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1 dation, catalepsy, or learning impairment in passive avoidance.
2 NT-3 did not influence locomotor activity or passive avoidance.
3 findings bridge rodent and human research on passive avoidance and behavioral inhibition and furnish
4 rotein mice worsened the memory retention in passive avoidance and novel object recognition tests, an
7 sions of the nucleus basalis interfered with passive avoidance and spatial memory-related behaviors.
8 tor subtypes in Xenopus oocytes; (2) improve passive avoidance and spatial Morris water task performa
11 s showed analogous behavior by adapting both passive avoidance behavior and behavioral inhibition to
12 he hippocampus with patients showing reduced passive avoidance behavior and inhibition across all thr
21 the open field and enhanced performance in a passive-avoidance fear conditioning task, which were bot
24 ferences in conditioned place preference and passive avoidance learning seen in Kal7(KO) mice are abr
25 n of recall of a weak version of the 1-trial passive avoidance learning task could be achieved by beh
26 l-induced deficits in spatial navigation and passive avoidance learning were investigated with a rat
27 contextual fear learning and enhances future passive avoidance learning, which may model certain beha
31 showed that 6-OHDA significantly reduced the passive avoidance memory performance, non-enzymatic (tot
33 nistration of GA significantly increased the passive avoidance memory, total thiol and GPx contents a
34 exhibited normal learning and memory in the passive avoidance, Morris water maze, and fear condition
35 (i.e., decreased burying behavior), but not passive avoidance of aversive stimuli (i.e., exploration
36 In these, a key behavioral assay comprises passive avoidance of potential threat and inhibition, bo
38 ing electrical footshocks exhibited enhanced passive avoidance (PA) learning when trained 24 h after
40 ormed similarly to wild-type controls in the passive avoidance paradigm, a test of aversive learning.
43 e exhibit normal learning and retention of a passive avoidance paradigm; however, they do not master
44 holinergic drugs have been shown to regulate passive avoidance performance via the amygdala, the neur
48 munity were able to learn active, as well as passive, avoidance protocols with shock as reinforcement
50 ng impairment and nesting behaviors based on passive avoidance, T-Maze, and nesting behavior tests.
51 maze, novel objective recognition, step-down passive avoidance, tail suspension, and sucrose preferen
52 , they performed significantly better in the passive avoidance task (255 +/- 36 s and 145 +/- 18 s in
54 ound impairment in acquisition of a standard passive avoidance task but failed to impair place learni
56 of impaired performance of fmr1 KO mice on a passive avoidance task is suggestive of a deficit in lea
57 ehavior in a novel open field and learning a passive avoidance task were assessed during nicotine tre
58 p-matched on IQ, gender, and age performed a passive avoidance task while undergoing functional MRI.
59 p-matched on IQ, gender, and age performed a passive avoidance task while undergoing functional MRI.
60 ated zero maze and impaired acquisition of a passive avoidance task, but normal behavior in open fiel
61 xhibited impaired performance in a long-term passive avoidance task, providing additional evidence fo
69 ce have no memory 21 d after training in the passive avoidance test, suggesting a pivotal role for ad
72 E3 or Apoe-/- mice to reach criterion during passive avoidance training, but castration did not modul
75 ale DISC1(D453G) mice displayed a deficit in passive avoidance, while neither males nor females exhib
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