 pocampus and dentate gyrus, substantia nigra pars compacta, ventral tegmental area, geniculate nucleu
286 on of the SN allowing readers to distinguish pars compacta ventralis and dorsalis from pars reticulat
287 ic terminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral tegmental area (V
288 wn as the nucleus tegmenti pedunculopontinus pars compacta was recognized as homologous to the mammal
289 s strongly expressed in the substantia nigra pars compacta, was present at lower levels in the striat
290 n of TRPM2 and TRPM4mRNA in substantia nigra pars compacta.We propose that ICAN is selectively activa
292 e-containing neurons in the substantia nigra pars compacta were located within the calbindin-rich zon
293 ost-commissural putamen and substantia nigra pars compacta were processed for tyrosine hydroxylase an
294 e dopaminergic cells in the substantia nigra pars compacta were unaffected by METH or haloperidol alo
295 more dorsolaterally in the substantia nigra pars compacta, whereas neurons inhibited by the stimuli
296 inergic (DA) neurons of the substantia nigra pars compacta, which are preferentially lost in human Pa
297 of the DYT1 gene within the substantia nigra pars compacta, which provides dopaminergic innervation t
298 specify mDA neurons of the substantia nigra pars compacta while the late Shh and Gli1 lineages maint
299 dopaminergic neurons in the substantia nigra pars compacta, with more than 40% of these neurons lost
300 educed neuron counts in the substantia nigra pars compacta, yet striatal medium spiny neuron dendriti
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