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1 se transcription product comprises the HIV-1 pathogen-associated molecular pattern.
2 NA as the principal innate immune-activating pathogen-associated molecular pattern.
3 ased levels of double-stranded RNA, a potent pathogen-associated molecular pattern.
4 nents of the fungal cell wall and key fungal pathogen-associated molecular patterns.
5 es microbes from self by detecting conserved pathogen-associated molecular patterns.
6 strong affinity between rSp0032 and several pathogen-associated molecular patterns.
7 ich were originally identified as sensors of pathogen-associated molecular patterns.
8 walls and is one of the well-studied microbe/pathogen-associated molecular patterns.
9 ecific subcellular compartments and can bind pathogen-associated molecular patterns.
10 for many of the responses of macrophages to pathogen-associated molecular patterns.
11 immune system uses various proteins to sense pathogen-associated molecular patterns.
12 ceptors that recognize microbial products or pathogen-associated molecular patterns.
13 y in plants can be triggered by microbe- and pathogen-associated molecular patterns.
14 he inflammatory response elicited by its own pathogen-associated molecular patterns.
15 ent with methyl jasmonate, Pep peptides, and pathogen-associated molecular patterns.
16 by innate immune cells recognizing conserved pathogen-associated molecular patterns.
17 increased susceptibility to danger- but not pathogen-associated molecular patterns.
18 cell death and discriminates danger- versus pathogen-associated molecular patterns.
19 by pattern recognition molecules recognizing pathogen-associated molecular patterns.
20 oglia recognize and are activated by various pathogen-associated molecular patterns.
21 fense-expressing plants after treatment with pathogen-associated molecular patterns.
22 o tumour-necrosis factor (TNF), IL-1beta and pathogen-associated molecular patterns.
23 of immunity triggered by host recognition of pathogen-associated molecular patterns.
24 agonist properties with typical features of pathogen-associated molecular patterns.
25 ons, such as TLRs, which allow them to sense pathogen-associated molecular patterns.
26 ) family, detect stress, danger stimuli, and pathogen-associated molecular patterns.
27 rich repeat domain responsible for detecting pathogen-associated molecular patterns, a membrane spann
30 ng lectin (encoded by MBL2) bind to specific pathogen-associated molecular patterns, activate the com
31 ody, a tissue of neural crest origin, detect pathogen associated molecular patterns and danger associ
32 though the role of NLRP3 in host response to pathogen associated molecular patterns and danger associ
33 TLRs are immune receptors that recognize pathogen associated molecular patterns and then signal a
34 the role of post-translational responses to pathogen-associated molecular pattern and damage-associa
35 a nonstructural protein encoded by EBV is a pathogen-associated molecular pattern and that it has im
36 nthomonas species, confirming that Ax21 is a pathogen-associated molecular pattern and that XA21 is a
37 crophages is activated by the recognition of pathogen-associated molecular patterns and by many struc
38 n integrates environmental cues derived from pathogen-associated molecular patterns and cell-intrinsi
40 proteins, NLRs, are intracellular sensors of pathogen-associated molecular patterns and damage-associ
42 metabolic profiles rather similar to that of pathogen-associated molecular patterns and discuss how s
43 role as a recognition molecule of exogenous pathogen-associated molecular patterns and initiator of
44 gal cell wall is decorated with multifarious pathogen-associated molecular patterns and is the main t
45 chaffeensis PBP provides novel insights into pathogen-associated molecular patterns and pathogenesis
46 litatively modulates macrophage responses to pathogen-associated molecular patterns and pathogens by
47 ses to microbes are the interactions between pathogen-associated molecular patterns and pattern recog
48 ng growth factor-beta through recognition of pathogen-associated molecular patterns and phosphatidyls
49 tracellular receptors that recognize various pathogen-associated molecular patterns and play crucial
51 inflammation through their ability to sense pathogen-associated molecular patterns and products of t
52 pattern recognition receptors that recognize pathogen-associated molecular patterns and signal throug
53 F1 inhibits ethylene biosynthesis induced by pathogen-associated molecular patterns and that Arabidop
56 ocessing in response to bacterial infection, pathogen-associated molecular patterns, and damage-assoc
57 xpress Toll-like receptors (TLRs), recognize pathogen-associated molecular patterns, and mediate the
58 through which signals from helper T cells or pathogen-associated molecular patterns are delivered.
60 tem detects HCV infection, including how HCV pathogen-associated molecular patterns are generated dur
63 h multiple signals, including recognition of pathogen-associated molecular patterns, as well as signa
66 is study demonstrates that in the absence of pathogen-associated molecular patterns, Batf3-dependent
67 g a lectin-like domain that binds the fungal pathogen-associated molecular pattern beta-glucan and se
68 e report that recognition of purified fungal pathogen-associated molecular pattern beta-glucan by hum
69 tent with the concept that bacteria modulate pathogen-associated molecular patterns by expression of
70 rly in the course of infection, detection of pathogen-associated molecular patterns by innate immune
72 y against microbes depends on recognition of pathogen-associated molecular patterns by innate recepto
74 vated upon cytosolic sensing of a variety of pathogen-associated molecular patterns by Nod-like recep
76 of signaling evoked by direct perception of pathogen-associated molecular patterns by plant cells un
77 n, innate immune cells are able to recognize pathogen-associated molecular patterns by receptors such
78 In innate immune sensing, the detection of pathogen-associated molecular patterns by recognition re
79 rgency myelopoiesis, involves recognition of pathogen-associated molecular patterns by the common mye
80 of danger-associated molecular patterns and pathogen-associated molecular patterns by the nervous sy
82 l capsids were sensed at the cell surface as pathogen-associated molecular patterns by Toll-like rece
85 ition receptors contain a binding domain for pathogen-associated molecular patterns coupled to a sign
86 loaded with Ag and surface modified with the pathogen-associated molecular pattern CpG oligodeoxynucl
87 stem recognizes both damage (or danger)- and pathogen-associated molecular patterns (DAMP and PAMP, r
88 flammation mediated by damage-associated and pathogen-associated molecular patterns (DAMPs and PAMPs)
89 late monophosphate (c-di-AMP) is an emerging pathogen-associated molecular pattern during intracellul
90 nd FLS2 mediate recognition of the bacterial pathogen-associated molecular patterns EF-Tu and flagell
92 generation of reactive oxygen species after pathogen-associated molecular pattern elicitor treatment
93 otein signaling is directly activated by the pathogen-associated molecular pattern flagellin peptide
94 require potent adjuvants, as they lack known pathogen-associated molecular patterns found in attenuat
96 immunity that specifically recognize diverse pathogen-associated molecular patterns from pathogens.
97 ugh the generation and accumulation of viral pathogen-associated molecular patterns has been well cha
98 chanisms governing the innate recognition of pathogen-associated molecular patterns have been well de
99 belonging to the classes of the damage- and pathogen-associated molecular patterns, i.e. oligogalact
100 e considered the primary cells to respond to pathogen associated molecular patterns in the liver, rec
102 sterile conditions to avoid the influence of pathogen-associated molecular patterns in subsequent exp
103 deficiency during activation with bacterial pathogen associated molecular patterns, including heat-k
104 mmunity are initiated upon the perception of pathogen-associated molecular patterns, including peptid
105 rates the role of ferrous iron (Fe(2+)) as a pathogen-associated molecular pattern-independent agonis
106 the host cell inflammasome response to other pathogen-associated molecular patterns, indicated by red
107 te immunity, we defined the effects of CS on pathogen-associated molecular pattern-induced (PAMP-indu
109 K4 activity was found to negatively regulate pathogen-associated molecular pattern-induced reactive o
111 Wnts also alter DC responses to microbe- or pathogen-associated molecular patterns, inhibiting proin
113 arising from the organismal distribution of pathogen-associated molecular patterns is a major cause
114 Innate immune cell activation triggered by pathogen-associated molecular patterns is mediated by va
115 ity is broad and based on the recognition of pathogen-associated molecular patterns, it is uniquely r
116 4 (NLRC4)/Ipaf is involved in recognition of pathogen-associated molecular patterns leading to caspas
117 or; PRR, pattern recognition receptor; PAMP, pathogen-associated molecular pattern; LPS, lipopolysacc
120 ouble-stranded (ds) RNA substrate, a typical pathogen-associated molecular pattern molecule, to induc
122 he patient's myeloid cells with a variety of pathogen-associated molecular pattern molecules (PAMPs)
125 act as an innate immune sensor of microbial pathogen-associated molecular pattern molecules (PAMPs)
126 er of pathogens, including helminths, act as pathogen-associated molecular pattern molecules (PAMPs),
127 primary HLSECs transfected with HCV-specific pathogen-associated molecular pattern molecules increase
129 I elicits NET formation by means of multiple pathogen-associated molecular patterns (most notably end
130 In response to proinflammatory cytokines and pathogen-associated molecular patterns, NF-kappaB activa
131 pattern recognition receptors in recognizing pathogen-associated molecular patterns of Mycobacterium
132 ost pattern recognition molecules that sense pathogen-associated molecular patterns of the invading p
133 ieu due to the robust production elicited by pathogen-associated molecular patterns on cells of the i
134 ine development, multivalent presentation of pathogen-associated molecular patterns on the particle s
135 elf-motifs within viral products (known as a pathogen-associated molecular pattern or PAMP) to trigge
136 masome complex requires cytosolic sensing of pathogen-associated molecular patterns or danger-associa
138 tput of Toll-like receptor (TLR) response to pathogen associated molecular patterns (PAMP), and (iii)
140 Plasmodium RNA as a previously unrecognized pathogen-associated molecular pattern (PAMP) capable of
141 eties of its natural Gram negative bacterial pathogen-associated molecular pattern (PAMP) counterpart
142 ts reveal c-di-AMP to be a key mycobacterial pathogen-associated molecular pattern (PAMP) driving hos
143 tional CTLs is specific to the nature of the pathogen-associated molecular pattern (PAMP) encountered
144 port that the exposure of beta-glucan, a key pathogen-associated molecular pattern (PAMP) located at
145 5' triphosphate (5'ppp) of the HCV RNA and a pathogen-associated molecular pattern (PAMP) motif locat
146 athogen recognition receptor that recognizes pathogen-associated molecular pattern (PAMP) motifs to d
148 RNA to deplete proteins involved in specific pathogen-associated molecular pattern (PAMP) recognition
149 sting that malaria parasites harbor a common pathogen-associated molecular pattern (PAMP) recognized
150 a subset of genes involved in immunity, and pathogen-associated molecular pattern (PAMP) signals rep
151 pidly biochemically activated in response to pathogen-associated molecular pattern (PAMP) stimulation
152 sence of a single Sh3bp2 cherubic allele and pathogen-associated molecular pattern (PAMP) stimulation
153 (LPS) of Gram-negative bacteria is a common pathogen-associated molecular pattern (PAMP) that induce
154 surface glycoprotein, VSV-G, was likely the pathogen-associated molecular pattern (PAMP) that initia
156 We show that XopN action in planta reduced pathogen-associated molecular pattern (PAMP)-induced gen
157 ouse bone marrow-derived dendritic cell (DC) pathogen-associated molecular pattern (PAMP)-induced pro
159 initial defense responses from the plant is pathogen-associated molecular pattern (PAMP)-triggered i
160 dicating that AvrPiz-t functions to suppress pathogen-associated molecular pattern (PAMP)-triggered i
161 osest homologue, CRH1, are also required for pathogen-associated molecular pattern (PAMP)-triggered i
162 1 combines with the LRR-RLKs FLS2 and EFR in pathogen-associated molecular pattern (PAMP)-triggered i
163 1-associated kinase 1 (BAK1) is required for pathogen-associated molecular pattern (PAMP)-triggered i
164 erium Pseudomonas syringae can suppress both pathogen-associated molecular pattern (PAMP)-triggered i
165 ntegral part of the signaling cascade during pathogen-associated molecular pattern (PAMP)-triggered i
166 prises membrane glycoproteins that recognize pathogen-associated molecular patterns (PAMP) and mediat
167 ed processes, such as hyperresponsiveness to pathogen-associated molecular patterns (PAMP), enhanced
169 an be elicited by infection (sepsis) through pathogen associated molecular patterns (PAMPs) or throug
175 Transmembrane receptors recognize conserved pathogen-associated molecular patterns (PAMPs) and activ
176 ein that regulates plant immune responses to pathogen-associated molecular patterns (PAMPs) and bacte
177 e stimulation by environmental or endogenous pathogen-associated molecular patterns (PAMPs) and dange
179 -I, MDA-5, and LGP2) recognize viral RNAs as pathogen-associated molecular patterns (PAMPs) and initi
181 udomonas syringae involves both detection of pathogen-associated molecular patterns (PAMPs) and recog
182 on of innate immunity through the release of pathogen-associated molecular patterns (PAMPs) and their
183 dritic cells (DCs) are directly activated by pathogen-associated molecular patterns (PAMPs) and under
187 n shown to close in response to bacteria and pathogen-associated molecular patterns (PAMPs) as part o
188 ptors (TLRs) exert a key role in recognizing pathogen-associated molecular patterns (PAMPs) but have
190 the generation of interferon (IFN)-inducing pathogen-associated molecular patterns (PAMPs) by influe
191 Innate immunity relies on the perception of pathogen-associated molecular patterns (PAMPs) by patter
192 s can detect invaders via the recognition of pathogen-associated molecular patterns (PAMPs) by patter
193 mponent of plant defense is the detection of pathogen-associated molecular patterns (PAMPs) by plasma
194 e potential pathogens via the recognition of pathogen-associated molecular patterns (PAMPs) by surfac
198 ing virion RNA display at least two distinct pathogen-associated molecular patterns (PAMPs) containin
200 (GH12) proteins act as virulence factors and pathogen-associated molecular patterns (PAMPs) in oomyce
201 yndrome (TRAPS), whilst both spontaneous and pathogen-associated molecular patterns (PAMPs) induced I
202 by recognizing microbial molecules known as pathogen-associated molecular patterns (PAMPs) inducing
204 n of pattern-recognition receptors (PRRs) by pathogen-associated molecular patterns (PAMPs) is in ess
206 ous ligand-binding ectodomains that perceive pathogen-associated molecular patterns (PAMPs) or damage
207 n-recognition receptors (PRRs) and cell wall pathogen-associated molecular patterns (PAMPs) orientate
209 gh Toll-like receptor 2 (TLR2); however, the pathogen-associated molecular patterns (PAMPs) responsib
211 mmunostimulatory factors and presentation of pathogen-associated molecular patterns (PAMPs) that are
212 stem of humans and other mammals responds to pathogen-associated molecular patterns (PAMPs) that are
213 icrobes relies on the detection of conserved pathogen-associated molecular patterns (PAMPs) that are
214 attern recognition receptors (PRRs) to sense pathogen-associated molecular patterns (PAMPs) that are
215 cleic acids constitute an important group of pathogen-associated molecular patterns (PAMPs) that effi
217 responses may be initiated by recognition of pathogen-associated molecular patterns (PAMPs) that stim
218 es to vaccines require direct recognition of pathogen-associated molecular patterns (PAMPs) through p
221 t imposed through high conservation of viral Pathogen-Associated Molecular Patterns (PAMPs), and by t
223 cular patterns (DAMPs), but not to microbial pathogen-associated molecular patterns (PAMPs), are repr
224 obial-associated molecular patterns (MAMPs), pathogen-associated molecular patterns (PAMPs), danger-a
225 s suppress the cytokine response to multiple pathogen-associated molecular patterns (PAMPs), includin
226 eptors) are used by glial cells to recognize pathogen-associated molecular patterns (PAMPs), mediate
227 receptors (RLRs) that are activated by viral pathogen-associated molecular patterns (PAMPs), such as
244 One important aspect is interaction with pathogen-associated molecular patterns (PAMPs)/danger-as
245 S in lungs from mice exposed to CS and viral pathogen-associated molecular patterns (PAMPs)/live viru
246 ibacters trigger plant immunity response via pathogen-associated molecular patterns (PAMPs, such as l
248 gellin) associated with microbial pathogens (pathogen-associated molecular patterns, PAMPs), whereas
253 Toll-like receptors (TLRs), which are early pathogen associated molecular pattern receptors, in path
254 ligands to test whether triggering multiple pathogen-associated molecular pattern receptors could en
255 ual advance, in that NLRX1 is a modulator of pathogen-associated molecular pattern receptors rather t
256 tiple receptors including insulin receptors, pathogen-associated molecular pattern receptors, cytokin
257 resulting from the GI-2 deletion may affect pathogen-associated molecular pattern recognition by Tol
258 drawn attention to a strong relationship of pathogen-associated molecular pattern recognition with a
261 he host distinguishes between danger- versus pathogen-associated molecular patterns remains unresolve
262 y innate immune cells that upon encountering pathogen-associated molecular patterns respond by produc
264 resent a class of previously uncharacterised pathogen-associated molecular patterns sensed by pattern
266 ies, particularly when CD4(+) T-cell help or pathogen-associated molecular pattern signals are limite
267 e receptor (NLR) proteins act as sensors for pathogen-associated molecular patterns, stress, or dange
269 istance by diminishing surface expression of pathogen-associated molecular patterns, such as flagella
270 ogen detection is triggered by perception of pathogen-associated molecular patterns, such as flagelli
271 nnate immune system, mainly mediated through pathogen-associated molecular patterns, such as flagelli
272 LRs) recognize pathogens by interacting with pathogen-associated molecular patterns, such as the phos
274 en Pseudomonas aeruginosa expresses not only pathogen-associated molecular patterns that activate NF-
275 ike receptor (TLR) signaling is triggered by pathogen-associated molecular patterns that mediate well
277 Despite the absence of exogenous microbial pathogen-associated molecular patterns, the Tfh cell res
279 ribute to cytokine production in response to pathogen associated molecular pattern-TLR-mediated stimu
281 rferon-driven events can lead to exposure of pathogen-associated molecular patterns to the host cytos
282 lated) electrolyte leakage and a compromised pathogen-associated molecular pattern-triggered immune r
283 duction elements that coordinate damage- and pathogen-associated molecular pattern-triggered immunity
284 r-triggered immunity (ETI) responses but not pathogen-associated molecular pattern-triggered immunity
285 s heterotrimeric G-protein complex modulates pathogen-associated molecular pattern-triggered immunity
286 r-triggered immunity (ETI) responses but not pathogen-associated molecular pattern-triggered immunity
287 Signaling components are shared between the pathogen-associated molecular pattern-triggered immunity
288 recognition receptor that is responsible for pathogen-associated molecular pattern-triggered immunity
290 cognition receptor complexes, which disrupts pathogen-associated molecular pattern-triggered immunity
291 Both proteasome subunits are required for pathogen-associated molecular pattern-triggered immunity
292 the proteasome is an essential component of pathogen-associated molecular pattern-triggered immunity
294 tahrcC mutant, and decreased expression of a pathogen-associated molecular pattern-triggered marker g
295 infection, indicating that HCV dsRNA was the pathogen-associated molecular pattern triggering TLR3 si
296 innate inflammasome responses recognize the pathogen-associated molecular patterns which lead into t
297 ccharides that comprise the cell wall act as pathogen associated molecular patterns, which govern the
298 is required for recognition and detection of pathogen associated molecular patterns, which results in
299 ce, systemic inflammation was induced by the pathogen associated molecular pattern zymosan (intraperi
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