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1 pproximately 25% of LQTS remains unexplained pathogenetically.
2 a similar array of features might be linked pathogenetically.
3 to identify putative circulating biomarkers pathogenetically and prognostically linked to bicuspid a
4 ter cases suggest that skeletal myxoid CS is pathogenetically distinct from its extraskeletal counter
7 s-Danlos syndrome (EDS) is a genetically and pathogenetically heterogeneous group of disorders of whi
9 support the concept that STAT3 activation is pathogenetically important in ALCL cells by deregulating
17 protein 43 (TDP-43) are RNA-binding proteins pathogenetically linked to amyotrophic lateral sclerosis
18 chia coli (ECOLI) PDC-E2 mimic that has been pathogenetically linked to PBC but lacks this motif has
19 sis (ET), and primary myelofibrosis (PMF) as pathogenetically related myeloproliferative disorders (M
20 ic pulmonary arterial hypertension (IPAH) is pathogenetically related to low levels of the vasodilato
22 ase-IV (DPPIV/CD26) as a novel, specific and pathogenetically relevant biomarker of CD34(+)/CD38( hor
23 ma and TNF-alpha) secretion and to a lack of pathogenetically relevant compensatory responses to othe
26 ise positional mapping and identification of pathogenetically relevant genes, we analysed of human ge
27 cted recent data that provide clinically and pathogenetically relevant insights or identify potential
28 d at least two genes (c-kit and PDGFRA) with pathogenetically relevant mutations have been identified
29 and illustrate the importance of delineating pathogenetically relevant subgroups within the "recurren
31 tive kinase activation and are thought to be pathogenetically relevant, implicating FLT3 as a plausib
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