ライフサイエンスコーパス: partial seizure

1 plex-partial seizures) or unimpaired (simple-partial seizures).

2 for tonic-clonic seizures than they are for partial seizures.

3 temporal and parietal regions than in simple partial seizures.

4 of 56 patients with drug-resistant, complex partial seizures.

5 y complex partial seizures and lastly simple partial seizures.

6 during complex-partial compared with simple-partial seizures.

7 ly greater in complex-partial than in simple-partial seizures.

8 set of obvious chronic recurrent spontaneous partial seizures.

9 cquired non-progressive cerebral lesions and partial seizures.

10 ity in up to 80% of patients with refractory partial seizures.

11 e epilepsy and medically intractable complex partial seizures.

12 lobe structures of 21 patients with complex partial seizures.

13 ages in 36 patients with intractable complex partial seizures.

14 ity in a rat model of focally evoked complex partial seizures.

15 Among patients with partial seizures, 35% respond poorly to available medica

16 Epilepsies were most often complex partial seizures (47%) and more than half were nonconvul

17 tion, of cerebral tissue in 10 patients with partial seizures and acquired lesions and 30 patients wi

18 vulsive state expressed mainly by continuous partial seizures and even new bouts of generalized seizu

19 in opioid neurotransmission associated with partial seizures and higher cognitive function, we inves

20 reat infantile spasms and refractory complex partial seizures and is in clinical trials to treat addi

21 ly generalized seizures, followed by complex partial seizures and lastly simple partial seizures.

22 vely tissue organization in 22 patients with partial seizures and MCD, with 30 control subjects.

23 of pregabalin, a drug used for treatment of partial seizures and neuropathic pain.

24 es and acquired lesions and 30 patients with partial seizures and normal MRI.

25 Individual analyses of the 30 patients with partial seizures and normal optimal MRI identified a sig

26 ed with SE during an unprovoked seizure were partial seizures and previous craniotomy.

27 imaging methods for identifying the cause of partial seizures, and can contribute to pre-surgical eva

28 , cingulate, and retrosplenial cortex during partial seizures, and increases in all of these regions

29 al and ictal states in patients with complex partial seizures, and the importance of the transition b

30 n, measured 2 hours after the first stage 2 (partial) seizure, appeared in neurons of the ipsilateral

31 Patients presenting with a history of only partial seizures are at low risk of subsequent tonic-clo

32 f the 10 patients with DNT had postoperative partial seizures arising in the ipsilateral hemisphere,

33 Epilepsy and Single Seizures) study had only partial seizures at randomisation.

34 and following seizures that impair (complex partial seizures) but not those that preserve (simple pa

35 onal activity and cerebral metabolism during partial seizures, but found increased neuronal activity

36 ed episodes of epigastric pain), and complex partial seizures consistent with temporal lobe epilepsy.

37 th no impairment of awareness (auras, simple partial seizures) continue, if there is a prior history

38 the frontal cortex during behaviorally mild partial seizures, contrasted with fast polyspike activit

39 sence, generalised tonic-clonic, and complex partial seizures, converge on the same set of anatomical

40 into temporal lobe structures cause complex partial seizures (CPS) and pathological changes observed

41 We studied 328 complex partial seizures (CPS) in 63 consecutive patients with t

42 have also been proposed as models of complex partial seizures (CPSs) following traumatic brain injury

43 We compared daily times of partial seizures determined by continuous electroencepha

44 The group did, however, continue to have partial seizures during follow-up, and modelling showed

45 Secondarily generalized seizures and complex partial seizures exhibited increased slow waves distribu

46 between seizure type (generalized or complex partial), seizure frequency, age of onset and duration o

47 Patients with complex partial seizures had lower GABA levels (1.03 mmol/kg of

48 ents who present with only simple or complex partial seizures have a poorly documented prognosis.

49 zure types and the rate of generalization of partial seizures, however rapid-eye-movement sleep seems

50 electroencephalography recordings during 63 partial seizures in 26 patients with surgically confirme

51 roved as an add-on drug for the treatment of partial seizures in adults with epilepsy.

52 or seizures in animals, analogous to complex partial seizures in humans, result in a consistent activ

53 s model mild absence seizures and/or complex partial seizures in humans, then an opportunity may exis

54 This remote effect of partial seizures may cause impaired cerebral functions,

55 ture hippocampus, possibly providing a novel partial seizure model in the developing rat.

56 eizures) but not those that preserve (simple partial seizures) normal consciousness and responsivenes

57 anxiety disorder and adjunctive therapy for partial seizures of epilepsy.

58 Migrating partial seizures of infancy is an early onset epileptic

59 The study has revealed that migrating partial seizures of infancy is associated with an expand

60 We report the case of a child with migrating partial seizures of infancy secondary to an activating m

61 Fourteen children with migrating partial seizures of infancy were reported during the 2 y

62 Migrating partial seizures of infancy, also known as epilepsy of i

63 roencephalograms in 35 patients with complex partial seizures of temporal lobe origin who were seizur

64 The differing effects of partial seizures on neurobehavioral recovery following a

65 ndent bilateral temporal lobe (IBTL) complex partial seizures on the intracranial electroencephalogra

66 alysis, reflecting the most common region of partial seizure onset.

67 e the frequency of further simple or complex partial seizures or a combination of both.

68 seizures and classified as impaired (complex-partial seizures) or unimpaired (simple-partial seizures

69 three different time periods: (i) during the partial seizure phase prior to generalization; (ii) duri

70 Partial seizures produce increased cerebral blood flow i

71 Specifically, complex partial seizures require evaluation of the frontal lobes

72 However, when partial seizures secondarily generalize, there are often

73 nts remained seizure free (apart from simple partial seizures [SPS]) at 5 years after surgery, and 47

74 with pharmacologically intractable, complex-partial seizures, surgical excision of the involved temp

75 ng number of genes have been identified with partial seizure syndromes.

76 pilepsy begins in adults with short-duration partial seizures that originate in the hippocampus.

77 erent types of chronic recurrent spontaneous partial seizures that worsen in frequency and duration o

78 c-clonic seizures (all patients) and complex partial seizures (three patients).

79 ormations of cortical development (MCDs) and partial seizures, to quantify the GABA(A)-central benzod

80 nisms of intractable epilepsy of the complex partial seizure type.

81 All patients had complex partial seizures, underwent amygdalohippocampectomy, and

82 Finally, we observed that complex-partial seizures were somewhat more common with onset in

83 nimal clonic, maximal tonic, or psychomotor (partial) seizures were determined in 16 different inbred

84 d in the same ictal sites as obvious complex partial seizures, were electrographically similar to rat

85 In patients with refractory partial seizures who are candidates for surgical treatme

86 eral temporal lobe epilepsy characterized by partial seizures with auditory disturbances.

87 The brain regions involved during partial seizures with secondary generalization have not

88 In agreement with findings from partial seizures without secondary generalization, cereb

89 ons of cerebral blood flow increases than in partial seizures without secondary generalization.

90 In the subset of 253 partial seizures without secondary generalized convulsio