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4 ne expression patterns in multibacillary and paucibacillary disease, no shift in T cell genes from Th
6 oche detected 95% of patients with more than paucibacillary (greater than 20 colonies) tuberculosis.
7 Clinical and microbiological reactivation of paucibacillary infection by TNF neutralization was assoc
10 del was developed with leprosy group (either paucibacillary leprosy [PB] or multibacillary leprosy [M
11 ent was started on WHO multidrug therapy for paucibacillary leprosy along with antiinflammatory drugs
12 tides showed IFN-gamma responses only in the paucibacillary leprosy and household contact groups, wit
15 a) secretion upon incubation with blood from paucibacillary leprosy patients, a group who limit M. le
17 gely of infected epithelioid macrophages and paucibacillary lesions contain T cells but few bacteria.
18 e sought to develop and characterize a novel paucibacillary model in mice, which develops necrotic lu
19 es in low-resource settings and the inherent paucibacillary nature of mycobacterial disease in childh
22 manifestations of Lyme disease; however, the paucibacillary nature of the spirochete in this environm
25 feron (IFN-gamma) responses from both TB and paucibacillary (PB) leprosy patients and from healthy ho
26 DO-LID detected larger proportions of MB and paucibacillary (PB) leprosy than the alternative, the St
27 inadequate methods of pathogen detection in paucibacillary pediatric samples and lack of a child-spe
29 , the test sensitivity remains suboptimal in paucibacillary specimens that are negative for acid-fast
32 notypic testing for complex drug resistance, paucibacillary sputum, response to treatment, and detect
34 patients, including those diagnosed with the paucibacillary, tuberculoid form of disease, can be clas
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