ライフサイエンスコーパス: pathogenicity island

1 uding those carrying virulence determinants (pathogenicity islands).

2 nd dependent upon a functional H. pylori cag pathogenicity island.

3 binding protein HilD encoded in a different pathogenicity island.

4 Type III secretion system encoded in the LEE pathogenicity island.

5 ion of an H. pylori host receptor by the cag pathogenicity island.

6 y using the genes encoded in the Francisella pathogenicity island.

7 tivation of the tcpPH promoter on the Vibrio pathogenicity island.

8 luding the vacuolating cytotoxin and the cag pathogenicity island.

9 iosynthetic assembly line encoded by the pks pathogenicity island.

10 suppression of the virulence-associated LEE pathogenicity island.

11 borne on the locus of enterocyte effacement pathogenicity island.

12 se II (MPII)) are encoded by the B. fragilis pathogenicity island.

13 glJ, or pdpC, three genes of the Francisella pathogenicity island.

14 open reading frames with characteristics of pathogenicity islands.

15 neated by CGH in addition to the three known pathogenicity islands.

16 e integrase proteins found on staphylococcal pathogenicity islands.

17 nce plasmid and, in some cases, the Shigella pathogenicity islands.

18 ial virulence loci encoded within Salmonella pathogenicity islands.

19 Staphylococcus aureus pathogenicity island 1 (SaPI1) is a mobile genetic eleme

20 r genes--hilA, invF, and ssrA--in Salmonella pathogenicity island 1 (SPI-1) and 2 (SPI-2), by binding

21 wo type III secretion systems (TTSSs) within pathogenicity island 1 (SPI-1) and island 2 (SPI-2).

22 and SC2, harboring variations in Salmonella pathogenicity island 1 (SPI-1) and SPI-2 and exhibiting

23 nterica serovar Typhimurium (S. Typhimurium) pathogenicity island 1 (SPI-1) encodes a type III secret

24 In Salmonella, the Salmonella pathogenicity island 1 (SPI-1) encodes a type three secr

25 aspase-1 activation and, in part, Salmonella pathogenicity island 1 (SPI-1) expression by Salmonella.

26 enhanced bacterial expression of Salmonella pathogenicity island 1 (SPI-1) genes and increased intra

27 cription of the flagellar and the Salmonella pathogenicity island 1 (SPI-1) regulons in a FliZ-depend

28 biquitination is triggered by the Salmonella pathogenicity island 1 (SPI-1) T3SS effectors SopB and S

29 ype III secretion systems (T3SSs)-Salmonella pathogenicity island 1 (SPI-1) T3SS, SPI-2 T3SS, and the

30 pecialized organelle known as the Salmonella pathogenicity island 1 (SPI-1) type 3 secretion system (

31 e numerous virulence genes within Salmonella pathogenicity island 1 (SPI-1), newly identified flagell

32 terica subsp. salamae encodes the Salmonella pathogenicity island 1 (SPI-1), SPI-2, and the locus of

33 H and mutS genes at the 3' end of Salmonella Pathogenicity Island 1 (SPI-1), ste fimbrial operon, and

34 Here, starting with the 35 kb Salmonella pathogenicity island 1 (SPI-1), we eliminated internal r

35 ecretion system (T3SS) encoded in Salmonella pathogenicity island 1 (SPI-1), while macrophage surviva

36 absence and presence of bile in a Salmonella pathogenicity island 1 (SPI-1)-dependent manner.

37 body interferes specifically with Salmonella pathogenicity island 1 (SPI-1)-dependent, but not SPI-1-

38 the Salmonella enterica serovar Typhimurium pathogenicity island 1 (SPI-1)-encoded TTSS are required

39 I secretion system encoded within Salmonella pathogenicity island 1 (SPI-1).

40 ependent on multiple genes within Salmonella pathogenicity island 1 (SPI-1).

41 s, many of which are found within Salmonella pathogenicity island 1 (SPI-1).

42 Salmonella pathogenicity island 1 (SPI1) and SPI4 have previously b

43 Infection with a Salmonella pathogenicity island 1 (SPI1) mutant did not result in m

44 ransferase Pat regulates genes on Salmonella Pathogenicity Island 1 (SPI1) that are required for the

45 ecretion system (TTSS) encoded by Salmonella pathogenicity island 1 (SPI1) that were previously shown

46 yphimurium uses a T3SS encoded by Salmonella pathogenicity island 1 (SPI1) to actively invade cells t

47 o the host cell cytoplasm via the Salmonella pathogenicity island 1 (SPI1) type III secretion system

48 rica serovar Typhimurium uses the Salmonella pathogenicity island 1 (SPI1) type III secretion system

49 aperone sigma(28), the removal of Salmonella pathogenicity island 1 (Spi1), the removal of flagellar

50 tion system (T3SS) encoded within Salmonella pathogenicity island 1 (SPI1).

51 S); the T3SS genes are carried on Salmonella pathogenicity island 1 (SPI1).

52 effect of fim gene expression on Salmonella pathogenicity island 1 (SPI1).

53 e III secretion system encoded on Salmonella pathogenicity island 1 (SPI1).

54 ecretion system (T3SS) encoded on Salmonella pathogenicity island 1 (SPI1).

55 um employing genes encoded within Salmonella Pathogenicity Island 1 (SPI1).

56 volved in cholera disease is the V. cholerae pathogenicity island 1 (VPI-1).

57 ibute to enteric infection (e.g., Salmonella pathogenicity island 1 [SPI-1], SPI-4, SPI-5, CS54, fliH

58 i cell entry was dependent on the Salmonella pathogenicity island 1 and Shigella mxi/spa type III sec

59 ical Salmonella enterica serovar Typhimurium pathogenicity island 1 basal body, determined using sing

60 A network encoded within Salmonella Pathogenicity Island 1 controls the expression of a type

61 nuclear leukocytes, SipA or other Salmonella pathogenicity island 1 effectors had no effect on induct

62 rium invA mutant defective in the Salmonella pathogenicity island 1 invasion apparatus was less capab

63 onella enterica invasion genes on Salmonella pathogenicity island 1 is under the control of the compl

64 t observed after infection with a Salmonella pathogenicity island 1 mutant deficient in type III secr

65 gellin translocation required the Salmonella Pathogenicity Island 1 Type III secretion system (SPI-1

66 A functional Salmonella pathogenicity island 1 type III secretion system appears

67 r infection required a functional salmonella pathogenicity island 1 type III secretion system but not

68 ns raise the possibility that the salmonella pathogenicity island 1 type III secretion system cannot

69 roteins secreted by the bacterial Salmonella pathogenicity island 1- and Salmonella pathogenicity isl

70 cretion systems (TTSS) encoded in Salmonella pathogenicity islands 1 and 2 (SPI-1 and SPI-2) that del

71 sion of representative genes from Salmonella pathogenicity islands 1 and 2 (SPI1 and SPI2) and the im

72 nds on the virulence determinants Salmonella pathogenicity islands 1 and 2, and it is characterized b

73 virulence determinants, including Salmonella Pathogenicity Islands 1, 2, 3, 4, and 6.

74 s (T3SSs) encoded in two distinct Salmonella pathogenicity islands, 1 and 2 (SPI1 and SPI2, respectiv

75 tellaria baicalensis, targets S. Typhimurium pathogenicity island-1 (SPI-1) type III secretion system

76 S. typhimurium has two T3SS: Salmonella pathogenicity island-1 (SPI1), which encodes the rod pro

77 The hilA gene on the Salmonella enterica pathogenicity island-1 encodes the key transcriptional r

78 We investigated the roles of Salmonella pathogenicity island 2 (SPI-2) and two SPI-2 effectors i

79 ls, bacteria failed to upregulate Salmonella pathogenicity island 2 (SPI-2) genes and did not form a

80 While the T3SS within Salmonella pathogenicity island 2 (SPI-2) has been previously shown

81 Our results demonstrated that the Salmonella pathogenicity island 2 (SPI-2) T3SS assembled into a fun

82 effect was dependent on an intact Salmonella pathogenicity island 2 (SPI-2) type 3 secretion system.

83 acuolar membrane pore made by the Salmonella pathogenicity island 2 (SPI-2) type III secretion system

84 ke putative C-ring protein of the Salmonella pathogenicity island 2 (SPI-2)-encoded T3SS.

85 B two-component system located on Salmonella pathogenicity island 2 (SPI-2).

86 virulence factors encoded by the Salmonella pathogenicity island 2 (SPI-2).

87 endent regulation of genes within Salmonella pathogenicity island 2 (SPI-2).

88 Because PhoP-regulated Salmonella pathogenicity island 2 (SPI2) genes are also repressed b

89 rison of candidate genes from the Salmonella Pathogenicity Island 2 (SPI2) locus was conducted in the

90 iesis in a process independent of Salmonella pathogenicity island 2 (SPI2) or flagellin.

91 We show herein that the Salmonella pathogenicity island 2 (SPI2) response regulator SsrB un

92 translocator and effector of the Salmonella pathogenicity island 2 (SPI2) type III secretion system,

93 (TLR)-dependent signals to induce Salmonella Pathogenicity Island 2 (SPI2), a locus required for intr

94 Vibrio pathogenicity island 2 (VPI-2), a 57-kb region found exc

95 sin (HlyA); C-II encodes a variant of Vibrio pathogenicity island 2 (VPI-2), and Vibrio seventh pande

96 lso preserve the transcription of Salmonella pathogenicity island 2 gene targets from the inhibitory

97 trate up-regulation of particular Salmonella pathogenicity island 2 genes (especially spiC) and incre

98 Salmonella virulence depends in part on its pathogenicity island 2 type III secretion system (SPI-2

99 ofessional phagocytes through the Salmonella pathogenicity island 2 type III secretion system (TTSS).

100 murium virulence determinant, the Salmonella pathogenicity island 2 type III secretion system, is req

101 The Salmonella Pathogenicity Island-2 (i.e. SPI-2) encodes a unique typ

102 Vibrio pathogenicity island-2 (VPI-2) is a 57-kb region integra

103 cteria in a process that required Salmonella pathogenicity island-2 and correlated with increased exp

104 nella pathogenicity island 1- and Salmonella pathogenicity island-2-encoded virulence systems.

105 ation of a number of fimbrial and Salmonella pathogenicity island 3 (SPI-3) and 5 genes, suggesting a

106 that Salmonella enterica serovar Typhimurium pathogenicity island 4 carries a type I secretion system

107 um requires a T6SS encoded within Salmonella pathogenicity island-6 (SPI-6).

108 Salmonella pathogenicity island 7 (SPI-7), found within some pathog

109 vi66 occurs in the cytotoxin-associated gene pathogenicity island, a genomic region known to be assoc

110 Streptococcus pneumoniae is an example of a pathogenicity island acquired through genetic recombinat

111 , is consistent with the notion that certain pathogenicity islands act cooperatively with the LEE isl

112 Genes in the cag pathogenicity island also contribute to the inflammatory

113 FIB plasmid harboring components of the ColV pathogenicity island and a multidrug resistance (MDR)-en

114 ses of H. pylori status, carriage of the cag pathogenicity island and assignment of H. pylori to phyl

115 These events are mediated by the cag pathogenicity island and by mitogen-activated protein ki

116 on the locus of enterocyte effacement (LEE) pathogenicity island and display high levels of multifun

117 is located between hrpR and avrE1 in the Hrp pathogenicity island and is carried in the functional cl

118 Virulence factors of H. pylori; the cag pathogenicity island and OipA affected IL-18 induction i

119 lbA and clbP, genes contained within the pks pathogenicity island and required for the synthesis of c

120 ew experimental methods, strain information, pathogenicity islands and external references that link

121 osa may be a useful strategy for identifying pathogenicity islands and novel virulence determinants.

122 mental conditions that induce the Salmonella pathogenicity islands and present a small RNA expression

123 med pUTI89 with many characteristics of UPEC pathogenicity islands and that likely arose due to horiz

124 the Type III secretion system of Salmonella pathogenicity islands and two component signal transduct

125 . pylori comB transformation system, the cag pathogenicity island, and another type IV secretion syst

126 re an initial characterization of this novel pathogenicity island, and we establish that it is stable

127 induction of bacteriophages, the movement of pathogenicity islands, and the expression of virulence f

128 elements such as genomic islands, prophages, pathogenicity islands, and the staphylococcal chromosoma

129 he EHEC locus of enterocyte effacement (LEE) pathogenicity island are known to contribute to this pat

130 Pathogenicity islands are sets of successive genes in a

131 m antibiotic class resistance and a putative pathogenicity island, arginine catabolic mobile element

132 and identify vacuolating cytotoxin A and cag pathogenicity island as the bacterial virulence determin

133 The tos operon is AT rich, resides on pathogenicity island aspV, and is not expressed under la

134 g the organism and strains harboring the cag pathogenicity island augment disease risk.

135 Bacterial virulence factors within the cag pathogenicity island, c-Abl tyrosine kinase, and interac

136 acaque model to study the effects of the cag pathogenicity island (cag PAI) on the H pylori host-path

137 igger inflammation in host cells via its cag pathogenicity island (cag PAI) type IV secretion system

138 Infections with cytotoxin-associated gene pathogenicity island (cag PAI)-containing strains are as

139 H. pylori strains harboring the cag pathogenicity island (cag+), which encodes CagA and a ty

140 trains of Helicobacter pylori (Hp) possess a pathogenicity island, cag, that encodes the effector pro

141 The T3SS is encoded in a pathogenicity island called the locus of enterocyte effa

142 oli are caused by isolates that also carry a pathogenicity island called the locus of enterocyte effa

143 effectors, are carried within a chromosomal pathogenicity island called the locus of enterocyte effa

144 These results show that all three pathogenicity islands can excise from the chromosome, wh

145 conferring antibiotic resistance, as well as pathogenicity islands, capsule loci and other variable t

146 SPI4 is a 24-kb pathogenicity island containing six open reading frames,

147 anscriptional regulator encoded on the SPI-2 pathogenicity-island, determines the switch between thes

148 occal serine-rich repeat protein (PsrP) is a pathogenicity island-encoded adhesin that mediates attac

149 tion of this regulator, designated PerA (for pathogenicity island-encoded regulator), we first examin

150 which is unusually sheathed by the large cag-pathogenicity-island-encoded protein CagY.

151 The cag-pathogenicity-island-encoded type IV secretion system of

152 This pathogenicity island encodes and expresses staphylococca

153 The locus of enterocyte effacement (LEE) pathogenicity island encodes many genes required for the

154 terohaemorrhagic Escherichia coli harbours a pathogenicity island encoding a type 3 secretion system

155 enome is invariably associated with the high-pathogenicity island, encoding the siderophore yersiniab

156 Here we show that their multiple pathogenicity islands form together a coherently organiz

157 ing directly to promoters on the Francisella Pathogenicity Island (FPI) and positively regulating the

158 The Francisella tularensis pathogenicity island (FPI) encodes many proteins that ar

159 regulated, including all of the Francisella pathogenicity island (FPI) genes.

160 ion system (T6SS) encoded by the Francisella pathogenicity island (FPI) is critical for the virulence

161 F. novicida DeltaiglB, a Francisella pathogenicity island (FPI) mutant, is deficient in phago

162 Further, Francisella pathogenicity island (FPI) protein expression was induce

163 dependent upon the regulation of Francisella pathogenicity island (FPI) virulence genes, which is poo

164 virulence is dependent upon the Francisella pathogenicity island (FPI), a cluster of genes that we s

165 stems from genes encoded on the Francisella pathogenicity island (FPI).

166 in the iglABCD operon in the Francisella sp. pathogenicity island (FPI).

167 eplicates using the genes in the Francisella pathogenicity island (FPI).

168 n surrounding pmrA or within the Francisella pathogenicity island (FPI).

169 rotein encoded by the duplicated Francisella pathogenicity island (FPI).

170 tic elements; including an ArdB encoded on a pathogenicity island from uropathogenic Escherichia coli

171 imilarities in their genomic organization to pathogenicity islands from other bacteria and are likely

172 A loss of cag pathogenicity island function was observed in 3 reisolat

173 interleukin 8 from AGS cells (to detect cag pathogenicity island function), neutral red uptake (to d

174 A (SspA) complex and the DNA-binding protein pathogenicity island gene regulator (PigR)-activates FPI

175 073, homologs of the Shigella flexneri SHI-2 pathogenicity island gene shiA, suppress the host inflam

176 drivers of the gain and loss of genes and of pathogenicity islands (gene clusters), which contribute

177 elements, but the natural deletion of 13 cag pathogenicity island genes and the truncation of CagA im

178 aken together, our data demonstrate that the pathogenicity island genes tested are essential for F. t

179 ups of motility and chemotaxis genes and for pathogenicity island genes, especially cagA, a predictor

180 nce factors including all of the Francisella pathogenicity island genes, LPS O-antigen synthetic gene

181 The LEE pathogenicity island has been acquired on multiple occas

182 Here we report that the staphylococcal pathogenicity islands have a dual role in gene transfer:

183 gulase-negative staphylococci, no associated pathogenicity islands have been found in the genome of S

184 Two genes within the T3SS pathogenicity island, herein named vttR(A) (locus tag A3

185 The Yersinia high-pathogenicity island (HPI) is common to multiple virulen

186 rinary E. coli isolates is the Yersinia high pathogenicity island (HPI), which directs the biosynthes

187 I secretion system encoded within Salmonella pathogenicity island I (SPI1).

188 Analysis of genetic variation in the LEE pathogenicity island identified 30 distinct LEE subtypes

189 esence of an incomplete or nonfunctional cag pathogenicity island in 18/276 genomes.

190 by genes located in a previously identified pathogenicity island in F. tularensis LVS.

191 However, conservation of tomA on a pathogenicity island in S. acidiscabies and S. turgidisc

192 thetic pathway and is located on a conserved pathogenicity island in S. scabies, S. turgidiscabies an

193 isparate genomic islands, defines VSP-1 as a pathogenicity island in V. cholerae, and implicates its

194 oded iron transport system is present within pathogenicity islands in all Shigella spp. and some path

195 The SaPIs are 14- to 17-kb mobile pathogenicity islands in staphylococci that carry genes

196 SaPI1 and SaPIbov1 are chromosomal pathogenicity islands in Staphylococcus aureus that carr

197 ther group we term Tn6022-like elements form pathogenicity islands in the Acinetobacter baumannii com

198 in bacterial pathogens (often manifested as pathogenicity islands in the recipient organism) and it

199 ate immune system utilizing genes encoding a pathogenicity island, including iglABCD, and instead uti

200 zontal gene transfer (HGT) are the classical pathogenicity islands, including the integrative and con

201 whereas IL-18 protein was OipA dependent-cag pathogenicity island independent, indicating that OipA r

202 stric epithelial cells, mediated via the cag pathogenicity island, induces N-terminally truncated Del

203 Analysis of the chromosome region revealed a pathogenicity island inserted between two tRNA sequences

204 olysin expression, and expression of a novel pathogenicity island involved in alpha-ketoglutarate met

205 ved in pathogenesis, the presence of the cag pathogenicity island is associated with increased gastri

206 amidinotransferase enzyme located in the Hrp pathogenicity island, is required for systemic infection

207 However, cag pathogenicity island knockout strains of H. pylori had v

208 A unique cluster of genes was found in the pathogenicity island-like emm region that included a nov

209 group B2-associated traits (including malX [pathogenicity-island marker], pap [P fimbriae] elements,

210 he EPEC locus of enterocyte effacement (LEE) pathogenicity island, non-LEE-encoded effector H1 (NleH1

211 BM96) encoding a novel AT was located in the pathogenicity island of avian pathogenic Escherichia col

212 o the exchangeable effector locus in the Hrp pathogenicity island of DC3000D28E.

213 shows similarity in content to a plasmid and pathogenicity island of human uropathogenic E. coli (UPE

214 e cupD gene cluster is located on the PAPI-1 pathogenicity island of strain PA14 and has probably bee

215 the yersiniabactin system found in the high-pathogenicity island of Yersinia sp. and is the first of

216 oxin (Proteus toxic agglutinin) and the high pathogenicity island of Yersinia spp.

217 ee-living photoautotroph share features with pathogenicity islands of parasitic bacteria, suggesting

218 ng PAO1 tRNA(Lys)-linked genomic island, the pathogenicity islands of strain PA14, and pKLC102 of clo

219 and the SprA1(AS) RNA antitoxin are within a pathogenicity island on opposite strands and possess a 3

220 rease activity and is independent of the cag pathogenicity island or VacA.

221 th unknown function, which were localized to pathogenicity islands or APEC O1's large virulence plasm

222 PMI2596-2605 are contained within the high-pathogenicity island, originally described in Yersinia p

223 n H. pylori infections in which both the cag pathogenicity island (PAI) and outer inflammatory protei

224 The Enterococcus faecalis pathogenicity island (PAI) encodes known virulence trait

225 e of a large virulence plasmid that houses a pathogenicity island (PAI) encoding a novel family of su

226 A 150-kb pathogenicity island (PAI) encoding several genes that c

227 jugative virulence plasmid harboring a 21-kb pathogenicity island (PAI) for growth in host macrophage

228 ional regulator was identified on the 153-kb pathogenicity island (PAI) found among virulent Enteroco

229 elicobacter pylori strains harboring the cag pathogenicity island (PAI) have been associated with mor

230 H. pylori strains bearing the cag pathogenicity island (PAI) induced higher levels of intr

231 es (aaiA-Y), which are localized to a 117 kb pathogenicity island (PAI) inserted at pheU.

232 ls infected with either cagA-negative or cag pathogenicity island (PAI) mutant.

233 es, at more than 11 Mb, and encodes a 100-kb pathogenicity island (PAI) shared with other plant-patho

234 the toxigenic vacA s1 allele, a complete cag pathogenicity island (PAI), cagA alleles containing mult

235 rry the Locus of Enterocyte Effacement (LEE) pathogenicity island (PAI), which encodes genes that med

236 e observed following infection with both cag pathogenicity island (PAI)-positive and -negative strain

237 form T4SS, in addition to the status of cag pathogenicity island (PAI).

238 b region of chromosomal DNA known as the cag pathogenicity island (PAI).

239 for the presence of a previously identified pathogenicity island (PAI).

240 tion is dependent on the presence of the cag pathogenicity island (PAI).

241 clustered in discrete genetic modules termed pathogenicity islands (PAI).

242 a strain deficient in the major pathway (cag pathogenicity island [PAI] encoded) for delivery of pept

243 in MM were used to determine distribution of pathogenicity islands (PAIs) across C. cellulans, which

244 Pathogenicity islands (PAIs) are a specific group of gen

245 Instability and excision of pathogenicity islands (PAIs) have already been described

246 re we investigate structural polymorphism in pathogenicity islands (PAIs) in Pseudomonas viridiflava,

247 thogen Pseudomonas viridiflava possesses two pathogenicity islands (PAIs) that share many gene homolo

248 Our results suggest that the pathogenicity island PAPI-1 may have evolved by acquisit

249 The pathogenicity island PAPI-1 of strain PA14 is a cluster

250 The large Pseudomonas aeruginosa pathogenicity island PAPI-1 of strain PA14 is a cluster

251 e one that was used for integration of small pathogenicity island PAPI-2 in strain PA14.

252 virulence factors has focussed upon the cag pathogenicity island, particularly its roles in regulati

253 esin encoded in the Streptococcus pneumoniae pathogenicity island psrP-secY2A2.

254 While the acquisition of the plasmid-encoded pathogenicity island (pXO1) and capsule genes (pXO2) rep

255 tructural gene, bslA, is located on the pXO1 pathogenicity island (pXO1-90) and bslA expression is bo

256 genes are indeed located within the deleted pathogenicity island region.

257 Among these, two pathogenicity islands, region of diversity 8a (RD8a), wh

258 One explanation for this discrepancy is that pathogenicity islands, regions of DNA found in some stra

259 The Francisella pathogenicity island, required for bacterial phagosome e

260 The Staphylococcus aureus pathogenicity island SaPI1 carries the gene for the toxi

261 thesis operon, the betA-betB operon, and the pathogenicity island, SaPI5, while virulence genes were

262 These genes, along with a putative pathogenicity island (SaPIBov) present uniquely in the c

263 Staphylococcus aureus pathogenicity islands (SaPIs) are genetic elements that

264 Staphylococcal pathogenicity islands (SaPIs) are the prototypical membe

265 that induce the cycle of some Staphylococcal pathogenicity islands (SaPIs) by binding to the SaPI-enc

266 Staphylococcal pathogenicity islands (SaPIs) carry superantigen and res

267 e clinically important Staphylococcus aureus pathogenicity islands (SaPIs) use this tactic to spread

268 Staphylococcus aureus pathogenicity islands (SaPIs), such as SaPI1, exploit sp

269 ansfer agents (GTAs), and the staphylococcal pathogenicity islands (SaPIs), the primary focus of this

270 ghly mobile phage satellites, staphylococcal pathogenicity islands (SaPIs), which carry and dissemina

271 ation of genetic elements known as S. aureus pathogenicity islands (SaPIs), which carry genes for sup

272 d virulence factors are encoded on S. aureus pathogenicity islands (SaPIs).

273 cells through the mobilization of S. aureus pathogenicity islands (SaPIs).

274 first evidence of a composite S. epidermidis pathogenicity island (SePI), the product of multiple ins

275 The pgm locus encodes, within a high-pathogenicity island, siderophore biosynthesis genes tha

276 e reductions in expression of the Salmonella pathogenicity island (SPI) 1 transcriptional regulator g

277 ystem SsrA/B, which is located on Salmonella Pathogenicity Island (SPI) 2.

278 Horizontal acquisition of Salmonella Pathogenicity Island (SPI)-2 permitted the expansion of

279 ion of genes encoded within and outside of a pathogenicity island (SPI-2), which is required for syst

280 la enterica serovar Typhimurium harbors five pathogenicity islands (SPI) required for infection in ve

281 ot only the already characterized Salmonella Pathogenicity Islands (SPI-1 to SPI-10) but also three n

282 The two T3SS are encoded on separate pathogenicity islands, SPI-1 and -2, with SPI-1 more str

283 557 and two neighbouring genes, located on a pathogenicity island termed SPI-16, resemble genes of th

284 n formation are encoded within a chromosomal pathogenicity island termed the locus of enterocyte effa

285 me non-O1/non-O139 strains have acquired the pathogenicity island that encodes the TCP, the role that

286 25-RE, which had a 15-kb deletion in the cag pathogenicity island that truncated CagA and eliminated

287 age therapy for bovine mastitis, we observed pathogenicity island transfer between S. aureus and L. m

288 iffering at 113 gene loci, including the cag pathogenicity island virulence genes.

289 Vibrio Pathogenicity Island (VPI)-1 was present; however, SXT/R

290 tor AphA at the tcpPH promoter on the Vibrio pathogenicity island (VPI).

291 tuent that increases disease risk is the cag pathogenicity island, which encodes a secretion system t

292 risk is the cytotoxin-associated gene (cag) pathogenicity island, which encodes a secretion system t

293 ments cancer risk is the strain-specific cag pathogenicity island, which encodes a type IV secretion

294 key virulence factor of H. pylori is the Cag pathogenicity island, which encodes a type IV secretion

295 icobacter pylori strains that harbor the cag pathogenicity island, which encodes a type IV secretion

296 these novel plasmids do not contain the pXO1 pathogenicity island, which in each instance is replaced

297 Virulence factors include the cag pathogenicity island, which induces proinflammatory, pro

298 H. pylori strains that possess the cag pathogenicity island, which translocates CagA into the h

299 One cancer-linked locus is the cag pathogenicity island, which translocates components of p

300 , EHEC represses flagellar genes and the LEE pathogenicity island while it activates the acid fitness