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1 er adhesin that is conserved within the ETEC pathovar.
2 hat some antigens are restricted to the ETEC pathovar.
3 0%), but absent in the other major X. oryzae pathovar.
4 ree Secretion System in Pseudomonas syringae pathovars.
5 d that blurs the distinction between E. coli pathovars.
6 issue specificity for individual species and pathovars.
7 dividual host species coevolving with single pathovars.
8 but not complete, overlap between these two pathovars.
9 to (Pto) representing particularly divergent pathovars.
10 tors that contribute to the formation of new pathovars.
11 to diverse TAL effectors from both X. oryzae pathovars.
13 e" confirmed the diverse history of the ETEC pathovar and provides a finer resolution of the E. coli
15 fector suites from two sequenced P. syringae pathovars and show that type III effector protein suites
16 cluster but before the divergence of modern pathovars and that some EELs underwent transpositions yi
19 sed by uropathogenic Escherichia coli (UPEC) pathovars belong to the most frequent infections in huma
20 sed by uropathogenic Escherichia coli (UPEC) pathovars belong to the most frequent infections in huma
24 wever, the recently emerged ST313 lineage II pathovar commonly causes systemic bacteremia in sub-Saha
25 cterium Pseudomonas syringae is divided into pathovars differing in host specificity, with P. syringa
26 he family of agn alleles in Escherichia coli pathovars encodes autotransporters that have been implic
27 nts and unique features of three unsequenced pathovars, enterotoxigenic E. coli, enteropathogenic E.
29 phenotypic markers, but only two of the six pathovars have been subjected to any genome sequencing p
30 protein in the T3SS of different P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P.
31 stence and virulence potential of an E. coli pathovar hybrid that blurs the distinction between E. co
32 other distinguishing feature between the two pathovars is their distinctive sets of transposable elem
33 f infection with either Pseudomonas syringae pathovar japonica (Psj) or Xanthomonas translucens patho
34 rast, the resistance to Pseudomonas syringae pathovar maculicola (RPM1) bacterial resistance gene is
35 ation with HR-eliciting Pseudomonas syringae pathovars measured by laser photoacoustic detection was
36 ultiple effector proteins from an individual pathovar more frequently and more intensely than host sp
37 e majority of deaths are attributable to one pathovar of E. coli, namely, enterotoxigenic E. coli.
40 newly analyzed and eight known PFPs from 12 pathovars of P. syringae, which belong to four genomospe
44 ne (COR) is a phytotoxin produced by several pathovars of Pseudomonas syringae and consists of corona
45 t only compromised nonhost resistance to few pathovars of Pseudomonas syringae and Xanthomonas campes
47 portant virulence factor produced by several pathovars of the bacterial pathogen Pseudomonas syringae
48 ance to another race of H. parasitica and to pathovars of the bacterial pathogen Pseudomonas syringae
50 i (ETEC) is an important pathogenic variant (pathovar) of E. coli in developing countries from a huma
51 e analyses with a phylogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a stro
52 hF gene was cloned from Pseudomonas syringae pathovar phaseolicola (PPH:) races 5 and 7, based on its
54 tomato hrpW were found in other P. syringae pathovars, Pseudomonas viridiflava, Ralstonia (Pseudomon
56 psis thaliana pathogens Pseudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by in
57 (serotypes A-C) and two sexually transmitted pathovars; serotypes D-K and lymphogranuloma venereum (L
61 ess and its prevalence in many X. campestris pathovars suggests that the Bs2 gene may be durable in t
63 invasive Escherichia coli (EIEC) is a unique pathovar that has a pathogenic mechanism nearly indistin
65 were more resistant to Pseudomonas syringae pathovar tomato (Pst) DC3000 infection implicating SCD1
67 pe Pi-0 is resistant to Pseudomonas syringae pathovar tomato (Pst) strain DC3000 expressing the T3S e
69 the bacterial pathogens Pseudomonas syringae pathovar tomato and Erwinia amylovora, respectively, and
70 odel bacterial pathogen Pseudomonas syringae pathovar tomato DC3000 (DC3000), which is pathogenic on
71 of the foliar pathogen, Pseudomonas syringae pathovar tomato DC3000 (hereafter PstDC3000), into the p
73 s in the plant pathogen Pseudomonas syringae pathovar tomato strain DC3000 possess characteristic pat
74 lence protein AvrPto of Pseudomonas syringae pathovar tomato, the agent of bacterial speck disease, a
76 e resistance in response to Pto (P. syringae pathovars tomato) DC3000(avrB), but not against Pto DC30
77 dentified as putative orthologs in these two pathovars using a reciprocal best-hit method, with 3,941
79 virulence factor from Xanthomonas campestris pathovar vesicatoria (Xcv) that is translocated into tom
80 ype III effector from Xanthomonas campestris pathovar vesicatoria (Xcv), suppresses symptom productio
81 -dependent promoters of Pseudomonas syringae pathovars were identified with a consensus sequence of 5
83 ted AvrBs2 proteins from the two Xanthomonas pathovars were strongly conserved and had predicted sequ
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