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1 he presurgical evaluation of drug-refractory partial epilepsy.
2 izing the epileptogenic region in refractory partial epilepsy.
3 indicative of the site of seizure origin in partial epilepsy.
4 nterictal SPECT images from 15 patients with partial epilepsy.
5 mporal areas following surgical treatment of partial epilepsy.
6 l electrodes in patients with drug-resistant partial epilepsies.
7 s the first-choice drug for the treatment of partial epilepsies.
8 to genetic susceptibility shared across the partial epilepsies.
9 for at least part of the heritability of the partial epilepsies.
10 of a consecutive series of 59 patients with partial epilepsy and a history of PIP, and 94 control pa
11 ed members in Family A (KCNA1 G724C) exhibit partial epilepsy and myokymia but no ataxic episodes, su
12 history of PIP, and 94 control patients with partial epilepsy and no history of PIP evaluated as inpa
13 ata were obtained, 96 had well characterized partial epilepsy and were enrolled, along with 27 contro
14 This disorder is the most common refractory partial epilepsy, and also the one most often treated su
15 erating seizures in patients with refractory partial epilepsy are referred to as the epileptogenic zo
16 onitoring with scalp EEG who had intractable partial epilepsy based on ictal electro-clinico-anatomic
17 eurotransmitter disturbances associated with partial epilepsy, detected and depicted (but not underst
18 oman with a history of medically intractable partial epilepsy, does not have an anatomical lesion but
19 sment, about 30% of patients with refractory partial epilepsy due to hippocampal sclerosis fail to be
20 isease risk, the role of common variation in partial epilepsies has not yet been explored in a well-p
23 10q24 may suggest it as a candidate gene for partial epilepsy in families where the disease has been
24 ight on mechanisms underlying a rare form of partial epilepsy in humans, autosomal dominant nocturnal
27 e aim of this study was to determine whether partial epilepsy is associated with abnormalities in hip
29 (b) an ictal SPECT image from a patient with partial epilepsy matched to itself after modification to
31 TCI is demonstrable in many cases of benign partial epilepsy of childhood, a disorder once thought t
32 of a common form of human epilepsy, complex partial epilepsy of temporal lobe origin, has establishe
34 s taking gabapentin compared with 14 complex partial epilepsy patients, matched for antiepileptic dru
36 p brain stimulation of anterior thalamus for partial epilepsy recently was approved in Europe and sev
37 ne receptor subunit genes cosegregate with a partial epilepsy syndrome known as autosomal dominant no
38 h influence risk for epilepsy shared amongst partial epilepsy syndromes, in 3445 patients and 6935 co
40 RS and DBS in patients with refractory focal/partial epilepsy was conducted using PubMed and Cochrane
42 have a modest role in predisposition to the partial epilepsies when considered across syndromes in E
43 ries of 20 adults with medically intractable partial epilepsy, where high-resolution brain MRI disclo
44 LGI1 have been linked to autosomal dominant partial epilepsy with auditory features (ADPEAF), an unu
46 of the causative gene in autosomal-dominant partial epilepsy with auditory features (ADPEAF, MIM 600
47 as adjuvant therapy in the US and Europe for partial epilepsy with or without secondary generalizatio
49 we describe a new family epilepsy syndrome, partial epilepsy with pericentral spikes, which we map t
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