ライフサイエンスコーパス: pathological condition

1 fucosyl isomeric species associated with the pathological condition.

2 lization of the wound, thereby reversing the pathological condition.

3 ular metabolism under both physiological and pathological conditions.

4 neurodegenerative diseases as well as other pathological conditions.

5 c lineage in vivo, both in physiological and pathological conditions.

6 the therapeutic spectrum of HU to additional pathological conditions.

7 lar activities, physiological processes, and pathological conditions.

8 ing the cardiac cycle under physiological or pathological conditions.

9 of the Kv7.4 channel under physiological and pathological conditions.

10 omes for degradation under physiological and pathological conditions.

11 for beta-A detection under physiological and pathological conditions.

12 ity, leading to autoimmune diseases or other pathological conditions.

13 hondria and released into bloodstream during pathological conditions.

14 ortical circuit function under normal and/or pathological conditions.

15 ead to uncontrolled cell division in several pathological conditions.

16 chemerin/ChemR23 system in physiological and pathological conditions.

17 However, this relationship is reversed under pathological conditions.

18 tors of immune responses in cancer and other pathological conditions.

19 maging due to its upregulation under various pathological conditions.

20 lly validated therapeutic target for several pathological conditions.

21 hese RNAs must remain unrecognized under non-pathological conditions.

22 rs linked to both antimicrobial immunity and pathological conditions.

23 f COX-2 mRNAs in human OA chondrocytes under pathological conditions.

24 g dendrite regeneration in physiological and pathological conditions.

25 angiogenesis plays a pivotal role in diverse pathological conditions.

26 and is associated with its dysregulation in pathological conditions.

27 FOXO3a/1/PGC-1alpha, under physiological and pathological conditions.

28 eripheral tissues, both in physiological and pathological conditions.

29 non, observed in different physiological and pathological conditions.

30 be activated under several physiological and pathological conditions.

31 shown to cooperate with TGF-beta in various pathological conditions.

32 e mTORC2 kinase under both physiological and pathological conditions.

33 tem in host defense and its dysregulation in pathological conditions.

34 d YAP and their diverse impact under several pathological conditions.

35 n be targeted to ameliorate dendrite loss in pathological conditions.

36 x protein that has a role in a wide range of pathological conditions.

37 iversity, and its deregulation can result in pathological conditions.

38 ulatory mechanisms could be influenced under pathological conditions.

39 extracellular space under tissue-damaging or pathological conditions.

40 ggable target to modulate angiogenesis under pathological conditions.

41 roliferative cells in both physiological and pathological conditions.

42 tential linear motifs that are altered under pathological conditions.

43 mpaired adaptive vascular growth under these pathological conditions.

44 o play a role in different physiological and pathological conditions.

45 may be inadequate and misleading under many pathological conditions.

46 nderstand redox biology in physiological and pathological conditions.

47 vide near real-time surveillance of multiple pathological conditions.

48 that access the central nervous system under pathological conditions.

49 role in various physiological functions and pathological conditions.

50 ing and plasticity, and how it is altered in pathological conditions.

51 is critical in the treatment of many neural pathological conditions.

52 alues to allow discrimination of normal from pathological conditions.

53 y thus be a useful tool to study healthy and pathological conditions.

54 sels is low, it increases considerably under pathological conditions.

55 eal therapeutic transgene, active only under pathological conditions.

56 ive disorders such as Alzheimer and/or other pathological conditions.

57 n sets the base for examining these cells in pathological conditions.

58 ough their receptors under physiological and pathological conditions.

59 been shown to vary between species and under pathological conditions.

60 is a way to enhance cognition in normal and pathological conditions.

61 ssessment of Alu expression alteration under pathological conditions.

62 production and removal in physiological and pathological conditions.

63 in zinc homeostasis under physiological and pathological conditions.

64 sm for restoring dopamine levels in relevant pathological conditions.

65 ed under oxidative stress play a key role in pathological conditions.

66 l neural stem cells (NSCs) respond to graded pathological conditions.

67 well as memory under both physiological and pathological conditions.

68 tore mitochondrial integrity under PD-linked pathological conditions.

69 issue hypoxia can occur in physiological and pathological conditions.

70 ranscription under diverse physiological and pathological conditions.

71 he innate immune response underlies numerous pathological conditions.

72 es neuronal function under physiological and pathological conditions.

73 evels are elevated in both physiological and pathological conditions.

74 pressed in a tissue-specific manner or under pathological conditions.

75 drial functions to embryonic development and pathological conditions.

76 ing that this transition might also occur in pathological conditions.

77 pression is likely targeted during different pathological conditions.

78 e seizures under different physiological and pathological conditions.

79 iological actions of CO in physiological and pathological conditions.

80 tegies to modify cell behavior in normal and pathological conditions.

81 een associated with cell death in many human pathological conditions.

82 tanding of angiogenesis in physiological and pathological conditions.

83 transferases (PRMTs) has been linked to many pathological conditions.

84 ells of other tissues under physiological or pathological conditions.

85 during a broad spectrum of physiological and pathological conditions.

86 their contribution to both physiological and pathological conditions.

87 utor to neurodegeneration caused by numerous pathological conditions.

88 ls that enhance GSIS under physiological and pathological conditions.

89 f gene expression in specific cells to treat pathological conditions.

90 programs of CNS myelination under normal and pathological conditions.

91 ally modulating a wide-range of inflammatory pathological conditions.

92 bute to prefrontal function under normal and pathological conditions.

93 e cell injury or activation in patients with pathological conditions.

94 involved in a plethora of physiological and pathological conditions.

95 ological processes in both physiological and pathological conditions.

96 e microenvironment of both physiological and pathological conditions.

97 mitochondrial function to physiological and pathological conditions.

98 s for memory storage under physiological and pathological conditions.

99 or regulators of T cell responses in several pathological conditions.

100 ancer and an increasing number of identified pathological conditions.

101 dvanced glycation end products under certain pathological conditions.

102 ession and activity in OA chondrocytes under pathological conditions.

103 -cell communication in normal physiology and pathological conditions.

104 th following a thrombotic event in normal or pathological conditions.

105 nt cellular lineages under physiological and pathological conditions.

106 erstanding of how these factors change under pathological conditions.

107 distances, are imperative in both normal and pathological conditions.

108 e is known about how this profile changes in pathological conditions.

109 tides significantly associated with discrete pathological conditions.

110 d metabolism signifies a key element in many pathological conditions.

111 ns are substantially reorganized during many pathological conditions.

112 lose during normal development as well as in pathological conditions.

113 involved in executive function in normal and pathological conditions.

114 enesis of delayed mucosal healing in certain pathological conditions.

115 pendent regulation in both physiological and pathological conditions.

116 ainst pathogens and whose imbalance leads to pathological conditions.

117 ates anti-inflammatory actions under various pathological conditions.

118 pment and their implications in a variety of pathological conditions.

119 dulin II has been implicated in a variety of pathological conditions [10-13], but the developmental f

120 However, the distinction between the pathological condition and the benign one is very challe

121 caused by altered sleep-awake patterns or by pathological conditions and can initiate or exacerbate h

122 f all body tissues, is indicative of several pathological conditions and cellular death.

123 ervous system (CNS) that become activated in pathological conditions and determine the fate of other

124 box 1 (HMGB1) can be released under chronic pathological conditions and initiate inflammatory cascad

125 rts to understand the role of miR-297c-5p in pathological conditions and its potential for facilitati

126 synthase that take place in the heart under pathological conditions and of their potential role in d

127 essential since it underlies a multitude of pathological conditions and therapeutic interventions.

128 perties of a cell reflect its biological and pathological conditions and there have been active resea

129 genesis in mice during development and under pathological conditions and to explore the underlying mo

130 nderstanding of differentiation anomalies in pathological conditions and will contribute to improving

131 It has also been associated with several pathological conditions and with gene expression regulat

132 rate and rapid detection of suspected bovine pathological conditions, and can largely enable rapid fi

133 entities that play roles in gene regulation, pathological conditions, and evolution.

134 of the immune response in physiological and pathological conditions, and it could be impaired during

135 ration vs differentiation in both normal and pathological conditions, and that JAK2 chemical inhibito

136 ew metabolic biomarkers in physiological and pathological conditions, and to study and evaluate treat

137 ies, e.g., actomyosin contractility and many pathological conditions are driven or followed by bendin

138 ms leading to hyperphosphorylation of Tau in pathological conditions are unknown.

139 ism and contribute to both physiological and pathological conditions as second messengers.

140 A complete understanding of pathological conditions as well as normal brain function

141 from latency and thus contributes to severe pathological conditions associated with HHV-6.

142 Under pathological conditions, ATP synthase hydrolyzes ATP to

143 Nucleotides are released in the heart under pathological conditions, but little is known about their

144 olic shifts in physiological adaptations and pathological conditions, but may be influenced by the nu

145 ylation has been observed in podocytes under pathological conditions, but the molecular mechanism lin

146 rsors of neuromelanin (NM) pigment and under pathological conditions can modify and damage macromolec

147 Alcoholic liver disease is a pathological condition caused by overconsumption of alco

148 s, cell competition may occur in a number of pathological conditions.Cell competition causes the remo

149 A cataract is a pathological condition characterized by the clouding of

150 pharmacological agents for the treatment of pathological conditions characterized by impaired mitoch

151 or novel pharmacological approaches in those pathological conditions characterized by persistent immu

152 emonstrates that reduction of miR-378 during pathological conditions contributes to cardiac remodelin

153 Under basal or pathological conditions, electron leakage for ROS produc

154 However, under pathological conditions, excessive activation of NMDARs

155 s that associate Activin/Nodal signalling to pathological conditions, focusing on cancer stem cells i

156 ere evaluated in physiological and simulated pathological conditions (FTR), and post-treatments.

157 s to nuclear mechanical state in physio- and pathological conditions, furthering our understanding of

158 and TFH cells to autoantibody profiles under pathological conditions has not been thoroughly investig

159 NAs (miRNAs), to a genetic predisposition to pathological conditions has remained unclear.

160 A number of pathological conditions have been linked to mutations in

161 e functions, neuropsychiatric disorders, and pathological conditions; hence understanding genes regul

162 pregulated in circulating blood in different pathological conditions; however, the characteristics an

163 ammatory diseases whose activity can lead to pathological conditions if unregulated.

164 How a pathological condition in a given tissue influences syst

165 helial Wnt signaling and BBB integrity under pathological conditions in adult mice.

166 al, mechanical, or chemical means alleviated pathological conditions in rat colitis and hypertension

167 HDAC6 dysfunction is associated with several pathological conditions in the central nervous system.

168 relevant for homeostasis, immunity, and also pathological conditions in the gastrointestinal tract.

169 SC/progenitor system under physiological and pathological conditions in vivo.

170 important when channels are activated under pathological conditions in which the pH is reduced, such

171 erapeutic applicability in a wide variety of pathological conditions including asthma, cancer, erecti

172 biogenesis have been associated with various pathological conditions including cancers.

173 serve as non-invasive biomarkers in several pathological conditions including inflammatory diseases.

174 ht the role of the GSK-3 isoforms in various pathological conditions including myocardial aging, isch

175 cumulative stressors can lead to an array of pathological conditions including posttraumatic stress d

176 sses, and have also been implicated in human pathological conditions (including cancer).

177 etimes NDM appears in association with other pathological conditions, including autoimmune diseases.

178 TGF-beta1 is involved in many pathological conditions, including autoimmune disorders,

179 Since several pathological conditions, including bacterial infections,

180 Its crucial role in many pathological conditions, including cancer and ischemic b

181 namics play an important role within several pathological conditions, including cancer and neurologic

182 biological processes under physiological and pathological conditions, including cancer, diabetes, hea

183 tion of protein under many physiological and pathological conditions, including cancer.

184 Hypoxia occurs in many pathological conditions, including chronic inflammation

185 reciate the role of necroptosis in different pathological conditions, including critical illnesses.

186 of genes involved in many physiological and pathological conditions, including development, metaboli

187 and oxygen species is implicated in certain pathological conditions, including diabetes mellitus.

188 gesting a role for this rhomboid protease in pathological conditions, including EGFR signaling.

189 isruption of this balance is associated with pathological conditions, including eosinophilic esophagi

190 sion of HERV-Ks has been linked to different pathological conditions, including HIV infection, but ou

191 tion (pRTK) has been associated with diverse pathological conditions, including human neoplasms.

192 mplicated in a spectrum of physiological and pathological conditions, including immune responses.

193 ing but also with several other, age-related pathological conditions, including inflammation, vascula

194 nd tyrosine nitration are present in several pathological conditions, including neurodegeneration, st

195 been associated with cell death in multiple pathological conditions, including neurodegeneration.

196 otential therapeutic agents for a variety of pathological conditions, including several types of canc

197 otential therapeutic agents for a variety of pathological conditions, including several types of canc

198 ntal inflammation is associated with several pathological conditions, including stillbirth and fetal

199 articipate in a variety of physiological and pathological conditions, including tumor suppression [2]

200 Under pathological conditions induced by chronic catecholamine

201 e 2 (MK2) pathway is involved in a series of pathological conditions (inflammation diseases and metas

202 Although multiple acute and chronic lung pathological conditions involve perturbations in sphingo

203 possible contribution of aberrant R-loops to pathological conditions is also discussed.

204 the level of circulating catalytic IgG under pathological conditions is an intrinsic property of each

205 of cerebral blood flow in physiological and pathological conditions is mediated by arteriolar SMCs.

206 hanism of how mTORC1 activation occurs under pathological conditions is not completely known.

207 r, its effect on cartilage homeostasis under pathological conditions is not known.

208 how LKB1 is regulated under physiological or pathological conditions is still unclear.

209 corneal tissue structure in homeostatic and pathological conditions is unknown.

210 deacetylation in normal brain functions and pathological conditions is unquestionable, yet the molec

211 ponse to a treatment or the development of a pathological condition, is proposed here.

212 let hyperreactivity, which is common in many pathological conditions, is associated with increased at

213 enic default pathway may be subverted during pathological conditions, leading to skeletal fragility a

214 hyaluronan in animal cells are indicators of pathological conditions like infection or malignancy.

215 e in DEG/ENaC biology, therefore influencing pathological conditions like ischemia.

216 ic therapeutic inhibitors targeting FXIII in pathological conditions like thrombosis.

217 HRH would be beneficial for life-threatening pathological conditions, like cardiac hypertrophy and he

218 Under pathological conditions, matrix metalloproteinases (MMPs

219 rine and autocrine cell signaling, and under pathological conditions may induce and/or accelerate cel

220 A wide spectrum of pathological conditions may result from the interaction

221 Since their profiles reflect pathological conditions, miRNAs have recently been propo

222 importance of calcineurin in both normal and pathological conditions, new tools that elucidate the sp

223 ation of tissues, cell divisions, aging, and pathological conditions observed in many cancers.

224 Under pathological conditions of dysregulated Ca(2+) cycling,

225 anism that likely limits tissue damage under pathological conditions of MBP-1 oversecretion.

226 he CNS is relevant to many physiological and pathological conditions of the CNS.

227 epresents a potential therapeutic target for pathological conditions of the muscle in which the stem

228 tion exerts a pervasive impact on normal and pathological conditions of the nervous system.

229 nsitive to elevated extracellular ATP during pathological conditions of urinary bladder inflammation

230 heir contribution to an increasing number of pathological conditions, opens a plethora of medicinal c

231 though histamine is normally associated with pathological conditions or allergic and anaphylactic rea

232 orphogenesis can contribute to disease under pathological conditions or can be altered to induce dise

233 ength of these currents becomes larger under pathological conditions or drug administrations.

234 mechanical signaling under physiological and pathological conditions, purely based on histological da

235 rus replication and led to hyperinduction of pathological condition-related genes late in infection.

236 Diagnosis and therapeutic interventions in pathological conditions rely upon clinical monitoring of

237 membrane homeostasis under physiological and pathological conditions remain largely unknown.

238 chanisms underlying the development of these pathological conditions remain undefined.

239 recise role of TAK1 in the adult heart under pathological conditions remains largely unknown.

240 tational activity is regulated in normal and pathological conditions remains largely unknown.

241 asis and may be a viable target for treating pathological conditions resulting from oxidative damage.

242 strating how the shift from physiological to pathological conditions results from slight perturbation

243 Due to low concentration levels, even in pathological conditions, sensitive cGMP detection remain

244 understand VGLUT's involvement in normal and pathological conditions.SIGNIFICANCE STATEMENT VGLUT3 is

245 levels, such as attention and arousal and in pathological conditions such as Alzheimer's disease.

246 rbed in wound healing and is dysregulated in pathological conditions such as atopic dermatitis.

247 n can be used to modulate senescence-related pathological conditions such as cancer, and the aging pr

248 deficiency has been associated with multiple pathological conditions such as depression, schizophreni

249 s current diffusion MR methods could monitor pathological conditions such as hyperexcitability, e.g.,

250 hanisms may play a maladaptive role in human pathological conditions such as in the fatigue and anore

251 , Ca(2+) influx was thought to occur only in pathological conditions such as ischaemia.

252 Pathological conditions such as ischemia or pressure ove

253 Upon cardiac pathological conditions such as ischemia, microenvironme

254 ible for astrocyte reactivity in progressive pathological conditions such as ND.

255 the internal milieu of the skin can lead to pathological conditions such as psoriasis.

256 Spreading depolarization occurs during pathological conditions such as stroke, brain injury, an

257 r processes such as cell differentiation and pathological conditions such as tumor cell migration.

258 Pathological conditions, such as a stroke, trigger neura

259 ytosolic accumulation of self-DNA results in pathological conditions, such as autoimmunity.

260 on the correlation between RAGE activity and pathological conditions, such as cancer, diabetes, cardi

261 nvolvement of human NAT enzymes in different pathological conditions, such as cancer, has encouraged

262 inhibitors may be useful in the treatment of pathological conditions, such as cancer, in which cerami

263 Under pathological conditions, such as continuous MAPK pathway

264 from food intake, social behavior, and even pathological conditions, such as gambling and drug addic

265 of the dural vasculature has been limited to pathological conditions, such as headaches, but little i

266 d during cell proliferation under normal and pathological conditions, such as liver regeneration and

267 lation are concurrent with physiological and pathological conditions, such as pregnancy and rheumatoi

268 y cognitive functions and to be disrupted in pathological conditions, such as schizophrenia.

269 s high altitude, air travel, and sports, and pathological conditions, such as solid tumors, have been

270 ndria indicates loss of their function under pathological conditions, such as stroke and brain trauma

271 ase in extracellular ATP concentration under pathological conditions, such as tissue damage or viscer

272 ion of contact inhibition is associated with pathological conditions, such as tumor spreading.

273 death that promotes inflammation in various pathological conditions, suggesting that it might be a p

274 Under pathological conditions, Tau dissociates from axonal mic

275 erfusion (I/R) injury is a relatively common pathological condition that can lead to multi-organ fail

276 CpG DNA) persists in tissues and blood under pathological conditions that are associated with enhance

277 ied mitochondrial fragmentation during acute pathological conditions that mimic severe, moderate, and

278 tiation and myelination for treatment of CNS pathological conditions that require remyelination.

279 rocytes under a variety of physiological and pathological conditions, the acute effects of seizures o

280 mune response in different physiological and pathological conditions, the aim of this work was to inv

281 At the same time, under these pathological conditions, the changes in muscle sensory n

282 al nervous system (CNS) has often focused on pathological conditions, the importance of neuroimmune c

283 ellular vesicles (EVs) and physiological and pathological conditions, the interest in EVs is exponent

284 functional exhaustion of satellite cells in pathological conditions, thereby maintaining the endogen

285 anges in cardiomyocytes in physiological and pathological conditions, thereby providing novel therape

286 While such channels are mostly active in pathological conditions, they have recently been shown t

287 Conversely, under pathological condition, this pathway relayed aberrant ce

288 ing evidence of function in health and under pathological conditions, this review will outline the gl

289 2) regulates blood vessel remodeling in many pathological conditions through differential effects on

290 ted to be involved in some physiological and pathological conditions through processing substrates, i

291 bute to the development of many diseases and pathological conditions through the excessive production

292 d protein metabolism under physiological and pathological conditions, underscoring the potential of t

293 ircadian clock genes under physiological and pathological conditions; we focus on their roles in regu

294 However, under pathological conditions when sulfite concentrations are

295 hich could be of relevance in the context of pathological conditions where spine densities and neural

296 rate that ANG2 acts as a Tie2 agonist in non-pathological conditions, whereas in the setting of infla

297 olved in a variety of cellular processes and pathological conditions, which makes it a promising phar

298 ture in 3D for different genetic mutants and pathological conditions will help uncover novel molecula

299 ve brain tissue under both physiological and pathological conditions with single-cell resolution and

300 alyzing their action against ROS produced by pathological conditions within the cells.