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1 s, globus pallidus and the substantia nigra (pars reticulata).
2 d ventral pallidum and the substantia nigra, pars reticulata.
3 ne reticular formation, and substantia nigra pars reticulata.
4 iatum, globus pallidus, and substantia nigra pars reticulata.
5 in the globus pallidus and substantia nigra pars reticulata.
6 on, as did the striatum and substantia nigra pars reticulata.
7 ication of D-amphetamine in substantia nigra pars reticulata.
8 botenic acid lesions of the substantia nigra pars reticulata.
9 cond microdialysis probe in substantia nigra pars reticulata.
10 ed GABAergic neurons in the substantia nigra pars reticulata.
11 sh pars compacta ventralis and dorsalis from pars reticulata.
12 ectly to the homolog of the substantia nigra pars reticulata.
13 to subthalamic nucleus and substantia nigra pars reticulata.
14 ganglia output neurons in substantia nigra, pars reticulata.
15 ion from globus pallidus or substantia nigra pars reticulata.
16 igra pars compacta (SNC) or substantia nigra pars reticulata.
17 in slices from rat or mouse substantia nigra pars reticulata.
18 rgic neurons of the VTA and substantia nigra pars reticulata.
19 hydroxylase (TH) positive cell bodies in the pars reticulata.
20 idus, the entopeduncular nucleus, and the SN pars reticulata.
21 he brainstem, including the substantia nigra pars reticulata.
22 la, and caudal parts of the substantia nigra pars reticulata.
23 nterpositus nucleus and the substantia nigra pars reticulata.
24 nificantly increased in the substantia nigra pars reticulata (13.5+/-4.1 and 26.3+/-2.9%, respectivel
25 ir in the SN of the non-lesioned hemisphere (pars reticulata: 14.8+/-1.19 O.D. control vs. 36+/-2.6 O
26 found that activity in the substantia nigra pars reticulata, a basal ganglia output, predictably dif
27 12 GABAergic neurons in the substantia nigra pars reticulata also were recorded and filled with bioti
28 eptors (D1+) project to the substantia nigra pars reticulata and are thought to facilitate movement.
29 atergic transmission in the substantia nigra pars reticulata and entopeduncular nucleus, two major ta
31 s from 684 globus pallidus, substantia nigra pars reticulata and subthalamic neurons recorded in inta
32 ethane anaesthesia from the substantia nigra pars reticulata and subthalamic nucleus along with corti
33 munoreactive nuclei were concentrated in the pars reticulata and the majority of labeled nigral neuro
36 and globus pallidus interna/substantia nigra pars reticulata, and infusion of recombinant glial deriv
37 urons in the substantia nigra pars compacta, pars reticulata, and pars lateralis), and 51% in nucleus
38 line output is initiated in substantia nigra pars reticulata, and this influence contributes to the e
39 riatum, globus pallidus and substantia nigra pars reticulata, and was not detectable in the subthalam
40 thesis that D1 receptors in substantia nigra pars reticulata are involved in the excitatory component
41 entopeduncular nucleus, and substantia nigra pars reticulata, areas of the basal ganglia receiving st
44 ng GABAergic neurons of the substantia nigra pars reticulata cannot be differentiated on the basis of
45 -dorsal-lateral part of the substantia nigra pars reticulata (cdlSNr), directly or indirectly through
47 ojection neurons target the substantia nigra pars reticulata (direct pathway) or the lateral globus p
48 ntal area, caudate putamen, substantia nigra pars reticulata, entorhinal cortex, central amygdala, la
49 in the globus pallidus and substantia nigra pars reticulata following lesions of the nigrostriatal t
50 afferents from striatum, globus pallidus, or pars reticulata have been shown to be mediated predomina
51 In the globus pallidus and substantia nigra pars reticulata, however, mGluR1a-ir was tightly cluster
53 ordings from neurons in the substantia nigra pars reticulata in rat brain slices and labeled them wit
54 reflex blinking is (1) the substantia nigra pars reticulata inhibits SC neurons, (2) the SC excites
56 in the globus pallidus and substantia nigra pars reticulata is caused by abnormal striatal activity.
57 via nigral D2 receptors, perhaps located on pars reticulata neurons themselves, to regulate basal ga
60 nnabinoid agonists, applied locally into the pars reticulata of substantia nigra (SNpr), could modula
62 pine-7,8-diol (SKF 38393) hydrochloride into pars reticulata of substantia nigra elicited a significa
63 profiles of neurons in the substantia nigra pars reticulata of the basal ganglia and in the superior
67 region of the hippocampus, substantia nigra pars reticulata, pallidum, and deep cerebellar nuclei.
70 timulation was identical in substantia nigra pars reticulata slices prepared from TKO and wild-type m
71 to inhibit output from the substantia nigra pars reticulata (SNpr) and internal pallidal segment (GP
72 nge in LFPs recorded in the substantia nigra pars reticulata (SNpr) and motor cortex (MCx) in the hem
73 , increases in motor cortex-substantia nigra pars reticulata (SNpr) coherence emerged in the 8-25 Hz
74 rior colliculus (SC) or the substantia nigra pars reticulata (SNpr) contralateral to the cortical les
76 rance of APP domains in rat substantia nigra pars reticulata (SNpR) neurons targeted for delayed dege
77 cular nucleus (EPN) and the substantia nigra pars reticulata (SNpr) on the intact side of the brain,
79 nculopontine nucleus to the substantia nigra pars reticulata (SNr) act on muscarinic acetylcholine re
80 he monkey, but not the rat, substantia nigra pars reticulata (SNr) also harbored a significant level
81 dy, we demonstrate that the substantia nigra pars reticulata (SNr) also provides a massive input to P
82 nch LSt projects to the GP, substantia nigra pars reticulata (SNr) and pars compacta (SNc), but not t
83 expressed GFRalpha-1 in the substantia nigra pars reticulata (SNR) and the ventral tegmental area (VT
84 ated the involvement of the substantia nigra pars reticulata (SNr) and the ventromedial nucleus of th
85 th GABAergic neurons in the substantia nigra pars reticulata (SNr) and with distal dendrites (in SNr)
86 c projection neurons in the substantia nigra pars reticulata (SNr) are key basal ganglia output neuro
87 evelopmental changes in the substantia nigra pars reticulata (SNr) associated with the expression of
90 he light microscope level, neurons of the SN pars reticulata (SNr) displayed moderate to strong immun
91 tory glutamatergic input to substantia nigra pars reticulata (SNr) from the subthalamic nucleus (STN)
93 argets of the striatum, the substantia nigra pars reticulata (SNr) has been hypothesized to play a ro
94 measured BG output from the substantia nigra pars reticulata (SNr) in mice while monitoring their mov
97 nt evidence that the monkey substantia nigra pars reticulata (SNr) in the basal ganglia represents st
100 l, a GABA agonist, into the substantia nigra pars reticulata (SNr) markedly reduced blink amplitude.
101 p recordings were made from substantia nigra pars reticulata (SNr) neurones in rat midbrain slices.
102 apped with non-dopaminergic substantia nigra pars reticulata (SNr) neurons and proximal dendrites of
103 ied altered the activity of substantia nigra pars reticulata (SNr) neurons of the basal ganglia (BG)
104 e activity and responses of substantia nigra pars reticulata (SNr) neurons to GABA, glutamate (GLU),
105 d the effects of DBS of the substantia nigra pars reticulata (SNr) on amygdala-kindled seizures.
106 , neurons downstream in the substantia nigra pars reticulata (SNr) only responded to Stop cues in tri
107 nglia output neurons in the substantia nigra pars reticulata (SNr) receive strong CARTir input from t
109 on dendrites located in the substantia nigra pars reticulata (SNr) than on those located in SNc, reve
110 histochemistry, in the rat substantia nigra pars reticulata (SNr) through an acute and persistent au
112 e, GABAergic neurons of the substantia nigra pars reticulata (SNr) tonically inhibit the target nucle
113 nd overlying brain, and the substantia nigra pars reticulata (SNr) were compared with contralateral b
114 A projection neurons of the substantia nigra pars reticulata (SNr), a key basal ganglia output nucleu
115 pattern is expressed in the substantia nigra pars reticulata (SNr), a main target of striatal efferen
116 ve GABAergic neurons of the substantia nigra pars reticulata (SNr), a major output of the basal gangl
117 excitatory influence on the substantia nigra pars reticulata (SNR), a major output structure of the b
118 a dual-output nucleus, the substantia nigra pars reticulata (SNr), and the presence of modulatory in
119 peduncular nucleus (EP) and substantia nigra pars reticulata (SNr), as does the acute injection of le
120 their midbrain target, the substantia nigra pars reticulata (SNr), at E14 in the mouse with a robust
121 in GABAergic neurons of the substantia nigra pars reticulata (SNr), but not in DA neurons of the subs
122 ckground strain mice in the substantia nigra pars reticulata (SNr), subthalamic nucleus (STN), rostro
124 ), globus pallidus (GP) and substantia nigra pars reticulata (SNr), together with the ipsilateral fro
125 bil (Meriones unguiculatus) substantia nigra pars reticulata (SNr), whose "neuronal" phenotype was co
130 s of NS cell bodies in the pars compacta and pars reticulata (TH immunohistochemistry and Cresyl viol
131 ra (SN) consists of GABAergic neurons of the pars reticulata that inhibit thalamic neurons and provid
132 peractivity in both STN and substantia nigra pars reticulata, the main output structure of basal gang
133 output projections from the substantia nigra pars reticulata to the deep layers of the superior colli
134 ens and 5-HT release in the substantia nigra pars reticulata, using a common stimulation in a single
135 d to a lesser extent in the substantia nigra pars reticulata, while no hybridization signal was detec
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